A condition in which the percentage of progressively motile sperm is abnormally low. In men, it is defined as
Movement characteristics of SPERMATOZOA in a fresh specimen. It is measured as the percentage of sperms that are moving, and as the percentage of sperms with productive flagellar motion such as rapid, linear, and forward progression.
Food that has been prepared and stored in a way to prevent spoilage.
The inability of the male to effect FERTILIZATION of an OVUM after a specified period of unprotected intercourse. Male sterility is permanent infertility.
Mature male germ cells derived from SPERMATIDS. As spermatids move toward the lumen of the SEMINIFEROUS TUBULES, they undergo extensive structural changes including the loss of cytoplasm, condensation of CHROMATIN into the SPERM HEAD, formation of the ACROSOME cap, the SPERM MIDPIECE and the SPERM TAIL that provides motility.
The thick, yellowish-white, viscid fluid secretion of male reproductive organs discharged upon ejaculation. In addition to reproductive organ secretions, it contains SPERMATOZOA and their nutrient plasma.
A condition of suboptimal concentration of SPERMATOZOA in the ejaculated SEMEN to ensure successful FERTILIZATION of an OVUM. In humans, oligospermia is defined as a sperm count below 20 million per milliliter semen.
A count of SPERM in the ejaculum, expressed as number per milliliter.
The ratio of the number of conceptions (CONCEPTION) including LIVE BIRTH; STILLBIRTH; and fetal losses, to the mean number of females of reproductive age in a population during a set time period.

Beta-endorphin in serum and seminal plasma in infertile men. (1/72)

AIM: To access beta-endorphin levels in serum as well as seminal plasma in different infertile male groups. METHODS: Beta-endorphin was estimated in the serum and seminal plasma by enzyme-linked immunosorbent assay (ELISA) method in 80 infertile men equally divided into four groups: non-obstructive azoospermia (NOA), obstructive azoospermia (OA), congenital bilateral absent vas deferens (CBVAD) and asthenozoospermia. The results were compared to those of 20 normozoospermic proven fertile men. RESULTS: There was a decrease in the mean levels of beta-endorphin in the seminal plasma of all successive infertile groups (mean +/- SD: NOA 51.30 +/- 27.37, OA 51.88 +/- 9.47, CBAVD 20.36 +/- 13.39, asthenozoospermia 49.26 +/- 12.49 pg/mL, respectively) compared to the normozoospermic fertile control (87.23 +/- 29.55 pg/mL). This relation was not present in mean serum level of beta-endorphin between four infertile groups (51.09 +/- 14.71, 49.76 +/- 12.4, 33.96 +/- 7.2, 69.1 +/- 16.57 pg/mL, respectively) and the fertile control group (49.26 +/- 31.32 pg/mL). The CBVAD group showed the lowest seminal plasma mean level of beta-endorphin. Testicular contribution of seminal beta-endorphin was estimated to be approximately 40%. Seminal beta-endorphin showed significant correlation with the sperm concentration (r = 0.699, P = 0.0188) and nonsignificant correlation with its serum level (r = 0.375, P = 0.185) or with the sperm motility percentage (r = 0.470, P = 0.899). CONCLUSION: The estimation of beta-endorphin alone is not conclusive to evaluate male reproduction as there are many other opiates acting at the hypothalamic pituitary gonadal axis.  (+info)

Association between the apolipoprotein B signal peptide gene insertion/deletion polymorphism and male infertility. (2/72)

In male mice heterozygous for a null apolipoprotein B (apoB), allele infertility was noticed. These data led us to investigate a possible role of APOB gene polymorphism and male infertility in humans. In this case-control study, we searched for an association between the insertion/deletion (I/D) polymorphism of the APOB gene and male infertility in 560 Slovene Caucasian men. The study group consisted of 310 infertile patients: 115 with azoospermia and 195 with oligoasthenoteratozoospermia (OAT) and a control group of 250 fertile men. We found a statistically significant difference in the genotype distribution between the two groups (chi2 = 6.315, P = 0.043). A separate analysis of azoospermic and OAT patients demonstrated that significant differences in genotype distribution were limited to the OAT group (chi2 = 7.011, P = 0.030). The presence of the D allele (DD or ID genotypes) conferred a 1.6 risk [chi2 = 6.089, P = 0.014, 95% confidence interval (95% CI) = 1.102-2.347] for male infertility in the OAT group of patients. We did not find a correlation between the I/D polymorphism genotypes and the clinical characteristics of infertile men: sperm concentration (P = 0.102), rapid progressive motility (P = 0.449), normal morphology (P = 0.085) and Johnsen score (P = 0.531). These data suggest that genetic variation in the signal peptide of the APOB gene (I/D polymorphism) might be a risk factor for the development of male infertility.  (+info)

Asthenoteratozoospermia in mice lacking testis expressed gene 18 (Tex18). (3/72)

Testis expressed gene 18 (Tex18) is a small gene with one exon of 240 bp, which is specifically expressed in male germ cells. The gene encodes for a protein of 80 amino acids with unknown domain. To investigate the function of (Tex18) gene, we generated mice with targeted disruption of the (Tex18) gene by homologous recombination. Homozygous mutant males on a mixed genetic background (C57BL/6J x 129/Sv) are fertile, while they are subfertile on the 129/Sv background, although mating is normal. We showed that Tex18(-/-) males are subfertile because of abnormal sperm morphology and reduced motility, which is called asthenoteratozoospermia, suggesting that (Tex18) affects sperm characteristics. Maturation of spermatids is unsynchronized and partially impaired in seminiferous tubules of Tex18(-/-) mice. Electron microscopical examination demonstrated abnormal structures of sperm head. In vivo experiments with sperm of Tex18(-/-) 129/Sv mice revealed that the migration of spermatozoa from the uterus into the oviduct is reduced. This result is supported by the observation that sperm motility, as determined by the computer-assisted semen analysis system, is significantly affected, compared to wild-type spermatozoa. Generation of transgenic mice containing Tex18-EGFP fusion construct revealed a high transcriptional activity of (Tex18) during spermiogenesis, a process with morphological changes of haploid germ cells and development to mature spermatozoa. These results indicate that (Tex18) is expressed predominantly during spermatid differentiation and subfertility of the male Tex18(-/-) mice on the 129/Sv background is due to the differentiation arrest, abnormal sperm morphology and reduced sperm motility.  (+info)

No evidence for an mtDNA role in sperm motility: data from complete sequencing of asthenozoospermic males. (4/72)

The first complete mitochondrial DNA (mtDNA) sequences (approximately 16,569 bp) in 20 patients with asthenozoospermia and a comparison with 23 new complete mtDNA sequences in teratoasthenozoospermic individuals, confirmed no sharing of specific polymorphisms or specific mitochondrial lineages between these individuals. This is strong evidence against the accepted claim of a major role played by mtDNA in male fertility, once supported by haplogroup association studies based on the screening of hypervariable region I. The hypothesis of maternally driven selection acting in male reproductive success must thus be treated with caution.  (+info)

Absence of tektin 4 causes asthenozoospermia and subfertility in male mice. (5/72)

Sperm flagellar motion is the outcome of a dynamic interplay between the axonemal cytoskeleton, the peri-axonemal accessory structures, and multiple regulatory networks that coordinate to produce flagellar beat and waveform. Tektins are conserved components of the flagellar proteome in evolutionarily diverse species and are believed to play essential roles in the mechanics of sperm motility. Using database mining, we identified multiple new paralogs of previously annotated tektins, including tektin 4 (TEKT4), which shares 77.1% identity with its nearest human homologue. Mouse Tekt4 is a germ cell-enriched gene, most abundantly expressed in haploid round spermatids in the testis, and the protein is localized to the sperm flagella. Male mice lacking TEKT4 on a 129S5/SvEvBrd inbred background are subfertile. Tekt4-null sperm exhibit drastically reduced forward progressive velocity and uncoordinated waveform propagation along the flagellum. In Tekt4-null sperm, flagellar ultrastructure is grossly unaltered as revealed by transmission electron microscopy. However, the ineffective flagellar strokes lead to approximately 10-fold higher consumption of intracellular ATP in Tekt4-null sperm as compared to wild-type, and null spermatozoa rapidly lose progressive motility when incubated for > or = 1.5 h. Our studies demonstrate that TEKT4 is necessary for the proper coordinated beating of the sperm flagellum and male reproductive physiology.  (+info)

Effect of cigarette smoking on semen quality of infertile men. (6/72)

INTRODUCTION: Defective sperm quality is a significant cause of infertility. It is known that cigarette smoking affects semen quality. Our aim was to compare the semen of infertile cigarette smokers with infertile non-smokers to study the effect of smoking on semen quality. METHODS: Semen samples of 100 cigarette smokers and 100 strictly non-smoking primary infertility patients were included in the study, following stringent exclusion criteria. Smokers were categorised as light, moderate and heavy smokers. Semen samples were examined for asthenozoospermia, oligozoospermia and teratozoospermia, according to World Health Organisation guidelines. RESULTS: 39 percent of non-smokers showed normozoospermia, while only three percent of smokers were normozoospermics. Light smokers predominantly showed asthenozoospermia. Heavy smokers showed asthenozoospermia, teratozoospermia and oligozoospermia. Statistical analysis using Fisher's exact test showed that the incidence of both isolated asthenozoospermia (p-value is 0.0015) and asthenozoospermia with teratozoospermia (p-value is 0.0106) among smokers was significant, in comparison to non-smokers. Overall impact of asthenozoospermia (p-value is less than 0.0001) and teratozoospermia (p-value is 0.0328) but not of oligozoospermia was observed on the semen quality in smokers, compared with non-smokers. CONCLUSION: Asthenozoospermia, the most common semen variable in our study, can be an early indicator of reduction in quality of semen, as seen in light smokers. In addition, heavy smoking produces teratozoospermia, which further reduces semen quality. Oligozoospermia may be due to factors other than smoking.  (+info)

Seminal plasma anti-Mullerian hormone level correlates with semen parameters but does not predict success of testicular sperm extraction (TESE). (7/72)

AIM: To assess seminal plasma anti-Mullerian hormone (AMH) level relationships in fertile and infertile males. METHODS: Eighty-four male cases were studied and divided into four groups: fertile normozoospermia (n = 16), oligoasthenoteratozoospermia (n = 15), obstructive azoospermia (OA) (n = 13) and non-obstructive azoospermia (NOA) (n = 40). Conventional semen analysis was done for all cases. Testicular biopsy was done with histopathology and fresh tissue examination for testicular sperm extraction (TESE) in NOA cases. NOA group was subdivided according to TESE results into unsuccessful TESE (n = 19) and successful TESE (n = 21). Seminal plasma AMH was estimated by enzyme linked immunosorbent assay (ELISA) and serum follicular stimulating hormone (FSH) was estimated in NOA cases only by radioimmunoassay (RIA). RESULTS: Mean seminal AMH was significantly higher in fertile group than in oligoasthenoteratozoospermia with significance (41.5 +/- 10.9 pmol/L vs. 30.5 +/- 10.3 pmol/L, P < 0.05). Seminal AMH was not detected in any OA patients. Seminal AMH was correlated positively with testicular volume (r = 0.329, P = 0.005), sperm count (r = 0.483, P = 0.007), sperm motility percent (r = 0.419, P = 0.021) and negatively with sperm abnormal forms percent (r = -0.413, P = 0.023). Nonsignificant correlation was evident with age (r = -0.155, P = 0.414) and plasma FSH (r = -0.014, P = 0.943). In NOA cases, seminal AMH was detectable in 23/40 cases, 14 of them were successful TESE (57.5%) and was undetectable in 17/40 cases, 10 of them were unsuccessful TESE (58.2%). CONCLUSION: Seminal plasma AMH is an absolute testicular marker being absent in all OA cases. However, seminal AMH has a poor predictability for successful testicular sperm retrieval in NOA cases.  (+info)

Superoxide dismutase and catalase activities in the seminal plasma of normozoospermic and asthenozoospermic Beagles. (8/72)

We measured the blood plasma testosterone (T) levels and superoxide dismutase (SOD) and catalase activities in the seminal plasma of the ejaculates of 5 normal (2-5 years old) and 5 asthenozoospermic (AZ-) (3-5 years old) Beagles. Sperm ejaculated by AZ-dogs was incubated for 3 hr in Eagle's MEM only (controls) or Eagle's MEM containing 100 units/ml of SOD or catalase. Sperm motility was examined during incubation. The mean (+/- SE) plasma T level of the AZ-dogs (1.2 +/- 0.2 ng/ml) was significantly lower than in the normal dogs (2.5 +/- 0.2 ng/ml) (P<0.005). The mean (+/- SE) seminal plasma SOD and catalase activities (18.8 +/- 1.9 and 0.5 +/- 0.1 unit/g protein, respectively) were significantly lower in the AZ-dogs than in the normal dog (43.3 +/- 2.5 and 2.2 +/- 0.4 unit/g protein, respectively) (P<0.001 and 0.01, respectively). The motility of sperm incubated in Eagle's MEM containing SOD or catalase was significantly higher than that of control sperm incubated in only Eagle's MEM after 2 or 3 hr of incubation (P<0.05). The results of this study indicate that poor T secretion by the testes and low antioxidant enzyme activities are related to AZ in the dog.  (+info)

Asthenozoospermia is a term used in the field of andrology, which is the study of male reproductive health. It refers to a condition where the majority of sperm in a semen sample have reduced motility, meaning they do not move normally or efficiently. This can make it more difficult for the sperm to reach and fertilize an egg, potentially leading to infertility issues.

To be more specific, asthenozoospermia is defined as having less than 40% of sperm with progressive motility, which means they move forward in a straight line or in a large circle. The condition can be caused by various factors, including genetic abnormalities, environmental toxins, infections, and structural issues with the sperm themselves.

It's worth noting that asthenozoospermia is often diagnosed through a semen analysis, which is a routine test used to assess male fertility. If you or someone you know has been diagnosed with this condition, it may be helpful to consult with a reproductive endocrinologist or andrologist who can provide more information and guidance on potential treatment options.

Sperm motility is the ability of sperm to move actively and effectively through the female reproductive tract towards the egg for fertilization. It is typically measured as the percentage of moving sperm in a sample, and their progressiveness or velocity. Normal human sperm motility is generally defined as forward progression of at least 25 micrometers per second, with at least 50% of sperm showing progressive motility. Reduced sperm motility, also known as asthenozoospermia, can negatively impact fertility and reproductive outcomes.

Preserved food, in a medical context, refers to food that has been treated or processed in order to inhibit spoilage and prolong its shelf life. This is typically achieved through methods such as canning, pickling, smoking, drying, or freezing. These processes work by reducing the moisture content, increasing acidity, or introducing chemicals that prevent the growth of bacteria, fungi, and other microorganisms that can cause foodborne illness.

While preserved foods can be a valuable source of nutrition, especially in situations where fresh food is not available, it's important to note that some preservation methods can also introduce harmful substances, such as sodium nitrite in cured meats or acrylamide in fried or baked starchy foods. Therefore, preserved foods should be consumed in moderation as part of a balanced diet.

Male infertility is a condition characterized by the inability to cause pregnancy in a fertile female. It is typically defined as the failure to achieve a pregnancy after 12 months or more of regular unprotected sexual intercourse.

The causes of male infertility can be varied and include issues with sperm production, such as low sperm count or poor sperm quality, problems with sperm delivery, such as obstructions in the reproductive tract, or hormonal imbalances that affect sperm production. Other factors that may contribute to male infertility include genetic disorders, environmental exposures, lifestyle choices, and certain medical conditions or treatments.

It is important to note that male infertility can often be treated or managed with medical interventions, such as medication, surgery, or assisted reproductive technologies (ART). A healthcare provider can help diagnose the underlying cause of male infertility and recommend appropriate treatment options.

Spermatozoa are the male reproductive cells, or gametes, that are produced in the testes. They are microscopic, flagellated (tail-equipped) cells that are highly specialized for fertilization. A spermatozoon consists of a head, neck, and tail. The head contains the genetic material within the nucleus, covered by a cap-like structure called the acrosome which contains enzymes to help the sperm penetrate the female's egg (ovum). The long, thin tail propels the sperm forward through fluid, such as semen, enabling its journey towards the egg for fertilization.

Semen is a complex, whitish fluid that is released from the male reproductive system during ejaculation. It is produced by several glands, including the seminal vesicles, prostate gland, and bulbourethral glands. Semen contains several components, including sperm (the male reproductive cells), as well as various proteins, enzymes, vitamins, and minerals. Its primary function is to transport sperm through the female reproductive tract during sexual intercourse, providing nutrients and aiding in the protection of the sperm as they travel toward the egg for fertilization.

Oligospermia is a medical term used to describe a condition in which the semen contains a lower than normal number of sperm. Generally, a sperm count of less than 15 million sperm per milliliter (ml) of semen is considered to be below the normal range.

Oligospermia can make it more difficult for a couple to conceive naturally and may require medical intervention such as intracytoplasmic sperm injection (ICSI) or in vitro fertilization (IVF). The condition can result from various factors, including hormonal imbalances, genetic abnormalities, varicocele, environmental factors, and certain medications.

It's important to note that oligospermia is not the same as azoospermia, which is a condition where there is no sperm present in the semen at all.

Sperm count, also known as sperm concentration, is the number of sperm present in a given volume of semen. The World Health Organization (WHO) previously defined a normal sperm count as at least 20 million sperm per milliliter of semen. However, more recent studies suggest that fertility may be affected even when sperm counts are slightly lower than this threshold. It's important to note that sperm count is just one factor among many that can influence male fertility. Other factors, such as sperm motility (the ability of sperm to move properly) and morphology (the shape of the sperm), also play crucial roles in successful conception.

The pregnancy rate is a measure used in reproductive medicine to determine the frequency or efficiency of conception following certain treatments, interventions, or under specific conditions. It is typically defined as the number of pregnancies per 100 women exposed to the condition being studied over a specified period of time. A pregnancy is confirmed when a woman has a positive result on a pregnancy test or through the detection of a gestational sac on an ultrasound exam.

In clinical trials and research, the pregnancy rate helps healthcare professionals evaluate the effectiveness of various fertility treatments such as in vitro fertilization (IVF), intrauterine insemination (IUI), or ovulation induction medications. The pregnancy rate can also be used to assess the impact of lifestyle factors, environmental exposures, or medical conditions on fertility and conception.

It is important to note that pregnancy rates may vary depending on several factors, including age, the cause of infertility, the type and quality of treatment provided, and individual patient characteristics. Therefore, comparing pregnancy rates between different studies should be done cautiously, considering these potential confounding variables.

... (or asthenospermia) is the medical term for reduced sperm motility. Complete asthenozoospermia, that is, 100 ... asthenozoospermia) than in men with oligozoospermia or teratozoospermia. Among men with asthenozoospermia, 31% were found to ... Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see ... found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. The sperm ...
Cause Isolated Male Infertility Due to Asthenozoospermia". The American Journal of Human Genetics. 105 (1): 198-212. doi: ... "Association of DNAH11 gene polymorphisms with asthenozoospermia in Northeast Chinese patients". Bioscience Reports. 39 (6). doi ...
"Mutations in dynein genes in patients affected by isolated non-syndromic asthenozoospermia". Hum. Reprod. 23 (8): 1957-62. doi: ... with primary ciliary dyskinesia and multiple morphological anomalies of the flagella that result in asthenozoospermia and male ...
"ADCY10 frameshift variant leading to severe recessive asthenozoospermia and segregating with absorptive hypercalciuria". Human ...
In mice, a deletion on mtDNA causes oligospermia and asthenozoospermia, resulting in infertility. Taken together, mtDNA ...
"ADCY10 frameshift variant leading to severe recessive asthenozoospermia and segregating with absorptive hypercalciuria". Human ...
"Asthenozoospermia in mice with targeted deletion of the sperm mitochondrion-associated cysteine-rich protein (Smcp) gene". ...
This procedure takes approximately 60 minutes and it is specially indicated in oligozoospermia, asthenozoospermia and abundant ...
... is usually confused with asthenozoospermia, which is the inability of the sperm to move even when alive. To check ...
Studies in humans show that an excess of circulating thyroid hormones during thyrotoxicosis results in asthenozoospermia, ...
Haplogroup T can predispose to asthenozoospermia (Ruiz-Pesini 2000). However, these findings have been disputed due to a small ...
... is a combination of: Asthenozoospermia (reduced sperm motility) and Oligozoospermia (low spermatozoon ...
It is used in cases where men have severe cases of oligozoospermia, asthenozoospermia or teratozoospermia, and where the couple ...
... and asthenozoospermia [i.e. sperm with reduced motility]). Infertility associated with a sSMC in women is ~7.5-fold less common ...
... and asthenozoospermia [sperm with reduced motility]); c) band 11 from the short arm of chromosome 14 (notated as (14)q11.1)( ...
... increase in sperm with abnormal morphology Asthenozoospermia - reduced sperm motility Necrozoospermia - all sperm in the ...
Asthenozoospermia (or asthenospermia) is the medical term for reduced sperm motility. Complete asthenozoospermia, that is, 100 ... asthenozoospermia) than in men with oligozoospermia or teratozoospermia. Among men with asthenozoospermia, 31% were found to ... Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see ... found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. The sperm ...
... cause asthenozoospermia and multiple midpiece defects in humans. ... Background: Asthenozoospermia is a major factor contributing to ... Biallelic mutations in ARMC12 cause asthenozoospermia and multiple midpiece defects in humans and mice J Med Genet. 2023 Feb;60 ... However, the role of ARMC12 in human asthenozoospermia remains unknown. Objective: To study the genetic defects in patients ... Conclusion: Our findings prove for the first time that defects in ARMC12 cause asthenozoospermia and multiple midpiece defects ...
Read all posts about Asthenozoospermia,Dr.Yogesh Chavan,MD(Ayu.Kerala), ...
Asthenozoospermia is a sperm disorder characterized by problems in sperm motility. The World Health Organization (WHO) ... Asthenozoospermia is a sperm disorder characterized by problems in sperm motility. The World Health Organization (WHO) ... Otherwise, the seminal sample will be diagnosed with asthenozoospermia.. Since in ICSI it is the specialist who introduces the ...
... Pelloni M;Paoli D;MAJOLI, MARTINA;Pallotti F; ... Molecular study of human sperm RNA: Ropporin and CABYR in asthenozoospermia / Pelloni, M; Paoli, D; Majoli, Martina; Pallotti, ... Molecular study of human sperm RNA: Ropporin and CABYR in asthenozoospermia / Pelloni, M; Paoli, D; Majoli, Martina; Pallotti, ...
... SALVOLINI, ... Patient(s): Twenty-nine infertile patients affected by idiopathic asthenozoospermia and 26 age-matched normospermic fertile ... Patient(s): Twenty-nine infertile patients affected by idiopathic asthenozoospermia and 26 age-matched normospermic fertile ... Nitric oxide synthase and tyrosine nitration in idiopathic asthenozoospermia: an immunohistochemical study / Salvolini, ...
Asthenozoospermia or asthenospermia is the decrease in the motility of the mans sperm and compromises fertility because it ... Causes of Asthenozoospermia. Although the causes of asthenozoospermia are not exactly known, we do know that sperm motility can ... How is Asthenozoospermia diagnosed?. Asthenozoospermia can be diagnosed through a simple seminogram or semen analysis. It is ... What is Asthenozoospermia?. Asthenozoospermia or asthenospermia is the decrease in the mobility of spermatozoa in men. ...
The Association Between Dairy Product Consumption and Asthenozoospermia Risk: A Hospital-Based Case-Control Study.DOCX ... Table_1_The Association Between Dairy Product Consumption and Asthenozoospermia Risk: A Hospital-Based Case-Control Study. . ... Background: Evidence of an association between dairy product and main related dairy nutrient intake, and the asthenozoospermia ... Methods: A hospital-based case-control study including 549 men with asthenozoospermia and 581 normozoospermic controls was ...
ANALISIS EKSPRESI MICRORNA (MIRNA) HSA-MIR-34C-3P DAN HSA-MIR-34C-5P PADA SPERMATOZOA PRIA ASTHENOZOOSPERMIA. *Darsini, Ninik ( ...
Asthenozoospermia. This is sperm that have a motility deficiency. Sperm need to move quickly and effectively through the ...
VD3 supplementation may modulate OS and affect sperm motility in men with asthenozoospermia and serum 25-OH-D3 < 30 ng/ml. Iran ... This randomized, triple-masking, placebo-controlled clinical trial conducted on 86 asthenozoospermia infertile men with serum ... Vitamin D3 Supplementation Effects on Spermatogram and Oxidative Stress Biomarkers in Asthenozoospermia Infertile Men: a ...
Low TEX40 expression is associated with asthenozoospermia. Title: Proteomic analyses reveal lower expression of TEX40 and ...
Nine men with asthenozoospermia vs. fertile donors. Sperm was washed in Hepes-buffered saline, motility tested by ...
Expression of HBD1 is reduced in individuals with asthenozoospermia and leukocytospermia. In one study, treatment with ...
Omu, A.E.; Dashti, H.; Al-Othman, S. Treatment of asthenozoospermia with zinc sulphate: Andrological, immunological and ...
Omu AE, Dashti H, Al-Othman S. Treatment of asthenozoospermia with zinc sulphate: andrological, immunological and obstetric ...
Male mice homozygous for a null allele display infertility, asthenozoospermia, teratozoospermia, and impaired spermiogenesis. ...
... with primary ciliary dyskinesia and multiple morphological anomalies of the flagella that result in asthenozoospermia and male ...
poor mobility (asthenozoospermia) *and a low number of spermatozoa with a normal shape (teratozoospermia). ...
... focused on the testicles have often been done in men suffering from fertility issues such as azoospermia or asthenozoospermia ...
Men with sub-fertility (asthenozoospermia, defined as total sperm motility below 40% and sperm concentration below 20 million/ ...
Sperms poor motility (Asthenozoospermia). The need for this treatment arises when a man ejaculates, but his fluid doesnt ... Asthenozoospermia and ICSI. This treatment, known as Intracytoplasmic Sperm Injection (IUI), is done when ...
ADCY10 frameshift variant leading to severe recessive asthenozoospermia and segregating with absorptive hypercalciuria. A ...
One such factor that can cause male infertility is asthenozoospermia. Asthenozoospermia is a ... What are The Causes of Asthenozoospermia Like women, there are many factors that can cause infertility in men. ...
Asthenozoospermia: Does IVF Or ICSI Help With Poor Sperm Motility? November 28th, 2023 ...
... was related to asthenozoospermia (a clinical manifestation of male infertility) by regulating WDR4 gene expression and the DNA ...
asthenozoospermia. Cyp19a1. Cyp19a1tm1Esi/Cyp19a1tm1Esi. involves: 129S6/SvEvTac. color key. MP:0001177. atelectasis. Cyp1a2. ...
Asthenozoospermia. Male sperm motility is abnormal (it is referred to the way it swims). Keep in mind, when the sperm does not ... With asthenozoospermia around 100% of the sperm, samples are not able to move properly. In addition, this problem occurs in ... Absolute asthenozoospermia. IVF with ICSI is the first treatment option suggested to the couple. ...
  • Sperm DNA fragmentation level is higher in men with sperm motility defects (asthenozoospermia) than in men with oligozoospermia or teratozoospermia. (wikipedia.org)
  • A total of 205 DNA samples from 48 oligozoospermia (OZ), 52 asthenozoospermia (AZ), 55 teratozoospermia (TZ) patients, and 50 normozoospermia (NZ) men were analyzed. (edu.au)
  • The Total Antioxidant Capacity in the semen of infertile patients with asthenozoospermia, oligozoospermia, teratozoospermia, and their combinations - as well as in other pathological conditions - is often reduced to a statistically significant degree compared to normal fertile men. (athenslab.gr)
  • Male mice homozygous for a null allele display infertility, asthenozoospermia, teratozoospermia, and impaired spermiogenesis. (jax.org)
  • Absolute asthenozoospermia and ICSI: What are the options? (wikipedia.org)
  • In Vitro Fertilization with Intracytoplasmic Sperm Injection (IVF+ICSI) offers the highest success rates to patients with asthenozoospermia, thanks to the fact that it prevents sperm from having to travel to fertilize the egg. (ingenes.com)
  • Asthenozoospermia: Does IVF Or ICSI Help With Poor Sperm Motility? (reuniterx.com)
  • Asthenozoospermia (or asthenospermia) is the medical term for reduced sperm motility. (wikipedia.org)
  • Asthenozoospermia or asthenospermia is the decrease in the motility of the man's sperm and compromises fertility because it prevents them from moving to the ovum and penetrating it. (ingenes.com)
  • Asthenozoospermia or asthenospermia is the decrease in the mobility of spermatozoa in men. (ingenes.com)
  • If the test sample ranks only 25-30% of the sperms in A grade, this reflects low sperm motility, referred to as asthenozoospermia or asthenospermia. (aasthafertility.com)
  • Asthenozoospermia is a sperm disorder characterized by problems in sperm motility . (invitra.com)
  • VD3 supplementation may modulate OS and affect sperm motility in men with asthenozoospermia and serum 25-OH-D3 (bvsalud.org)
  • Asthenozoospermia - Sperm with reduced motility and consequently poor quality. (nordicalagos.org)
  • Patient(s): Twenty-nine infertile patients affected by idiopathic asthenozoospermia and 26 age-matched normospermic fertile donors. (univpm.it)
  • Complete asthenozoospermia, that is, 100% immotile spermatozoa in the ejaculate, is reported at a frequency of 1 of 5000 men. (wikipedia.org)
  • Conclusion(s): Our results support the hypothesis that increased NOS activity and an excess of tyrosine nitration may affect the functional characteristics of spermatozoa in idiopathic asthenozoospermia. (univpm.it)
  • In conclusion, kallikrein is a useful drug in the treatment of asthenozoospermia only when the sperm concentration is not decreased below the optimal value. (tau.ac.il)
  • Asthenozoospermia is a major factor contributing to male infertility. (nih.gov)
  • Methods: A hospital-based case-control study including 549 men with asthenozoospermia and 581 normozoospermic controls was carried out in the infertility clinics of Shengjing Hospital of China Medical University between June, 2020 and December, 2020. (figshare.com)
  • This randomized, triple-masking, placebo- controlled clinical trial conducted on 86 asthenozoospermia infertile men with serum 25 hydroxy vitamin D3 (25-OH-D3) infertility clinic of Ahvaz Jahad Daneshgahi, Iran . (bvsalud.org)
  • Naturally occurring mutations in this gene are associated with primary ciliary dyskinesia and multiple morphological anomalies of the flagella that result in asthenozoospermia and male infertility. (nih.gov)
  • One such factor that can cause male infertility is asthenozoospermia. (drrukkayal.com)
  • PHENOTYPE: Homozygous null mice display male infertility with asthenozoospermia. (utsouthwestern.edu)
  • Otherwise, the seminal sample will be diagnosed with asthenozoospermia. (invitra.com)
  • Asthenozoospermia is the most common seminal disorder and compromises fertility because it prevents sperm from traveling to the egg and penetrating it. (ingenes.com)
  • The objective is to evaluate the effects of vitamin D3 (VD3) on sperm parameters and seminal and serum oxidative stress (OS) biomarkers in asthenozoospermia infertile men . (bvsalud.org)
  • Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see Primary ciliary dyskinesia) and necrozoospermia. (wikipedia.org)
  • To study the genetic defects in patients with asthenozoospermia. (nih.gov)
  • Our findings prove for the first time that defects in ARMC12 cause asthenozoospermia and multiple midpiece defects in humans. (nih.gov)
  • A total of 125 patients with asthenozoospermia and 120 men with proven fertility were recruited. (nih.gov)
  • This method is used to facilitate fertility in couples with moderate asthenozoospermia and is only recommended when the woman is under 35 years of age. (ingenes.com)
  • The asthenozoospermia procedure should always be accompanied by advice for a healthy lifestyle, which in some cases can help restore fertility. (ingenes.com)
  • Asthenozoospermia: reduced sperm mobility. (wistim.com)
  • However, the role of ARMC12 in human asthenozoospermia remains unknown. (nih.gov)
  • Molecular study of human sperm RNA: Ropporin and CABYR in asthenozoospermia. (uniroma1.it)
  • Nitric oxide synthase and tyrosine nitration in idiopathic asthenozoospermia: an immunohistochemical study. (univpm.it)
  • Conclusion: This study provided limited evidence of an association between the intake of total dairy products and the main related dairy nutrients including protein, fat, calcium, saturated fatty acids, and phosphorus, and the asthenozoospermia risk. (figshare.com)
  • Results: After adjustment for potential confounders, we found no statistically significant associations between the intake of total dairy products and asthenozoospermia risk (OR T3vs.T1 = 1.19, 95%CI = 0.85-1.67). (figshare.com)
  • Of note, after stratification by body mass index (BMI), and the saturated fatty acids consumption from dairy was significantly associated with a higher asthenozoospermia risk (OR T3vs.T1 = 1.76, 95%CI = 1.01-3.09) among participants with a BMI below 25 kg/m 2 . (figshare.com)
  • Among men with asthenozoospermia, 31% were found to have high levels of DNA fragmentation. (wikipedia.org)
  • found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. (wikipedia.org)
  • An unconditional multiple logistic regression was used to estimate the odds ratios (ORs) and the corresponding 95% confidence intervals (CIs) for asthenozoospermia risk. (figshare.com)
  • With asthenozoospermia around 100% of the sperm, samples are not able to move properly. (gemivf.com)
  • Idiopathic asthenozoospermia, a common factor in male infertility, is characterized by altered sperm motility function in fresh ejaculate. (nih.gov)
  • This study provides valuable findings on a causative relationship between LRRC23 mutations and male infertility due to asthenozoospermia. (elifesciences.org)
  • Naturally occurring mutations in this gene are associated with primary ciliary dyskinesia and multiple morphological anomalies of the flagella that result in asthenozoospermia and male infertility. (nih.gov)
  • Clinically, azoospermia and asthenozoospermia are more common, especially asthenozoospermia. (diureticspill.com)
  • Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see Primary ciliary dyskinesia) and necrozoospermia. (wikipedia.org)
  • Overall, our results indicate that busulfan causes structural damage to the seminiferous tubules, which can lead to asthenozoospermia, and that Piwil1 gene is a marker gene of asthenozoospermia. (arccjournals.com)
  • However it should not be confused with Asthenozoospermia where sperms are non-motile but they are not dead. (indiraivf.com)
  • If in a sperm sample 40 percent of sperms are immotile then the diagnosis is asthenozoospermia. (drvikram.com)
  • 11. Absence of annulus in human asthenozoospermia: case report. (nih.gov)
  • Healthy lifestyle habits, including a varied and balanced diet and moderate exercise, can help improve certain mild asthenozoospermia problems in men. (invitra.com)
  • Although the β-defensin 126 (DEFB126) protein is associated with asthenozoospermia, DEFB126 gene polymorphisms have not been extensively studied. (nih.gov)
  • Therefore, the association between DEFB126 gene polymorphisms and asthenozoospermia requires further investigation. (nih.gov)
  • Screening was performed by semen analysis, karyotype analysis, and Y microdeletion detection, and 102 fertile men and 106 men with asthenozoospermia in Chengdu, China, were selected for DEFB126 gene sequence analyses. (nih.gov)
  • Asthenozoospermia is a condition where the sperm is so weak that it cannot successfully enter the woman's body and combine with the egg to form an embryo. (diureticspill.com)
  • To explore the histological etiology of asthenozoospermia, we measured the sperm quality index (SQI) and Piwil1 expression in the testes of three types of asthenozoospermia roosters: field, artificial and normal. (arccjournals.com)
  • It is important to note that once men find themselves with asthenozoospermia during pregnancy preparation, they do not have to be overly anxious and can effectively improve by taking relevant measures. (diureticspill.com)