A genus (formerly part of Rhus genus) of shrubs, vines, or trees that yields a highly allergenic oleoresin which causes a severe contact dermatitis (DERMATITIS, TOXICODENDRON). The most toxic species are Toxicodendron vernix (poison sumac), T. diversilobum (poison oak), and T. radicans (poison ivy). T. vernicifera yields a useful varnish from which certain enzymes (laccases) are obtained.
An allergic contact dermatitis caused by exposure to plants of the genus Toxicodendron (formerly Rhus). These include poison ivy, poison oak, and poison sumac, all plants that contain the substance urushiol, a potent skin sensitizing agent. (From Dorland, 27th ed)
A chronic inflammatory genetically determined disease of the skin marked by increased ability to form reagin (IgE), with increased susceptibility to allergic rhinitis and asthma, and hereditary disposition to a lowered threshold for pruritus. It is manifested by lichenification, excoriation, and crusting, mainly on the flexural surfaces of the elbow and knee. In infants it is known as infantile eczema.
Any inflammation of the skin.
A contact dermatitis due to allergic sensitization to various substances. These substances subsequently produce inflammatory reactions in the skin of those who have acquired hypersensitivity to them as a result of prior exposure.
Rare, chronic, papulo-vesicular disease characterized by an intensely pruritic eruption consisting of various combinations of symmetrical, erythematous, papular, vesicular, or bullous lesions. The disease is strongly associated with the presence of HLA-B8 and HLA-DR3 antigens. A variety of different autoantibodies has been detected in small numbers in patients with dermatitis herpetiformis.
A chronic inflammatory disease of the skin with unknown etiology. It is characterized by moderate ERYTHEMA, dry, moist, or greasy (SEBACEOUS GLAND) scaling and yellow crusted patches on various areas, especially the scalp, that exfoliate as dandruff. Seborrheic dermatitis is common in children and adolescents with HIV INFECTIONS.
A recurrent contact dermatitis caused by substances found in the work place.

Secondary bacterial infections complicating skin lesions. (1/7)

Secondary bacterial infection in skin lesions is a common problem. This review summarises a series of studies of the microbiology of several of these infections: scabies, psoriasis, poison ivy, atopic dermatitis, eczema herpeticum and kerion. Staphylococcus aureus and group A beta-haemolytic streptococci were the most prevalent aerobes and were isolated from all body sites. In contrast, organisms that reside in the mucous membranes close to the lesions predominated in infections next to these membranes. In this fashion, enteric gram-negative bacilli and Bacteroides spp. were found most often in buttock and leg lesions. The probable sources of these organisms are the rectum and vagina, where they normally reside. Group A beta-haemolytic streptococci, pigmented Prevotella and Porphyromonas spp. and Fusobacterium spp. were most commonly found in lesions of the head, face, neck and fingers. These organisms probably reached these sites from the oral cavity, where they are part of the normal flora. This review highlights the polymicrobial aerobic-anaerobic microbiology of secondarily infected skin lesions.  (+info)

CURRENT CONCEPTS IN DERMATOLOGY. III. THE USE OF RADIOTHERAPY AND CORTICOSTEROIDS. (2/7)

The use of ionizing radiation and corticosteroids is discussed, in this third and final part of a review of diseases of the skin. Radiation is being used less extensively because superior methods of treatment are available for many conditions which formerly were frequently treated by this modality. The concept of applying the radiation at the level of the basic pathologic process has been developed into clinical practice by the use of generators which can produce very soft (or superficial) ionizing radiation. Topical or systemic corticosteroids do not cure skin diseases but produce dramatic suppression of signs and symptoms. For best results consideration must be given to the diagnosis, the natural history of the disease to be treated, the method of administration and a search for possible contraindications to the use of these steroids. Basic dermatological principles (removal of offending agents, bland soothing applications, sedation, etc.) must be adhered to. The corticosteroids are not a panacea in the treatment of skin disease.  (+info)

Biomass and toxicity responses of poison ivy (Toxicodendron radicans) to elevated atmospheric CO2. (3/7)

Contact with poison ivy (Toxicodendron radicans) is one of the most widely reported ailments at poison centers in the United States, and this plant has been introduced throughout the world, where it occurs with other allergenic members of the cashew family (Anacardiaceae). Approximately 80% of humans develop dermatitis upon exposure to the carbon-based active compound, urushiol. It is not known how poison ivy might respond to increasing concentrations of atmospheric carbon dioxide (CO(2)), but previous work done in controlled growth chambers shows that other vines exhibit large growth enhancement from elevated CO(2). Rising CO(2) is potentially responsible for the increased vine abundance that is inhibiting forest regeneration and increasing tree mortality around the world. In this 6-year study at the Duke University Free-Air CO(2) Enrichment experiment, we show that elevated atmospheric CO(2) in an intact forest ecosystem increases photosynthesis, water use efficiency, growth, and population biomass of poison ivy. The CO(2) growth stimulation exceeds that of most other woody species. Furthermore, high-CO(2) plants produce a more allergenic form of urushiol. Our results indicate that Toxicodendron taxa will become more abundant and more "toxic" in the future, potentially affecting global forest dynamics and human health.  (+info)

Dermoscopy of black-spot poison ivy. (4/7)

Black-spot poison ivy is an uncommon presentation of poison ivy (Toxicodendron) allergic contact dermatitis. A 78-year-old sought evaluation of a black spot present on her right hand amid pruritic vesicles. The presentation of a black spot on the skin in a clinical context suggesting poison ivy is indicative of black-spot poison ivy. Dermoscopy revealed a jagged, centrally homogeneous, dark brown lesion with a red rim. A skin sample was obtained and compared against a poison ivy standard using ultra-fast liquid chromatography-tandem mass spectrometry (UFLC-MS/MS). This finding confirmed the presence of multiple urushiol congeners in the skin sample. Black-spot poison ivy may be added to the list of diagnoses that show a specific dermoscopic pattern.  (+info)

Urushiol (poison ivy)-triggered suppressor T cell clone generated from peripheral blood. (5/7)

Allergic contact dermatitis to Toxicodendron radicans (poison ivy) is mediated by the hapten urushiol. An urushiol-specific, interleukin 2 (IL-2)-dependent T cell clone (RLB9-7) was generated from the peripheral blood of a patient with a history of allergic contact dermatitis to T. radicans. This clone proliferated specifically to both leaf extract and pure urushiol. Although the clone had the phenotype CD3+CD4+CD8+, proliferation to antigen was blocked by anti-CD8 and anti-HLA-A, B, C, but not by anti-CD4, suggesting that CD4 was not functionally associated with the T cell receptor. Furthermore, studies with antigen-presenting cells from MHC-typed donors indicated that the clone was MHC class 1 restricted. RLB9-7 was WT31 positive, indicating it bears the alpha beta T cell receptor. The clone lacked significant natural killer cell activity and produced only low levels of IL-2 or gamma-interferon upon antigen stimulation. Addition of RLB9-7 to autologous peripheral blood mononuclear cells in the presence of urushiol inhibited the pokeweed mitogen-driven IgG synthesis. This suppression was resistant to irradiation (2,000 rad) and was not seen when RLB9-7 was added to allogeneic cells, even in the presence of irradiated autologous antigen-presenting cells, suggesting that suppression was MHC restricted and not mediated by nonspecific soluble factors. However, RLB9-7 cells in the presence of urushiol inhibited the synthesis of tetanus toxoid-specific IgG by autologous lymphocytes, indicating that the suppression, although triggered specifically by urushiol, was nonspecific.  (+info)

Keratinocyte intercellular adhesion molecule-1 (ICAM-1) expression precedes dermal T lymphocytic infiltration in allergic contact dermatitis (Rhus dermatitis). (6/7)

The ability of small molecules such as urushiol, present as a wax on the poison ivy leaf surface, to cause allergic contact dermatitis (rhus dermatitis) has fascinated immunologists for decades. Current dogma suggests that these epicutaneously applied catechol-containing molecules serve as haptens to conjugate with larger proteins via reactive o-quinone intermediates. These complexes are then recognized as foreign antigens by the immune system and elicit a hypersensitivity reaction. Phorbol ester can directly induce cultured keratinocyte (KC) intercellular adhesion molecule-1 (ICAM-1) expression via a protein kinase C (PK-C)-dependent mechanism. As urushiol is also a known PK-C agonist, we asked if topical application of a poison ivy/oak mixture could directly induce epidermal KC ICAM-1 expression. During the pre-erythematous phase of this reaction (4 to 20 hours), epidermal KCs expressed ICAM-1; this "initiation phase" preceded the appearance of activated memory T lymphocytes in the papillary dermis, and thus appeared to be nonlymphokine mediated. A near-contiguous cellular-adhesion molecular network was identified by ICAM-1 staining of basal KCs, dermal dendrocytes, and endothelial cells. During the second 24-hour period with the onset of erythema and edema, there was an "amplification phase" of more intense KC ICAM-1 expression coupled with relatively weak KC HLA-DR expression that coincided with dermal and epidermal T-cell infiltration. This suggests the presence of lymphokines, such as gamma interferon, during the amplification phase because of KC HLA-DR expression. On cultured KCs, urushiol directly induced ICAM-1 expression but not HLA-DR. Thus, in addition to functioning as an antigenic hapten, urushiol directly induces KC ICAM-1 expression. The KC ICAM-1 expression may then alter the dynamic trafficking of memory T cells in the epidermis, so as to initiate cutaneous inflammation in a nonantigen specific manner. This initiation phase is followed by T-cell infiltration and consequent lymphokine production that significantly amplifies the original stimulus. Thus much can still be learned about the molecular pathophysiology of this common type of cutaneous inflammation.  (+info)

Gas plant (Dictamnus albus) phytophotodermatitis simulating poison ivy. (7/7)

A 48-year-old man presented with an itchy rash that resembled superficial burns or cane marks on his left forearm; similar lesions had appeared every summer for 5 years. Poison ivy dermatitis had been the initial diagnosis, but the patient knew that this plant was absent from his well tended garden. A visit to the garden revealed the gas plant Dictamnus albus, and occlusive patch testing with leaf cuttings produced a reaction after the skin was exposed to sunlight. Gas plant phytophotodermatitis was diagnosed.  (+info)

"Toxicodendron" is a genus of flowering plants in the family Anacardiaceae, also known as the cashew family. This genus includes several species that are well-known for causing allergic reactions in humans, particularly through contact with their sap or urushiol-containing parts of the plant. The most common and notorious species in this genus is Toxicodendron radicans, also known as poison ivy, poison oak, and poison sumac. These plants can cause an itchy, blistering rash upon contact with the skin, which is a type of allergic reaction called contact dermatitis. The severity of the reaction can vary from person to person, depending on their sensitivity to urushiol and the amount of exposure they have had to the plant.

Toxicodendron dermatitis is a type of contact dermatitis that results from exposure to plants belonging to the Toxicodendron genus, which includes poison ivy, poison oak, and poison sumac. The reaction is caused by an oily resin called urushiol found in these plants. When the oil comes into contact with the skin, it can cause an allergic reaction that leads to a red, itchy rash, often with blisters or weeping lesions.

The rash usually appears within 12-72 hours after exposure and can last for several weeks. The severity of the reaction varies from person to person, depending on their sensitivity to urushiol and the amount of contact they had with the plant. In addition to direct skin contact, urushiol can also be spread through secondary sources such as clothing, pets, or tools that have come into contact with the plant.

Prevention measures include avoiding contact with Toxicodendron plants, wearing protective clothing and gloves when working in areas where these plants may be present, and washing skin and clothing thoroughly with soap and water after exposure. In some cases, medical treatment may be necessary to manage symptoms and prevent complications.

Atopic dermatitis is a chronic, inflammatory skin condition that is commonly known as eczema. It is characterized by dry, itchy, and scaly patches on the skin that can become red, swollen, and cracked over time. The condition often affects the skin on the face, hands, feet, and behind the knees, and it can be triggered or worsened by exposure to certain allergens, irritants, stress, or changes in temperature and humidity. Atopic dermatitis is more common in people with a family history of allergies, such as asthma or hay fever, and it often begins in infancy or early childhood. The exact cause of atopic dermatitis is not fully understood, but it is thought to involve a combination of genetic and environmental factors that affect the immune system and the skin's ability to maintain a healthy barrier function.

Dermatitis is a general term that describes inflammation of the skin. It is often characterized by redness, swelling, itching, and tenderness. There are many different types of dermatitis, including atopic dermatitis (eczema), contact dermatitis, seborrheic dermatitis, and nummular dermatitis.

Atopic dermatitis is a chronic skin condition that often affects people with a family history of allergies, such as asthma or hay fever. It typically causes dry, scaly patches on the skin that can be extremely itchy.

Contact dermatitis occurs when the skin comes into contact with an irritant or allergen, such as poison ivy or certain chemicals. This type of dermatitis can cause redness, swelling, and blistering.

Seborrheic dermatitis is a common condition that causes a red, itchy rash, often on the scalp, face, or other areas of the body where oil glands are located. It is thought to be related to an overproduction of oil by the skin's sebaceous glands.

Nummular dermatitis is a type of eczema that causes round, coin-shaped patches of dry, scaly skin. It is more common in older adults and often occurs during the winter months.

Treatment for dermatitis depends on the underlying cause and severity of the condition. In some cases, over-the-counter creams or lotions may be sufficient to relieve symptoms. Prescription medications, such as corticosteroids or immunosuppressants, may be necessary in more severe cases. Avoiding triggers and irritants can also help prevent flare-ups of dermatitis.

Allergic contact dermatitis is a type of inflammatory skin reaction that occurs when the skin comes into contact with a substance (allergen) that the immune system recognizes as foreign and triggers an allergic response. This condition is characterized by redness, itching, swelling, blistering, and cracking of the skin, which usually develops within 24-48 hours after exposure to the allergen. Common allergens include metals (such as nickel), rubber, medications, fragrances, and cosmetics. It is important to note that a person must first be sensitized to the allergen before developing an allergic response upon subsequent exposures.

Dermatitis herpetiformis (DH) is a chronic, autoimmune blistering skin disorder that is characterized by the presence of symmetrical, pruritic (itchy), papulo-vesicular (papules and small fluid-filled blisters) eruptions on the extensor surfaces of the body, such as the elbows, knees, buttocks, and shoulders. It is often associated with gluten sensitivity or celiac disease, a condition that causes an abnormal immune response to gluten, a protein found in wheat, barley, and rye.

The exact cause of DH is not fully understood, but it is believed to result from the interaction between genetic, environmental, and immunological factors. The disorder is characterized by the presence of IgA antibodies in the skin, which trigger an immune response that leads to the formation of the characteristic rash.

DH is typically treated with a gluten-free diet, which can help to control the symptoms and prevent complications such as malabsorption and nutritional deficiencies. Medications such as dapsone may also be used to control the itching and blistering associated with the disorder. In some cases, topical corticosteroids or other anti-inflammatory medications may be prescribed to help manage symptoms.

It is important to note that DH is a chronic condition that requires ongoing management and monitoring. People with DH should work closely with their healthcare provider to develop an appropriate treatment plan and monitor their progress over time.

Seborrheic dermatitis is a common, inflammatory skin condition that mainly affects the scalp, face, and upper part of the body. It causes skin irritation, flaking, and redness, often in areas where the skin is oily or greasy. The exact cause of seborrheic dermatitis is not fully understood, but it appears to be related to a combination of genetic, environmental, and microbial factors.

The symptoms of seborrheic dermatitis can vary in severity and may include:

* Greasy or flaky scales on the scalp, eyebrows, eyelashes, ears, or beard
* Redness and inflammation of the skin
* Itching, burning, or stinging sensations
* Yellow or white crusty patches on the scalp or other affected areas
* Hair loss (in severe cases)

Seborrheic dermatitis is a chronic condition that tends to flare up and then subside over time. While there is no cure for seborrheic dermatitis, various treatments can help manage the symptoms and prevent complications. These may include medicated shampoos, topical creams or ointments, and lifestyle changes such as stress reduction and avoiding triggers that worsen symptoms.

It is important to note that seborrheic dermatitis should not be confused with other skin conditions, such as psoriasis or eczema, which may have similar symptoms. A healthcare professional can provide a proper diagnosis and recommend appropriate treatment options based on the individual's specific needs.

Occupational dermatitis is a specific type of contact dermatitis that results from exposure to certain substances or conditions in the workplace. It can be caused by direct contact with chemicals, irritants, or allergens present in the work environment. This condition typically affects the skin on the hands and forearms but can also involve other areas of the body, depending on the nature of the exposure.

There are two main types of occupational dermatitis:

1. Irritant contact dermatitis (ICD): This type occurs when the skin comes into direct contact with an irritating substance, leading to redness, swelling, itching, and sometimes blistering. Common irritants include solvents, detergents, oils, and other industrial chemicals.
2. Allergic contact dermatitis (ACD): This type is a result of an allergic reaction to a specific substance. The immune system identifies the allergen as harmful and mounts a response, causing skin inflammation. Common allergens include latex, metals (such as nickel), and certain plants (like poison ivy).

Prevention measures for occupational dermatitis include using appropriate personal protective equipment (PPE) like gloves, masks, and aprons, as well as practicing good hygiene, such as washing hands regularly and avoiding touching the face with contaminated hands. If you suspect you have developed occupational dermatitis, consult a healthcare professional for proper diagnosis and treatment.

... that occurs from exposure to members of the plant genus Toxicodendron. In North America, this includes poison ivy, poison oak, ... Toxicodendron dermatitis is an allergic contact dermatitis (allergic phytodermatitis) ... Clinical and immunologic features of systemic contact dermatitis from ingestion of Rhus (Toxicodendron). Contact Dermatitis. ... Contact dermatitis from Rhus toxicodendron in a homeopathic remedy. J Am Acad Dermatol. 2004 Jan. 50(1):150-1. [QxMD MEDLINE ...
Urushiol-induced contact dermatitis (also called Toxicodendron dermatitis or Rhus dermatitis) is a type of allergic contact ... Dermatitis from poison sumac is less common. Urushiol causes an eczematous contact dermatitis characterized by redness, ... dermatitis caused by the oil urushiol found in various plants, most notably sumac family species of the genus Toxicodendron: ... Urushiol-induced contact dermatitis is caused by contact with a plant or any other object containing urushiol oil. The oil ...
Poison ivy (Toxicodendron) dermatitis. http://www.uptodate.com/contents/search. Accessed June 10, 2020. ... Contact dermatitis Enlarge image Close Contact dermatitis. Contact dermatitis. Illustration of contact dermatitis on different ... Contact dermatitis. Mayo Clinic; 2019.. *Kermott CA, et al., eds. Poison ivy rash. In: Mayo Clinic Book of Home Remedies. 2nd ... Poison ivy allergic contact dermatitis. Mayo Clinic; 2019.. *Outsmarting poison ivy and other poisonous plants. U.S. Food and ...
Toxicodendron (rhus) allergic contact dermatitis: (poison ivy, oak, or sumac). In: Buttaravoli P, Leffler SM, Herrington RR, ... Contact dermatitis and patch testing. In: Dinulos JGH, ed. Habifs Clinical Dermatology. 7th ed. Philadelphia, PA: Elsevier; ... Plant-induced dermatitis. In: Auerbach PS, Cushing TA, Harris NS, eds. Auerbachs Wilderness Medicine. 7th ed. Philadelphia, PA ... The oil can also stick to animal fur, which explains why people often contract the skin irritation (dermatitis) from their ...
Wilderness & Environmental Medicine: "Toxicodendron dermatitis: Poison ivy, oak, and sumac.". *British Journal of Dermatology ... Seborrheic Dermatitis. Seborrheic dermatitis is the single most common rash of adults. When it occurs in infancy, it is ... Eczema (Dermatitis). Eczema (sometimes called "dermatitis") is a genetic condition associated with itchy, dry skin. It usually ... There are many types of eczema, and many types include the word "dermatitis" (in dermatology, dermatitis is another word for ...
In the U.S., the most common dermatitis-causing plant genus is the Toxicodendron (formerly Rhus). Approximately 50%-75% of the ... Plant dermatitis is an allergic inflammatory skin reaction in response to the oils of poisonous plants. ... U.S. adult population are susceptible to skin reactions upon exposure to Toxicodendron oil or oleoresin, called urushiol. ... In the U.S., the most common dermatitis-causing plant genus is Toxicodendron (formerly Rhus) (i.e., poison ivy, poison oak, and ...
Categories: Dermatitis, Toxicodendron Image Types: Photo, Illustrations, Video, Color, Black&White, PublicDomain, ...
Poison ivy and a related Toxicodendron species are considered a public health concern because they cause contact dermatitis, an ... These are different types of dermatitis caused by exposure to plants:. *Mechanical dermatitis occurs when the skin is cut, ... Irritant dermatitis can happen when an irritating chemical in the plant makes contact with the skin. This makes up about 80% of ... The classic allergenic dermatitis of these plants is caused by urushiol. Contrary to popular belief, for urushiol to come into ...
2001 Apr;6(7):3-5. "Treatment of toxicodendron dermatitis (poison ivy and poison oak)," by JD Guin. http://www.ncbi.nlm.nih.gov ...
Toxicodendron (rhus) allergic contact dermatitis: (poison ivy, oak, or sumac). In: Buttaravoli P, Leffler SM, Herrington RR, ... Contact dermatitis and patch testing. In: Dinulos JGH, ed. Habifs Clinical Dermatology. 7th ed. Philadelphia, PA: Elsevier; ... Plant-induced dermatitis. In: Auerbach PS, Cushing TA, Harris NS, eds. Auerbachs Wilderness Medicine. 7th ed. Philadelphia, PA ... The oil can also stick to animal fur, which explains why people often contract the skin irritation (dermatitis) from their ...
In the U.S., the most common dermatitis-causing plant genus is the Toxicodendron (formerly Rhus). Approximately 50%-75% of the ... Update: Plant Dermatitis Among Active Component Service Members, U.S. Armed Forces, 2010-2020. ... Plant dermatitis is an allergic inflammatory skin reaction in response to the oils of poisonous plants. ... U.S. adult population are susceptible to skin reactions upon exposure to Toxicodendron oil or oleoresin, called urushiol. ...
Toxicodendron Contact Dermatitis: A Case Report and Brief Review. https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC7733371/ ...
... remains the best approach to preventing Toxicodendron dermatitis.. If ingested, poison ivy can produce severe gastrointestinal ... Contact dermatitis can also result from mango ingestion without first removing the fruit skin, or from lacquer tree sap (from ... A major factor in poison plant dermatitis is the failure to recognize the plant. In general, all of the plants discussed here ... Barring complications, the dermatitis is self-limiting, lasting from 1 to 3 weeks. The skin usually recovers unblemished, ...
Poison Ivy: It is also known as Toxicodendron Radicans, poison ivy can cause contact dermatitis when in contact with the human ...
DERMATITIS, TOXICODENDRON). The most toxic species are Toxicodendron vernix (poison sumac), T. diversilobum (poison oak), and T ... Exploring the mango-poison ivy connection: the riddle of discriminative plant dermatitis. Contact Dermatitis. 2005 Jan; 52(1):3 ... "Toxicodendron" is a descriptor in the National Library of Medicines controlled vocabulary thesaurus, MeSH (Medical Subject ... Biomass and toxicity responses of poison ivy (Toxicodendron radicans) to elevated atmospheric CO2. Proc Natl Acad Sci U S A. ...
Toxicodendron) genus details, photos and seasonal allergies, Camden county, North Carolina ... Toxicodendron)? Research Poison-Ivy, Poison-Oak, Poison-Sumac ( ... delayed hypersensitivity or contact dermatitis 24 to 48 hours ... Poison-Ivy, Poison-Oak, Poison-Sumac (Toxicodendron). Home « North Carolina « Camden « Toxicodendron ... Poison-Ivy, Poison-Oak, Poison-Sumac (Toxicodendron) is a genus of the ANACARDIACEAE family.. This genus includes the following ...
Toxicodendron vernicifluum is a deciduous Tree growing to 15 m (49ft) by 10 m (32ft). See above for USDA hardiness. It is hardy ... from a burning plant or even contact with pets or animals that have touched the plant can cause severe allergic dermatitis in ... Toxicodendron vernicifluum is a deciduous Tree growing to 15 m (49ft) by 10 m (32ft). See above for USDA hardiness. It is hardy ... Rhus toxicodendron. Eastern Poison Oak. Shrub. 0.6. 6-9 LMH. N. DM. 0. 2. 2. ...
Dermatitis 2020 Mar/Apr; 31(2):99-105 18. Urushiol compounds detected in Toxicodendron-labeled consumer products using mass ...
All parts of the plant are toxic, an allergic contact dermatitis. About 70% of adults who contact poison oak or poison ivy ( ... The accepted name is Toxicodendron diversilobum, formerly Rhus diversiloba. * Native to western parts of Washington, Oregon, ... Toxicodendron radicans) will develop a rash in a delayed fashion from about 8 hr minimum to 2 weeks later. The rash is due to ...
Up to 25% of firefighters may need to leave the fireline because of severe toxicodendron dermatitis.136 This group would ... The true prevalence of dermatitis among florists is unknown.117 Irritant dermatitis outweighs allergic contact dermatitis in ... Frullania and lichen dermatitis accounts for 12% of occupational dermatitis in outdoor workers.137 The dermatitis is worse in ... 132 No study demonstrated any relationship between atopy and dermatitis. Most importantly, most cases of hand dermatitis were ...
toxicodendron dermatitis DOID:3819 * secondary corneal edema DOID:11032 * eversion of lacrimal punctum ...
Dermatitis, Toxicodendron. 2. + + 24. Dermatitis, Photoallergic. 2. + + 25. Asthma. 2. + + 26. Hypersensitivity, Immediate. 2. ...
Dermatitis shows itself in painful and long-continued swellings and eruptions. In the worst case, smoke inhaled by burning ... Poison Oak according to Wikipedia can cause Toxicodendron diversilobum.. Ssin contact first causes itching; then evolves into ... According to Wikipedia: Urushiol-induced contact dermatitis is the allergic reaction caused by poison ivy. In extreme cases, a ... According to Wikipedia, in terms of its potential to cause urushiol-induced contact dermatitis, poison sumac is more toxic than ...
Stay clear of Toxicodendron plants. If you come in contact with it, quickly washing the contact point with soap and water can ... In medical terms, this rash is called contact dermatitis. Its used to describe anything that causes skin swelling after ... Poison ivy and its relatives-poison oak and poison sumac-belong to the genus Toxicodendron. These plants are commonly found ... Poison ivy-induced contact dermatitis is still common in North America and affects around 50 million Americans every year. ...
Western poison-ivy (Toxicodendron rydbergii, alternatively Rhus radicans) - CAUSES DERMATITIS. **Interior or Woods rose (Rosa ... Western poison-ivy (Toxicodendron rydbergii, alternatively Rhus radicans) - CAUSES DERMATITIS. GRASSES & GRASSLIKE PLANTS (not ...
Discover the 1st step of our experiment on poison ivy induced dermatitis on NativeSkin®. Watch our timelaps video to see what ... Gladman, Aaron C. "Toxicodendron Dermatitis: Poison Ivy, Oak, and Sumac." Wilderness and environmental medecine, vol. 17, no. 2 ... The major allergen responsible for Toxicodendron dermatitis is the urushiol, contained in the oleoresinous sap of the plant (3) ... In the United States, the most common allergic contact dermatitis is caused by Toxicodendron species such as poison ivy, poison ...
2020). Toxicodendron contact dermatitis: A case report and brief review. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7733371/ ... 2023). Toxicodendron toxicity. https://www.ncbi.nlm.nih.gov/books/NBK557866/. *Monroe J, et al. ( ...

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