Neuralgic syndromes which feature chronic or recurrent FACIAL PAIN as the primary manifestation of disease. Disorders of the trigeminal and facial nerves are frequently associated with these conditions.
A syndrome characterized by recurrent episodes of excruciating pain lasting several seconds or longer in the sensory distribution of the TRIGEMINAL NERVE. Pain may be initiated by stimulation of trigger points on the face, lips, or gums or by movement of facial muscles or chewing. Associated conditions include MULTIPLE SCLEROSIS, vascular anomalies, ANEURYSMS, and neoplasms. (Adams et al., Principles of Neurology, 6th ed, p187)
Pain in nerves, frequently involving facial SKIN, resulting from the activation the latent varicella-zoster virus (HERPESVIRUS 3, HUMAN). The two forms of the condition preceding the pain are HERPES ZOSTER OTICUS; and HERPES ZOSTER OPHTHALMICUS. Following the healing of the rashes and blisters, the pain sometimes persists.
Intense or aching pain that occurs along the course or distribution of a peripheral or cranial nerve.
Severe or complete loss of facial muscle motor function. This condition may result from central or peripheral lesions. Damage to CNS motor pathways from the cerebral cortex to the facial nuclei in the pons leads to facial weakness that generally spares the forehead muscles. FACIAL NERVE DISEASES generally results in generalized hemifacial weakness. NEUROMUSCULAR JUNCTION DISEASES and MUSCULAR DISEASES may also cause facial paralysis or paresis.
The 7th cranial nerve. The facial nerve has two parts, the larger motor root which may be called the facial nerve proper, and the smaller intermediate or sensory root. Together they provide efferent innervation to the muscles of facial expression and to the lacrimal and SALIVARY GLANDS, and convey afferent information for TASTE from the anterior two-thirds of the TONGUE and for TOUCH from the EXTERNAL EAR.
The 5th and largest cranial nerve. The trigeminal nerve is a mixed motor and sensory nerve. The larger sensory part forms the ophthalmic, mandibular, and maxillary nerves which carry afferents sensitive to external or internal stimuli from the skin, muscles, and joints of the face and mouth and from the teeth. Most of these fibers originate from cells of the TRIGEMINAL GANGLION and project to the TRIGEMINAL NUCLEUS of the brain stem. The smaller motor part arises from the brain stem trigeminal motor nucleus and innervates the muscles of mastication.
Diseases of the ninth cranial (glossopharyngeal) nerve or its nuclei in the medulla. The nerve may be injured by diseases affecting the lower brain stem, floor of the posterior fossa, jugular foramen, or the nerve's extracranial course. Clinical manifestations include loss of sensation from the pharynx, decreased salivation, and syncope. Glossopharyngeal neuralgia refers to a condition that features recurrent unilateral sharp pain in the tongue, angle of the jaw, external auditory meatus and throat that may be associated with SYNCOPE. Episodes may be triggered by cough, sneeze, swallowing, or pressure on the tragus of the ear. (Adams et al., Principles of Neurology, 6th ed, p1390)
Pain associated with a damaged PUDENDAL NERVE. Clinical features may include positional pain with sitting in the perineal and genital areas, sexual dysfunction and FECAL INCONTINENCE and URINARY INCONTINENCE.
Observable changes of expression in the face in response to emotional stimuli.
Mechanical compression of nerves or nerve roots from internal or external causes. These may result in a conduction block to nerve impulses (due to MYELIN SHEATH dysfunction) or axonal loss. The nerve and nerve sheath injuries may be caused by ISCHEMIA; INFLAMMATION; or a direct mechanical effect.
Muscles of facial expression or mimetic muscles that include the numerous muscles supplied by the facial nerve that are attached to and move the skin of the face. (From Stedman, 25th ed)
Surgical interruption of a spinal or cranial nerve root. (From Dorland, 28th ed)
An attenuated vaccine used to prevent and/or treat HERPES ZOSTER, a disease caused by HUMAN HERPESVIRUS 3.
Pain in the facial region including orofacial pain and craniofacial pain. Associated conditions include local inflammatory and neoplastic disorders and neuralgic syndromes involving the trigeminal, facial, and glossopharyngeal nerves. Conditions which feature recurrent or persistent facial pain as the primary manifestation of disease are referred to as FACIAL PAIN SYNDROMES.
Surgery performed to relieve pressure from MICROVESSELS that are located around nerves and are causing NERVE COMPRESSION SYNDROMES.
Diseases of the facial nerve or nuclei. Pontine disorders may affect the facial nuclei or nerve fascicle. The nerve may be involved intracranially, along its course through the petrous portion of the temporal bone, or along its extracranial course. Clinical manifestations include facial muscle weakness, loss of taste from the anterior tongue, hyperacusis, and decreased lacrimation.
The facial skeleton, consisting of bones situated between the cranial base and the mandibular region. While some consider the facial bones to comprise the hyoid (HYOID BONE), palatine (HARD PALATE), and zygomatic (ZYGOMA) bones, MANDIBLE, and MAXILLA, others include also the lacrimal and nasal bones, inferior nasal concha, and vomer but exclude the hyoid bone. (Jablonski, Dictionary of Dentistry, 1992, p113)
Junction between the cerebellum and the pons.
General or unspecified injuries to the soft tissue or bony portions of the face.
The anterior portion of the head that includes the skin, muscles, and structures of the forehead, eyes, nose, mouth, cheeks, and jaw.

Craniofacial pain and motor function: pathogenesis, clinical correlates, and implications. (1/24)

Many structural, behavioral, and pharmacological interventions imply that favorable treatment effects in musculoskeletal pain states are mediated through the correction of muscle function. The common theme of these interventions is captured in the popular idea that structural or psychological factors cause muscle hyperactivity, muscle overwork, muscle fatigue, and ultimately pain. Although symptoms and signs of motor dysfunction can sometimes be explained by changes in structure, there is strong evidence that they can also be caused by pain. This new understanding has resulted in a better appreciation of the pathogenesis of symptoms and signs of the musculoskeletal pain conditions, including the sequence of events that leads to the development of motor dysfunction. With the improved understanding of the relationship between pain and motor function, including the inappropriateness of many clinical assumptions, a new literature emerges that opens the door to exciting therapeutic opportunities. Novel treatments are expected to have a profound impact on the care of musculoskeletal pain and its effect on motor function in the not-too-distant future.  (+info)

Musculoskeletal disorders of the neck and shoulders in female sewing machine operators: prevalence, incidence, and prognosis. (2/24)

OBJECTIVES: To assess the occurrence and persistence of two restrictively defined neck-shoulder disorders among sewing machine operators. To assess factors associated with the development of neck-shoulder disorder and prognostic factors for remaining a case, when disorders were already present. METHODS: In an initial group of 243 sewing machine operators, 178 were followed up for 2 years. At baseline and at 1 and 2 years follow up the participants underwent a clinical examination of the neck and arms and filled in a questionnaire about current musculoskeletal complaints. Clinical criteria for two main neck-shoulder disorders were defined: rotator cuff tendinitis and myofascial pain syndrome. A baseline control group consisted of 357 women with varied non-repetitive work. RESULTS: At baseline the overall prevalence of myofascial pain syndrome and rotator cuff tendinitis was 15.2% and 5.8% among sewing machine operators compared with 9.0% and 2.2%, respectively, among controls. The presence of the disorders was strongly associated with a self perception of poor general health. Although myofascial pain syndrome showed a U shaped association with years as a sewing machine operator, rotator cuff tendinitis was absent among the newest recruits and present among 15% of the women with more than 20 years as a sewing machine operator. Besides years as a sewing machine operator, the risk of having a neck-shoulder disorder at baseline was significantly associated with high stress (prevalence ratio (PR)=2.54; 95% confidence interval (95% CI) 1.28 to 5.05) when adjusted for age, body mass index, smoking, living alone with children, job strain, and social support from colleagues and supervisors. Only one of 13 participants with rotator cuff tendinitis at baseline recovered during follow up. Myofascial pain syndrome showed a much more fluctuating tendency. Low social support (RR 3.72; 95% CI 1.22 to 11.30) and smoking (RR 3.93; 95% CI 1.33 to 11.58) were associated with the development of neck-shoulder disorders, which was also associated with neck-shoulder pain score and living alone with children. CONCLUSION: Rotator cuff tendinitis showed a higher degree of persistence than myofascial pain syndrome. Both disorders highly influenced the perception of general health. Women who lived alone with children, were smokers, or experienced low support from colleagues and supervisors had a higher risk of contracting a neck-shoulder disorder.  (+info)

Myofascial pain syndrome in farmers--a comprehensive approach to treatment. (3/24)

There is evidence that chronic pain disorders such as Myofascial Pain Syndrome (MPS), resulting from repeated biomechanical stress caused by ergonomic hazards. e.g. trauma and overuse of the muscles, often occur in agricultural workers. Hypothetically, the neuropathic character of MPS makes the disease unresponsive to the typical analgesics. Accordingly, in this study three trials of treatment in patients with MPS were performed and compared. The first trial (I) was based on rehabilitation, while the second (II) was based on treatment with sertraline, an antidepressive, serotoninergic drug. For third trial (III), rehabilitation plus the above-mentioned administration of sertraline, were applied. Altogether, 49 patients were recruited to the trials. Control group consisted of 23 persons. Response to the treatment was assessed according to the criteria of neuropsychological tests MADRS and BDI. The MPS syndrome was found to be relatively common in Polish farmers and formed 12.7% of all chronic pain syndromes diagnosed in the Institute of Agricultural Medicine during 18 months. All the patients with MPS showed mood disorders in the baseline assessment by the neuropsychological tests. Patients from groups I, II, and III declared improvement after two months of the treatment (77%, 80% and 93% respectively). In the neuropsychological tests, only patients treated with rehabilitation and sertraline (group III) showed statistically significant improvement in comparison with baseline assessment both after one month and after two months of the observation. Thus, rehabilitation and serotoninergic system modification might be a good solution in the management of MPS.  (+info)

Randomised trial of acupuncture compared with conventional massage and "sham" laser acupuncture for treatment of chronic neck pain. (4/24)

OBJECTIVES: To compare the efficacy of acupuncture and conventional massage for the treatment of chronic neck pain. DESIGN: Prospective, randomised, placebo controlled trial. SETTING: Three outpatient departments in Germany. PARTICIPANTS: 177 patients aged 18-85 years with chronic neck pain. INTERVENTIONS: Patients were randomly allocated to five treatments over three weeks with acupuncture (56), massage (60), or "sham" laser acupuncture (61). MAIN OUTCOME MEASURES: PRIMARY OUTCOME MEASURE: maximum pain related to motion (visual analogue scale) irrespective of direction of movement one week after treatment. SECONDARY OUTCOME MEASURES: range of motion (3D ultrasound real time motion analyser), pain related to movement in six directions (visual analogue scale), pressure pain threshold (pressure algometer), changes of spontaneous pain, motion related pain, global complaints (seven point scale), and quality of life (SF-36). Assessments were performed before, during, and one week and three months after treatment. Patients' beliefs in treatment were assessed. RESULTS: One week after five treatments the acupuncture group showed a significantly greater improvement in motion related pain compared with massage (difference 24.22 (95% confidence interval 16.5 to 31.9), P=0.0052) but not compared with sham laser (17.28 (10.0 to 24.6), P=0.327). Differences between acupuncture and massage or sham laser were greater in the subgroup who had had pain for longer than five years (n=75) and in patients with myofascial pain syndrome (n=129). The acupuncture group had the best results in most secondary outcome measures. There were no differences in patients' beliefs in treatment. CONCLUSIONS: Acupuncture is an effective short term treatment for patients with chronic neck pain, but there is only limited evidence for long term effects after five treatments.  (+info)

Neurogenic pain relief by repetitive transcranial magnetic cortical stimulation depends on the origin and the site of pain. (5/24)

OBJECTIVE: Drug resistant neurogenic pain can be relieved by repetitive transcranial magnetic stimulation (rTMS) of the motor cortex. This study was designed to assess the influence of pain origin, pain site, and sensory loss on rTMS efficacy. PATIENTS AND METHODS: Sixty right handed patients were included, suffering from intractable pain secondary to one of the following types of lesion: thalamic stroke, brainstem stroke, spinal cord lesion, brachial plexus lesion, or trigeminal nerve lesion. The pain predominated unilaterally in the face, the upper limb, or the lower limb. The thermal sensory thresholds were measured within the painful zone and were found to be highly or moderately elevated. Finally, the pain level was scored on a visual analogue scale before and after a 20 minute session of "real" or "sham" 10 Hz rTMS over the side of the motor cortex corresponding to the hand on the painful side, even if the pain was not experienced in the hand itself. RESULTS: and discussion: The percentage pain reduction was significantly greater following real than sham rTMS (-22.9% v -7.8%, p = 0.0002), confirming that motor cortex rTMS was able to induce antalgic effects. These effects were significantly influenced by the origin and the site of pain. For pain origin, results were worse in patients with brainstem stroke, whatever the site of pain. This was consistent with a descending modulation within the brainstem, triggered by the motor corticothalamic output. For pain site, better results were obtained for facial pain, although stimulation was targeted on the hand cortical area. Thus, in contrast to implanted stimulation, the target for rTMS procedure in pain control may not be the area corresponding to the painful zone but an adjacent one. Across representation plasticity of cortical areas resulting from deafferentation could explain this discrepancy. Finally, the degree of sensory loss did not interfere with pain origin or pain site regarding rTMS effects. CONCLUSION: Motor cortex rTMS was found to result in a significant but transient relief of chronic pain, influenced by pain origin and pain site. These parameters should be taken into account in any further study of rTMS application in chronic pain control.  (+info)

The epidemiology of chronic syndromes that are frequently unexplained: do they have common associated factors? (6/24)

BACKGROUND: Syndromes for which no physical or pathological changes can be found tend to be researched and managed in isolation although hypotheses suggest that they may be one entity. The objectives of our study were to investigate the co-occurrence, in the general population, of syndromes that are frequently unexplained and to evaluate whether they have common associated factors. METHODS: We conducted a population-based cross-sectional survey that included 2,299 subjects who were registered with a General Medical Practice in North-west England and who completed full postal questionnaires (response rate 72%). The study investigated four chronic syndromes that are frequently unexplained: chronic widespread pain, chronic oro-facial pain, irritable bowel syndrome, and chronic fatigue. Validated instruments were used to measure the occurrence of syndromes and to collect information on a variety of associated factors: demographic (age, gender), psychosocial (anxiety, depression, illness behaviour), life stressors, and reporting of somatic symptoms. RESULTS: We found that 587 subjects (27%) reported one or more syndromes: 404 (18%) reported one, 134 (6%) reported two, 34 (2%) reported three, and 15 (1%) reported all four syndromes. The occurrence of multiple syndromes was greater than would be expected by chance (P < 0.001). There were factors that were common across syndromes: female gender [odds ratio (OR) = 1.8; 95% confidence interval (95% CI) 1.5-2.2], high levels of aspects of health anxiety like health worry preoccupation (OR = 3.5; 95% CI 2.8-4.4) and reassurance seeking behaviour (OR = 1.4; 95% CI 1.1-1.7), reporting of other somatic symptoms (OR = 3.6; 95% CI 2.9-4.4), and reporting of recent adverse life events (OR = 2.3; 95% CI 1.9-2.8). CONCLUSION: This study has shown that chronic syndromes that are frequently unexplained co-occur in the general population and share common associated factors. Primary care practitioners need to be aware of these characteristics so that management is appropriate at the outset.  (+info)

Diferential diagnosis in atypical facial pain: a clinical study. (7/24)

OBJECTIVE: To evaluate a sample of patients with atypical facial pain (AFP) in comparison to patients with symptomatic facial pain (SFP). METHOD: 41 patients with previous diagnostic of AFP were submitted to a standardized evaluation protocol, by a multidisciplinary pain team. RESULTS: 21 (51.2%) were considered AFP and 20 (48.8%) (SFP) received the following diagnosis: 8 (40.0%) had temporomandibular disorders (TMD); 3 (15.0%) had TMD associated to systemic disease (fibromyalgia, systemic erythematosus lupus); 4 (20.0%) had neuropathy after ear, nose and throat (ENT) surgery for petroclival tumor; 2 (10.0%) had Wallenberg syndrome; 1 (5.0%) had intracranial tumor; 1 (5.0%) had oral cancer (epidermoid carcinoma), and 1 (5.0%) had burning mouth syndrome (BMS) associated to fibromyalgia. Spontaneous descriptors of pain were not different between AFP and SFP groups (p=0.82). Allodynia was frequent in SFP (p=0.05) and emotion was the triggering factor most prevalent in AFP (p=0.06). AFP patients had more traumatic events previously to pain (p=0.001). CONCLUSION: AFP patients had more: a) traumatic events previously to pain onset, and b) emotions as a triggering factor for pain. These data support the need of trained health professionals in multidisciplinary groups for the accurate diagnosis and treatment of these patients.  (+info)

A pilot study investigating the effects of fast left prefrontal rTMS on chronic neuropathic pain. (8/24)

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Facial neuralgia is a general term that refers to painful conditions affecting the facial nerves. It is often used to describe two specific disorders: trigeminal neuralgia and glossopharyngeal neuralgia.

1. Trigeminal neuralgia (TN), also known as tic douloureux, is a chronic pain condition that affects the trigeminal nerve, one of the major nerves of the face. The trigeminal nerve is responsible for sensations in the face and motor functions such as biting and chewing. Trigeminal neuralgia causes intense, stabbing, electric shock-like pain in the areas of the face where the branches of the nerve are distributed: the lower jaw, upper jaw, and cheek. The pain usually affects one side of the face, is triggered by light touch or other stimuli, and can last from a few seconds to several minutes.

2. Glossopharyngeal neuralgia (GPN) is a similar but less common condition that involves the glossopharyngeal nerve, which is responsible for sensations in the throat, tongue, and ear on one side of the face. GPN causes sharp, stabbing pain in these areas, often triggered by swallowing, talking, or coughing.

Both trigeminal neuralgia and glossopharyngeal neuralgia can be debilitating and significantly impact a person's quality of life. The exact cause of these conditions is not always clear, but they are often associated with nerve compression by blood vessels or tumors, age-related changes in the nerves and blood vessels, multiple sclerosis, or other underlying medical conditions. Treatment options may include medications to manage pain, surgical procedures to decompress the affected nerves, or, in some cases, radiofrequency ablation or gamma knife radiosurgery to destroy a portion of the nerve and reduce pain signals.

Trigeminal neuralgia is a chronic pain condition that affects the trigeminal nerve, which is one of the largest nerves in the head. It carries sensations from the face to the brain.

Medically, trigeminal neuralgia is defined as a neuropathic disorder characterized by episodes of intense, stabbing, electric shock-like pain in the areas of the face supplied by the trigeminal nerve (the ophthalmic, maxillary, and mandibular divisions). The pain can be triggered by simple activities such as talking, eating, brushing teeth, or even touching the face lightly.

The condition is more common in women over 50, but it can occur at any age and in either gender. While the exact cause of trigeminal neuralgia is not always known, it can sometimes be related to pressure on the trigeminal nerve from a nearby blood vessel or other causes such as multiple sclerosis. Treatment typically involves medications, surgery, or a combination of both.

Postherpetic neuralgia (PHN) is a type of neuralgia, which is defined as pain in the distribution of a nerve or nerves. Specifically, PHN is a neuropathic pain condition that develops after an individual has had herpes zoster, also known as shingles. Shingles is caused by the reactivation of the varicella-zoster virus, which lies dormant in the nervous system following chickenpox infection.

PHN is characterized by persistent burning pain, often accompanied by sensory abnormalities such as numbness, tingling, or itching, in the area of the body where shingles occurred. The pain can be severe and debilitating, significantly impacting a person's quality of life. PHN primarily affects older adults and individuals with weakened immune systems.

The exact cause of PHN is not fully understood, but it is believed to result from damage to the affected nerves and their surrounding tissues during the shingles infection. This damage can lead to altered nerve function and increased sensitivity to stimuli, resulting in chronic pain. Treatment for PHN typically involves a combination of medications, such as antidepressants, anticonvulsants, or opioids, as well as topical treatments, physical therapy, and lifestyle modifications to help manage the pain and improve quality of life.

Neuralgia is a type of pain that occurs along the pathway of a nerve, often caused by damage or irritation to the nerve. It is typically described as a sharp, stabbing, burning, or electric-shock like pain that can be severe and debilitating. Neuralgia can affect any nerve in the body, but it most commonly occurs in the facial area (trigeminal neuralgia) or in the nerves related to the spine (postherpetic neuralgia). The pain associated with neuralgia can be intermittent or constant and may be worsened by certain triggers such as touch, temperature changes, or movement. Treatment for neuralgia typically involves medications to manage pain, as well as other therapies such as nerve blocks, surgery, or lifestyle modifications.

Facial paralysis is a loss of facial movement due to damage or dysfunction of the facial nerve (cranial nerve VII). This nerve controls the muscles involved in facial expressions, such as smiling, frowning, and closing the eyes. Damage to one side of the facial nerve can cause weakness or paralysis on that side of the face.

Facial paralysis can result from various conditions, including:

1. Bell's palsy - an idiopathic (unknown cause) inflammation of the facial nerve
2. Trauma - skull fractures, facial injuries, or surgical trauma to the facial nerve
3. Infections - Lyme disease, herpes zoster (shingles), HIV/AIDS, or bacterial infections like meningitis
4. Tumors - benign or malignant growths that compress or invade the facial nerve
5. Stroke - damage to the brainstem where the facial nerve originates
6. Congenital conditions - some people are born with facial paralysis due to genetic factors or birth trauma

Symptoms of facial paralysis may include:

* Inability to move one or more parts of the face, such as the eyebrows, eyelids, mouth, or cheeks
* Drooping of the affected side of the face
* Difficulty closing the eye on the affected side
* Changes in saliva and tear production
* Altered sense of taste
* Pain around the ear or jaw
* Speech difficulties due to weakened facial muscles

Treatment for facial paralysis depends on the underlying cause. In some cases, such as Bell's palsy, spontaneous recovery may occur within a few weeks to months. However, physical therapy, medications, and surgical interventions might be necessary in other situations to improve function and minimize complications.

The facial nerve, also known as the seventh cranial nerve (CN VII), is a mixed nerve that carries both sensory and motor fibers. Its functions include controlling the muscles involved in facial expressions, taste sensation from the anterior two-thirds of the tongue, and secretomotor function to the lacrimal and salivary glands.

The facial nerve originates from the brainstem and exits the skull through the internal acoustic meatus. It then passes through the facial canal in the temporal bone before branching out to innervate various structures of the face. The main branches of the facial nerve include:

1. Temporal branch: Innervates the frontalis, corrugator supercilii, and orbicularis oculi muscles responsible for eyebrow movements and eyelid closure.
2. Zygomatic branch: Supplies the muscles that elevate the upper lip and wrinkle the nose.
3. Buccal branch: Innervates the muscles of the cheek and lips, allowing for facial expressions such as smiling and puckering.
4. Mandibular branch: Controls the muscles responsible for lower lip movement and depressing the angle of the mouth.
5. Cervical branch: Innervates the platysma muscle in the neck, which helps to depress the lower jaw and wrinkle the skin of the neck.

Damage to the facial nerve can result in various symptoms, such as facial weakness or paralysis, loss of taste sensation, and dry eyes or mouth due to impaired secretion.

The trigeminal nerve, also known as the fifth cranial nerve or CNV, is a paired nerve that carries both sensory and motor information. It has three major branches: ophthalmic (V1), maxillary (V2), and mandibular (V3). The ophthalmic branch provides sensation to the forehead, eyes, and upper portion of the nose; the maxillary branch supplies sensation to the lower eyelid, cheek, nasal cavity, and upper lip; and the mandibular branch is responsible for sensation in the lower lip, chin, and parts of the oral cavity, as well as motor function to the muscles involved in chewing. The trigeminal nerve plays a crucial role in sensations of touch, pain, temperature, and pressure in the face and mouth, and it also contributes to biting, chewing, and swallowing functions.

The glossopharyngeal nerve, also known as the ninth cranial nerve (CN IX), is primarily responsible for providing motor innervation to the stylopharyngeus muscle and sensory innervation to parts of the pharynx, middle ear, and posterior tongue. It also plays a role in the reflexive control of heart rate via the baroreceptors located in the carotid sinus.

Glossopharyngeal nerve diseases refer to conditions that affect the function of this nerve, leading to various symptoms. These diseases can be classified into two main categories: peripheral and central. Peripheral disorders are caused by damage or injury to the nerve itself, while central disorders result from problems in the brainstem where the glossopharyngeal nerve originates.

Some examples of glossopharyngeal nerve diseases include:

1. Glossopharyngeal neuralgia: A rare condition characterized by severe, stabbing pain in the throat, ear, or tongue, often triggered by swallowing or talking. This disorder may be caused by compression of the nerve by blood vessels or other structures.

2. Infections: Bacterial and viral infections can cause inflammation and damage to the glossopharyngeal nerve, leading to dysfunction. Examples include Lyme disease, herpes zoster (shingles), and meningitis.

3. Tumors: Benign or malignant growths in the head and neck region can compress and injure the glossopharyngeal nerve, resulting in symptoms related to its dysfunction.

4. Trauma: Direct trauma to the neck or skull base can damage the glossopharyngeal nerve, causing various deficits depending on the severity of the injury.

5. Neurological disorders: Conditions such as multiple sclerosis and stroke can affect the central connections of the glossopharyngeal nerve in the brainstem, leading to dysfunction.

6. Genetic conditions: Rare genetic disorders like Moersch-Woltman syndrome (also known as stiff person syndrome) can involve the glossopharyngeal nerve and cause symptoms related to its dysfunction.

Symptoms of glossopharyngeal nerve dysfunction may include difficulty swallowing, hoarseness, loss of taste on the back of the tongue, decreased sensation in the throat or ear, and pain in the neck, throat, or ear. Treatment for these conditions depends on the underlying cause and may involve medications, surgery, or other interventions to address the specific problem.

Pudendal Neuralgia is a chronic pain condition characterized by the irritation or damage to the pudendal nerve, which supplies sensation and innervation to the perineum, genital region, and lower rectum. The symptoms often include burning pain, numbness, tingling, or shooting pain in these areas, which can be worsened by sitting or certain movements. It is important to note that Pudendal Neuralgia is not the same as Pudendal Nerve Entrapment (PNE), although PNE can lead to Pudendal Neuralgia. The diagnosis of this condition typically involves a thorough physical examination, medical history, and sometimes specialized tests like nerve blocks or electromyography (EMG) studies.

A facial expression is a result of the contraction or relaxation of muscles in the face that change the physical appearance of an individual's face to convey various emotions, intentions, or physical sensations. Facial expressions can be voluntary or involuntary and are a form of non-verbal communication that plays a crucial role in social interaction and conveying a person's state of mind.

The seven basic facial expressions of emotion, as proposed by Paul Ekman, include happiness, sadness, fear, disgust, surprise, anger, and contempt. These facial expressions are universally recognized across cultures and can be detected through the interpretation of specific muscle movements in the face, known as action units, which are measured and analyzed in fields such as psychology, neurology, and computer vision.

Nerve compression syndromes refer to a group of conditions characterized by the pressure or irritation of a peripheral nerve, causing various symptoms such as pain, numbness, tingling, and weakness in the affected area. This compression can occur due to several reasons, including injury, repetitive motion, bone spurs, tumors, or swelling. Common examples of nerve compression syndromes include carpal tunnel syndrome, cubital tunnel syndrome, radial nerve compression, and ulnar nerve entrapment at the wrist or elbow. Treatment options may include physical therapy, splinting, medications, injections, or surgery, depending on the severity and underlying cause of the condition.

Facial muscles, also known as facial nerves or cranial nerve VII, are a group of muscles responsible for various expressions and movements of the face. These muscles include:

1. Orbicularis oculi: muscle that closes the eyelid and raises the upper eyelid
2. Corrugator supercilii: muscle that pulls the eyebrows down and inward, forming wrinkles on the forehead
3. Frontalis: muscle that raises the eyebrows and forms horizontal wrinkles on the forehead
4. Procerus: muscle that pulls the medial ends of the eyebrows downward, forming vertical wrinkles between the eyebrows
5. Nasalis: muscle that compresses or dilates the nostrils
6. Depressor septi: muscle that pulls down the tip of the nose
7. Levator labii superioris alaeque nasi: muscle that raises the upper lip and flares the nostrils
8. Levator labii superioris: muscle that raises the upper lip
9. Zygomaticus major: muscle that raises the corner of the mouth, producing a smile
10. Zygomaticus minor: muscle that raises the nasolabial fold and corner of the mouth
11. Risorius: muscle that pulls the angle of the mouth laterally, producing a smile
12. Depressor anguli oris: muscle that pulls down the angle of the mouth
13. Mentalis: muscle that raises the lower lip and forms wrinkles on the chin
14. Buccinator: muscle that retracts the cheek and helps with chewing
15. Platysma: muscle that depresses the corner of the mouth and wrinkles the skin of the neck.

These muscles are innervated by the facial nerve, which arises from the brainstem and exits the skull through the stylomastoid foramen. Damage to the facial nerve can result in facial paralysis or weakness on one or both sides of the face.

Rhizotomy is a surgical procedure where the root(s) of a nerve are cut. It is often used to treat chronic pain, spasticity, or other neurological symptoms that have not responded to other treatments. In some cases, only a portion of the nerve root may be severed (selective rhizotomy), while in others the entire root may be cut (root transaction). The specific nerves targeted during a rhizotomy depend on the individual patient's condition and symptoms.

This procedure is typically performed by a neurosurgeon, and it can be done through an open surgical approach or using minimally invasive techniques such as endoscopic or percutaneous approaches. After the surgery, patients may require physical therapy to help regain strength and mobility in the affected area. Potential risks of rhizotomy include numbness, weakness, and loss of reflexes in the areas served by the severed nerves.

The Herpes Zoster vaccine, also known as the shingles vaccine, is a preventive measure against the reactivation of the varicella-zoster virus (VZV) in individuals who have previously had chickenpox. The vaccine contains a live but weakened form of VZV that boosts the immune system's ability to recognize and fight off the virus, thereby reducing the risk of developing shingles and its complications. It is typically administered as a single dose for people aged 50 and older, or as a two-dose series for those aged 19 and older who have weakened immune systems.

Facial pain is a condition characterized by discomfort or pain felt in any part of the face. It can result from various causes, including nerve damage or irritation, injuries, infections, dental problems, migraines, or sinus congestion. The pain can range from mild to severe and may be sharp, dull, constant, or intermittent. In some cases, facial pain can also be associated with other symptoms such as headaches, redness, swelling, or changes in sensation. Accurate diagnosis and treatment of the underlying cause are essential for effective management of facial pain.

Microvascular decompression surgery (MVD) is a surgical procedure used to alleviate the symptoms of certain neurological conditions, such as trigeminal neuralgia and hemifacial spasm. The primary goal of MVD is to relieve pressure on the affected cranial nerve by placing a small pad or sponge between the nerve and the blood vessel that is causing compression. This procedure is typically performed under a microscope, hence the term "microvascular."

During the surgery, the neurosurgeon makes an incision behind the ear and creates a small opening in the skull (a craniotomy) to access the brain. The surgeon then identifies the affected nerve and the blood vessel that is compressing it. Using specialized instruments under the microscope, the surgeon carefully separates the blood vessel from the nerve and places a tiny pad or sponge between them to prevent further compression.

The benefits of MVD include its high success rate in relieving symptoms, minimal impact on surrounding brain tissue, and lower risk of complications compared to other surgical options for treating these conditions. However, as with any surgery, there are potential risks and complications associated with MVD, including infection, bleeding, cerebrospinal fluid leakage, facial numbness, hearing loss, balance problems, and very rarely, stroke or death.

It is essential to consult a qualified neurosurgeon for a thorough evaluation and discussion of the risks and benefits of microvascular decompression surgery before making a treatment decision.

Facial nerve diseases refer to a group of medical conditions that affect the function of the facial nerve, also known as the seventh cranial nerve. This nerve is responsible for controlling the muscles of facial expression, and it also carries sensory information from the taste buds in the front two-thirds of the tongue, and regulates saliva flow and tear production.

Facial nerve diseases can cause a variety of symptoms, depending on the specific location and extent of the nerve damage. Common symptoms include:

* Facial weakness or paralysis on one or both sides of the face
* Drooping of the eyelid and corner of the mouth
* Difficulty closing the eye or keeping it closed
* Changes in taste sensation or dryness of the mouth and eyes
* Abnormal sensitivity to sound (hyperacusis)
* Twitching or spasms of the facial muscles

Facial nerve diseases can be caused by a variety of factors, including:

* Infections such as Bell's palsy, Ramsay Hunt syndrome, and Lyme disease
* Trauma or injury to the face or skull
* Tumors that compress or invade the facial nerve
* Neurological conditions such as multiple sclerosis or Guillain-Barre syndrome
* Genetic disorders such as Moebius syndrome or hemifacial microsomia

Treatment for facial nerve diseases depends on the underlying cause and severity of the symptoms. In some cases, medication, physical therapy, or surgery may be necessary to restore function and relieve symptoms.

The facial bones, also known as the facial skeleton, are a series of bones that make up the framework of the face. They include:

1. Frontal bone: This bone forms the forehead and the upper part of the eye sockets.
2. Nasal bones: These two thin bones form the bridge of the nose.
3. Maxilla bones: These are the largest bones in the facial skeleton, forming the upper jaw, the bottom of the eye sockets, and the sides of the nose. They also contain the upper teeth.
4. Zygomatic bones (cheekbones): These bones form the cheekbones and the outer part of the eye sockets.
5. Palatine bones: These bones form the back part of the roof of the mouth, the side walls of the nasal cavity, and contribute to the formation of the eye socket.
6. Inferior nasal conchae: These are thin, curved bones that form the lateral walls of the nasal cavity and help to filter and humidify air as it passes through the nose.
7. Lacrimal bones: These are the smallest bones in the skull, located at the inner corner of the eye socket, and help to form the tear duct.
8. Mandible (lower jaw): This is the only bone in the facial skeleton that can move. It holds the lower teeth and forms the chin.

These bones work together to protect vital structures such as the eyes, brain, and nasal passages, while also providing attachment points for muscles that control chewing, expression, and other facial movements.

The cerebellopontine angle (CPA) is a narrow space located at the junction of the brainstem and the cerebellum, where the pons and cerebellum meet. This region is filled with several important nerves, blood vessels, and membranous coverings called meninges. The CPA is a common site for various neurological disorders because it contains critical structures such as:

1. Cerebellum: A part of the brain responsible for coordinating muscle movements, maintaining balance, and fine-tuning motor skills.
2. Pons: A portion of the brainstem that plays a role in several vital functions, including facial movements, taste sensation, sleep regulation, and respiration.
3. Cranial nerves: The CPA is home to the following cranial nerves:
* Vestibulocochlear nerve (CN VIII): This nerve has two components - cochlear and vestibular. The cochlear part is responsible for hearing, while the vestibular part contributes to balance and eye movement.
* Facial nerve (CN VII): This nerve controls facial expressions, taste sensation in the anterior two-thirds of the tongue, salivary gland function, and lacrimation (tear production).
4. Blood vessels: The CPA contains critical blood vessels like the anterior inferior cerebellar artery (AICA), which supplies blood to various parts of the brainstem, cerebellum, and cranial nerves.
5. Meninges: These are protective membranes surrounding the brain and spinal cord. In the CPA, the meninges include the dura mater, arachnoid mater, and pia mater.

Disorders that can affect the structures in the cerebellopontine angle include acoustic neuromas (vestibular schwannomas), meningiomas, epidermoids, and arteriovenous malformations. These conditions may cause symptoms such as hearing loss, tinnitus (ringing in the ears), vertigo (dizziness), facial weakness or numbness, difficulty swallowing, and imbalance.

Facial injuries refer to any damage or trauma caused to the face, which may include the bones of the skull that form the face, teeth, salivary glands, muscles, nerves, and skin. Facial injuries can range from minor cuts and bruises to severe fractures and disfigurement. They can be caused by a variety of factors such as accidents, falls, sports-related injuries, physical assaults, or animal attacks.

Facial injuries can affect one or more areas of the face, including the forehead, eyes, nose, cheeks, ears, mouth, and jaw. Common types of facial injuries include lacerations (cuts), contusions (bruises), abrasions (scrapes), fractures (broken bones), and burns.

Facial injuries can have significant psychological and emotional impacts on individuals, in addition to physical effects. Treatment for facial injuries may involve simple first aid, suturing of wounds, splinting or wiring of broken bones, reconstructive surgery, or other medical interventions. It is essential to seek prompt medical attention for any facial injury to ensure proper healing and minimize the risk of complications.

In medical terms, the face refers to the front part of the head that is distinguished by the presence of the eyes, nose, and mouth. It includes the bones of the skull (frontal bone, maxilla, zygoma, nasal bones, lacrimal bones, palatine bones, inferior nasal conchae, and mandible), muscles, nerves, blood vessels, skin, and other soft tissues. The face plays a crucial role in various functions such as breathing, eating, drinking, speaking, seeing, smelling, and expressing emotions. It also serves as an important identifier for individuals, allowing them to be recognized by others.

"Facial Neuralgia Resources". Trigeminal Neuralgia Resources / Facial Neuralgia Resources. Archived from the original on 8 July ... "trigeminal neuralgia, type 2" or trigeminal neuralgia with concomitant pain), based on a recent classification of facial pain. ... Trigeminal neuralgia (TN or TGN), also called Fothergill disease, tic douloureux, or trifacial neuralgia is a long-term pain ... Trigeminal neuralgia at Curlie Trigeminal Neuralgia at NHS Choices (Articles with short description, Short description is ...
"Facial Neuralgia Resources". Trigeminal Neuralgia Resources / Facial Neuralgia Resources. Archived from the original on 8 July ... A patient resource group for trigeminal neuralgia (which has been described as the most painful condition in existence) have ...
"Neuralgia-inducing cavitational osteonecrosis (NICO). Osteomyelitis in 224 jawbone samples from patients with facial neuralgia ... Neuralgia-inducing cavitational osteonecrosis (NICO) is a diagnosis whereby a putative jawbone cavitation causes chronic facial ... Bouquot JE, Christian J (April 1995). "Long-term effects of jawbone curettage on the pain of facial neuralgia". J. Oral ... Roberts AM, Person P (October 1979). "Etiology and treatment of idiopathic trigeminal and atypical facial neuralgias". Oral ...
Trigeminal neuralgia is another example of a cause of facial pain. Neuralgia refers to pain in the distribution of a nerve (or ... "atypical facial neuralgia" as synonyms of AFP. The ICHD-2 lists diagnostic criteria for "persistent idiopathic facial pain" ( ... Cranial neuralgias and central causes of facial pain; Introduction". IHS. Retrieved 7 May 2013. "Trigeminal Neuralgia and ... Atypical facial pain (AFP) is a type of chronic facial pain which does not fulfill any other diagnosis. There is no consensus ...
The first practitioner to see facial neuralgia patients is often a dentist who may lack deep training in facial neurology. Thus ... TN "Trigeminal Neuralgia Description / Definition", [US] Facial Pain Association, "TN (Trigeminal Neuralgia) Description / ... Atypical trigeminal neuralgia (ATN), or type 2 trigeminal neuralgia, is a form of trigeminal neuralgia, a disorder of the fifth ... The long-time first drug of choice for facial neuralgia has been carbamazepine, an anti-seizure agent. Due to the significant ...
"Adjuvant topical therapy with a cannabinoid receptor agonist in facial postherpetic neuralgia". Journal der Deutschen ... Calabrò RS, Gervasi G, Marino S, Mondo PN, Bramanti P (May 2010). "Misdiagnosed chronic pelvic pain: pudendal neuralgia ... or post-herpetic neuralgia (12 men, 7 women)-whose symptoms spanned eight pain categories ("burning", "osteoarticular", " ...
When severe facial pain is purported to be caused by osteonecrosis, the term NICO, for neuralgia-inducing cavitational ... Bouquot JE; Christian J (1995). "Long-term effects of jawbone curettage on the pain of facial neuralgia". J. Oral Maxillofac. ... Neville BW; Damn D; Allen C; Bouquot JE (1995). "Facial pain and neuromuscular diseases.". Oral and maxillofacial pathology. pp ... Adams WR; Spolnik KJ; Bouquot JE (1999). "Maxillofacial osteonecrosis in a patient with multiple "idiopathic" facial pains". ...
Pancoast was also among the first to section facial nerve to relieve neuralgia. On the eye, Pancoast operated upon cataracts. ...
When he reaches the doctor's house he pretends to be suffering from facial neuralgia. The doctor is suspicious but gives Danny ...
Do TM, Unis GD, Kattar N, Ananth A, McCoul ED (October 2020). "Neuromodulators for Atypical Facial Pain and Neuralgias: A ... Moderate evidence exists in favor of amitriptyline use for atypical facial pain. Amitriptyline is ineffective for HIV- ...
In March 2011, she experienced an episode of viral trigeminal neuralgia, which resulted in temporary facial paralysis. She ...
He was often short of money and in 1925, he suffered a facial neuralgia with severe pain. From 1932, Liljefors lived at ...
Neuralgia: she suffered from facial pain which was treated with morphine and chloral and led to addiction. The pain was so ...
His clinical interests include Stereotactic and functional neurosurgery, facial pain, trigeminal neuralgia, and gamma knife ... Facial Nerve Pain, Facial Pain, Glioblastoma, Headache disorders, Laminectomy, Lower back pain, Movement Disorders, Multiple ... He is now an Advisory board member of Facial Pain Association Vycor Medical, Inc and Stimwave, Inc. and an editorial board ... "Peripheral Neurostimulation for Treatment of Intractable Occipital Neuralgia". Journal of Neurosurgery. Konstantin V. Slavin, M ...
He found Shackleton complaining of back pains and severe facial neuralgia, and asking for a painkilling drug. In a brief ...
Cacciopoli allegedly received his moniker "Tommy Twitch" because he suffered from facial neuralgia, an uncontrollable muscle ...
Headache or facial pain, as in the descriptive former term "neuralgia-inducing" (cavitational osteonecrosis). Fibromyalgia. ... Former and colloquial names include Osteonecrosis of the jaws (ONJ), cavitations, dry or wet socket, and NICO (Neuralgia- ...
Suffering from facial Neuralgia, he surrendered with the remnants of Lee's army at Appomattox Court House in April 1865. In ...
In August 2011, Khan admitted that he suffers from trigeminal neuralgia, a facial nerve disorder commonly known as the "suicide ... "Salman Khan on his facial nerve disorder: Fans do not care about that". Hindustan Times. 19 May 2017. Retrieved 29 March 2022 ...
... that this abnormal impingement of the nerve might be the cause of the painful facial condition known as trigeminal neuralgia. ... Peter J. Jannetta, Pioneering Neurosurgeon on Facial Pain, Dies at 84". The New York Times. Shelton, Mark L. (1989). Working in ... Peter J. Jannetta, pioneering neurosurgeon on facial pain, dies at 84". The New York Times. Retrieved April 16, 2016. Raap, ... In addition to helping trigeminal neuralgia patients, the procedure became a treatment option for several related conditions. ...
... "facial neuralgia", which would today be diagnosed as trigeminal neuralgia. His biographer, Joseph Brent, says that when in the ...
De Quincey suffered neuralgic facial pain, "trigeminal neuralgia" - "attacks of piercing pain in the face, of such severity ... By his own testimony, de Quincey first used opium in 1804 to relieve his neuralgia; he used it for pleasure, but no more than ... that they sometimes drive the victim to suicide." He reports using opium first in 1804 to relieve his neuralgia. Thus, as with ...
... neurosurgeons and who developed a new brain procedure to eliminate facial spasms and facial pain caused by trigeminal neuralgia ...
It has other "off-label" medical purposes, such as treating severe facial pain, such as that caused by trigeminal neuralgia.[ ... Paralysis of the facial muscles Failure of the cervix Blinking frequently Anti-cancer drug delivery A very rare form of ... Difficulty swallowing or speaking Facial weakness on both sides of the face Blurred or double vision Dropping eyelids Trouble ...
... was used in the treatment of facial and other neuralgias. It also proved valuable in some cases of malarial fever, and was ...
... facial neuralgia, insomnia, and to combat laryngitis and all the ailments of the respiratory airways. Stocks in Milan, A. ... facial hyperkinesia, as well as other neuroses originating from both the central and peripheral nervous systems, neuralgia of ...
... a plant containing an oil once used for toothaches and facial neuralgia Achillea ptarmica or European pellitory, bastard ...
... minimally invasive endoscopy-assisted microvascular decompression in trigeminal neuralgia (facial pain) and facial hemispasm ( ... involuntary facial twitching) as well as the surgery of acoustic neuromas (tumors of the vestibular nerves), tumors of the ...
On the advice of his doctor Samuel Habershon in the aftermath of an attack of facial neuralgia, Gladstone stayed at Cannes from ...
This was quite an important discovery because surgeons would often cut this nerve as an attempted cure for facial neuralgia, ... Bell's palsy: a unilateral idiopathic paralysis of facial muscles due to a lesion of the facial nerve. Bell's phenomenon: A ... In 1821, he described in the trajectory of the facial nerve and a disease, Bell's Palsy which led to the unilateral paralysis ... This paper held Bell's most famous discovery, that the facial nerve or seventh cranial nerve is a nerve of muscular action. ...

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