'Gingival neoplasms' are abnormal, uncontrolled growths of tissue originating from the gingiva, which can be benign (non-cancerous) or malignant (cancerous), often manifesting as swellings, ulcerations, or masses within the oral cavity.
Oral tissue surrounding and attached to TEETH.
Excessive growth of the gingiva either by an increase in the size of the constituent cells (GINGIVAL HYPERTROPHY) or by an increase in their number (GINGIVAL HYPERPLASIA). (From Jablonski's Dictionary of Dentistry, 1992, p574)
'Gingival diseases' is a general term for conditions affecting the soft tissues surrounding and supporting the teeth, primarily characterized by inflammation, bleeding, redness, or swelling, which can progress to periodontal disease if left untreated.
Non-inflammatory enlargement of the gingivae produced by factors other than local irritation. It is characteristically due to an increase in the number of cells. (From Jablonski's Dictionary of Dentistry, 1992, p400)
Generalized or localized diffuse fibrous overgrowth of the gingival tissue, usually transmitted as an autosomal dominant trait, but some cases are idiopathic and others produced by drugs. The enlarged gingiva is pink, firm, and has a leather-like consistency with a minutely pebbled surface and in severe cases the teeth are almost completely covered and the enlargement projects into the oral vestibule. (Dorland, 28th ed)
Inflammation of gum tissue (GINGIVA) without loss of connective tissue.
An abnormal extension of a gingival sulcus not accompanied by the apical migration of the epithelial attachment.
Abnormal enlargement or overgrowth of the gingivae brought about by enlargement of existing cells.
A numerical rating scale for classifying the periodontal status of a person or population with a single figure which takes into consideration prevalence as well as severity of the condition. It is based upon probe measurement of periodontal pockets and on gingival tissue status.
An index which scores the degree of dental plaque accumulation.
Inflammation and loss of connective tissues supporting or surrounding the teeth. This may involve any part of the PERIODONTIUM. Periodontitis is currently classified by disease progression (CHRONIC PERIODONTITIS; AGGRESSIVE PERIODONTITIS) instead of age of onset. (From 1999 International Workshop for a Classification of Periodontal Diseases and Conditions, American Academy of Periodontology)
Surgical excision of the gingiva at the level of its attachment, thus creating new marginal gingiva. This procedure is used to eliminate gingival or periodontal pockets or to provide an approach for extensive surgical interventions, and to gain access necessary to remove calculus within the pocket. (Dorland, 28th ed)
Pathological processes involving the PERIODONTIUM including the gum (GINGIVA), the alveolar bone (ALVEOLAR PROCESS), the DENTAL CEMENTUM, and the PERIODONTAL LIGAMENT.
Tumors or cancer of the PANCREAS. Depending on the types of ISLET CELLS present in the tumors, various hormones can be secreted: GLUCAGON from PANCREATIC ALPHA CELLS; INSULIN from PANCREATIC BETA CELLS; and SOMATOSTATIN from the SOMATOSTATIN-SECRETING CELLS. Most are malignant except the insulin-producing tumors (INSULINOMA).
A species of gram-negative, anaerobic, rod-shaped bacteria originally classified within the BACTEROIDES genus. This bacterium produces a cell-bound, oxygen-sensitive collagenase and is isolated from the human mouth.
An abnormal extension of a gingival sulcus accompanied by the apical migration of the epithelial attachment and bone resorption.
New abnormal growth of tissue. Malignant neoplasms show a greater degree of anaplasia and have the properties of invasion and metastasis, compared to benign neoplasms.
A wedge-shaped collar of epithelial cells which form the attachment of the gingiva to the tooth surface at the base of the gingival crevice.
Chronic inflammation and loss of PERIODONTIUM that is associated with the amount of DENTAL PLAQUE or DENTAL CALCULUS present. Chronic periodontitis occurs mostly in adults and was called adult periodontitis, but this disease can appear in young people.
The structures surrounding and supporting the tooth. Periodontium includes the gum (GINGIVA), the alveolar bone (ALVEOLAR PROCESS), the DENTAL CEMENTUM, and the PERIODONTAL LIGAMENT.
Neoplasms containing cyst-like formations or producing mucin or serum.
Surgical reshaping of the gingivae and papillae for correction of deformities (particularly enlargements) and to provide the gingivae with a normal and functional form, the incision creating an external bevel. (Dorland, 28th ed)
Loss or destruction of periodontal tissue caused by periodontitis or other destructive periodontal diseases or by injury during instrumentation. Attachment refers to the periodontal ligament which attaches to the alveolar bone. It has been hypothesized that treatment of the underlying periodontal disease and the seeding of periodontal ligament cells enable the creating of new attachment.
A film that attaches to teeth, often causing DENTAL CARIES and GINGIVITIS. It is composed of MUCINS, secreted from salivary glands, and microorganisms.
Tumors or cancer of the SKIN.
Techniques used to expose dental surface below the gingival margin in order to obtain better dental impression during periodental and peri-implant applications. The retraction of the gingival tissue can be achieved surgically (e.g., laser gingivectomy and rotary curettage) or chemically with a retraction cord.
Two or more abnormal growths of tissue occurring simultaneously and presumed to be of separate origin. The neoplasms may be histologically the same or different, and may be found in the same or different sites.
An acute or chronic GINGIVITIS characterized by redness and swelling, NECROSIS extending from the interdental papillae along the gingival margins, PAIN; HEMORRHAGE, necrotic odor, and often a pseudomembrane. The condition may extend to the ORAL MUCOSA; TONGUE; PALATE; or PHARYNX. The etiology is somewhat unclear, but may involve a complex of FUSOBACTERIUM NUCLEATUM along with spirochetes BORRELIA or TREPONEMA.
Pain in the adjacent areas of the teeth.
A condition sometimes occurring after tooth extraction, particularly after traumatic extraction, resulting in a dry appearance of the exposed bone in the socket, due to disintegration or loss of the blood clot. It is basically a focal osteomyelitis without suppuration and is accompanied by severe pain (alveolalgia) and foul odor. (Dorland, 28th ed)
Acute or chronic inflammation of tissues surrounding the apical portion of a tooth, associated with the collection of pus, resulting from infection following pulp infection through a carious lesion or as a result of an injury causing pulp necrosis. (Dorland, 27th ed)
Localized circumscribed purulent area of inflammation in the periodontal tissue. It is a derivative of marginal periodontitis and commonly associated with suprabony and infrabony pockets and interradicular involvements, in contrast to periapical abscess which is attributable to pulp necrosis.
Inflammation of the DENTAL PULP, usually due to bacterial infection in dental caries, tooth fracture, or other conditions causing exposure of the pulp to bacterial invasion. Chemical irritants, thermal factors, hyperemic changes, and other factors may also cause pulpitis.
Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)

Autocrine and paracrine motility factors and their involvement in invasiveness in a human oral carcinoma cell line. (1/136)

Invasive potentials of malignant cancer cells are regulated by cell motility factors. To examine the regulation of motility and invasiveness in oral squamous carcinoma, we investigated autocrine- and/or paracrine-acting cell motility factors, using a newly established human cell line (IF cells) from oral squamous cell carcinoma, which has highly invasive and metastatic characteristics. Conditioned medium derived from IF cells stimulated cell scattering and migration of GB-d1 gallbladder carcinoma cells, indicating that IF cells secreted cell motility factors. Using antibodies, IF-derived cell motility factors proved to be transforming growth factor (TGF)-alpha and TGF-beta1. Antibodies against TGF-alpha and TGF-beta1 inhibited autonomous migration of the IF cells. On the other hand, in vitro invasion of IF cells was strongly enhanced by hepatocyte growth factor (HGF) but only slightly by TGF-alpha and TGF-beta1. The conditioned medium from fibroblasts enhanced in vitro invasion of IF cells, an event abrogated by anti-HGF antibody, but not by antibodies against TGF-alpha and TGF-beta1. Importantly, IF cells secreted a factor inducing HGF production in fibroblasts and the factor was identified as interleukin-1, which means that a mutual interaction exists between tumour cells and fibroblasts, as mediated by the HGF/HGF-inducer loop. These results indicate that IF cells utilize TGF-alpha and TGF-beta1 as autocrine-acting motility factors and HGF as a paracrine-acting motility factor, and that invasiveness of IF cells is particularly stimulated by HGF derived from stromal fibroblasts. Utilization of multiple cell motility/invasion factors that act in distinct pathways may confer highly invasive and metastatic potentials in IF oral squamous carcinoma cells.  (+info)

Undefined complications of parathyroid adenoma, parathyroid hyperplasia (primary hyperparathyroidism), thyroid follicular adenoma, thyroid papillary carcinoma, temporal astrocytoma, cerebellar meningioma, and hemangioma of external auditory meatus and oral papilloma. (2/136)

A 59-year-old woman who had parathyroid adenoma, parathyroid hyperplasia, thyroid follicular adenoma, thyroid papillary carcinoma, astrocytoma of the right temporal lobe, cerebellar meningioma, capillary hemangioma of the left external auditory meatus and papilloma of the left upper gingiva is reported. Dynamic magnetic resonance imaging, computed tomography with contrast-enhancement and gastrofiberscopy revealed no remarkable findings in the pituitary, pancreas, adrenals, stomach or duodenum. Similar lesions were not found in any family members. Defect of the causative genes of multiple endocrine neoplasia types I and IIa, MENIN and RET was not detected. Further follow-up of this patient and family members is needed.  (+info)

Deep extension from carcinoma arising from the gingiva: CT and MR imaging features. (3/136)

BACKGROUND AND PURPOSE: CT and MR imaging are useful for evaluating the extension of carcinomas in the face and neck. We evaluated the involvement by carcinoma arising from the gingiva (ie, gingival cancer) by using CT and MR imaging. METHODS: We retrospectively examined 122 patients with squamous cell carcinoma (SCCA) in the lower (88 patients) and upper (34 patients) gingiva. Extension of SCCA into the spaces of the face and neck was evaluated with CT and MR imaging, and findings were surgically confirmed. RESULTS: Spread into the face and neck spaces occurred in 58% of patients. The buccal space was the most common site of spread, occurring in 42% of the lower and of 47% of the upper gingival cancers. Spread into the masticator space occurred from the lower gingival cancers in the molar region (20%) but not from the anterior region. Masticator space involvement from the upper gingiva was rare (4%). The retromolar triangle and buccal space immediately anterior to the ramus served as a corridor for cancer extension from the lower gingiva into the masticator space. The sublingual space (11%) was a less common site of spread from the lower gingiva. CONCLUSION: Gingival cancers spread into the masticator, buccal, and sublingual spaces depending on the primary sites in the oral cavity. An understanding of the face and neck-space anatomy is important in diagnosing cancer extension in the oral cavity gingiva and in treating patients with such disease.  (+info)

Gingival metastasis from gallbladder cancer. (4/136)

Gallbladder cancer is generally diagnosed at an advanced stage. The liver is the most commonly invaded organ by direct extension and/or metastasis, followed by regional lymph nodes. Oral soft tissue metastasis is extremely unusual. This report describes the case of a 62-year-old woman diagnosed with advanced metastatic gallbladder cancer, who initially presented with abdominal pain. Diagnosis of gallbladder cancer was made about 3 months after her symptoms developed, when a laparoscopic cholecystectomy was performed because of the suspicion of gallstones. Liver metastasis was also discovered during surgery. A postoperative investigation revealed additional lung and bone metastases. A visible left gingival tumor was found on physical examination and was confirmed as gallbladder cancer metastasis by compatible histopathology 1 month after surgery. The patient responded poorly to chemotherapy and unfortunately died 5 months after the diagnosis. The clinical presentation of gallbladder cancer was relatively typical, apart from the unusual gingival metastasis. The medical literature contains quite a few examples of metastatic lesions located strictly in the oral soft tissue, however no case of gallbladder cancer metastasizing to the oral soft tissue has been previously reported.  (+info)

Prenatal diagnosis of congenital epulis: a case report. (5/136)

Congenital epulis or congenital granular cell tumor, is a benign tumor that has rarely been diagnosed prenatally. We report a case of a large congenital epulis diagnosed at 26 weeks of gestation that increased in size during gestation. Color and power Doppler ultrasound examination showed marked blood flow in the tumor. The tumor could be resected completely following Cesarean section and histologically examined. We discuss the prenatal diagnosis and histogenesis of congenital epulis.  (+info)

Expression of E-cadherin, alpha-catenin, and beta-catenin in the process of lymph node metastasis in oral squamous cell carcinoma. (6/136)

Regional lymph node metastasis is a very important prognostic indicator. In the metastatic process, reduction in cell to cell adhesion including E-cadherin-catenin cell adhesion complex is an essential step. We investigated immunohistochemical expression of E-cadherin, alpha-catenin and beta-catenin in 159 tissue samples from patients with oral squamous cell carcinoma and examined the correlation between their expressions and the presence of regional lymph node metastasis. Significantly greater reduction in expression levels of E-cadherin, alpha-catenin and beta-catenin was found in the metastatic group (n=64) compared to the nonmetastatic group (n=95) (P=0.007, 0.001, 0.001, respectively). However, there was no significant correlation between their expressions and the features of the regional metastasis, the number of metastatic lymph nodes or the presence of extracapsular metastasis. These data suggest that evaluation of the immunohistochemical expression of E-cadherin, alpha-catenin and beta-catenin is extremely valuable for the diagnosis of metastatic occurrence.  (+info)

Cemento-ossifying fibroma of mandibular gingiva: single case report. (7/136)

We report a case of a woman presenting a giant cementoossifying fibroma depending of the mandibular gingivae. The evolution of the process was 20 years. Cemento-ossifying fibroma is a relatively rare tumour classified between fibroosseous lesions. This lesion appears within the bone although in some occasions it involves the gingivae soft tissues, as the case we present. It is a slow-growing and well-defined tumorous lesion, because of this, it is considered as a benign lesion. The histologic findings alone may be similar to other pathologies such as osteoblastoma, low-grade osteosarcoma and particularly to fibrous dysplasia. An accurate diagnosis requires careful clinical, radiological and histological correlation in order to make an optimal treatment and an excellent outcome.  (+info)

Malignant ameloblastic fibro-odontoma in a dog. (8/136)

An 11-year-old male Collie was presented with a swelling of the face caused by tumor masses arising from the gingiva. Postmortem examination revealed metastases to the lymph nodes, lung, liver, and orbital cavity. Histologically, the tumor represented a combination of fibrosarcomatous proliferation, pulpal mesenchyme, and undifferentiated odontogenic epithelium, with a follicular or plexiform growth pattern. In addition, the follicular areas of the tumor showed a biphasic character, and there were numerous apoptotic cells in plexiform areas. Furthermore, acidophilic material resembling dysplastic dentine or enamel matrix was observed in the metastatic lesion in the lung. Based on the histological characters, the present case was diagnosed as malignant ameloblastic fibro-odontoma. This study is the first known description of a possible malignant ameloblastic fibro-odontoma in a dog with metastasis to distant organs.  (+info)

Gingival neoplasms refer to abnormal growths or tumors that occur in the gingiva, which are the part of the gums that surround the teeth. These growths can be benign (non-cancerous) or malignant (cancerous). Benign neoplasms include conditions such as fibromas, papillomas, and hemangiomas, while malignant neoplasms are typically squamous cell carcinomas.

Gingival neoplasms can present with a variety of symptoms, including swelling, bleeding, pain, and loose teeth. They may also cause difficulty with chewing, speaking, or swallowing. The exact cause of these neoplasms is not always known, but risk factors include tobacco use, alcohol consumption, poor oral hygiene, and certain viral infections.

Diagnosis of gingival neoplasms typically involves a thorough clinical examination, including a dental exam and biopsy. Treatment options depend on the type and stage of the neoplasm, but may include surgery, radiation therapy, chemotherapy, or a combination of these approaches. Regular dental check-ups and good oral hygiene practices can help to detect gingival neoplasms at an early stage and improve treatment outcomes.

Gingiva is the medical term for the soft tissue that surrounds the teeth and forms the margin of the dental groove, also known as the gum. It extends from the mucogingival junction to the base of the cervical third of the tooth root. The gingiva plays a crucial role in protecting and supporting the teeth and maintaining oral health by providing a barrier against microbial invasion and mechanical injury.

Gingival overgrowth, also known as gingival hyperplasia or hypertrophy, refers to an abnormal enlargement or growth of the gum tissue (gingiva) surrounding the teeth. This condition can be caused by various factors, including poor oral hygiene, certain medications (such as phenytoin, cyclosporine, and calcium channel blockers), genetic predisposition, and systemic conditions like vitamin C deficiency or leukemia.

Gingival overgrowth can lead to several complications, such as difficulty in maintaining oral hygiene, which may result in periodontal disease, tooth decay, bad breath, and potential loss of teeth. In some cases, the enlarged gum tissue may also cause discomfort or pain during speaking, chewing, or brushing. Treatment for gingival overgrowth typically involves improving oral hygiene, adjusting medications if possible, and undergoing surgical procedures to remove the excess gum tissue. Regular dental check-ups and cleanings are essential in managing and preventing this condition.

Gingival diseases are infections or inflammations that affect the gingiva, which is the part of the gum around the base of the teeth. These diseases can be caused by bacteria found in dental plaque and can lead to symptoms such as redness, swelling, bleeding, and receding gums. If left untreated, gingival diseases can progress to periodontal disease, a more serious condition that can result in tooth loss. Common types of gingival diseases include gingivitis and periodontitis.

Gingival hyperplasia is a condition characterized by an abnormal growth or enlargement of the gingiva (gum tissue). This condition can be caused by various factors, including bacterial infection, certain medications (such as phenytoin, cyclosporine, and nifedipine), systemic diseases (such as leukemia, vitamin C deficiency, and Crohn's disease), and genetic disorders.

The enlarged gum tissue can be uncomfortable, irritated, and prone to bleeding, especially during brushing or flossing. It may also make it difficult to maintain good oral hygiene, which can increase the risk of dental caries and periodontal disease. Treatment for gingival hyperplasia typically involves improving oral hygiene, controlling any underlying causes, and in some cases, surgical removal of the excess tissue.

Gingival fibromatosis is a benign (non-cancerous) condition characterized by the excessive growth of gum (gingival) tissue. The overgrowth can affect one or both the maxilla (upper jaw) and mandible (lower jaw) and can lead to various dental and oral health issues, such as difficulty in chewing, speaking, and maintaining proper oral hygiene.

The etiology of gingival fibromatosis can be divided into two categories: hereditary and acquired. Hereditary gingival fibromatosis is often associated with genetic mutations, while acquired gingival fibromatosis can result from factors like chronic inflammation due to poor oral hygiene, certain medications (such as phenytoin, cyclosporine, or nifedipine), and systemic conditions (like leukemia).

The management of gingival fibromatosis typically involves surgical removal of the excess tissue. However, recurrence is common due to the condition's tendency for regrowth. Regular follow-ups with a dental professional are essential to monitor any potential regrowth and maintain good oral hygiene.

Gingivitis is a mild form of gum disease (periodontal disease) that causes irritation, redness, swelling and bleeding of the gingiva, or gums. It's important to note that it is reversible with good oral hygiene and professional dental treatment. If left untreated, however, gingivitis can progress to a more severe form of gum disease known as periodontitis, which can result in tissue damage and eventual tooth loss.

Gingivitis is most commonly caused by the buildup of plaque, a sticky film of bacteria that constantly forms on our teeth. When not removed regularly through brushing and flossing, this plaque can harden into tartar, which is more difficult to remove and contributes to gum inflammation. Other factors like hormonal changes, poor nutrition, certain medications, smoking or a weakened immune system may also increase the risk of developing gingivitis.

A gingival pocket, also known as a sulcus, is a small space or groove between the gum tissue (gingiva) and the tooth. It's a normal anatomical structure found in healthy teeth and gums, and it measures about 1-3 millimeters in depth. The purpose of the gingival pocket is to allow for the movement of the gum tissue during functions such as eating, speaking, and swallowing.

However, when the gums become inflamed due to bacterial buildup (plaque) or other factors, the pocket can deepen, leading to the formation of a pathological gingival pocket. Pathological pockets are typically deeper than 3 millimeters and may indicate the presence of periodontal disease. These pockets can harbor harmful bacteria that can cause further damage to the gum tissue and bone supporting the tooth, potentially leading to tooth loss if left untreated.

Gingival hypertrophy is a condition characterized by an abnormal enlargement or overgrowth of the gingiva (gum tissue). This can be caused due to various reasons such as inflammation from poor oral hygiene, certain medications like phenytoin and cyclosporine, or systemic conditions such as pregnancy, leukemia, and vitamin C deficiency.

The enlarged gums may appear swollen, red, and bleed easily. They can also cover the teeth, making cleaning difficult, which can further worsen the inflammation. Depending on the cause, treatment options may include improving oral hygiene, changing medications, or undergoing surgical procedures to remove the excess tissue.

The Periodontal Index (PI) is not a current or widely used medical/dental term. However, in the past, it was used to describe a method for assessing and measuring the severity of periodontal disease, also known as gum disease.

Developed by Henry H. Klein and colleagues in 1978, the Periodontal Index was a scoring system that evaluated four parameters: gingival inflammation, gingival bleeding, calculus (tartar) presence, and periodontal pocket depths. The scores for each parameter ranged from 0 to 3, with higher scores indicating worse periodontal health. The overall PI score was the sum of the individual parameter scores, ranging from 0 to 12.

However, due to its limited ability to predict future disease progression and the introduction of more comprehensive assessment methods like the Community Periodontal Index (CPI) and the Basic Periodontal Examination (BPE), the use of the Periodontal Index has become less common in dental practice and research.

The dental plaque index (DPI) is a clinical measurement used in dentistry to assess the amount of dental plaque accumulation on a person's teeth. It was first introduced by Silness and Löe in 1964 as a method to standardize the assessment of oral hygiene and the effectiveness of oral hygiene interventions.

The DPI is based on a visual examination of the amount of plaque present on four surfaces of the teeth, including the buccal (cheek-facing) and lingual (tongue-facing) surfaces of both upper and lower first molars and upper and lower incisors. The examiner assigns a score from 0 to 3 for each surface, with higher scores indicating greater plaque accumulation:

* Score 0: No plaque detected, even after probing the area with a dental explorer.
* Score 1: Plaque detected by visual examination and/or probing but is not visible when the area is gently dried with air.
* Score 2: Moderate accumulation of soft deposits that are visible upon visual examination before air drying, but which can be removed by scraping with a dental explorer.
* Score 3: Abundant soft matter, visible upon visual examination before air drying and not easily removable with a dental explorer.

The DPI is calculated as the average score of all surfaces examined, providing an overall measure of plaque accumulation in the mouth. It can be used to monitor changes in oral hygiene over time or to evaluate the effectiveness of different oral hygiene interventions. However, it should be noted that the DPI has limitations and may not accurately reflect the presence of bacterial biofilms or the risk of dental caries and gum disease.

Periodontitis is a severe form of gum disease that damages the soft tissue and destroys the bone supporting your teeth. If left untreated, it can lead to tooth loss. It is caused by the buildup of plaque, a sticky film of bacteria that constantly forms on our teeth. The body's immune system fights the bacterial infection, which causes an inflammatory response. If the inflammation continues for a long time, it can damage the tissues and bones that support the teeth.

The early stage of periodontitis is called gingivitis, which is characterized by red, swollen gums that bleed easily when brushed or flossed. When gingivitis is not treated, it can advance to periodontitis. In addition to plaque, other factors that increase the risk of developing periodontitis include smoking or using tobacco products, poor oral hygiene, diabetes, a weakened immune system, and genetic factors.

Regular dental checkups and good oral hygiene practices, such as brushing twice a day, flossing daily, and using an antimicrobial mouth rinse, can help prevent periodontitis. Treatment for periodontitis may include deep cleaning procedures, medications, or surgery in severe cases.

A gingivectomy is a dental procedure that involves the surgical removal or reshaping of the gum tissue (gingiva) to improve the health and appearance of the teeth and gums. This procedure is typically performed when the gums have become swollen, inflamed, or infected due to periodontal disease, which can cause the gums to recede and expose the tooth roots. By removing the affected gum tissue, a gingivectomy can help to eliminate pockets of bacteria and promote healthy gum growth.

During the procedure, a dental surgeon will use local anesthesia to numb the area and then carefully cut away the excess gum tissue using specialized instruments. The surgeon may also smooth and reshape the remaining gum tissue to create a more even and aesthetically pleasing appearance. After the procedure, patients may experience some discomfort, swelling, or bleeding, but these symptoms can typically be managed with over-the-counter pain medications and careful oral hygiene practices.

It's important to note that while a gingivectomy can help to improve the health of the gums and teeth, it is not a substitute for good oral hygiene habits. Regular brushing, flossing, and dental checkups are essential for maintaining healthy teeth and gums over the long term.

According to the American Academy of Periodontology, periodontal diseases are chronic inflammatory conditions that affect the tissues surrounding and supporting the teeth. These tissues include the gums, periodontal ligament, and alveolar bone. The primary cause of periodontal disease is bacterial plaque, a sticky film that constantly forms on our teeth.

There are two major stages of periodontal disease:

1. Gingivitis: This is the milder form of periodontal disease, characterized by inflammation of the gums (gingiva) without loss of attachment to the teeth. The gums may appear red, swollen, and bleed easily during brushing or flossing. At this stage, the damage can be reversed with proper dental care and improved oral hygiene.
2. Periodontitis: If left untreated, gingivitis can progress to periodontitis, a more severe form of periodontal disease. In periodontitis, the inflammation extends beyond the gums and affects the deeper periodontal tissues, leading to loss of bone support around the teeth. Pockets filled with infection-causing bacteria form between the teeth and gums, causing further damage and potential tooth loss if not treated promptly.

Risk factors for developing periodontal disease include poor oral hygiene, smoking or using smokeless tobacco, genetic predisposition, diabetes, hormonal changes (such as pregnancy or menopause), certain medications, and systemic diseases like AIDS or cancer. Regular dental check-ups and good oral hygiene practices are crucial for preventing periodontal disease and maintaining overall oral health.

Pancreatic neoplasms refer to abnormal growths in the pancreas that can be benign or malignant. The pancreas is a gland located behind the stomach that produces hormones and digestive enzymes. Pancreatic neoplasms can interfere with the normal functioning of the pancreas, leading to various health complications.

Benign pancreatic neoplasms are non-cancerous growths that do not spread to other parts of the body. They are usually removed through surgery to prevent any potential complications, such as blocking the bile duct or causing pain.

Malignant pancreatic neoplasms, also known as pancreatic cancer, are cancerous growths that can invade and destroy surrounding tissues and organs. They can also spread (metastasize) to other parts of the body, such as the liver, lungs, or bones. Pancreatic cancer is often aggressive and difficult to treat, with a poor prognosis.

There are several types of pancreatic neoplasms, including adenocarcinomas, neuroendocrine tumors, solid pseudopapillary neoplasms, and cystic neoplasms. The specific type of neoplasm is determined through various diagnostic tests, such as imaging studies, biopsies, and blood tests. Treatment options depend on the type, stage, and location of the neoplasm, as well as the patient's overall health and preferences.

"Porphyromonas gingivalis" is a gram-negative, anaerobic, rod-shaped bacterium that is commonly found in the oral cavity and is associated with periodontal disease. It is a major pathogen in chronic periodontitis, which is a severe form of gum disease that can lead to destruction of the tissues supporting the teeth, including the gums, periodontal ligament, and alveolar bone.

The bacterium produces several virulence factors, such as proteases and endotoxins, which contribute to its pathogenicity. It has been shown to evade the host's immune response and cause tissue destruction through various mechanisms, including inducing the production of pro-inflammatory cytokines and matrix metalloproteinases.

P. gingivalis has also been linked to several systemic diseases, such as atherosclerosis, rheumatoid arthritis, and Alzheimer's disease, although the exact mechanisms of these associations are not fully understood. Effective oral hygiene practices, including regular brushing, flossing, and professional dental cleanings, can help prevent the overgrowth of P. gingivalis and reduce the risk of periodontal disease.

A periodontal pocket is a pathological space or gap that develops between the tooth and the surrounding gum tissue (gingiva) as a result of periodontal disease. This condition is also known as a "periodontal depth" or "probing depth." It is measured in millimeters using a dental probe, and it indicates the level of attachment loss of the gingival tissue to the tooth.

In a healthy periodontium, the sulcus (the normal space between the tooth and gum) measures 1-3 mm in depth. However, when there is inflammation due to bacterial accumulation, the gums may become red, swollen, and bleed easily. As the disease progresses, the sulcus deepens, forming a periodontal pocket, which can extend deeper than 3 mm.

Periodontal pockets provide an environment that is conducive to the growth of harmful bacteria, leading to further tissue destruction and bone loss around the tooth. If left untreated, periodontal disease can result in loose teeth and eventually tooth loss. Regular dental check-ups and professional cleanings are essential for maintaining healthy gums and preventing periodontal pockets from developing or worsening.

Neoplasms are abnormal growths of cells or tissues in the body that serve no physiological function. They can be benign (non-cancerous) or malignant (cancerous). Benign neoplasms are typically slow growing and do not spread to other parts of the body, while malignant neoplasms are aggressive, invasive, and can metastasize to distant sites.

Neoplasms occur when there is a dysregulation in the normal process of cell division and differentiation, leading to uncontrolled growth and accumulation of cells. This can result from genetic mutations or other factors such as viral infections, environmental exposures, or hormonal imbalances.

Neoplasms can develop in any organ or tissue of the body and can cause various symptoms depending on their size, location, and type. Treatment options for neoplasms include surgery, radiation therapy, chemotherapy, immunotherapy, and targeted therapy, among others.

Epithelial attachment is a general term that refers to the point where epithelial cells, which are the cells that line the outer surfaces of organs and blood vessels, adhere or attach to an underlying structure. In the context of the mouth and teeth, epithelial attachment is often used to describe the connection between the gum tissue (gingiva) and the tooth surface.

In a healthy mouth, the gingival tissue fits tightly around each tooth, forming a protective seal that helps prevent bacteria and other harmful substances from entering the spaces between the teeth and gums. This tight seal is maintained by specialized epithelial cells called junctional epithelial cells, which form a barrier between the oral environment and the underlying connective tissue.

When the gingival tissue becomes inflamed due to factors such as poor oral hygiene or certain medical conditions, the epithelial attachment can become compromised, leading to a condition known as gingivitis. If left untreated, gingivitis can progress to periodontal disease, which is characterized by the destruction of the tissues that support the teeth, including the bone and connective tissue.

In summary, epithelial attachment refers to the point where epithelial cells adhere to an underlying structure, and in the context of oral health, it describes the connection between the gum tissue and the tooth surface.

Chronic periodontitis is a type of gum disease that is characterized by the inflammation and infection of the tissues surrounding and supporting the teeth. It is a slow-progressing condition that can lead to the destruction of the periodontal ligament and alveolar bone, which can result in loose teeth or tooth loss if left untreated.

Chronic periodontitis is caused by the buildup of dental plaque and calculus (tartar) on the teeth, which harbor bacteria that release toxins that irritate and inflame the gums. Over time, this chronic inflammation can lead to the destruction of the periodontal tissues, including the gingiva, periodontal ligament, and alveolar bone.

The signs and symptoms of chronic periodontitis include:

* Red, swollen, or tender gums
* Bleeding gums during brushing or flossing
* Persistent bad breath (halitosis)
* Receding gums (exposure of the tooth root)
* Loose teeth or changes in bite alignment
* Deep periodontal pockets (spaces between the teeth and gums)

Risk factors for chronic periodontitis include poor oral hygiene, smoking, diabetes, genetics, and certain medications. Treatment typically involves a thorough dental cleaning to remove plaque and calculus, followed by additional procedures such as scaling and root planing or surgery to eliminate infection and promote healing of the periodontal tissues. Good oral hygiene practices, regular dental checkups, and quitting smoking are essential for preventing chronic periodontitis and maintaining good oral health.

The periodontium is a complex structure in the oral cavity that surrounds and supports the teeth. It consists of four main components:
1. Gingiva (gums): The pink, soft tissue that covers the crown of the tooth and extends down to the neck of the tooth, where it meets the cementum.
2. Cementum: A specialized, calcified tissue that covers the root of the tooth and provides a surface for the periodontal ligament fibers to attach.
3. Periodontal ligament (PDL): A highly vascular and cell-rich connective tissue that attaches the cementum of the tooth root to the alveolar bone, allowing for tooth mobility and absorption of forces during chewing.
4. Alveolar bone: The portion of the jawbone that contains the sockets (alveoli) for the teeth. It is a spongy bone with a rich blood supply that responds to mechanical stresses from biting and chewing, undergoing remodeling throughout life.

Periodontal diseases, such as gingivitis and periodontitis, affect the health and integrity of the periodontium, leading to inflammation, bleeding, pocket formation, bone loss, and ultimately tooth loss if left untreated.

Neoplasms: Neoplasms refer to abnormal growths of tissue that can be benign (non-cancerous) or malignant (cancerous). They occur when the normal control mechanisms that regulate cell growth and division are disrupted, leading to uncontrolled cell proliferation.

Cystic Neoplasms: Cystic neoplasms are tumors that contain fluid-filled sacs or cysts. These tumors can be benign or malignant and can occur in various organs of the body, including the pancreas, ovary, and liver.

Mucinous Neoplasms: Mucinous neoplasms are a type of cystic neoplasm that is characterized by the production of mucin, a gel-like substance produced by certain types of cells. These tumors can occur in various organs, including the ovary, pancreas, and colon. Mucinous neoplasms can be benign or malignant, and malignant forms are often aggressive and have a poor prognosis.

Serous Neoplasms: Serous neoplasms are another type of cystic neoplasm that is characterized by the production of serous fluid, which is a thin, watery fluid. These tumors commonly occur in the ovary and can be benign or malignant. Malignant serous neoplasms are often aggressive and have a poor prognosis.

In summary, neoplasms refer to abnormal tissue growths that can be benign or malignant. Cystic neoplasms contain fluid-filled sacs and can occur in various organs of the body. Mucinous neoplasms produce a gel-like substance called mucin and can also occur in various organs, while serous neoplasms produce thin, watery fluid and commonly occur in the ovary. Both mucinous and serous neoplasms can be benign or malignant, with malignant forms often being aggressive and having a poor prognosis.

Gingivoplasty is a surgical procedure in dentistry that involves the reshaping or contouring of the gingiva (gums). This procedure is typically performed for aesthetic purposes, to improve the appearance of gums that are uneven or have an irregular shape. It can also be done to remove excess gum tissue that may be covering too much of a tooth, making it appear shorter than the other teeth.

Gingivoplasty is often recommended as a part of periodontal treatment to ensure the proper fit and function of dental restorations or to manage and prevent gum disease. The procedure involves removing and reshaping the gingival tissue to create a more aesthetically pleasing and healthy gum line.

It's important to note that while gingivoplasty can improve the appearance of the gums, it does not address any underlying issues related to gum disease or bone loss. Additional periodontal treatments may be necessary to address these concerns.

Periodontal attachment loss (PAL) is a clinical measurement in dentistry that refers to the amount of connective tissue attachment between the tooth and its surrounding supportive structures (including the gingiva, periodontal ligament, and alveolar bone) that has been lost due to periodontal disease. It is typically expressed in millimeters and represents the distance from the cementoenamel junction (CEJ), which is the point where the tooth's crown meets the root, to the bottom of the periodontal pocket.

Periodontal pockets are formed when the gums detach from the tooth due to inflammation and infection caused by bacterial biofilms accumulating on the teeth. As the disease progresses, more and more of the supporting structures are destroyed, leading to increased pocket depths and attachment loss. This can eventually result in loose teeth and even tooth loss if left untreated.

Therefore, periodontal attachment loss is an important indicator of the severity and progression of periodontal disease, and its measurement helps dental professionals assess the effectiveness of treatment interventions and monitor disease status over time.

Dental plaque is a biofilm or mass of bacteria that accumulates on the surface of the teeth, restorative materials, and prosthetic devices such as dentures. It is initiated when bacterial colonizers attach to the smooth surfaces of teeth through van der Waals forces and specific molecular adhesion mechanisms.

The microorganisms within the dental plaque produce extracellular polysaccharides that help to stabilize and strengthen the biofilm, making it resistant to removal by simple brushing or rinsing. Over time, if not regularly removed through oral hygiene practices such as brushing and flossing, dental plaque can mineralize and harden into tartar or calculus.

The bacteria in dental plaque can cause tooth decay (dental caries) by metabolizing sugars and producing acid that demineralizes the tooth enamel. Additionally, certain types of bacteria in dental plaque can cause periodontal disease, an inflammation of the gums that can lead to tissue damage and bone loss around the teeth. Regular professional dental cleanings and good oral hygiene practices are essential for preventing the buildup of dental plaque and maintaining good oral health.

Skin neoplasms refer to abnormal growths or tumors in the skin that can be benign (non-cancerous) or malignant (cancerous). They result from uncontrolled multiplication of skin cells, which can form various types of lesions. These growths may appear as lumps, bumps, sores, patches, or discolored areas on the skin.

Benign skin neoplasms include conditions such as moles, warts, and seborrheic keratoses, while malignant skin neoplasms are primarily classified into melanoma, squamous cell carcinoma, and basal cell carcinoma. These three types of cancerous skin growths are collectively known as non-melanoma skin cancers (NMSCs). Melanoma is the most aggressive and dangerous form of skin cancer, while NMSCs tend to be less invasive but more common.

It's essential to monitor any changes in existing skin lesions or the appearance of new growths and consult a healthcare professional for proper evaluation and treatment if needed.

Gingival retraction techniques are clinical procedures used in dentistry to temporarily retract the gingiva (gums) and expose the teeth's crown surfaces, especially the portion that is located below the gumline, known as the cementoenamel junction (CEJ), for a more precise impression making. This procedure is essential during the fabrication of dental restorations such as crowns, bridges, and dentures to ensure a proper fit and avoid future problems like recurrent decay or periodontal disease.

There are two main categories of gingival retraction techniques: mechanical and chemical methods. Mechanical methods involve the use of specific instruments, such as gingival retraction cords or plastic retraction devices, to physically displace the gum tissue from the tooth's preparation margin. In contrast, chemical methods rely on solutions containing astringents (like aluminum chloride or ferric sulfate) that cause the gingival tissues to contract and retract.

In some cases, a combination of both mechanical and chemical techniques may be employed for optimal retraction and hemostasis control. The choice of technique depends on factors like the patient's oral condition, the type and complexity of restoration being made, and the clinician's preference.

Multiple primary neoplasms refer to the occurrence of more than one primary malignant tumor in an individual, where each tumor is unrelated to the other and originates from separate cells or organs. This differs from metastatic cancer, where a single malignancy spreads to multiple sites in the body. Multiple primary neoplasms can be synchronous (occurring at the same time) or metachronous (occurring at different times). The risk of developing multiple primary neoplasms increases with age and is associated with certain genetic predispositions, environmental factors, and lifestyle choices such as smoking and alcohol consumption.

Necrotizing ulcerative gingivitis (NUG), also known as trench mouth or acute necrotizing ulcerative gingivostomatitis, is a severe and painful form of gingivitis that is characterized by the presence of necrosis (tissue death) and ulcers in the gum tissue. It is caused by a combination of factors, including poor oral hygiene, stress, smoking, and a weakened immune system. The condition is often associated with the presence of certain types of bacteria that produce toxins that can damage the gum tissue.

NUG is characterized by the sudden onset of symptoms such as severe pain, bleeding, bad breath, and a grayish-white or yellowish film covering the gums. The gums may also appear bright red, swollen, and shiny, and may bleed easily when brushed or touched. In some cases, the condition can progress to involve other areas of the mouth, such as the lining of the cheeks and lips.

NUG is typically treated with a combination of professional dental cleaning, antibiotics to eliminate the bacterial infection, and pain management. It is important to maintain good oral hygiene practices to prevent recurrence of the condition. If left untreated, NUG can lead to more serious complications such as tooth loss or spread of the infection to other parts of the body.

A toothache is defined as pain or discomfort in or around a tooth, usually caused by dental cavities, gum disease, tooth fracture, or exposed tooth roots. The pain may be sharp and stabbing, throbbing, or constant and dull. It can also be aggravated by hot, cold, sweet, or sour foods and drinks, or by biting or chewing. Toothaches are serious and should not be ignored as they can be a sign of more significant dental issues that require immediate professional attention from a dentist.

"Dry socket" is a common term used in dentistry to describe a condition that can occur after a tooth extraction. The medical term for dry socket is "alveolar osteitis." This condition arises when the blood clot that forms in the socket where the tooth was removed becomes dislodged or fails to form properly, exposing the bone and nerves underneath.

Dry socket can be quite painful, causing a throbbing sensation that may radiate to the ear, neck, or temple. It can also lead to bad breath and an unpleasant taste in the mouth. The exact cause of dry socket is not entirely clear, but several factors may increase the risk, including smoking, poor oral hygiene, using birth control pills, and having a history of dry socket.

Treatment for dry socket typically involves cleaning the socket and placing a medicated dressing to promote healing and relieve pain. Over-the-counter pain medications and warm compresses may also help alleviate discomfort. It is essential to follow your dentist's instructions carefully to prevent complications and promote proper healing.

A periapical abscess is a localized infection that occurs at the tip of the tooth's root, specifically in the periapical tissue. This tissue surrounds the end of the tooth's root and helps anchor the tooth to the jawbone. The infection is usually caused by bacteria that enter the pulp chamber of the tooth as a result of dental caries (tooth decay), periodontal disease, or trauma that damages the tooth's protective enamel layer.

The infection leads to pus accumulation in the periapical tissue, forming an abscess. The symptoms of a periapical abscess may include:

1. Pain and tenderness in the affected tooth, which can be throbbing or continuous
2. Swelling in the gums surrounding the tooth
3. Sensitivity to hot, cold, or pressure on the tooth
4. Fever, general malaise, or difficulty swallowing (in severe cases)
5. A foul taste in the mouth or bad breath
6. Tooth mobility or loosening
7. Formation of a draining sinus tract (a small opening in the gums that allows pus to drain out)

Periapical abscesses require dental treatment, which typically involves removing the infected pulp tissue through root canal therapy and cleaning, shaping, and sealing the root canals. In some cases, antibiotics may be prescribed to help control the infection, but they do not replace the necessary dental treatment. If left untreated, a periapical abscess can lead to severe complications, such as the spread of infection to other parts of the body or tooth loss.

A periodontal abscess is a localized collection of pus in the tissues surrounding and supporting the teeth, caused by an infection. It's typically characterized by symptoms such as pain, swelling, redness, and sometimes drainage of pus from the affected area. The infection usually arises from dental plaque that accumulates on the teeth and gums, leading to periodontal disease. If left untreated, a periodontal abscess can result in tissue destruction, bone loss, and even tooth loss. Treatment typically involves draining the abscess, removing any infected tissue, and providing oral hygiene instruction to prevent future infections. In some cases, antibiotics may also be prescribed to help clear up the infection.

Pulpitis is a dental term that refers to the inflammation of the pulp, which is the soft tissue inside the center of a tooth that contains nerves, blood vessels, and connective tissue. The pulp helps to form the dentin, the hard layer beneath the enamel. Pulpitis can result from tooth decay, dental trauma, or other factors that cause damage to the tooth's protective enamel and dentin layers, exposing the pulp to irritants and bacteria.

There are two types of pulpitis: reversible and irreversible. Reversible pulpitis is characterized by mild inflammation that can be treated and potentially reversed with dental intervention, such as a filling or root canal treatment. Irreversible pulpitis, on the other hand, involves severe inflammation that cannot be reversed, and typically requires a root canal procedure to remove the infected pulp tissue and prevent further infection or damage to the tooth.

Symptoms of pulpitis may include tooth sensitivity to hot or cold temperatures, pain or discomfort when biting down or applying pressure to the tooth, and in some cases, spontaneous or radiating pain. If left untreated, pulpitis can lead to more serious dental issues, such as abscesses or bone loss around the affected tooth.

An encyclopedia is a comprehensive reference work containing articles on various topics, usually arranged in alphabetical order. In the context of medicine, a medical encyclopedia is a collection of articles that provide information about a wide range of medical topics, including diseases and conditions, treatments, tests, procedures, and anatomy and physiology. Medical encyclopedias may be published in print or electronic formats and are often used as a starting point for researching medical topics. They can provide reliable and accurate information on medical subjects, making them useful resources for healthcare professionals, students, and patients alike. Some well-known examples of medical encyclopedias include the Merck Manual and the Stedman's Medical Dictionary.

10] Gingival melanoacanthomas may be evident as solitary or multiple. [11] Oral melanoacanthoma may be bilateral. [12] It may ... The cause of melanoacanthoma is unknown, but most instances appear to represent a benign neoplasm. Irritation or trauma to the ... A gingival melanoacanthoma was believed to be caused by teeth-whitening strips. [34] ... Tapia JL, Quezada D, Gaitan L, Hernandez JC, Paez C, Aguirre A. Gingival melanoacanthoma: case report and discussion of its ...
Examples include neoplasms of the gingival or alveolar mucosa (usually squamous cell carcinoma),: 299 conditions which cause ... A variant of the periodontal abscess is the gingival abscess, which is limited to the gingival margin, has a quicker onset, and ... A healthy gingival pocket will contain bacteria and some calculus kept in check by the immune system. As the pocket deepens, ... The gingival papilla is swollen, tender and bleeds when touched. The pain occurs during and after eating, and may slowly ...
O Gingival overgrowth,O Gingival recession,O Gingivitis,O Glabellar hemangioma,O Glabellar reflex,O Glandular cell neoplasm,O ... O Neoplasm of the inner ear,O Neoplasm of the large intestine,O Neoplasm of the larynx,O Neoplasm of the lip,O Neoplasm of the ... O Neoplasm of the middle ear,O Neoplasm of the nail,O Neoplasm of the nervous system,O Neoplasm of the nose,O Neoplasm of the ... O Neoplasm of the colon,O Neoplasm of the ear,O Neoplasm of the endocrine system,O Neoplasm of the eye,O Neoplasm of the ...
Benign Breast Neoplasm, Fibula Fracture, LOW Turnover Osteopathy, Back Pain, Hiatus Hernia, Pain in Extremity, Mastitis, ... Gingival Disorder, Pancytopenia, Helicobacter Infection, Osteopenia, Hypoaesthesia, Stress, Upper Respiratory Tract Congestion ... Lung Neoplasm, Biopsy Cervix, Patellofemoral Pain Syndrome, Ovarian Cyst, Biopsy Breast, Cervical Dysplasia, Bursitis, Meniscus ... Lung Neoplasm, Skin Disorder, Anxiety, Osteoarthritis, Arthralgia, Spinal Osteoarthritis, Hypertension, Endometrial Atrophy ...
Peripheral odontogenic fibroma: a rare gingival neoplasm with clinico-pathological differential diagnosis.. Kumar, Rajesh; ...
Keywords : Gingival diseases.; Gingival neoplasms.; Infant newborn.. · abstract in Portuguese · text in English · pdf in ...
The purpose of this study is to elucidate the association between PM2.5 and oral neoplasm, including oral potentially malignant ... gingival, floor of mouth, palate, other parts of mouth, oropharyngeal, hypopharyngeal and unspecified pharyngeal cancer. OPMD ... The Association between Ambient Fine Particulate Matter and Oral Neoplasm among Smokers and Betel Quids Chewers Details 2021 - ... 2. Geographically gradients of oral neoplasms by county/city. (a) rate (%) of OPMD and OC cases (b) rate (%) of OPMD cases (c) ...
Gingival Hemorrhage (28). *Gingival Hyperplasia (9). *Gingival Hypertrophy (7). *Gingival Neoplasms (1) ...
Gingival Neoplasms Medicine & Life Sciences 48% * Literature Medicine & Life Sciences 28% * Staining and Labeling Medicine & ...
... and benign and malignant neoplasms of the minor salivary glands (eg, see Salivary Gland Neoplasms). ... Stokes SM, Sherban M, Carle L, Dryden S. A rapidly enlarging gingival mass in an 11-year-old boy. J Am Dent Assoc. 2016 Jan. ... Drug-induced gingival enlargement: an overview. Compend Contin Educ Dent. 2013 May. 34 (5):330-6. [QxMD MEDLINE Link]. ... Gingival fibromatosis with hypertrichosis syndrome: Case series of rare syndrome. Indian J Dent Res. 2017 Jul-Aug. 28 (4):457- ...
Keywords: oral cancer; squamous cell carcinoma; gingival neoplasms; gingival recession; early detection of cancer. ... Keywords: acute myeloid leukemia; flow cytometry; gingival hyperplasia. Gingival Squamous Cell Carcinoma: a Case Report. Luiz ... Generalised Leukaemic Gingival Enlargement: a Case Report. Mechery Reenesh, Singh Munishwar, Saroj Kumar Rath. J Oral ... Keywords: gingival crevicular fluid; inflammation; nitric oxide; periodontitis; saliva. Giant Cell Fibroma in Children: Report ...
Plasma Cell Neoplasms. 13 (.2%). Dental And Gingival Conditions. 12 (.19%). Gastrointestinal Stenosis And Obstr.... 12 (.19%) ...
Cyclophosphamide is an alkylating agent used as a chemotherapeutic agent in hematologic neoplasms. It produces DNA toxicity and ... The main side effects of cyclosporine are hypertension, gingival hyperplasia, skin rash, tremor, and nephrotoxicity. Blood ... The main side effects are hypertension, gingival hyperplasia, skin rash, tremor, and nephrotoxicity. ... Monoclonal gammopathies may appear in the context of a hematologic neoplasm (multiple myeloma, B cell leukemias, etc.); in the ...
... rapidly enlarging gingival mass with a histologic appearance of an undifferentiated neoplasm. Immunoperoxidase studies revealed ... rapidly enlarging gingival mass with a histologic appearance of an undifferentiated neoplasm. Immunoperoxidase studies revealed ... rapidly enlarging gingival mass with a histologic appearance of an undifferentiated neoplasm. Immunoperoxidase studies revealed ... rapidly enlarging gingival mass with a histologic appearance of an undifferentiated neoplasm. Immunoperoxidase studies revealed ...
Blastic plasmacytoid dendritic cell neoplasm: A rare case of gingival lesion with leukaemic presentation, file e0feeaa7-7b53- ...
Oral mucosa: gingival squamous hyperplasia (0/50, 8/50, 24/50, 24/50); gingival cystic keratinizing hyperplasia (0/50, 4/50, 9/ ... The increased incidences of carcinoma of the urethra, alveolar/bronchiolar neoplasms and cystic keratinizing epithelioma of the ... Oral mucosa: gingival squamous hyperplasia (2/50, 21/50, 24/50, 31/50); gingival cystic keratinizing hyperplasia (0/50, 4/50, ... Oral Mucosa: gingival squamous cell carcinoma (1/50, 5/50, 4/50, 5/50) Thyroid gland: follicular cell adenoma (0/50, 3/50, 4/50 ...
1.2.6.4.1. gingival hyperplasia- infiltration of cancer cells into gingival tissues ... malignant neoplasm:. 1.2.6.1.1. excess # of abnormal, immature WBCs. 1.2.6.2. unknown causes ... 1.5.1.3.1. spontaneous bleeding (ie nose bleeds, gingival bleeding w/out inflammation) ...
Create healthcare diagrams like this example called Gingival Enlargement in minutes with SmartDraw. SmartDraw includes 1000s of ... Gingival Enlargement. Create healthcare diagrams like this example called Gingival Enlargement in minutes with SmartDraw. ... Gingival Enlargement. Anterior view of teeth and gums afflicted with gingival enlargement.. LifeART Collection Images Copyright ...
Here, we report a case of NHL manifesting as a gingival mass in a 45-year-old HIV-positive female patient. ... Although uncommon, it is essential to be wary of this neoplasm since intraoral manifestations may be the first clinical ... Gingival mass in acquired immune deficiency syndrome patient: An unusual manifestation. p. 78. ... Careful evaluation of patients presenting with solitary atypical gingival mass can lead to early detection of HIV disease. ...
Tooth Wear Loose Teeth Teething Tooth Damage Bone Loss Postoperative Pain Cosmetic Peridontal Problems Dental Tissue Neoplasm ... Facial Pain Mouth Lesions Mouth Ulcer Tooth Abnormalities as Seen in Diabetes Tooth Decay Caused by Acid Attack Gingival ...
The mostly viral-related neoplasms seen in these sufferers are oropharyngeal and dental malignancies, whereas nearly all not- ... gingival hyperplasia, xerostomia or halitosis. Immunosuppressive therapy after kidney transplantation escalates the threat of ...
Gingival Neoplasms. *Leukoplakia, Oral. *Lip Neoplasms. *Palatal Neoplasms. *Salivary Gland Neoplasms. *Tongue Neoplasms ... "Tongue Neoplasms" is a descriptor in the National Library of Medicines controlled vocabulary thesaurus, MeSH (Medical Subject ... This graph shows the total number of publications written about "Tongue Neoplasms" by people in this website by year, and ... Below are the most recent publications written about "Tongue Neoplasms" by people in Profiles. ...
B) Gingival leukemic infiltrate in newly diagnosed acute myelogenous leukemia patient. Note the evidence of extensive gingival ... Figure 8‐2 Acute leukemia can be considered "naturally metastatic" in that the neoplasm arises within the white blood cell ... the dental tray should overlap the gingival margins of the teeth and still avoid unnecessary contact with the gingiva). ...
Neoplasms benign, malignant and unspecified (incl. cysts and polyps) Skin papilloma 1 Herpes virus infections include PTs: Oral ... gingival bleeding, dry mouth, dysphagia, gastrointestinal disorder, haematochezia Hepatobiliary disorders Aspartate ...
The authors investigated the effect of LL-37 on the gene regulation of human gingival fibroblasts (HGFs). During this study, it ... in patients with solid neoplasms. Kapoor et al. reports that the median overall survival of the patients who received ... Concomitant use of antibiotics and immune checkpoint inhibitors in patients with solid neoplasms: Retrospective data from real- ... Antimicrobial Peptide LL-37 on Gene Expression of Chemokines and 29 Toll-like Receptor-Associated Proteins in Human Gingival ...

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