Pleurisy
Tuberculosis, Pleural
Pleural Effusion
Carrageenan
Pleura
Adenosine Deaminase
Pleural Effusion, Malignant
Exudates and Transudates
Thoracic Wall
Lung
Beneficial effects of raxofelast (IRFI 016), a new hydrophilic vitamin E-like antioxidant, in carrageenan-induced pleurisy. (1/281)
1. Peroxynitrite is a strong oxidant that results from reaction between NO and superoxide. It has been recently proposed that peroxynitrite plays a pathogenetic role in inflammatory processes. Here we have investigated the therapeutic efficacy of raxofelast, a new hydrophilic vitamin E-like antioxidant agent, in rats subjected to carrageenan-induced pleurisy. 2. In vivo treatment with raxofelast (5, 10, 20 mg kg(-1) intraperitoneally 5 min before carrageenan) prevented in a dose dependent manner carrageenan-induced pleural exudation and polymorphonuclear migration in rats subjected to carrageenan-induced pleurisy. Lung myeloperoxidase (MPO) activity and malondialdehyde (MDA) levels, as well as histological organ injury were significantly reduced by raxofelast. 3. Immunohistochemical analysis for nitrotyrosine, a footprint of peroxynitrite, revealed a positive staining in lungs from carrageenan-treated rats. No positive nitrotyrosine staining was found in the lungs of the carrageenan-treated rats, which received raxofelast (20 mg kg 1) treatment. 4. Furthermore, in vivo raxofelast (5, 10, 20 mg kg(-1)) treatment significantly reduced peroxynitrite formation as measured by the oxidation of the fluorescent dihydrorhodamine 123, prevented the appearance of DNA damage, the decrease in mitochondrial respiration and partially restored the cellular level of NAD+ in ex vivo macrophages harvested from the pleural cavity of rats subjected to carrageenan-induced pleurisy. 5. In conclusion, our study demonstrates that raxofelast, a new hydrophilic vitamin E-like antioxidant agent, exerts multiple protective effects in carrageenan-induced acute inflammation. (+info)Bronchial carcinoma in patients with pre-existing unilateral lung disease. (2/281)
Forty-six cases of primary bronchogenic carcinoma occurring in patients with other unilateral pleuropulmonary diseases were studied. In 37 cases (80-4%) carcinoma developed in the previously healthy lung. All but one squamous-cell carcinoma and all of five undifferentiated small-cell carcinomas developed in the previously healthy lung while 7 of 15 adenocarcinomas were in the lung with impaired ventilation. It is suggested that the bronchial epithelium of the healthy lung is more exposed to exogenous carcinogens than that of the diseased, underventilated lung, resulting in a higher risk of development of squamous-cell and undifferentiated small-cell carcinoma. (+info)Effect of a novel non-steroidal anti-inflammatory drug (M-5011) on cytokine levels in rats with monosodium urate crystal- induced pleurisy. (3/281)
We evaluated the effects of a new non-steroidal anti-inflammatory drug (NSAID), d-2-[4-(3-methyl-2-thienyl)phenyl]propionic acid (M-5011), and indomethacin on the production of arachidonate metabolites and pro-inflammatory cytokines in male Sprague-Dawley rats with monosodium urate crystal (MSU)-induced pleurisy. Levels of tumor necrosis factor (TNF), interleukin (IL)-1 and IL-6 in the pleural exudate were determined by biological assays, while prostaglandin E2 (PGE2), leukotriene B4 (LTB4) and cytokine-induced chemoattractant-1 (CINC-1) levels were quantified by enzyme immunoassays. Orally administered M-5011 (5 mg/kg) decreased the pleural exudate volume at 3 and 4 hr after MSU injection. Indomethacin (10 mg/kg) decreased the volume at 3-5 hr. These drugs reduced the number of leukocytes in the pleural cavity at 6 hr. Both NSAIDs also reduced the content of PGE2 in the exudate without affecting LTB4 levels. Increased productions of both IL-6 and CINC-1 in the exudate were reduced by pretreatment with M-5011 or indomethacin, and TNF levels in the exudate were increased by pretreatment of these drugs. Thus, M-5011 inhibits the production of both IL-6 and CINC-1 at lower doses than those of indomethacin, and the inhibitory effect of M-5011 on CINC-1, but not IL-6, may partly contribute to the inhibition of leukocyte infiltration in rats with MSU-induced pleurisy. (+info)Bilateral pleuritis caused by Legionella micdadei. (4/281)
A 58-year-old woman was hospitalized because of progressive respiratory distress. She had a history of myasthenia gravis and invasive thymoma. After thymectomy, she had been administered oral prednisolone and intrathoracic anti-cancer drugs postoperatively. Her chest radiograph revealed bilateral pleural effusions. Legionella micdadei (L. micdadei) was isolated from the pleural effusions, and she was diagnosed as pleuritis caused by L. micdadei. She died despite intensive therapy with mechanical ventilation, drainage tube in the chest and intravenous erythromycin. Although only two cases of Legionellosis caused by L. micdadei have been reported in Japan, clinicians should be aware of L. micdadei as one of the candidates for infection in immunosuppressed hosts. (+info)Suppressive effect of distinct bradykinin B2 receptor antagonist on allergen-evoked exudation and leukocyte infiltration in sensitized rats. (5/281)
1. Bradykinin is suggested to play a role in the pathophysiology of several acute and chronic diseases, including allergic disorders such as asthma. In the present study, we have investigated the importance of bradykinin in mediating allergic inflammation in rats. 2. To this end we have tested the effects of the B2 receptor antagonists Hoe 140, FR173657 or FR172357 on the pleural inflammatory response triggered by intrapleural (i.pl.) injection of allergen (ovalbumin, 12 microg cavity(-1)) in 14 day-actively sensitized Wistar rats. Analysis of the pleural fluid effluent revealed a sequence of mast cell-dependent inflammatory events, including early protein exudation and neutrophilia and late pleural eosinophil influx. 3. Local treatment with Hoe 140 (0.1 and 1 microg cavity(-1)), FR173657 (1 and 10 microg cavity(-1)) or FR172357 (1 and 10 microg cavity(-1)) inhibited dose-dependently allergen-induced mast cell activation with impairment of pleural plasma leakage, neutrophil accumulation and late eosinophil influx. 4. Moreover, the B2 receptor antagonists also dose-dependently inhibited the allergic like inflammatory pleurisy triggered by bradykinin (50 microg cavity(-1)), which is characterized by acute mast cell degranulation, protein leakage and pleural eosinophil infiltration. 5. Taken together, our findings provide substantial evidence to suggest that bradykinin acting on its B2 receptors play a critical role in mediating allergic mast cell-dependent inflammation in rats, and suggest that B2 receptor antagonists may be useful therapeutically to control allergic dysfunction. (+info)Bradykinin down-regulates LPS-induced eosinophil accumulation in the pleural cavity of mice through type 2-kinin receptor activation: a role for prostaglandins. (6/281)
1. The role of both exogenously administered and endogenously generated bradykinin (BK) on LPS-induced eosinophil accumulation in the mice pleural cavity was investigated by means of treatment with BK selective receptor agonists/antagonists and captopril. 2. Intrathoracic (i.t.) injection of LPS (250 ng cavity(-1)) induced eosinophil influx at 24 h as previously described (Bozza et al., 1993). Pretreatment with the B1 receptor antagonist des-Arg9-[leu-8]BK (0.025 and 0.25 nmol cavity(-1)) showed no effect on this phenomenon, whereas pretreatment with the B2 receptor antagonists, NPC 17731 (0.025 and 0.25 nmol cavity(-1)) or HOE 140 (2.5 nmol cavity(-1)), increased LPS-induced eosinophil influx. Accordingly, pretreatment with captopril at 10 mg kg(-1) i.p., inhibited eosinophil infiltration induced by LPS in the pleural cavity, suggesting that endogenous BK is down-regulating LPS-induced eosinophil accumulation. 3. BK administered at 15 and 25 nmol cavity(-1), i.t. or i.p. also inhibited LPS-induced eosinophil accumulation. BK alone had no effect on the basal number of leucocytes in the pleural or peritoneal cavity in doses up to 25 nmol cavity(-1). Nevertheless, when injected at doses of 50 and 100 nmol cavity(-1) BK induced leucocyte influx characterized by neutrophil and eosinophil accumulation at 24 h. 4. Similarly to what was observed with BK, a specific B2 receptor agonist, Tyr8BK, administered at 0.25 nmol cavity(-1) i.p., significantly inhibited the eosinophil influx induced by LPS. 5. The mechanism by which B2 receptor agonists inhibit LPS-induced eosinophil accumulation was investigated by pretreating the animals with indomethacin or a selective cyclooxygenase-2 inhibitor, NS-398. Pretreatment with either indomethacin or NS-398 had no effect on eosinophil influx induced by LPS alone, but those drugs were able to restore the LPS-induced eosinophil influx in Tyr8BK (0.25 nmol cavity(-1)) injected mice. 6. In conclusion, endogenously generated bradykinin seems to modulate, through activation of B2 receptors, eosinphil accumulation induced by LPS via a mechanism dependent on prostanoid synthesis. (+info)Role of IL-6 in the pleurisy and lung injury caused by carrageenan. (7/281)
In the present study we used IL-6 knockout mice (IL-6KO) to evaluate the role of IL-6 in the inflammatory response caused by injection of carrageenan into the pleural space. Compared with carrageenan-treated IL-6 wild-type (IL-6WT) mice, carrageenan-treated IL-6KO mice exhibited a reduced degree of pleural exudation and polymorphonuclear cell migration. Lung myeloperoxidase activity and lipid peroxidation were significantly reduced in IL-6KO mice compared with those in IL-6WT mice treated with carrageenan. Immunohistochemical analysis for nitrotyrosine and poly(A)DP-ribose polymerase revealed a positive staining in lungs from carrageenan-treated IL-6WT mice. No positive staining for nitrotyrosine or PARS was found in the lungs of the carrageenan-treated IL-6KO mice. Staining of lung tissue sections obtained from carrageenan-treated IL-6WT mice with an anti-cyclo-oxygenase-2 Ab showed a diffuse staining of the inflamed tissue. Furthermore, expression of inducible nitric oxide synthase was found mainly in the macrophages of the inflamed lungs from carrageenan-treated IL-6WT mice. The intensity and degree of the staining for cyclo-oxygenase-2 and inducible nitric oxide synthase were markedly reduced in tissue sections obtained from carrageenan-treated IL-6KO mice. Most notably, the degree of lung injury caused by carrageenan was also reduced in IL-6KO mice. Treatment of IL-6WT mice with anti-IL-6 (5 microg/day/mouse at 24 and 1 h before carrageenan treatment) also significantly attenuated all the above indicators of lung inflammation. Taken together, our results clearly demonstrate that IL-6KO mice are more resistant to the acute inflammation of the lung caused by carrageenan injection into the pleural space than the corresponding WT mice. (+info)The protective role of endogenous melatonin in carrageenan-induced pleurisy in the rat. (8/281)
Peroxynitrite, a potent cytotoxic oxidant formed by the reaction of nitric oxide (NO) with the superoxide anion, was recently proposed to play a major pathogenic role in the inflammatory process. Here we have investigated the effects of endogenous melatonin, a known scavenger of peroxynitrite, in rats subjected to carrageenan-induced pleurisy. Endogenous melatonin was depleted in rats maintained on 24 h light cycle for 1 wk. In vivo depletion of endogenous melatonin enhanced the carrageenan-induced degree of pleural exudation and polymorphonuclear leukocyte migration in rats subjected to carrageenan-induced pleurisy. Lung myeloperoxidase activity and lipid peroxidation were significantly increased in melatonin-deprived rats. However, the inducible NO synthase in lung samples was unaffected by melatonin depletion. Immunohistochemical analysis for nitrotyrosine revealed a positive staining in lungs from carrageenan-treated rats that was markedly enhanced in melatonin-deprived rats. Furthermore, melatonin depletion significantly increased peroxynitrite formation as measured by the oxidation of the fluorescent dye dihydrorhodamine 123, enhanced DNA damage and the decrease in mitochondrial respiration and reduced the cellular levels of NAD+ in macrophages harvested from the pleural cavity of rats subjected to carrageenan-induced pleurisy. In vivo treatment with exogenous melatonin (15 mg/kg intraperitoneal) significantly reversed the effects of melatonin depletion. Thus, endogenous melatonin plays an important protective role against carrageenan-induced local inflammation. (+info)Pleurisy is a medical condition characterized by inflammation of the pleura, which are the thin membranes that surround the lungs and line the inside of the chest cavity. The pleura normally produce a small amount of lubricating fluid that allows for smooth movement of the lungs during breathing. However, when they become inflamed (a condition known as pleuritis), this can cause pain and difficulty breathing.
The symptoms of pleurisy may include sharp chest pain that worsens with deep breathing or coughing, shortness of breath, cough, fever, and muscle aches. The pain may be localized to one area of the chest or may radiate to other areas such as the shoulders or back.
Pleurisy can have many different causes, including bacterial or viral infections, autoimmune disorders, pulmonary embolism (a blood clot that travels to the lungs), and certain medications or chemicals. Treatment typically involves addressing the underlying cause of the inflammation, as well as managing symptoms such as pain and breathing difficulties with medications such as nonsteroidal anti-inflammatory drugs (NSAIDs) or opioids. In some cases, more invasive treatments such as thoracentesis (removal of fluid from the chest cavity) may be necessary.
Pleural Tuberculosis is a form of extrapulmonary tuberculosis (EPTB) that involves the infection and inflammation of the pleura, which are the thin membranes that surround the lungs and line the inside of the chest cavity. This condition is caused by the Mycobacterium tuberculosis bacterium, which can spread through the air when an infected person coughs, sneezes, or talks.
In pleural tuberculosis, the bacteria reach the pleura either through direct extension from a nearby lung infection or via bloodstream dissemination. The infection can cause the pleura to become inflamed and produce excess fluid, leading to pleural effusion. This accumulation of fluid in the pleural space can cause chest pain, coughing, and difficulty breathing.
Diagnosis of pleural tuberculosis typically involves a combination of medical history, physical examination, imaging studies such as chest X-rays or CT scans, and laboratory tests such as acid-fast bacilli (AFB) smear microscopy, culture, and nucleic acid amplification tests (NAATs) to detect the presence of M. tuberculosis in the pleural fluid or tissue samples.
Treatment of pleural tuberculosis typically involves a standard course of anti-tuberculosis therapy (ATT), which includes a combination of multiple antibiotics such as isoniazid, rifampin, ethambutol, and pyrazinamide. The duration of treatment may vary depending on the severity of the infection and the patient's response to therapy. In some cases, surgical intervention may be necessary to drain the pleural effusion or remove the infected pleura.
Pleural effusion is a medical condition characterized by the abnormal accumulation of fluid in the pleural space, which is the thin, fluid-filled space that surrounds the lungs and lines the inside of the chest wall. This space typically contains a small amount of fluid to allow for smooth movement of the lungs during breathing. However, when an excessive amount of fluid accumulates, it can cause symptoms such as shortness of breath, coughing, and chest pain.
Pleural effusions can be caused by various underlying medical conditions, including pneumonia, heart failure, cancer, pulmonary embolism, and autoimmune disorders. The fluid that accumulates in the pleural space can be transudative or exudative, depending on the cause of the effusion. Transudative effusions are caused by increased pressure in the blood vessels or decreased protein levels in the blood, while exudative effusions are caused by inflammation, infection, or cancer.
Diagnosis of pleural effusion typically involves a physical examination, chest X-ray, and analysis of the fluid in the pleural space. Treatment depends on the underlying cause of the effusion and may include medications, drainage of the fluid, or surgery.
Carriageenans are a family of linear sulfated polysaccharides that are extracted from red edible seaweeds. They have been widely used in the food industry as thickening, gelling, and stabilizing agents. In the medical field, they have been studied for their potential therapeutic applications, such as in the treatment of gastrointestinal disorders and inflammation. However, some studies have suggested that certain types of carriageenans may have negative health effects, including promoting inflammation and damaging the gut lining. Therefore, more research is needed to fully understand their safety and efficacy.
The pleura is the medical term for the double-layered serous membrane that surrounds the lungs and lines the inside of the chest cavity. The two layers of the pleura are called the parietal pleura, which lines the chest cavity, and the visceral pleura, which covers the surface of the lungs.
The space between these two layers is called the pleural cavity, which contains a small amount of lubricating fluid that allows the lungs to move smoothly within the chest during breathing. The main function of the pleura is to protect the lungs and facilitate their movement during respiration.
Adenosine Deaminase (ADA) is an enzyme that plays a crucial role in the immune system by helping to regulate the levels of certain chemicals called purines within cells. Specifically, ADA helps to break down adenosine, a type of purine, into another compound called inosine. This enzyme is found in all tissues of the body, but it is especially active in the immune system's white blood cells, where it helps to support their growth, development, and function.
ADA deficiency is a rare genetic disorder that can lead to severe combined immunodeficiency (SCID), a condition in which babies are born with little or no functional immune system. This makes them extremely vulnerable to infections, which can be life-threatening. ADA deficiency can be treated with enzyme replacement therapy, bone marrow transplantation, or gene therapy.
Malignant pleural effusion is a medical condition characterized by the abnormal accumulation of fluid in the pleural space (the area between the lungs and the chest wall) due to the spread of malignant (cancerous) cells from a primary tumor located elsewhere in the body. This type of effusion is typically associated with advanced-stage cancer, and it can cause symptoms such as shortness of breath, coughing, and chest pain. The presence of malignant pleural effusion often indicates a poor prognosis, and treatment is generally focused on palliating symptoms and improving quality of life.
Exudates and transudates are two types of bodily fluids that can accumulate in various body cavities or tissues as a result of injury, inflammation, or other medical conditions. Here are the medical definitions:
1. Exudates: These are fluids that accumulate due to an active inflammatory process. Exudates contain high levels of protein, white blood cells (such as neutrophils and macrophages), and sometimes other cells like red blood cells or cellular debris. They can be yellow, green, or brown in color and may have a foul odor due to the presence of dead cells and bacteria. Exudates are often seen in conditions such as abscesses, pneumonia, pleurisy, or wound infections.
Examples of exudative fluids include pus, purulent discharge, or inflammatory effusions.
2. Transudates: These are fluids that accumulate due to increased hydrostatic pressure or decreased oncotic pressure within the blood vessels. Transudates contain low levels of protein and cells compared to exudates. They are typically clear and pale yellow in color, with no odor. Transudates can be found in conditions such as congestive heart failure, liver cirrhosis, or nephrotic syndrome.
Examples of transudative fluids include ascites, pleural effusions, or pericardial effusions.
It is essential to differentiate between exudates and transudates because their underlying causes and treatment approaches may differ significantly. Medical professionals often use various tests, such as fluid analysis, to determine whether a fluid sample is an exudate or transudate.
Thoracoscopy is a surgical procedure in which a thoracoscope, a type of endoscope, is inserted through a small incision between the ribs to examine the lungs and pleural space (the space surrounding the lungs). It allows the surgeon to directly view the chest cavity, take biopsies, and perform various operations. This procedure is often used in the diagnosis and treatment of pleural effusions, lung cancer, and other chest conditions.
The thoracic wall refers to the anatomical structure that surrounds and protects the chest cavity or thorax, which contains the lungs, heart, and other vital organs. It is composed of several components:
1. Skeletal framework: This includes the 12 pairs of ribs, the sternum (breastbone) in the front, and the thoracic vertebrae in the back. The upper seven pairs of ribs are directly attached to the sternum in the front through costal cartilages. The lower five pairs of ribs are not directly connected to the sternum but are joined to the ribs above them.
2. Muscles: The thoracic wall contains several muscles, including the intercostal muscles (located between the ribs), the scalene muscles (at the side and back of the neck), and the serratus anterior muscle (on the sides of the chest). These muscles help in breathing by expanding and contracting the ribcage.
3. Soft tissues: The thoracic wall also contains various soft tissues, such as fascia, nerves, blood vessels, and fat. These structures support the functioning of the thoracic organs and contribute to the overall stability and protection of the chest cavity.
The primary function of the thoracic wall is to protect the vital organs within the chest cavity while allowing for adequate movement during respiration. Additionally, it provides a stable base for the attachment of various muscles involved in upper limb movement and posture.
A lung is a pair of spongy, elastic organs in the chest that work together to enable breathing. They are responsible for taking in oxygen and expelling carbon dioxide through the process of respiration. The left lung has two lobes, while the right lung has three lobes. The lungs are protected by the ribcage and are covered by a double-layered membrane called the pleura. The trachea divides into two bronchi, which further divide into smaller bronchioles, leading to millions of tiny air sacs called alveoli, where the exchange of gases occurs.
Thoracic neoplasms refer to abnormal growths or tumors that develop in the thorax, which is the area of the body that includes the chest and lungs. These neoplasms can be benign (non-cancerous) or malignant (cancerous). Malignant thoracic neoplasms are often referred to as lung cancer, but they can also include other types of cancer such as mesothelioma, thymoma, and esophageal cancer.
Thoracic neoplasms can cause various symptoms depending on their location and size. Common symptoms include coughing, chest pain, shortness of breath, hoarseness, and difficulty swallowing. Treatment options for thoracic neoplasms depend on the type, stage, and location of the tumor, as well as the patient's overall health. Treatment may include surgery, radiation therapy, chemotherapy, targeted therapy, or a combination of these approaches.
Pleurisy
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Pleurisy - Wikipedia
Pleurisy: MedlinePlus Medical Encyclopedia
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Herbal Remedies for Pleurisy, Ayurvedic Treatment - Causes & Symptoms
ICD-9 Code 012.02 -Tuberculous pleurisy bacteriological or histological examination results unknown (at present)- Codify by AAPC
Mycobacterium tuberculosis Rv3615c is a highly immunodominant antigen and specifically induces potent Th1-type immune responses...
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Pleura17
- Pleurisy is an inflammation of the lining of the lungs and chest (the pleura) that leads to chest pain when you take a breath or cough. (medlineplus.gov)
- When you have pleurisy, the normally smooth surfaces lining the lung (the pleura) become rough. (medlineplus.gov)
- Dry Pleurisy - It occurs when the pleura is embedded with fibrous tissue. (planetayurveda.com)
- Diagnosis Bilang isang komplikasyon ng pamamaga ng pleurisy, maaaring mangyari ang mga pagdirikit ng pleura. (paggaling.com)
- Ito ay dahil sa mababaw na paghinga sa isang wet pleurisy, na sanhi ng pleura at ang lung pleura na mahiga sa ibabaw ng bawat isa nang mas matagal kaysa sa dati. (paggaling.com)
- Mga kasingkahulugan sa isang mas malawak na kahulugan Mga kasingkahulugan: pleurisy, pleurisy, pleurisy pamamaga Teknikal na termino: pleuritis Kahulugan Ang pleura ay isang layer ng balat na sumasakop sa mga baga mula sa labas at nilalagay ang ribcage mula sa loob. (paggaling.com)
- Although it is normally smooth, if this lining becomes inflamed, the surface of the pleura becomes rough and irritated, causing the condition known as pleurisy . (rxhealthmed.ca)
- Doctors usually check for a sound caused by pleurisy, often described as a pleural 'rub,' by listening for the creaky, scratchy sound of the pleura rubbing together during a breath. (rxhealthmed.ca)
- Pleurisy is an inflammation of the pleura, which are the tissues that line the chest cavity and the outside of the lungs. (medicalnewstoday.com)
- Pleurisy occurs when the lung pleura become inflamed, rough, and sticky. (medicalnewstoday.com)
- Pleurisy in inflammation of the Pleura, causing pain when Breathing due to loss of lubrication. (medicinetraditions.com)
- Pleurisy is inflammation in the pleura membrane of chest cavity and lungs. (shankongacupuncture.com)
- When a patient has pleurisy , the normally smooth lining of the lung (the pleura ) becomes rough. (wikidoc.org)
- Pleurisy means inflammation of the pleura. (babylonhealth.com)
- In people with pleurisy, the two layers of pleura get inflamed (red and swollen). (lunghealth.ca)
- A wide variety of different factors may make the pleura inflamed resulting in lung pleurisy. (trueremedies.com)
- The last possibility indicates a transition from fibrinous pleurisy to aqueous pleura. (drozkandemirhan.com)
Pleuritis3
- Pleurisy, also known as pleuritis, is inflammation of the membranes that surround the lungs and line the chest cavity (pleurae). (wikipedia.org)
- Pleurisy is also known as Pleuritis. (planetayurveda.com)
- Pleurisy, also known as pleuritis, is a condition in which inflammation appears in the pleural membrane of the chest cavity and lungs. (trueremedies.com)
Inflammation5
- Pleurisy may develop when you have lung inflammation due to infection, such as a viral infection, pneumonia , or tuberculosis . (medlineplus.gov)
- Pancreatitis, an inflammation of the pancreas, can also lead to pleurisy. (rxhealthmed.ca)
- A "primary" pleurisy is an inflammation that happens in the pleural tissues themselves, from a germ that attacked them directly, or from an injury or growth. (lunghealth.ca)
- Coughing or heavy breathing may make your chest hurt and feel sensitive when pleurisy inflammation is present. (safe4cure.com)
- It is particularly valued for its ability to ease symptoms of pleurisy, a painful inflammation of the lining around the lungs. (medikonda.com)
Tuberculous pleurisy9
- ICD-9 code 012.02 for Tuberculous pleurisy bacteriological or histological examination results unknown (at present) is a medical classification as listed by WHO under the range -TUBERCULOSIS (010-018). (aapc.com)
- Tuberculous pleurisy (TP) is one of the most common extra-pulmonary tuberculosis forms. (smj.rs)
- Tuberculous pleurisy occurs when Mycobacterium tuberculosis antigen is released from a ruptured caseous focus into the pleural space causing hyperinflammatory response with a rapid influx of lymphocytes. (smj.rs)
- ADA in pleural punctate is a fast, efficient, and economical way for clarifying the etiology of a pleural effusion such as tuberculous pleurisy and treatment response during the follow up period. (smj.rs)
- Lymphadenitis and tuberculous pleurisy (TP) are the two most common extra-pulmonary forms [ 1 ],[ 2 ]. (smj.rs)
- Diagnosis of TB is challenging in patients with tuberculous pleurisy without a coexisting parenchymal lesion as they are sputum negative. (smj.rs)
- Blind closed pleural biopsy is the most sensitive diagnostic test for tuberculous pleurisy [ 10 ]. (smj.rs)
- alackofinformationabouttheetiology obtainedfromeachparticipantbefore Tuberculous pleurisy ofpleuraleffusionsinmostArabcoun- anyintervieworclinicalexamination Pleuraleffusionswerediagnosedas triesincludingQatar.Onlyafewstudies wasconducted. (who.int)
- 6. patients without lung parenchymal destruction (such as tuberculous pleurisy). (who.int)
Pneumonia5
- If pleurisy is associated with a lung infection (e.g., tuberculosis or pneumonia), then cough, shortness of breath, and fever are common. (rxhealthmed.ca)
- The most common cause of pleurisy is viral infection such as the flu or pneumonia. (lunghealth.ca)
- Generally, both main types often develop in people with high susceptibility to respiratory tract infections, tuberculosis, and pneumonia but dry pleurisy is more common than wet pleurisy. (trueremedies.com)
- Butterfly Weed provides relief for those suffering from pleurisy, as well as pneumonia, asthma and intercostal rheumatism. (earthbeatseeds.com)
- The TB fluid is usually clear but may be opalescent as in case of bacterial pleurisy in para-pneumonic effusion with pneumonia [ 5 ]. (smj.rs)
Symptoms8
- Depending on its cause, pleuritic chest pain may be accompanied by other symptoms: Dry cough Fever and chills Rapid, shallow breathing Shortness of breath Fast heart rate Sore throat followed by pain and swelling in the joints Pleurisy is often associated with complications that affect the pleural space. (wikipedia.org)
- Contact your provider if you have symptoms of pleurisy. (medlineplus.gov)
- In some cases the symptoms of pleurisy and their complication is not clear. (planetayurveda.com)
- People with symptoms of pleurisy should see a doctor. (medicalnewstoday.com)
- These symptoms could be caused by pleurisy, or they could be caused by another disease. (lunghealth.ca)
- If you have these symptoms, or if you think you have pleurisy, see your healthcare provider right away. (lunghealth.ca)
- Pleurisy is treated with medicines, surgery and other methods to lesson symptoms and to treat the cause. (lunghealth.ca)
- The symptoms of dry (fibrinous) pleurisy may be very mild, especially in cases of tuberculosis. (drozkandemirhan.com)
Symptom of pleurisy2
- The defining symptom of pleurisy is a sudden sharp, stabbing, burning or dull pain in the right or left side of the chest during breathing, especially when one inhales and exhales. (wikipedia.org)
- The main symptom of pleurisy is pain in the chest. (medlineplus.gov)
Lungs7
- Sometimes, a collection of fluid around the lungs may occur - this is called wet pleurisy . (rxhealthmed.ca)
- Without the buildup of fluid between the lungs and the lining, the condition is known as dry pleurisy . (rxhealthmed.ca)
- Asbestos fibres, which can stay in the air for a long time and can be inhaled into the lungs, have been linked to pleurisy and lung cancer. (rxhealthmed.ca)
- Pleurisy root was considered by the North American Indians to be one of the "Great White Father's best gifts to the children of nature" because of its specific action for the lungs. (vitalerbs.com)
- To limit the pain of pleurisy, limiting the movement of the lungs may be recommended by your healthcare provider. (lunghealth.ca)
- A condition where a membrane in lungs gets inflamed is called pleurisy. (geoherbal.com)
- Wet pleurisy is the condition in which the fluid exudes from the inflamed tissue into the space between the chest cavity and lungs. (trueremedies.com)
Including viral2
- Pleurisy can be caused by a variety of conditions, including viral or bacterial infections, autoimmune disorders, and pulmonary embolism. (wikipedia.org)
- Many conditions can lead to pleurisy, including viral and bacterial infections. (medicalnewstoday.com)
Skip1
- If you are looking for a natural alternative to medicine that may treat lung pleurisy at home naturally, do not skip this article of 52 Best Home Remedies For Pleurisy Pain. (trueremedies.com)
Responsible for pleurisy2
- A variety of conditions are responsible for pleurisy. (planetayurveda.com)
- The patient's history including any diseased condition or any medication which may be responsible for pleurisy. (planetayurveda.com)
Occurs3
- Pleural effusion involving fibrinous exudates in the fluid may be called fibrinous pleurisy, which sometimes occurs as a later stage of pleurisy. (wikipedia.org)
- Effusive Pleurisy - It occurs when the space of pleural layers filled with extra fluid, this condition is called pleural effusion. (planetayurveda.com)
- Secondly, purulent pleurisy occurs when the space between the pleural layers get filled with dead cells, also known as pus. (trueremedies.com)
Cause pleurisy3
- Environmental factors such as asbestos in the air can also cause pleurisy. (rxhealthmed.ca)
- A clot in a blood vessel of the lung, called a pulmonary embolism, can cause pleurisy. (rxhealthmed.ca)
- A pulmonary embolus (clot in the lung) or lung cancer can also cause pleurisy. (babylonhealth.com)
Purulent Pleurisy3
- Purulent Pleurisy - It is most dangerous type of pleurisy. (planetayurveda.com)
- If the excess fluid of wet pleurisy is infectious with pus formation, it is said to be empyema or purulent pleurisy. (trueremedies.com)
- In fact, wet pleurisy is classified into two types: effusive pleurisy and purulent pleurisy. (trueremedies.com)
Effusive Pleurisy1
- Firstly, effusive pleurisy appears when the space between the pleural layers is filled with exuded fluids. (trueremedies.com)
Cough1
- Pleurisy Root is also a valuable herbal remedy and is an excellent support for respiratory infections as well as chronic infections of the chest with stagnant phlegm and a dry cough. (earthbeatseeds.com)
ASCLEPIAS TUBEROSA2
- Pleurisy Root (Asclepias tuberosa) Dried Root All Natural Herbal Supplements GMO-FREE VEGAN MADE. (secrets.shop)
- Pleurisy root powder, derived from the root of the Asclepias tuberosa plant, has been traditionally used for its medicinal properties. (medikonda.com)
Respiratory infections1
- Early treatment of bacterial respiratory infections can prevent pleurisy. (medlineplus.gov)
Diagnosis1
- Diagnosis of TP should meet the following criteria: acid-fast bacilli (AFB) staining or Löwenstein-Jensen cultures, pleural biopsy culture, and histology (granuloma-like changes in pleural biopsy samples and the exclusion of pleurisy due to other causes) [ 9 ]. (smj.rs)
Butterfly Weed1
- Pleurisy weed is better known in parts of the country as butterfly weed. (dragonherbarium.com)
Complications1
- Pleurisy complications are considered as secondary conditions. (planetayurveda.com)
Herbal1
- Planet Ayurveda provides effective herbal remedies such as Praanrakshak Churna, Curcumin Capsules, Giloy Capsules & Tulsi Capsules for ayurvedic treatment of pleurisy. (planetayurveda.com)
Fluid5
- In some cases of pleurisy, excess fluid builds up in the pleural space. (wikipedia.org)
- Pleurisy can cause fluid to collect inside the chest. (medlineplus.gov)
- In wet pleurisy, this space can fill up with fluid that can get infected. (lunghealth.ca)
- If the fluid remains unchanged by disease, it is said to be dry pleurisy. (trueremedies.com)
- On the contrary, if the fluid heightens abnormally, it is said to be wet pleurisy. (trueremedies.com)
Sharp1
- People with pleurisy tend to experience sharp pain when breathing. (medicalnewstoday.com)
Medicines1
- Herbalist Matthew Wood shares that "Native Americans considered Pleurisy Root to be one of the most valuable medicines in treating lung disorders. (earthbeatseeds.com)
Treatment3
- Treatment depends on the cause of the pleurisy. (medlineplus.gov)
- If the pleurisy is related to an infection, fighting the infection is the most common treatment. (rxhealthmed.ca)
- Click to learn right side chest pain, tightness in chest, left/right side chest pain, pain in middle of chest, pleurisy treatment, anxiety chest pain. (safe4cure.com)
Pain3
- This can relieve the pain of pleurisy. (wikipedia.org)
- Before taking a deep look at how to stop lung pleurisy using home remedies for pleurisy pain, let's learn some basic information about pleurisy first. (trueremedies.com)
- Pleurisy root powder can help reduce chest pain, coughing, and promote the expulsion of mucus. (medikonda.com)
Root Powder1
- Medikonda Nutrients is the Largest Manufacturer, Wholesale Supplier, Bulk Distributor, Exporter of USDA Organic Pleurisy Root Powder. (medikonda.com)
Marshmallow1
- A few of the medicinal plants are ginseng, pleurisy root, snake root, blood root, blue flag and marshmallow. (yourdictionary.com)
Remedies1
- There is a wide range of home remedies which are extremely effective for lung pleurisy. (trueremedies.com)
Descriptor1
- Pleurisy" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (wakehealth.edu)
Diagnostic1
- Doctors use a combination of physical examination and diagnostic tests to diagnose pleurisy. (rxhealthmed.ca)
Common2
- Pleurisy is a common disease that many people suffer from in their lives nowadays. (trueremedies.com)
- After the colonists learned of this remedy from the Native Americans, it became a popular folk remedy for respiratory disorders, particularly pleurisy, hence its common name. (earthbeatseeds.com)
Patients4
- We know to look for the malar rash, but patients can also have small joint arthritis, nephritis, proteinuria, or pleurisy. (medscape.com)
- Aux États-Unis, lorsque le séjour en unité des soins intensifs est prolongé, les patients peuvent être transférés vers un CHSLD. (who.int)
- Dans la pratique, les patients qui ont besoin de soins de longue durée en Turquie sont hospitalisés en unités de soins intensifs. (who.int)
- Une proportion importante des lits réservés aux unités de soins intensifs en Turquie sont utilisés pour les soins de longue durée aux patients atteints de problèmes complexes. (who.int)