Prosopagnosia
Agnosia
Face
Recognition (Psychology)
Pattern Recognition, Visual
Temporal Lobe
Occipital Lobe
Cerebrum
Functional delineation of the human occipito-temporal areas related to face and scene processing. A PET study. (1/65)
By measuring regional cerebral blood flow using PET, we delineated the roles of the occipito-temporal regions activated by faces and scenes. We asked right-handed normal subjects to perform three tasks using facial images as visual stimuli: in the face familiar/unfamiliar discrimination (FF) task, they discriminated the faces of their friends and associates from unfamiliar ones; in the face direction discrimination (FD) task, they discriminated the direction of each unfamiliar face; in the dot location discrimination (DL) task, they discriminated the location of a red dot on a scrambled face. The activity in each task was compared with that in the control fixation (CF) task, in which they fixated on the centre of a display without visual stimuli. The DL task activated the occipital cortices and posterior fusiform gyri bilaterally. During the FD task, the activation extended anteriorly in the right fusiform gyrus and laterally to the right inferior temporal cortex. The FF task further activated the right temporal pole. To examine whether the activation due to faces was face-specific, we used a scene familiar/unfamiliar discrimination (SF) task, in which the subjects discriminated familiar scenes from unfamiliar ones. Our results suggest that (i) the occipital cortices and posterior fusiform gyri non-selectively respond to faces, scrambled faces and scenes, and are involved mainly in the extraction of physical features of complex visual images; (ii) the right inferior temporal/fusiform gyrus responds selectively to faces but not to non-face stimuli and is involved in the visual processing related to face perception, whereas the bilateral parahippocampal gyri and parieto-occipital junctions respond selectively to scenes and are involved in processing related to scene perception; and (iii) the right temporal pole is activated during the discrimination of familiar faces and scenes from unfamiliar ones, and is probably involved in the recognition of familiar objects. (+info)Selective sparing of face learning in a global amnesic patient. (2/65)
OBJECTIVE: To test the hypothesis that visual memory for faces can be dissociated from visual memory for topographical material. METHOD: A patient who developed a global amnesic syndrome after acute carbon monoxide poisoning is described. A neuroradiological examination documented severe bilateral atrophy of the hippocampi. RESULTS: Despite a severe anterograde memory disorder involving verbal information, abstract figures, concrete objects, topographical scenes, and spatial information, the patient was still able to learn previously unknown human faces at a normal (and, in some cases, at a higher) rate. CONCLUSIONS: Together with previous neuropsychological evidence documenting selective sparing of topographical learning in otherwise amnesic patients, this case is indicative of the fact that the neural circuits involved in face recognition are distinct from those involved in the recognition of other visuoperceptual material (for example, topographical scenes). (+info)Face recognition in age related macular degeneration: perceived disability, measured disability, and performance with a bioptic device. (3/65)
AIMS: (1) To explore the relation between performance on tasks of familiar face recognition (FFR) and face expression difference discrimination (FED) with both perceived disability in face recognition and clinical measures of visual function in subjects with age related macular degeneration (AMD). (2) To quantify the gain in performance for face recognition tasks when subjects use a bioptic telescopic low vision device. METHODS: 30 subjects with AMD (age range 66-90 years; visual acuity 0.4-1.4 logMAR) were recruited for the study. Perceived (self rated) disability in face recognition was assessed by an eight item questionnaire covering a range of issues relating to face recognition. Visual functions measured were distance visual acuity (ETDRS logMAR charts), continuous text reading acuity (MNRead charts), contrast sensitivity (Pelli-Robson chart), and colour vision (large panel D-15). In the FFR task, images of famous people had to be identified. FED was assessed by a forced choice test where subjects had to decide which one of four images showed a different facial expression. These tasks were repeated with subjects using a bioptic device. RESULTS: Overall perceived disability in face recognition did not correlate with performance on either task, although a specific item on difficulty recognising familiar faces did correlate with FFR (r = 0.49, p<0.05). FFR performance was most closely related to distance acuity (r = -0.69, p<0.001), while FED performance was most closely related to continuous text reading acuity (r = -0.79, p<0.001). In multiple regression, neither contrast sensitivity nor colour vision significantly increased the explained variance. When using a bioptic telescope, FFR performance improved in 86% of subjects (median gain = 49%; p<0.001), while FED performance increased in 79% of subjects (median gain = 50%; p<0.01). CONCLUSION: Distance and reading visual acuity are closely associated with measured task performance in FFR and FED. A bioptic low vision device can offer a significant improvement in performance for face recognition tasks, and may be useful in reducing the handicap associated with this disability. There is, however, little evidence for a correlation between self rated difficulty in face recognition and measured performance for either task. Further work is needed to explore the complex relation between the perception of disability and measured performance. (+info)Slowly progressive defect in recognition of familiar people in a patient with right anterior temporal atrophy. (4/65)
We report the case of a patient (C.O.) who showed a selective defect in the recognition of familiar people, with very mild disease progression during a period of 30 months resulting from focal atrophy of the right temporal lobe. On formal neuropsychological testing, C.O. obtained high scores on tests of general intelligence, episodic memory, language, executive functions, selective attention, visual recognition and visual-spatial abilities. On more specific tasks of familiar and unfamiliar face recognition, C.O. scored above the controls' means on perceptual tests, but obtained highly pathological results on amnesic-associative tests. His disorder of recognition of familiar people was not due to loss of person-specific information, since he obtained highly abnormal naming scores when presented with photographs of famous people but borderline or mildly abnormal scores in a tasks in which he had to name celebrities from verbal definitions. On the other hand, C.O.'s recognition disorder could not be considered to be a form of 'associative prosopagnosia' since a similar defect was observed when he was requested to access information about famous persons through their voice rather than their face. Two alternative interpretations are advanced to explain C.O.'s inability to access his relatively spared person-specific knowledge not only through the person's face but also through the person's voice. The first hypothesis is that, before accessing the person-specific information, unimodal recognition channels must converge into a multimodal, non-verbal person-recognition system and that the right anterior temporal cortices play a crucial role in this integrative activity. The second hypothesis is that the face-recognition units have privileged access to person-specific semantic knowledge and that other recognition subsystems require coactivation of the face-recognition units in order to access person-specific semantic information. (+info)Hyperfamiliarity for unknown faces after left lateral temporo-occipital venous infarction: a double dissociation with prosopagnosia. (5/65)
Right hemisphere dominance in face processing is well established and unilateral right inferior temporo-occipital damage can result in prosopagnosia. Here, we describe a 21-year-old right-handed woman with acute impairment in face recognition that selectively concerned unfamiliar faces, following a focal left lateral temporo-occipital venous infarct. She was severely impaired in discerning that unknown people seen in everyday life were unfamiliar, although she had no difficulty recognizing familiar people. Thus, she had no prosopagnosia, but abnormal 'hyperfamiliarity' for unknown faces. Her difficulty was not accompanied by delusions or deficits in discrimination, identification or memory for faces. Standard neuropsychological testing showed that her recognition of familiar faces was entirely normal. By contrast, her sense of personally knowing faces was severely impaired when unknown faces evoked weak signals of familiarity based on spurious cues, to the extent that she would misattribute fame to faces that were unknown but to which she had been incidentally exposed on a prior occasion. Priming experiments also revealed that, unlike normal subjects, she made familiarity judgements without accessing semantic identity representations. Moreover, in face recognition tests, she generally showed bias in that she relied more on right-hemisphere strategies to identify global traits and less on left-hemisphere processes compared with healthy subjects. This case provides novel evidence for a differential contribution of the two hemispheres to face recognition. Hyperfamiliarity for unknown faces might arise from an imbalance between reciprocal hemispheric functions in face recognition, with relative hypoactivation of left hemisphere processes but hyperactivation of right-hemisphere processes for retrieving stored associations about people, linking seen faces to representations of affective and personal relevance. Hence, abnormal bias in attributing some personal meaning to unknown faces could be evoked by spurious signals of familiarity based on irrelevant affective associations in the right hemisphere. (+info)A network of occipito-temporal face-sensitive areas besides the right middle fusiform gyrus is necessary for normal face processing. (6/65)
Neuroimaging studies have identified at least two bilateral areas of the visual extrastriate cortex that respond more to pictures of faces than objects in normal human subjects in the middle fusiform gyrus [the 'fusiform face area' (FFA)] and, more posteriorly, in the inferior occipital cortex ['occipital face area' (OFA)], with a right hemisphere dominance. However, it is not yet clear how these regions interact which each other and whether they are all necessary for normal face perception. It has been proposed that the right hemisphere FFA acts as an isolated ('modular') processing system for faces or that this region receives its face-sensitive inputs from the OFA in a feedforward hierarchical model of face processing. To test these proposals, we report a detailed neuropsychological investigation combined with a neuroimaging study of a patient presenting a deficit restricted to face perception, consecutive to bilateral occipito-temporal lesions. Due to the asymmetry of the lesions, the left middle fusiform gyrus and the right inferior occipital cortex were damaged but the right middle fusiform gyrus was structurally intact. Using functional MRI, we disclosed a normal activation of the right FFA in response to faces in the patient despite the absence of any feedforward inputs from the right OFA, located in a damaged area of cortex. Together, these findings show that the integrity of the right OFA is necessary for normal face perception and suggest that the face-sensitive responses observed at this level in normal subjects may arise from feedback connections from the right FFA. In agreement with the current literature on the anatomical basis of prosopagnosia, it is suggested that the FFA and OFA in the right hemisphere and their re-entrant integration are necessary for normal face processing. (+info)Impaired configurational processing in a case of progressive prosopagnosia associated with predominant right temporal lobe atrophy. (7/65)
F.G., a 71-year-old right-handed man, presented with a slowly progressive deterioration in his ability to recognize faces of familiar and famous persons, contrasting with the relative preservation of other cognitive domains. His primary face perception skills were intact. Along with his face-recognition deficit, F.G. also exhibited a mild visual agnosia. A more detailed analysis of his performance on visuoperceptual tests revealed a selective deficit in retrieving the configurational representation of complex visual entities and an over-reliance on analysing individual features. Quantitative volumetric measurements of his temporal lobe structures showed a prevalent atrophy of the right fusiform gyrus and parahippocampal cortex. The results of the present study suggest that a right temporal variant of frontotemporal lobar degeneration may be characterized over a period of several years by an impaired configurational processing of visually complex entities in the absence of any semantic deficit. (+info)A modulatory role for facial expressions in prosopagnosia. (8/65)
Brain-damaged patients experience difficulties in recognizing a face (prosopagnosics), but they can still recognize its expression. The dissociation between these two face-related skills has served as a keystone of models of face processing. We now report that the presence of a facial expression can influence face identification. For normal viewers, the presence of a facial expression influences performance negatively, whereas for prosopagnosic patients, it improves performance dramatically. Accordingly, although prosopagnosic patients show a failure to process the facial configuration in the interest of face identification, that ability returns when the face shows an emotional expression. Accompanying brain-imaging results indicate activation in brain areas (amygdala, superior temporal sulcus, parietal cortex) outside the occipitotemporal areas normally activated for face identification and lesioned in these patients. This finding suggests a modulatory role of these areas in face identification that is independent of occipitotemporal face areas. (+info)Prosopagnosia is a neurological disorder characterized by the inability to recognize or remember faces, even those of familiar people such as family members and friends. This condition often results from brain damage, particularly to the fusiform gyrus area located in the temporal lobe, which is responsible for facial recognition and memory.
Individuals with prosopagnosia may have difficulty distinguishing between faces, sometimes even mistaking their own reflection or confusing family members with strangers. However, they can still recognize people through other means, such as voice, hairstyle, clothing, or gait. Prosopagnosia can be congenital (present at birth) or acquired due to brain injury or disease.
There are two main types of prosopagnosia: developmental (or congenital) and acquired. Developmental prosopagnosia is present from birth and tends to run in families, suggesting a genetic component. Acquired prosopagnosia occurs after brain damage due to stroke, trauma, or degenerative diseases like dementia.
Prosopagnosia can significantly impact social interactions and relationships, causing distress and isolation for those affected. Currently, there is no cure for this condition; however, various strategies and techniques can help individuals with prosopagnosia cope and improve their face recognition abilities.
Agnosia is a medical term that refers to the inability to recognize or comprehend the meaning or significance of sensory stimuli, even though the specific senses themselves are intact. It is a higher-level cognitive disorder, caused by damage to certain areas of the brain that are responsible for processing and interpreting information from our senses.
There are different types of agnosia, depending on which sense is affected:
* Visual agnosia: The inability to recognize or identify objects, faces, or shapes based on visual input.
* Auditory agnosia: The inability to understand spoken language or recognize sounds, even though hearing is intact.
* Tactile agnosia: The inability to recognize objects by touch, despite normal tactile sensation.
* Olfactory and gustatory agnosia: The inability to identify smells or tastes, respectively, even though the senses of smell and taste are functioning normally.
Agnosia can result from various causes, including stroke, brain injury, infection, degenerative diseases, or tumors that damage specific areas of the brain involved in sensory processing and interpretation. Treatment for agnosia typically focuses on rehabilitation and compensation strategies to help individuals adapt to their deficits and improve their quality of life.
In medical terms, the face refers to the front part of the head that is distinguished by the presence of the eyes, nose, and mouth. It includes the bones of the skull (frontal bone, maxilla, zygoma, nasal bones, lacrimal bones, palatine bones, inferior nasal conchae, and mandible), muscles, nerves, blood vessels, skin, and other soft tissues. The face plays a crucial role in various functions such as breathing, eating, drinking, speaking, seeing, smelling, and expressing emotions. It also serves as an important identifier for individuals, allowing them to be recognized by others.
Visual pattern recognition is the ability to identify and interpret patterns in visual information. In a medical context, it often refers to the process by which healthcare professionals recognize and diagnose medical conditions based on visible signs or symptoms. This can involve recognizing the characteristic appearance of a rash, wound, or other physical feature associated with a particular disease or condition. It may also involve recognizing patterns in medical images such as X-rays, CT scans, or MRIs.
In the field of radiology, for example, visual pattern recognition is a critical skill. Radiologists are trained to recognize the typical appearances of various diseases and conditions in medical images. This allows them to make accurate diagnoses based on the patterns they see. Similarly, dermatologists use visual pattern recognition to identify skin abnormalities and diseases based on the appearance of rashes, lesions, or other skin changes.
Overall, visual pattern recognition is an essential skill in many areas of medicine, allowing healthcare professionals to quickly and accurately diagnose medical conditions based on visible signs and symptoms.
The temporal lobe is one of the four main lobes of the cerebral cortex in the brain, located on each side of the head roughly level with the ears. It plays a major role in auditory processing, memory, and emotion. The temporal lobe contains several key structures including the primary auditory cortex, which is responsible for analyzing sounds, and the hippocampus, which is crucial for forming new memories. Damage to the temporal lobe can result in various neurological symptoms such as hearing loss, memory impairment, and changes in emotional behavior.
The occipital lobe is the portion of the cerebral cortex that lies at the back of the brain (posteriorly) and is primarily involved in visual processing. It contains areas that are responsible for the interpretation and integration of visual stimuli, including color, form, movement, and recognition of objects. The occipital lobe is divided into several regions, such as the primary visual cortex (V1), secondary visual cortex (V2 to V5), and the visual association cortex, which work together to process different aspects of visual information. Damage to the occipital lobe can lead to various visual deficits, including blindness or partial loss of vision, known as a visual field cut.
The cerebrum is the largest part of the brain, located in the frontal part of the skull. It is divided into two hemispheres, right and left, which are connected by a band of nerve fibers called the corpus callosum. The cerebrum is responsible for higher cognitive functions such as thinking, learning, memory, language, perception, and consciousness.
The outer layer of the cerebrum is called the cerebral cortex, which is made up of gray matter containing billions of neurons. This region is responsible for processing sensory information, generating motor commands, and performing higher-level cognitive functions. The cerebrum also contains several subcortical structures such as the thalamus, hypothalamus, hippocampus, and amygdala, which play important roles in various brain functions.
Damage to different parts of the cerebrum can result in a range of neurological symptoms, depending on the location and severity of the injury. For example, damage to the left hemisphere may affect language function, while damage to the right hemisphere may affect spatial perception and visual-spatial skills.
Neuropsychological tests are a type of psychological assessment that measures cognitive functions, such as attention, memory, language, problem-solving, and perception. These tests are used to help diagnose and understand the cognitive impact of neurological conditions, including dementia, traumatic brain injury, stroke, Parkinson's disease, and other disorders that affect the brain.
The tests are typically administered by a trained neuropsychologist and can take several hours to complete. They may involve paper-and-pencil tasks, computerized tasks, or interactive activities. The results of the tests are compared to normative data to help identify any areas of cognitive weakness or strength.
Neuropsychological testing can provide valuable information for treatment planning, rehabilitation, and assessing response to treatment. It can also be used in research to better understand the neural basis of cognition and the impact of neurological conditions on cognitive function.
Prosopagnosia
Head injury
Inferior temporal gyrus
Tamy Glauser
Inferior longitudinal fasciculus
Carme Junyent
Prosopamnesia
Memory and social interactions
Covert facial recognition
Robert Gascoyne-Cecil, 3rd Marquess of Salisbury
Kim Seon-ho
Stephen Fry
Phonagnosia
Super recogniser
Oliver Sacks
Visual agnosia
Steve Wozniak
Jim Woodring
Agnosia
Chuck Close
Lee Min-ki
Fred W. Mast
Brad Pitt
Humpty Dumpty
Cerebral achromatopsia
999: Nine Hours, Nine Persons, Nine Doors
Face perception
John Hickenlooper
Object recognition (cognitive science)
Syndrome of subjective doubles
Prosopagnosia - Wikipedia
Prosopagnosia | National Institute of Neurological Disorders and Stroke
prosopagnosia Archives - ScienceAGoGo
Prosopagnosia - EyeWiki
OMG! Shenaz Treasury reveals she has prosopagnosia, says 'can't recognise faces' | Hindi Movie News - Bollywood - Times of India
prosopagnosia Archives - Nethervoice
Posterior Cerebral Artery Stroke: Background, Anatomy, Pathophysiology
Congenital prosopagnosia is associated with a genetic variation in the oxytocin receptor (OXTR) gene: An exploratory study -...
Prosopagnosia Archives | Big Green Pen
prosopagnosia | Brifly - News Simplified, Brifly
Prosopagnosia - An overview | Capsule Health
Prosopagnosia - The Foundation for Art & Healing
Glenn Stok on HubPages
Hospital Records
A novel PSEN1 (S230N) mutation causing early-onset Alzheimer's Disease associated with prosopagnosia, hoarding, and...
Dr Ashok Jansari on BBC The One Show talking about Prosopagnosia - Ashok Jansari
Memory | MedlinePlus
neurology - Why does prosopagnosia (face blindness) only affect recognition of faces? - Medical Sciences Stack Exchange
Bornstein B[au] - Search Results - PubMed
Jim Woodring - Wikipedia
Richard Russell - Research
I never forget a face! | BPS
Neuroscience
November | 2010 | Adam Blatner
Dame Jane Goodall | Academy of Achievement
Electrical Stimulation of Human Fusiform Face-Selective Regions Distorts Face Perception | Journal of Neuroscience
Frontiers | Worth a Glance: Using Eye Movements to Investigate the Cognitive Neuroscience of Memory
Developmental9
- Developmental prosopagnosia (DP), also called congenital prosopagnosia (CP), is a face-recognition deficit that is lifelong, manifesting in early childhood, and that cannot be attributed to acquired brain damage. (wikipedia.org)
- Further, something like dementia can impact facial recognition which is a subclass called developmental prosopagnosia, and that obviously carries a host of other impairments. (stackexchange.com)
- Prosopagnosia is a disorder that can be caused by trauma, genetics from birth, and developmental conditions. (stackexchange.com)
- 2003). Developmental or congenital prosopagnosia may be the result of prenatal brain damage, or the consequence of a genetic inheritance (Duchaine et al. (bps.org.uk)
- 2010). The prevalence of developmental prosopagnosia in the population may be 2 per cent (Kennerknecht et al. (bps.org.uk)
- Two recent studies show this is a common experience for people with a brain disorder called " developmental prosopagnosia " - or as it is more informally known, faceblindness. (edgehill.ac.uk)
- Corrina talks about her and the group's current research on the under-reported but highly prevalent disorder - developmental prosopagnosia. (tu-dresden.de)
- The talk will also highlight evidence for a neurobiological explanation of developmental prosopagnosia, an inherited, congenital impairment in the visual recognition of familiar faces in the absence of any structural brain damage. (nih.gov)
- Developmental Prosopagnosia: a window to content-specific processing. (bvsalud.org)
Congenital3
- In congenital prosopagnosia, the individual never adequately develops the ability to recognize faces. (wikipedia.org)
- Congenital prosopagnosia appears to run in families, which makes it likely to be the result of a genetic mutation or deletion. (nih.gov)
- Face-recognition deficits, referred to with the term prosopagnosia (i.e., face blindness), may manifest during development in the absence of any brain injury (from here the term congenital prosopagnosia, CP). (nih.gov)
Type of prosopagnosia2
- Additionally, an unusual type of prosopagnosia, known as "mirror-image" prosopagnosia, presents with the patient not recognizing their own face in the mirror or photographs. (aao.org)
- This type of prosopagnosia is lifelong, in contrast to "acquired prosopagnosia" which can develop after a brain injury. (edgehill.ac.uk)
Diagnose prosopagnosia2
- Because there are no proven diagnostic methods, one often relies on the clinical history to help diagnose prosopagnosia. (aao.org)
- So, how should we diagnose prosopagnosia instead? (edgehill.ac.uk)
Associative prosopagnosia6
- It is subdivided into apperceptive and associative prosopagnosia. (wikipedia.org)
- Associative prosopagnosia has typically been used to describe cases of acquired prosopagnosia with spared perceptual processes but impaired links between early face perception processes and the semantic information humans hold about people in our memories. (wikipedia.org)
- Right anterior temporal regions may also play a critical role in associative prosopagnosia. (wikipedia.org)
- Associative prosopagnosia is thought to be due to impaired functioning of the parahippocampal gyrus. (wikipedia.org)
- Associative prosopagnosia is defined as inability to recognize or apply any meaning to the face, despite perceiving it. (aao.org)
- [8] Because the BFRT and CFMT are both evaluating facial matching but not recognition, these tests are better served to evaluate associative prosopagnosia, as these patients are still able to perceive faces without difficulty. (aao.org)
Apperceptive prosopagnosia5
- Apperceptive prosopagnosia has typically been used to describe cases of acquired prosopagnosia with some of the earliest processes in the face perception system. (wikipedia.org)
- The brain areas thought to play a critical role in apperceptive prosopagnosia are right occipital temporal regions. (wikipedia.org)
- Apperceptive prosopagnosia is believed to be associated with impaired fusiform gyrus. (wikipedia.org)
- Apperceptive prosopagnosia is defined as the inability to even perceive and cognitively process the face. (aao.org)
- A patient with apperceptive prosopagnosia will claim they are unable to even perceive a face at all, let alone recognize who the face belongs to. (aao.org)
Super-recognisers1
- In fact, using the Cambridge Face Memory Test, the researchers found that their 'super-recognisers' performed an equal number of standard deviations above the population mean (2 SD), as those suffering prosopagnosia perform below the mean. (bps.org.uk)
Fusiform gyrus3
- The brain area usually associated with prosopagnosia is the fusiform gyrus, which activates specifically in response to faces. (wikipedia.org)
- Prosopagnosia is thought to be the result of abnormalities, damage, or impairment in the right fusiform gyrus, a fold in the brain that appears to coordinate the neural systems that control facial perception and memory. (stackexchange.com)
- Prosopagnosia can be caused by accidents that damage parts of the brain like the fusiform gyrus - the core part of a broad network of regions involved in processing images of faces. (discovermagazine.com)
Disorder6
- Prosopagnosia (also known as face blindness or facial agnosia) is a neurological disorder characterized by the inability to recognize faces. (nih.gov)
- Prosopagnosia can be socially debilitating as individuals with the disorder often have difficulty recognizing family members and close friends. (nih.gov)
- Prosopagnosia, also known as face blindness, refers to the neuro-ophthalmic disorder in which a patient has difficulty perceiving or recognizing faces. (aao.org)
- In a new article for The Conversation, Senior Lecturer in Psychology Dr Edwin Burns explores the brain disorder prosopagnosia, also known as faceblindness. (edgehill.ac.uk)
- Acquired prosopagnosia as a face-specific disorder: Ruling out the general visual similarity account. (mpg.de)
- The traditional view of this face blindness disorder-prosopagnosia in scientific parlance-has held that it arises from deficits in visual perception. (nih.gov)
Facial4
- In addition, apperceptive sub-types of prosopagnosia struggle recognizing facial emotion. (wikipedia.org)
- Some evidence has been shown that compensatory treatments such as verbalizing facial details may be effective in retraining these patients to enhance facial recognition, but due to the limited evidence, this remains a controversial tool for the treatment of prosopagnosia. (aao.org)
- Brad Pitt discusses his experience with facial blindness, a rare condition also known as prosopagnosia. (briflynews.com)
- If prosopagnosia does arise from damage to the fusiform gynus, why are the effects merely limited to facial perception? (stackexchange.com)
Recognise faces1
- IAmA Person living with Prosopagnosia, which basically means I am unable to recognise faces. (capsulehealth.one)
Faces9
- For those with prosopagnosia, the method for recognizing faces depends on the less sensitive object-recognition system. (wikipedia.org)
- Depending upon the degree of impairment, some people with prosopagnosia may only have difficulty recognizing familiar faces, while others will be unable to discriminate between unknown faces. (nih.gov)
- Why does prosopagnosia (face blindness) only affect recognition of faces? (stackexchange.com)
- I read a short story involving prosopagnosia ( Wikipedia ), which harms or fully removes a person's ability to remember and recognize faces, and I've since been doing reading to better understand the condition. (stackexchange.com)
- Acquired prosopagnosia occurs following brain damage, with sufferers losing the ability to recognise familiar faces, while sometimes retaining object-recognition ability (e.g. (bps.org.uk)
- However, alternative 'domain-general' theories suggest that prosopagnosia represents a breakdown in the processes associated with the recognition of objects for which the sufferer possesses expertise, in this case faces (e.g. (bps.org.uk)
- She did well in school despite an unusual neurological condition, known as prosopagnosia, which makes it difficult to recognize faces. (achievement.org)
- They have a condition called prosopagnosia , or face blindness, which makes them incredibly bad at recognising faces , despite their normal eyesight, memory, intelligence, and ability to recognise other objects. (discovermagazine.com)
- That's consistent with earlier data, which suggests that the right half of the brain is more important than the left when it comes to recognising faces and that damage to the ILF as a result of multiple sclerosis can induce symptoms of prosopagnosia. (discovermagazine.com)
Sufferers1
- It sounds like a nightmare, but this is the reality that sufferers of prosopagnosia (PA) - otherwise known as face blindness - have to contend with everyday. (scienceagogo.com)
Diagnosis4
- The diagnosis of prosopagnosia is made clinically. (aao.org)
- I first realized I may have a form of Prosopagnosia when I was receiving a diagnosis of autism. (artandhealing.org)
- However, the Harvard research and that by my own lab found that many prosopagnosia cases would not meet the criteria currently required for a diagnosis. (edgehill.ac.uk)
- Why do people with prosopagnosia perform too well on medical tests to get a diagnosis? (edgehill.ac.uk)
Neural1
- They are the equivalent of cables that link a network of computers together and in people born with prosopagnosia, these neural cables are shredded or missing, even though the individual machines work just fine. (discovermagazine.com)
Deficits1
- Although no tests were reported to confirm this, it would be consistent with the face-specific deficits in prosopagnosia. (bps.org.uk)
People2
- Acquired prosopagnosia with spared perceptual processes but impaired links between early face perception processes and the semantic information we hold about people in our memories. (capsulehealth.one)
- The Harvard lab proposes we should diagnose people with prosopagnosia if they score in the bottom 16% of the general population on the two face recognition tests. (edgehill.ac.uk)
Lesion3
- [5] When there is a lesion that occurs at some level of this processing, the patient complains of prosopagnosia. (aao.org)
- However, if a patient with prosopagnosia presents with left-sided homonymous hemianopsia, there should be concern for a right-sided occipital lesion. (aao.org)
- 11. Looking beyond the face area: lesion network mapping of prosopagnosia. (nih.gov)
Disorders2
- Consider participating in a clinical trial so clinicians and scientists can learn more about prosopagnosia and related disorders. (nih.gov)
- See also our prosopagnosia page for more information about disorders of face perception. (brunel.ac.uk)
Develops1
- If his results pan out, they could also give researchers an avenue for understanding how prosopagnosia develops. (discovermagazine.com)
Recognition1
- The opposite of prosopagnosia is the skill of superior face recognition ability. (wikipedia.org)
Autism1
- Some degree of prosopagnosia is often present in children with autism and Asperger's syndrome and may be the cause of impaired social development. (nih.gov)
Condition1
- For the moment, if you want to find out more about prosopagnosia, I highly recommend Cecilia Burman's excellent site on what it's like to live with the condition. (discovermagazine.com)
Processes1
- Acquired prosopagnosia with some of the earliest processes in the face perception system. (capsulehealth.one)
Result1
- Prosopagnosia can result from stroke, traumatic brain injury (TBI), or certain neurodegenerative diseases. (nih.gov)
Understand1
- In order to understand what Prosopagnosia means to me and to try and live my best life, I developed a series of coping mechanisms and strategies. (artandhealing.org)
Memory2
- Prosopagnosia is not related to memory dysfunction, memory loss, impaired vision, or learning disabilities. (nih.gov)
- An animator's experience of Prosopagnosia is explored through the contents of a memory box. (artandhealing.org)
Area1
- Animation of the fusiform area , the area damaged in prosopagnosia. (capsulehealth.one)
Index1
- These are taken from a questionnaire called the prosopagnosia index , first developed by a British research group in 2015. (edgehill.ac.uk)
Results1
- Acquired prosopagnosia results from occipito-temporal lobe damage and is most often found in adults. (wikipedia.org)