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*  Fields Institute Slides - Sarma
Limitations of Thalamic Relay: Insights into Motor Signal ... fields institute slides sarma home about us people contacts programs activities proposals applications honours prizes fellowships publications resources and facilities search lecture slides october performance limitations of thalamic relay insights into motor signal processing parkinson s disease and deep brain stimulation sridevi sarma johns hopkins university this web presentation contains the slides of a lecture given at the fields institute on may as part of the parkinson s disease workshop you may browse the slides in the presentation a browser capable of displaying png graphics is required or you may download a higher resolution printer ready version in pdf format requires acrobat reader shown slide large size previous slide next slide switch to small size...;large
*  Infantile striato thalamic degeneration | Disease | News & Events | Office of Rare Diseases Research
Infantile striato thalamic degeneration. Disease. News Events. ... Office of Rare Diseases Research ORDR-NCATS. Your browser does ... ORDR Office of Rare Diseases Research. Menu Close. Diseases....
*  Thalamic syndrome
Thalamic syndrome. Advocacy and Support ... the condition "Thalamic syndrome" for support, advocacy or ... the condition "Thalamic syndrome" for support, advocacy or...
*  Right Thalamic Bleed - Stroke - MedHelp
Right Thalamic Bleed - Stroke - MedHelp. Full Site. ... > Stroke > Right Thalamic Bleed Aa. She was found unconscious in...
*  Surgical Procedures for Parkinson's Disease | Blake Medical Center | Bradenton, FL
follow-up of subthalamic deep brain stimulation in Parkinson's ... -term follow-up of thalamic deep brain stimulation for essential...
*  Central post-stroke pain | Disease | Your Questions Answered | Office of Rare Diseases Research (ORD
Office of Rare Diseases Research ORDR-NCATS. Your browser does ... ORDR Office of Rare Diseases Research. Menu Close. Diseases. ... Genetic and Rare Diseases Information Center GARD. About GARD. ... Central post-stroke pain. Disease. Your Questions Answered. Office of Rare Diseases Research ORDR-NCATS. Your browser does not support javascript: Search for gard here, Search for news-and-events here. Department of Health Human Services. National Institutes of Health. NCATS. NCATS. Recursos en espa ol ORDR Home Help FAQ Contact Us. Search. Search. ORDR Office of Rare Diseases Research. Diseases. Genetic and Rare Diseases Information Center GARD. About GARD. Tips for the Undiagnosed. Contact GARD. Feedback for GARD. Enfermedades en espa ol. Search for Genetic Rare Diseases Search. Enfermedades en espa ol Browse Diseases A. View Diseases Categories Browse Diseases with FDA Approved Medical Products. Resources. Organization Search. Rare Diseases Resources. Genetics Resources. Genetic Testing and Treatment. R...
*  Project: Probing disrupted cortico-thalamic interactions in autism spectrum disorders
disrupted cortico-thalamic interactions in autism spectrum ... disrupted cortico-thalamic interactions in autism spectrum ... neurodevelopmental diseases such as Rett Syndrome and autism...
*  Frontiers | Orexin antagonists for neuropsychiatric disease: progress and...
2011). Orexins/hypocretins act in the posterior paraventricular thalamic nucleus during ... Orexin antagonists for neuropsychiatric disease: progress and potential pitfalls. Jiann ... Orexin antagonists for neuropsychiatric disease: progress and potential pitfalls. Front. ... or suppress dysregulated orexin function in neuropsychiatric and neurological disease ......
*  Memory in multiple sclerosis is linked to glutamate concentration in grey...
... thalamic and cingulate regions in RRMS patients. As in other neuropsychiatric diseases,29 ... Entering thalamic MTR and thalamic DIR lesions to the model revealed that thalamic [Glu] ... Thalamic atrophy and cognition in multiple sclerosis. Neurology 2007;69:1213-23. ... Considering the thalamic and hippocampal regions, no group differences in glutamate and ......
*  Plus it
TBE infection is characterized by a biphasic course of the disease in 74% of patients, ... 7 but enhancement of a thalamic lesion was described in one patient5 and slight ......
*  The Interface of Mechanics and Nociception in Joint Pathophysiology: Insights...
6). There is similar evidence for thalamic reorganization in TMJ pain in humans [82] and ... Clinically, TMJ disorder presents with many different disease pathologies, including pain ... A Disease of the Joint as an Organ," Arthritis Rheumatol., 64(6), pp. 1697-1707. [ ... A Disease of the Joint as an Organ," Arthritis Rheumatol., 64(6), pp. 1697-1707. [ ......
Labels: benefits CHRONIC DISEASE, BRAINFOG, CDC, CFS/ME, CHRONIC DISEASE, DIAGNOSING, ... nerve conduction which stimulates a plastic response on the cortical side of the thalamic ... celiac disease or no gastrointestinal disease at all.. The researchers found that the ... or life threatening disease". # Prof Racaniello: the CDC has stumbled when tackling CFS, ......
*  detlaphiltdic: February 2008
In an ever-lengthening list of recently reported "miracle cures" for Alzheimer's disease ... as well as in the ventral thalamic nuclei and the somatosensory and motor cortices, ... having normal olfaction and no brain diseases, were recruited. During fMRI, a women's ......

No data available that match "Thalamic Diseases"

(1/96) An 18-mer peptide fragment of prosaposin ameliorates place navigation disability, cortical infarction, and retrograde thalamic degeneration in rats with focal cerebral ischemia.

It was previously reported that prosaposin possesses neurotrophic activity that is ascribed to an 18-mer peptide comprising the hydrophilic sequence of the rat saposin C domain. To evaluate the effect of the 18-mer peptide on ischemic neuronal damage, the peptide was infused in the left lateral ventricle immediately after occlusion of the left middle cerebral artery (MCA) in stroke-prone spontaneously hypertensive (SP-SH) rats. The treatment ameliorated the ischemia-induced space navigation disability and cortical infarction and prevented secondary thalamic degeneration in a dose-dependent manner. In culture experiments, treatment with the 18-mer peptide attenuated free radical-induced neuronal injury at low concentrations (0.002 to 2 pg/mL), and the peptide at higher concentrations (0.2 to 20 ng/mL) protected neurons against hypoxic insult. Furthermore, a saposin C fragment comprising the 18-mer peptide bound to synaptosomal fractions of the cerebral cortex, and this binding decreased at the 1st day after MCA occlusion and recovered to the preischemic level at the 7th day after ischemia. These findings suggest that the 18-mer peptide ameliorates neuronal damage in vivo and in vitro through binding to the functional receptor, although the cDNA encoding prosaposin receptor has not been determined yet.  (+info)

(2/96) Bilateral enhancing thalamic lesions in a 10 year old boy: case report.

A young boy presented with monoparesis of the left arm. MRI disclosed bilateral enhancing thalamic lesions. Biopsy results and subsequent clinical history were most compatible with postinfectious or acute disseminated encephalomyelitis. This represents one of the first cases of acute disseminated encephalomyelitis affecting the thalami, established by biopsy. This uncommon disease entity is reviewed and how it may affect the deep grey matter is described.  (+info)

(3/96) MR spectroscopy of bilateral thalamic gliomas.

This study reports the MR spectroscopic patterns of two patients with bithalamic glioma. In one patient, phosphorus (31P) MR spectroscopy was performed. In both patients, the proton MR spectroscopic scans showed an increased creatine-phosphocreatine peak in the tumor. In the patient who underwent 31P-MR spectroscopy, an increased phosphocreatine peak was also observed. This group of thalamic tumors may be distinguished from other gliomas clinically, radiologically, and metabolically.  (+info)

(4/96) A case of thalamic syndrome: somatosensory influences on visual orientation.

The ability to set a straight line to the perceived gravitational vertical (subjective visual vertical, SVV) was investigated in a 21 year old woman with long standing left hemihypaesthesia due to a posterior thalamic infarct. The putative structures involved were the somatosensory and vestibular thalamus (VPL, VPM) and associative (pulvinar) thalamus. The SVV was normal when seated upright. When lying on her right side, line settings deviated about 17 degrees to the right, which is the normal A-effect. When lying on the hypaesthetic side the mean SVV remained close to true vertical-that is, the A-effect was absent, and there was a large increase in variability of the SVV settings. The findings support the view that the body tilt-induced bias of the SVV (A-effect) is largely mediated by somatosensory afferents. The finding that the A-effect was absent only when lying on the hypaesthetic side suggests that, during body tilt, the somatosensory system participates in visuogravitational orientation.  (+info)

(5/96) The clinical efficacy of neuroendoscope in surgical treatment for deafferentation pain.

Spinal cord stimulation (SCS) is one of the most minimally invasive and effective treatments for intractable pain. We report the efficacy of a very small diameter neuroendoscope on setting the electrode to the proper site in the epidural space. Our cases include thalamic hemorrhage, and each patient had unilateral intractable pain on L1 or less as the main complaint. They had been treated for over two years in other hospitals, but no significant relief was achieved. Because each patient had been given frequent epidural blocks, the adhesion in the epidural space was expected. In Group A (3 cases), we used very small diameter neuroendoscope to dissect adhesion in the epidural space and to make optimal space for lead placement under direct vision. Conventional lead placement under fluoroscopy was performed in Group B (3 cases). Medtronic's PISCES lead system was used for SCS. In Group A, stimulation and pain regions matched in all cases, and good pain relief was also achieved. In Group B, however, stimulation and pain regions matched incompletely and the increase in stimulation caused stimulation on the pain-free side.  (+info)

(6/96) Bilateral simultaneous thalamic hemorrhages--case report.

A 54-year-old male presented with bilateral simultaneous thalamic hemorrhages manifesting as semicoma, tetraplegia, and skew deviation. Magnetic resonance imaging and angiography demonstrated no lesions responsible for the bleeding. Coagulant factors were within normal ranges. The cause of these hemorrhages was considered to be hypertension. Conservative treatment was performed. He was discharged with serious neurological deficits.  (+info)

(7/96) Influenza A-associated encephalopathy with bilateral thalamic necrosis in Japan.

Two cases of acute encephalopathy in young children clearly showed evidence of influenza A virus infection and bilateral thalamic lesions. Influenza-associated encephalopathy with bilateral thalamic lesions has mostly been reported in Japan; it differs from Reye's syndrome in several respects. Other factors in addition to influenza virus infection may have contributed to the etiology of encephalopathy in our case patients.  (+info)

(8/96) Preoperative shunts in thalamic tumours.

Thirty one patients with thalamic glioma underwent a pre-tumour resection shunt surgery. The procedure was uneventful in 23 patients with relief from symptoms of increased intracranial pressure. Eight patients worsened after the procedure. The level of sensorium worsened from excessively drowsy state to unconsciousness in seven patients. Three patients developed hemiparesis, 4 developed paresis of extra-ocular muscles and altered pupillary reflexes, and 1 developed incontinence of urine and persistent vomiting. Alteration in the delicately balanced intracranial pressure and movements in the tumour and vital adjacent brain areas could be the probable cause of the worsening in the neurological state in these 8 patients. On the basis of these observations and on review of literature, it is postulated that the ventricular dilatation following an obstruction in the path of the cerebrospinal fluid flow by a tumour could be a natural defense phenomenon of the brain.  (+info)

What diseases can be cured by stem cells and how would the cells cure those specific diseases?

I need to know which diseases can be cured by Stem Cell treatment. I know that Parkinson's disease can be cured by this form of treatment but I need two other examples. I also need to know exactly how the cells will treat those specific diseases such as how the stem cells would target the motor cortex, causing a more sufficient amount of dopamine to be formed when treating Parkinson's disease.


There are many hurdles before stem cell implantation can be a potential cure or treatment for Parkinson's disease.  At this point skin cells have been used but the long term results are not in yet.

Other conditions with a cure potential include diabetes type 1 (juvenile diabetes), ALS, Huntington's disease, Becker Muscular dystrophy (BMD), Down Syndrome, adenpsone deaminase deficiency (ADA-SCID), Gaucher disease type III, Schechman-Bodian-Diamons syndromw (SBDS)

There is stem cell treatment for Crohn's disease.

Additional reading (I can't do all of your homework)

Proprietaty stem cells can prevent vision loss - retina protection:

You can also read this one about PD:

The 2nd part of your question can be searched in a normal fashion.  If I find time, I'll check it too.

What diseases would have the symptom of coughing up blood or blood in the phlegm?

This is not a symptom of my own so please don't tell me to go to the doctor, it is for a piece of work I have to do for College.

If you could tell me the name of a disease/ diseases that cause this, preferably not consumption or TB, a little about it and other symptoms it would be a massive help!
If you could also include treatment options and how serious a disease it is that would also be fantastic!

Hope you can help! Thanks!

First: spitting up blood is clinically known as: HEMOPTYSIS (bloody sputum, spit)
Yes, pneumonia is the most likely, but......
The following is from my medical e-book (I'm a nursing student)

"Blood in the sputum (hemoptysis) is most often seen in clients with chronic bronchitis or lung cancer. Clients with tuberculosis, pulmonary infarction, bronchial adenoma, or lung abscess may have grossly bloody sputum."
Also the end stage of cycstic fibrosis will present with hemoptysis.

a biggie in the hospital is:
A pulmonary embolism (PE) is a collection of particulate matter (solids, liquids, or gaseous substances) that enters venous circulation and lodges in the pulmonary vessels. Large emboli obstruct pulmonary blood flow, leading to decreased systemic oxygenation, pulmonary tissue hypoxia, and potential death. Any substance can cause an embolism, but a blood clot is the most common.

Pulmonary embolism is the most common acute pulmonary disease (90%) among hospitalized clients. In most people with PE, a blood clot from a deep vein thrombosis (DVT) breaks loose from one of the veins in the legs or the pelvis. The thrombus breaks off, travels through the vena cava and right side of the heart, and then lodges in a smaller blood vessel in the lung. Platelets collect with the embolus, triggering the release of substances that cause blood vessel constriction. Widespread pulmonary vessel constriction and pulmonary hypertension impair gas exchange. Deoxygenated blood shunts into the arterial circulation, causing hypoxemia. About 12% of clients with PE do not have hypoxemia.

Pulmonary embolism affects at least 500,000 people a year in the United States, about 10% of whom die. Many die within 1 hour of the onset of symptoms or before the diagnosis has even been suspected.

For clients with a known risk for PE, small doses of prophylactic subcutaneous heparin may be prescribed every 8 to 12 hours. Heparin prevents excessive coagulation in clients immobilized for a prolonged period, after trauma or surgery, or when restricted to bedrest. Occasionally, a drug to reduce platelet aggregation, such as clopidogrel (Plavix), is used in place of heparin.

A smaller one that popped up in the book:
Goodpasture's syndrome is an autoimmune disorder in which autoantibodies are made against the glomerular basement membrane and neutrophils. The two organs with the most damage are the lungs and the kidney. Lung damage is manifested as pulmonary hemorrhage. Kidney damage manifests as glomerulonephritis that may rapidly progress to complete renal failure (see Chapters 74 and 75). Unlike other autoimmune disorders, Goodpasture's syndrome occurs most often in adolescent or young adult men. The exact cause or triggering agent is unknown.

Goodpasture's syndrome usually is not diagnosed until serious lung and/or kidney problems are present. Manifestations include shortness of breath, hemoptysis (bloody sputum), decreased urine output, weight gain, generalized nondependent edema, hypertension, and tachycardia. Chest x-rays show areas of consolidation. The most common cause of death is uremia as a result of renal failure.

Spontaneous resolution of Goodpasture's syndrome has occurred but is rare. Interventions focus on reducing the immune-mediated damage and performing some type of renal supportive therapy.

What diseases are there that make children grow old?

I just watched an episode of Mystery Diagnosis, where a boy in England is growing old before his time. The kid is 8 yrs old, and has the looks and the bones of an elderly man. I know that one of them is called Hutchinsons disease, but this kid had something else, Is there other diseases that have the same or similar symptoms, and what are the names of those diseases?

What diseases can you get from cutting yourself with a rusty knife?

This is a question from a growing nurse. I've always wondered if you really can get a disease from a rusty knife or any rust and what kind of diseases you can get.

staph aureus and staph epidermidis  are commonly found on the skin and are responsible for
most infected wounds.  methicillin resistant staph aureus (MRSA) is becoming a serious
problem.   tetanus (clostridium tetani) is also a possibility but is usually not a problem with superficial
cuts that bleed a lot.  infected wounds not treated properly can become gangrenous (clostridium
perfringens).  clostridium bacteria are anaerobic which means that require a lack of oxygen to
grow.  poor circulation or elevating an infected foot may lead to gangrene due to the lack of oxygen
in the infected area.  if a person touches the cut with unclean hands, e. coli could infect the wound.

What are some diseases that your cells get?

I'm doing this project for school and I have to a write-up on two diseases that your cells get that effect your whole body, but I'm having trouble thinking of actual diseases to write about. Does anyone have any interesting diseases that fit this criteria? Thanks!

Sickle Cell disease, mostly found in African Americans. 
Cancer, mostly found in "anyone and everyone".
I wish you well on your report.

How were these diseases prevented or cured in the 1600 to early 1700s?


How were some of these diseases dealt with in the 1600s? If there was no cure or anything to prevent the diseases to happen can you explain why and what resulted in these situations? Thanks!

Inoculation was sometimes used to prevent smallpox but basically either you lived or you died.  Most survived chickenpox & measles but there were those who died or were left scarred or with damage to the vision or nervous system.  Malaria was a disease of the tropics and is found in parts of Africa, Asia, the Middle East, Central and South America, Hispaniola, and Oceania.  Mostly people died.  

The 1600s were in the 17th century & the 1700s were in the 18th century.   Do some online research.

What are some diseases similar to tuberculosis and could be mistaken as Tuberculosis?

So I am researching tuberculosis, and I have to answer the question:

If it isn't your exact disease, what else could it be?

So essentially I have to find diseases with the same signs and symptoms, ones that may act in the same way or cause your body to look the same way as tuberculosis.

I then have to talk about how they are similar and why. Any help would be great. Thanks.

So other mycobacterium would look the same on stains:
Mycobacterium bovis
Mycobacterium kansasii
and other mycobacteria

Other bacteria can cause similar lung disease (but look different on stain and culture):
Rhodococcus equii
and other bacteria

What kinds of diseases can you get from using a public restroom?

I've always heard that you can catch diseases from sitting on public toilets but no one has ever told me what kind you can get.
So I'm curious what kind of diseases have people gotten from using public restrooms?

You'd have a better chance of being hit by lightning. It's almost impossible to catch any kind of disease from a toilet, for the following reasons:
1. Infections don't live long outside the body...they need a host. Toilet seats are bad hosts.
2. They need warm wet areas to live.
3. Most infections enter the body through breaks in the skin or openings...
4. Even though your anus is an opening the intestines is a hostile enviroment for infection
5. You don't sit there long enough

You CAN get skin infections...I got one. They're not deadly, just annoying.