Spasm
Spasms, Infantile
Hemifacial Spasm
Esophageal Spasm, Diffuse
Ergonovine
Angina Pectoris, Variant
Facial Muscles
Bradykinin-induced bronchospasm in the rat in vivo: a role for nitric oxide modulation. (1/169)
Bradykinin has an important role in asthma pathogenesis, but its site of action is unclear. It was previously reported by the authors that bradykinin causes a dose-dependent reduction in dynamic compliance but little change in total lung resistance. This suggested that bradykinin may have a preferential effect in the distant lung. The purpose of the current investigation was to better characterize the effects of bradykinin on pulmonary resistance in rodents and explore the role of nitric oxide release in modulating the effect of bradykinin. Airway constriction was induced in the rats by aerosol administration of bradykinin with or without treatments with the inhaled bradykinin-2 receptor antagonist, Hoe 140 or the nitric oxide synthase inhibitors N(G)-nitro-L-arginine methylester or N(G)-monomethyl-L-arginine. Total lung resistance was partitioned into tissue and airway resistance by using the alveolar capsule method. Bradykinin induced a significant increase in both resistances. Hoe 140 abolished the response to bradykinin. The nitric oxide synthase inhibitors enhanced the bronchoconstricting response. In conclusion, the bradykinin response in the rats was not only localized to conducting airways but also involved a relatively selective tissue reaction. Bradykinin-induced bronchospasm in the rat is solely due to activation of bradykinin-2 receptor. Further, it was shown that nitric oxide significantly modulates the bronchospasm caused by bradykinin, suggesting that nitric oxide is an important modulator of airways responsiveness to bradykinin. (+info)Reduction of exercise-induced asthma in children by short, repeated warm ups. (2/169)
AIM: To study the effect of a warm up schedule on exercise-induced asthma in asthmatic children to enable them to engage in asthmogenic activities. METHOD: In the first study, peak flows during and after three short, repeated warm up schedules (SRWU 1, 2, and 3), identical in form but differing in intensity, were compared in 16 asthmatic children. In the second study the efficiency of the best of these SRWU schedules was tested on 30 young asthmatic children. Children performed on different days a 7 minute run alone (EX1) or the same run after an SRWU (EX2). RESULTS: The second study showed that for most children (24/30) the fall in peak flow after EX2 was less than that after EX1. The percentage fall in peak flow after EX2 was significantly correlated with the percentage change in peak flow induced by SRWU2 (r = 0.68). The children were divided into three subgroups according to the change in peak flow after SRWU2: (G1: increase in peak flow; G2: < 15% fall in peak flow; G3: > 15% fall in peak flow). Only the children in the G3 subgroup did not show any gain in peak flow after EX2 compared with EX1. CONCLUSION: The alteration in peak flow at the end of the SRWU period was a good predictor of the occurrence of bronchoconstriction after EX2. An SRWU reduced the decrease in peak flow for most of the children (24/30) in this series, thus reducing subsequent post-exercise deep bronchoconstriction. (+info)Reactive airways dysfunction and systemic complaints after mass exposure to bromine. (3/169)
Occasionally children are the victims of mass poisoning from an environmental contaminant that occurs due to an unexpected common point source of exposure. In many cases the contaminant is a widely used chemical generally considered to be safe. In the following case, members of a sports team visiting a community for an athletic event were exposed to chemicals while staying at a local motel. Bromine-based sanitizing agents and other chemicals such as hydrochloric acid, which were used in excess in the motel's swimming pool, may have accounted for symptoms experienced by the boy reported here and at least 16 other adolescents. Samples of pool water contained excess bromine (8.2 microg/mL; ideal pool bromine concentration is 2-4 microg/mL). Symptoms and signs attributable to bromine toxicity included irritative skin rashes; eye, nose, and throat irritation; bronchospasm; reduced exercise tolerance; fatigue; headache; gastrointestinal disturbances; and myalgias. While most of the victims recovered within a few days, the index case and several other adolescents had persistent or recurrent symptoms lasting weeks to months after the exposure. (+info)Regulation of baseline cholinergic tone in guinea-pig airway smooth muscle. (4/169)
1. We quantified baseline cholinergic tone in the trachealis of mechanically ventilated guinea-pigs and determined the influence of vagal afferent nerve activity on this parasympathetic tone. 2. There was a substantial amount of baseline cholinergic tone in the guinea-pig trachea, eliciting contractions of the trachealis that averaged 24.6 +/- 3.5 % (mean +/- s.e.m.) of the maximum attainable contraction. This tone was essentially abolished by vagotomy or ganglionic blockade, suggesting that it was dependent upon on-going pre-ganglionic input arising from the central nervous system. 3. Cholinergic tone in the trachealis could be markedly and rapidly altered (either increased or decreased) by changes in ventilation (e. g. cessation of ventilation; hyperpnoea; slow, deep breathing) and by lung distention (via positive end-expiratory pressure). These effects were not accompanied by marked alterations in blood gases and were abolished by vagotomy or atropine. By contrast, tachykinin receptor antagonists, which abolished capsaicin-induced bronchospasm, were without effect on baseline cholinergic tone. This and other evidence suggests that capsaicin-sensitive nerves have little if any influence on baseline parasympathetic tone. Likewise, while activation of afferent nerves innervating the larynx can alter airway parasympathetic nerve activity, transection of the superior laryngeal nerves was without effect on baseline cholinergic tone. 4. Cutting the vagus nerves caudal to the recurrent laryngeal nerves, thus leaving the preganglionic parasympathetic innervation of the trachealis intact but disrupting all afferent nerves innervating the lungs and intrapulmonary airways, abolished baseline cholinergic tone in the trachea. Sham vagotomy or cutting the vagi caudal to the lungs did not reduce baseline cholinergic tone. 5. The results indicate that baseline airway cholinergic nerve activity is necessarily dependent upon afferent nerve activity arising from the intrapulmonary airways and lungs. More specifically, the data are consistent with the hypothesis that on-going activity arising from the nerve terminals of intrapulmonary rapidly adapting receptors determines the level of baseline airway cholinergic tone. (+info)Bronchodilating effects of bambuterol on bronchoconstriction in guinea pigs. (5/169)
AIM: To study the effects of bambuterol (Bam) on bronchoconstriction in guinea pigs. METHODS: Bronchospasm induced by histamine aerosol, lung resistance (RL) and dynamic lung compliance (Cdyn) changes induced by ovalbumin aerosol in vivo, isolated resting lung parenchyma strips, and carbamylcholine-induced tracheal constriction in vitro in guinea pig were investigated. RESULTS: Bam dose-dependently prolonged the time to histamine-induced collapse, ED50 values (95% confidence limits) of Bam intragastric gavage (i.g.) after 1 h, 4 h, and 24 h were 0.74 (0.60-0.91), 0.75 (0.61-0.91) and 1.00 (0.77-1.30) mg.kg-1, respectively. Bam 2 or 10 mg.kg-1 i.g. 2 h before ovalbumin aerosol partly or almost completely inhibited bronchial challenge of ovalbumin-induced change of RL and Cdyn. Bam 0.1-1.0 mumol.L-1 gave a weak relaxation on isolated tracheal strips induced by carbamylcholine and failed to relax the isolated resting lung parenchyma strips in guinea pig. CONCLUSION: Bam showed a long-acting bronchodilation by its slow metabolism in vivo. (+info)Monosodium glutamate and asthma. (6/169)
Allen et al. (1987) conducted oral monosodium glutamate (MSG) challenges with 32 asthmatic volunteers and reported that 14 reacted to MSG. Another study by Moneret-Vautrin (1987) also reported MSG-induced asthma attacks in 2 of 30 asthmatic patients. Four additional studies have been conducted and none has confirmed the results of the above authors. These studies, by Schwartzstein et al. (1987), Germano (1991), Woods et al. (1998) and Woessner et al. (1999), challenged a total of 45 patients who gave a history of asthma attacks in oriental restaurants. None of these patients experienced asthmatic reactions after ingesting MSG (one-sided confidence interval of 0-0.066). Another 109 asthmatic patients, without a history of asthma in oriental restaurants, also did not react to ingestion of MSG (one-sided confidence interval of 0-0.027). With a confidence interval < 0.05 there is a >95% probability that MSG history-negative asthmatic patients are not sensitive to MSG. For the MSG history-positive asthmatics, 45 patients, in well-performed studies, underwent negative challenges to MSG, contrasting with two studies reporting positive challenges. Allen et al. (1987) and Moneret-Vautrin (1987), who reported positive MSG challenge results, performed studies with the following characteristics: 1) single blinded, conducted after discontinuing essential antiasthma medications; 2) used effort-dependent peak expiratory flow rate measurement of lung function; 3) added AM bronchodilators in some patients; 4) ignored wandering baselines on the placebo challenge days; and 5) conducted some challenges in the AM and some at night. In summary, the existence of MSG-induced asthma, even in history-positive patients, has not been established conclusively. (+info)Leukotriene-receptor antagonists. Role in asthma management. (7/169)
OBJECTIVE: To examine the role of leukotriene-receptor antagonists (LTRAs) in management of asthma. QUALITY OF EVIDENCE: Most data were derived from randomized, double-blind, controlled trials. MAIN MESSAGE: Leukotrienes appear to have an important role in the pathophysiology of asthma, including airway inflammation. Leukotriene-receptor antagonists are effective in improving asthma control end points, such as allergen, ASA, and exercise challenge, in clinical models of asthma. In chronic asthma, LTRA administration reduces asthma symptoms and rescue beta 2-agonist use, changes that are paralleled by improvements in lung function. Both zafirlukast and montelukast decrease circulating levels of eosinophils and could have other useful anti-inflammatory properties. Administration of LTRAs allows doses of inhaled corticosteroids to be reduced. Currently available LTRAs are free of serious side effects and are available as oral formulations. CONCLUSIONS: Leukotriene-receptor antagonists belong to a new class of asthma medication. While inhaled corticosteroids remain first-line therapy for managing chronic asthma, LTRAs should be considered for patients with ASA-sensitive asthma; as adjunct therapy when low to moderate doses of inhaled steroid alone provide incomplete control; or as adjunct therapy to allow reduction in doses of inhaled corticosteroids. (+info)Effects of fenoterol and ipratropium on respiratory resistance of asthmatics after tracheal intubation. (8/169)
We have studied the effects of a beta-agonist, fenoterol, and a cholinergic antagonist, ipratropium, on post-intubation total respiratory system resistance (Rrs) in asthmatics who developed increased Rrs after tracheal intubation. Sixteen stable asthmatics in whom Rrs increased after intubation were allocated randomly to receive either 10 puffs of fenoterol (group F) or 10 puffs of ipratropium (group IB) via a metered dose inhaler 5 min after intubation. Anaesthesia was induced and maintained with propofol i.v. Rrs was recorded before treatment and again 5, 15 and 30 min after treatment. Rrs decreased significantly from pretreatment values by mean 53 (SD 8)%, 53 (7)% and 58 (6)% at 5, 15 and 30 min, respectively, in group F, but declined by only 12 (6)%, 15 (4)% and 17 (5)% in group IB. At all times after treatment, patients in the fenoterol group had significantly lower Rrs values than those in the ipratropium group. We conclude that increased Rrs after tracheal intubation in asthmatics can be reduced effectively by treatment with fenoterol. A secondary finding of our study was that even after induction of anaesthesia with propofol, patients with a history of asthma may develop high Rrs. (+info)Bronchial spasm refers to a sudden constriction or tightening of the muscles in the bronchial tubes, which are the airways that lead to the lungs. This constriction can cause symptoms such as coughing, wheezing, and difficulty breathing. Bronchial spasm is often associated with respiratory conditions such as asthma, chronic obstructive pulmonary disease (COPD), and bronchitis. In these conditions, the airways are sensitive to various triggers such as allergens, irritants, or infections, which can cause the muscles in the airways to contract and narrow. This can make it difficult for air to flow in and out of the lungs, leading to symptoms such as shortness of breath, wheezing, and coughing. Bronchial spasm can be treated with medications that help to relax the muscles in the airways and open up the airways, such as bronchodilators and anti-inflammatory drugs.
A spasm is a sudden, involuntary contraction or tightening of a muscle, group of muscles, or a hollow organ such as the ureter or bronchi. Spasms can occur as a result of various factors including muscle fatigue, injury, irritation, or abnormal nerve activity. They can cause pain and discomfort, and in some cases, interfere with normal bodily functions. For example, a spasm in the bronchi can cause difficulty breathing, while a spasm in the ureter can cause severe pain and may lead to a kidney stone blockage. The treatment for spasms depends on the underlying cause and may include medication, physical therapy, or lifestyle changes.
Infantile spasms, also known as West syndrome, is a rare but serious type of epilepsy that affects infants typically between 4-8 months of age. The spasms are characterized by sudden, brief, and frequent muscle jerks or contractions, often involving the neck, trunk, and arms. These spasms usually occur in clusters and may cause the infant to bend forward or stretch out. Infantile spasms can be a symptom of various underlying neurological conditions and are often associated with developmental delays and regression. Early recognition and treatment are crucial for improving outcomes.
Hemifacial spasm is a neuromuscular disorder characterized by involuntary, irregular contractions or twitching of the muscles on one side of the face. These spasms typically begin around the eye and may progress to involve the muscles of the lower face, including those around the mouth.
The primary cause of hemifacial spasm is pressure on or irritation of the facial nerve (cranial nerve VII) as it exits the brainstem, often due to a blood vessel or tumor. This pressure can lead to abnormal electrical signals in the facial nerve, resulting in uncontrolled muscle contractions.
In some cases, hemifacial spasm may be associated with other conditions such as multiple sclerosis or Bell's palsy. Treatment options for hemifacial spasm include medications to help relax the muscles, botulinum toxin (Botox) injections to paralyze the affected muscles temporarily, and, in rare cases, surgical intervention to relieve pressure on the facial nerve.
Coronary vasospasm refers to a sudden constriction (narrowing) of the coronary arteries, which supply oxygenated blood to the heart muscle. This constriction can reduce or block blood flow, leading to symptoms such as chest pain (angina) or, in severe cases, a heart attack (myocardial infarction). Coronary vasospasm can occur spontaneously or be triggered by various factors, including stress, smoking, and certain medications. It is also associated with conditions such as coronary artery disease and variant angina. Prolonged or recurrent vasospasms can cause damage to the heart muscle and increase the risk of cardiovascular events.
Diffuse Esophageal Spasm (DES) is a motility disorder of the esophagus, which is the muscular tube that connects the throat to the stomach. In DES, the esophagus involuntarily and uncoordinately contracts, causing difficulty swallowing (dysphagia), chest pain, and sometimes regurgitation of food or liquids.
The term "diffuse" refers to the fact that these spasms can occur throughout the entire length of the esophagus, rather than being localized to a specific area. The exact cause of diffuse esophageal spasm is not known, but it may be associated with abnormalities in the nerve cells that control muscle contractions in the esophagus.
Diagnosis of DES typically involves a combination of medical history, physical examination, and specialized tests such as esophageal manometry or ambulatory 24-hour pH monitoring. Treatment options may include medications to relax the esophageal muscles, lifestyle modifications such as avoiding trigger foods, and in some cases, surgery.
Ergonovine is a medication that belongs to a class of drugs called ergot alkaloids. It is derived from the ergot fungus and is used in medical settings as a uterotonic agent, which means it causes the uterus to contract. Ergonovine is often used after childbirth to help the uterus return to its normal size and reduce bleeding.
Ergonovine works by binding to specific receptors in the smooth muscle of the uterus, causing it to contract. It has a potent effect on the uterus and can also cause vasoconstriction (narrowing of blood vessels) in other parts of the body. This is why ergonovine is sometimes used to treat severe bleeding caused by conditions such as uterine fibroids or ectopic pregnancy.
Like other ergot alkaloids, ergonovine can have serious side effects if not used carefully. It should be administered under the close supervision of a healthcare provider and should not be used in women with certain medical conditions, such as high blood pressure or heart disease. Ergonovine can also interact with other medications, so it's important to inform your healthcare provider of all medications you are taking before receiving this drug.
Angina pectoris, variant (also known as Prinzmetal's angina or vasospastic angina) is a type of chest pain that results from reduced blood flow to the heart muscle due to spasms in the coronary arteries. These spasms cause the arteries to narrow, temporarily reducing the supply of oxygen-rich blood to the heart. This can lead to symptoms such as chest pain, shortness of breath, and fatigue.
Variant angina is typically more severe than other forms of angina and can occur at rest or with minimal physical exertion. It is often treated with medications that help relax the coronary arteries and prevent spasms, such as calcium channel blockers and nitrates. In some cases, additional treatments such as angioplasty or bypass surgery may be necessary to improve blood flow to the heart.
It's important to note that chest pain can have many different causes, so it is essential to seek medical attention if you experience any symptoms of angina or other types of chest pain. A healthcare professional can help determine the cause of your symptoms and develop an appropriate treatment plan.
Facial muscles, also known as facial nerves or cranial nerve VII, are a group of muscles responsible for various expressions and movements of the face. These muscles include:
1. Orbicularis oculi: muscle that closes the eyelid and raises the upper eyelid
2. Corrugator supercilii: muscle that pulls the eyebrows down and inward, forming wrinkles on the forehead
3. Frontalis: muscle that raises the eyebrows and forms horizontal wrinkles on the forehead
4. Procerus: muscle that pulls the medial ends of the eyebrows downward, forming vertical wrinkles between the eyebrows
5. Nasalis: muscle that compresses or dilates the nostrils
6. Depressor septi: muscle that pulls down the tip of the nose
7. Levator labii superioris alaeque nasi: muscle that raises the upper lip and flares the nostrils
8. Levator labii superioris: muscle that raises the upper lip
9. Zygomaticus major: muscle that raises the corner of the mouth, producing a smile
10. Zygomaticus minor: muscle that raises the nasolabial fold and corner of the mouth
11. Risorius: muscle that pulls the angle of the mouth laterally, producing a smile
12. Depressor anguli oris: muscle that pulls down the angle of the mouth
13. Mentalis: muscle that raises the lower lip and forms wrinkles on the chin
14. Buccinator: muscle that retracts the cheek and helps with chewing
15. Platysma: muscle that depresses the corner of the mouth and wrinkles the skin of the neck.
These muscles are innervated by the facial nerve, which arises from the brainstem and exits the skull through the stylomastoid foramen. Damage to the facial nerve can result in facial paralysis or weakness on one or both sides of the face.
Blepharospasm is a medical condition characterized by involuntary spasms and contractions of the muscles around the eyelids. These spasms can cause frequent blinkings, eye closure, and even difficulty in keeping the eyes open. In some cases, the spasms may be severe enough to interfere with vision, daily activities, and quality of life.
The exact cause of blepharospasm is not fully understood, but it is believed to involve abnormal functioning of the basal ganglia, a part of the brain that controls movement. It can occur as an isolated condition (known as essential blepharospasm) or as a symptom of other neurological disorders such as Parkinson's disease or dystonia.
Treatment options for blepharospasm may include medication, botulinum toxin injections, surgery, or a combination of these approaches. The goal of treatment is to reduce the frequency and severity of the spasms, improve symptoms, and enhance the patient's quality of life.
Bronchoconstriction
Sudden infant death with dysgenesis of the testes syndrome
John Auer
Band on the Run
Bronchospasm
Anaphylaxis
Pathophysiology of asthma
Frank Chance
Nancy Hogshead-Makar
Health effects of electronic cigarettes
Assassination of James A. Garfield
Respiratory risks of indoor swimming pools
Anticholinergic
Exercise-induced bronchoconstriction
Flat-chested kitten syndrome
Causes of cancer pain
Cholinergic crisis
Cyclopentolate
Pilocarpine
Malaoxon
Isoetarine
Cramp fasciculation syndrome
Ipratropium bromide
Reslizumab
Kounis syndrome
Buteyko method
Rodney Hogg
Chronic cough
Dextromethorphan
Chlormethine
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Asthma17
- If you have allergy-induced asthma, your bronchial tubes will constrict when you inhale substances that trigger an allergic reaction. (healthline.com)
- You're at higher risk of developing asthma, a common cause of bronchial spasms, if you have another allergic condition. (healthline.com)
- The rising prevalence of asthma drives the value of the bronchial spasms treatment market size. (expertmarketresearch.com)
- The contraction and inflammation of bronchial muscles are due to the associated pulmonary disorders like emphysema, asthma, bronchitis, or chronic obstructive pulmonary disease (COPD). (expertmarketresearch.com)
- The bronchial spasms treatment market is driven by the increasing prevalence and incidence of respiratory disorders like emphysema, asthma, chronic bronchitis, and chronic obstructive pulmonary disorder. (expertmarketresearch.com)
- Not all viral infections trigger the bronchial spasms and inflammation with the same severity in asthma. (thestar.com.my)
- The herb is used with great success in the treatment of asthma and associated conditions due to its power to relieve spasms in the bronchial tubes. (healthy.net)
- It is thus used in bronchial asthma, bronchitis and whooping cough. (healthy.net)
- Bronchial asthma is a common disease both in children as well as in grown ups. (yogawiz.com)
- In Bronchial asthma is characterized the patient suffers repeated attacks of difficulty exhaling. (yogawiz.com)
- In Bronchial Asthma is that it is only a temporary functional narrowing. (yogawiz.com)
- Another crucial feature in Bronchial asthma is that the air passages start to exhibit unwarranted response by spasm and inflammation to all sorts of stimuli. (yogawiz.com)
- The three cardinal symptoms of Bronchial asthma are sneezing, runny nose and blocked or stuffy nose. (yogawiz.com)
- In Bronchial asthma this secretion is sticky and is expectorated with a lot of difficulty. (yogawiz.com)
- Asthma is a breathing problem due to narrowing of the airways (bronchial tubes) that allow air to move in and out of the lungs . (hdkino.org)
- Bronchospasm definition is - constriction of the air passages of the lung (as in asthma) by spasmodic contraction of the bronchial muscles. (papercritters.com)
- A 46-year-old man with a history of bronchial asthma and chronic sinusitis presented to our hospital with chest pain. (bvsalud.org)
Bronchospasm4
- When that happens, it's called a bronchial spasm, or a bronchospasm. (healthline.com)
- These airways can be narrowed due to the accumulation of mucus , spasm of the muscles that surround these airways (bronchospasm), or swelling of the lining of the airways. (hdkino.org)
- This muscular spasm also known as bronchospasm can result in further airway narrowing. (medicinenet.com)
- bronchospasm: [ spazm ] 1. (papercritters.com)
Mucus5
- Depending on the cause of your bronchial spasms, your bronchial glands may also produce more mucus. (healthline.com)
- Bronchitis is a term that describes inflammation of the bronchial tubes (bronchi and the smaller branches termed bronchioles ) that results in excessive secretions of mucus into the tubes, leading to tissue swelling that can narrow or close off bronchial tubes. (medicinenet.com)
- Cilia perform the function of moving particles and fluid (usually mucus) over the lining surface in such structures as the trachea, bronchial tubes, and nasal cavities to keep these hollow structures clear of particles and fluids. (medicinenet.com)
- Chronic coughing develops as the body attempts to open and clear the bronchial airways of particles and mucus or as an overreaction to ongoing inflammation. (medicinenet.com)
- It not only may help provide alleviation from upper respiratory infections caused by bacteria , but it can help get rid of mucus from the lungs and help relax bronchial spasms. (readersdigest.ca)
Bronchitis5
- Bronchitis is another common cause of bronchial spasms. (healthline.com)
- These infections can lead to acute bronchitis and bronchial spasms. (healthline.com)
- The number of goblet cells in the bronchial membrane increases in chronic bronchitis or after chronic exposure to mild irritants. (healthy.net)
- Albuterol also can be helpful in patients with emphysema and chronic bronchitis when symptoms are partially related to spasm of the airways' muscles. (hdkino.org)
- With long-standing inflammation, as can be seen in chronic bronchitis, this muscular spasm and inflammation result in a fixed, nonreversible narrowing of the airway, and the condition is termed chronic obstructive pulmonary disease ( COPD ). (medicinenet.com)
Inflammation1
- Inflammation of the bronchioles develops and causes smooth muscles in lung tissue to contract (spasm), further obstructing airflow. (msdmanuals.com)
Airway2
- In bronchial spams, the muscles around the bronchial tubes contract or inflame and obstruct the airway flow. (expertmarketresearch.com)
- As a calcium-dependent myogenic response could be involved, we examined the effect of a potent voltage-dependent calcium channel (VDC) antagonist, nifedipine (20 mg administered sublingually) versus placebo, on the DI-induced change in plethysmographic specific airway conductance (SGaw) in six asthmatic patients and six healthy controls both before and after a bronchial challenge with methacholine (MCh). (nih.gov)
Airways1
- Albuterol sulphate dilates bronchial airways by relaxing the muscles that surround the airways. (hdkino.org)
Lungs2
- Supports the free flow of air through the lungs by helping to prevent the constriction of the bronchials and promoting the relaxation of the bronchial tubes that are in spasm. (wholeworldbotanicals.com)
- Bronchial tubes extend from the trachea and terminate at the alveoli in the lungs . (medicinenet.com)
Cough2
- You may also cough a lot when your bronchial tubes are constricted. (healthline.com)
- A nonproductive cough, particularly at night, may mean spasms in the bronchial tubes caused by irritation. (hartfordhospital.org)
Broncho2
- Broncho (prefix) + spasm (suffix) b. (papercritters.com)
- Broncho: relating to the bronchi, spasm: involuntary contraction c. 2. (papercritters.com)
Symptoms5
- What are the symptoms of bronchial spasms? (healthline.com)
- Wheezing is one of the most common symptoms of a bronchial spasm. (healthline.com)
- If you're experiencing bronchial spasms, your doctor will probably ask you about your current symptoms and medical history. (healthline.com)
- The most common symptoms of bronchial spasms include wheezing, pain in the chest, difficulty in breathing and coughing. (expertmarketresearch.com)
- Prefix Definition: bronchial tube Example: bronchiolectasis Alternate Notation: bronchi/o ( including the quote, if possible ) about the symptoms and how it treated. (papercritters.com)
Contraction1
- A disorder characterized by a sudden contraction of the smooth muscles of the bronchial wall. (embl.de)
Muscles3
- Normally, the muscles surrounding your bronchial tubes are thin and smooth, and they allow air to flow easily. (healthline.com)
- spasms usually occur when the nerves supplying muscles are irritated, and are often accompanied by pain. (papercritters.com)
- Constriction of the bronchial muscles and other reference data is for informational Purposes. (papercritters.com)
Bronchi1
- The reason for the word "Bronchial" is owing to the fact that the fundamental problem lies in the bronchi. (yogawiz.com)
Acute1
- This can be attributed to its immediate therapeutic effect in treating acute or chronic bronchial spasms. (expertmarketresearch.com)
Disease1
- Treatment interventions for bronchial spasms depend on the pathophysiology and the severity of the disease. (expertmarketresearch.com)
Occur1
- Bronchial spasms occur when your bronchial tubes become inflamed. (healthline.com)
Allergies1
- People suffering from other allergies like food allergies or eczema are also expected to develop bronchial spasms. (expertmarketresearch.com)
Develop1
- The body cannot long bear the extreme acidity which is the environment of such spasms and will develop the holes or ulcerations which then do appear upon the allopathic testings and suggest to the chirurgeon that which is to be excised. (lawofone.info)
Production1
- Both histamine production and bronchial spasms increase with magnesium deficiency. (healthybliss.net)
Common1
- Bronchial spasms are among the most common pulmonary disorders. (expertmarketresearch.com)
Treatment1
- North America will hold a significant share of the bronchial spasms treatment market. (expertmarketresearch.com)
Tests1
- They may also conduct other tests to learn what's causing your bronchial spasms. (healthline.com)
Cases1
- In many cases, bronchial spasms are treatable or preventable. (healthline.com)
Patient1
- This narrative review describes the main applications of de la ultrasonografÃa en ultrasound in anesthesia, ultrasound-guided techniques, and current trends in the perioperative anesthetic management of anestesia the surgical patient. (bvsalud.org)