Toxicodendron
Dermatitis, Toxicodendron
Dermatitis, Atopic
Dermatitis, Allergic Contact
Dermatitis Herpetiformis
Dermatitis, Seborrheic
Secondary bacterial infections complicating skin lesions. (1/7)
Secondary bacterial infection in skin lesions is a common problem. This review summarises a series of studies of the microbiology of several of these infections: scabies, psoriasis, poison ivy, atopic dermatitis, eczema herpeticum and kerion. Staphylococcus aureus and group A beta-haemolytic streptococci were the most prevalent aerobes and were isolated from all body sites. In contrast, organisms that reside in the mucous membranes close to the lesions predominated in infections next to these membranes. In this fashion, enteric gram-negative bacilli and Bacteroides spp. were found most often in buttock and leg lesions. The probable sources of these organisms are the rectum and vagina, where they normally reside. Group A beta-haemolytic streptococci, pigmented Prevotella and Porphyromonas spp. and Fusobacterium spp. were most commonly found in lesions of the head, face, neck and fingers. These organisms probably reached these sites from the oral cavity, where they are part of the normal flora. This review highlights the polymicrobial aerobic-anaerobic microbiology of secondarily infected skin lesions. (+info)CURRENT CONCEPTS IN DERMATOLOGY. III. THE USE OF RADIOTHERAPY AND CORTICOSTEROIDS. (2/7)
The use of ionizing radiation and corticosteroids is discussed, in this third and final part of a review of diseases of the skin. Radiation is being used less extensively because superior methods of treatment are available for many conditions which formerly were frequently treated by this modality. The concept of applying the radiation at the level of the basic pathologic process has been developed into clinical practice by the use of generators which can produce very soft (or superficial) ionizing radiation. Topical or systemic corticosteroids do not cure skin diseases but produce dramatic suppression of signs and symptoms. For best results consideration must be given to the diagnosis, the natural history of the disease to be treated, the method of administration and a search for possible contraindications to the use of these steroids. Basic dermatological principles (removal of offending agents, bland soothing applications, sedation, etc.) must be adhered to. The corticosteroids are not a panacea in the treatment of skin disease. (+info)Biomass and toxicity responses of poison ivy (Toxicodendron radicans) to elevated atmospheric CO2. (3/7)
Contact with poison ivy (Toxicodendron radicans) is one of the most widely reported ailments at poison centers in the United States, and this plant has been introduced throughout the world, where it occurs with other allergenic members of the cashew family (Anacardiaceae). Approximately 80% of humans develop dermatitis upon exposure to the carbon-based active compound, urushiol. It is not known how poison ivy might respond to increasing concentrations of atmospheric carbon dioxide (CO(2)), but previous work done in controlled growth chambers shows that other vines exhibit large growth enhancement from elevated CO(2). Rising CO(2) is potentially responsible for the increased vine abundance that is inhibiting forest regeneration and increasing tree mortality around the world. In this 6-year study at the Duke University Free-Air CO(2) Enrichment experiment, we show that elevated atmospheric CO(2) in an intact forest ecosystem increases photosynthesis, water use efficiency, growth, and population biomass of poison ivy. The CO(2) growth stimulation exceeds that of most other woody species. Furthermore, high-CO(2) plants produce a more allergenic form of urushiol. Our results indicate that Toxicodendron taxa will become more abundant and more "toxic" in the future, potentially affecting global forest dynamics and human health. (+info)Dermoscopy of black-spot poison ivy. (4/7)
Black-spot poison ivy is an uncommon presentation of poison ivy (Toxicodendron) allergic contact dermatitis. A 78-year-old sought evaluation of a black spot present on her right hand amid pruritic vesicles. The presentation of a black spot on the skin in a clinical context suggesting poison ivy is indicative of black-spot poison ivy. Dermoscopy revealed a jagged, centrally homogeneous, dark brown lesion with a red rim. A skin sample was obtained and compared against a poison ivy standard using ultra-fast liquid chromatography-tandem mass spectrometry (UFLC-MS/MS). This finding confirmed the presence of multiple urushiol congeners in the skin sample. Black-spot poison ivy may be added to the list of diagnoses that show a specific dermoscopic pattern. (+info)Urushiol (poison ivy)-triggered suppressor T cell clone generated from peripheral blood. (5/7)
Allergic contact dermatitis to Toxicodendron radicans (poison ivy) is mediated by the hapten urushiol. An urushiol-specific, interleukin 2 (IL-2)-dependent T cell clone (RLB9-7) was generated from the peripheral blood of a patient with a history of allergic contact dermatitis to T. radicans. This clone proliferated specifically to both leaf extract and pure urushiol. Although the clone had the phenotype CD3+CD4+CD8+, proliferation to antigen was blocked by anti-CD8 and anti-HLA-A, B, C, but not by anti-CD4, suggesting that CD4 was not functionally associated with the T cell receptor. Furthermore, studies with antigen-presenting cells from MHC-typed donors indicated that the clone was MHC class 1 restricted. RLB9-7 was WT31 positive, indicating it bears the alpha beta T cell receptor. The clone lacked significant natural killer cell activity and produced only low levels of IL-2 or gamma-interferon upon antigen stimulation. Addition of RLB9-7 to autologous peripheral blood mononuclear cells in the presence of urushiol inhibited the pokeweed mitogen-driven IgG synthesis. This suppression was resistant to irradiation (2,000 rad) and was not seen when RLB9-7 was added to allogeneic cells, even in the presence of irradiated autologous antigen-presenting cells, suggesting that suppression was MHC restricted and not mediated by nonspecific soluble factors. However, RLB9-7 cells in the presence of urushiol inhibited the synthesis of tetanus toxoid-specific IgG by autologous lymphocytes, indicating that the suppression, although triggered specifically by urushiol, was nonspecific. (+info)Keratinocyte intercellular adhesion molecule-1 (ICAM-1) expression precedes dermal T lymphocytic infiltration in allergic contact dermatitis (Rhus dermatitis). (6/7)
The ability of small molecules such as urushiol, present as a wax on the poison ivy leaf surface, to cause allergic contact dermatitis (rhus dermatitis) has fascinated immunologists for decades. Current dogma suggests that these epicutaneously applied catechol-containing molecules serve as haptens to conjugate with larger proteins via reactive o-quinone intermediates. These complexes are then recognized as foreign antigens by the immune system and elicit a hypersensitivity reaction. Phorbol ester can directly induce cultured keratinocyte (KC) intercellular adhesion molecule-1 (ICAM-1) expression via a protein kinase C (PK-C)-dependent mechanism. As urushiol is also a known PK-C agonist, we asked if topical application of a poison ivy/oak mixture could directly induce epidermal KC ICAM-1 expression. During the pre-erythematous phase of this reaction (4 to 20 hours), epidermal KCs expressed ICAM-1; this "initiation phase" preceded the appearance of activated memory T lymphocytes in the papillary dermis, and thus appeared to be nonlymphokine mediated. A near-contiguous cellular-adhesion molecular network was identified by ICAM-1 staining of basal KCs, dermal dendrocytes, and endothelial cells. During the second 24-hour period with the onset of erythema and edema, there was an "amplification phase" of more intense KC ICAM-1 expression coupled with relatively weak KC HLA-DR expression that coincided with dermal and epidermal T-cell infiltration. This suggests the presence of lymphokines, such as gamma interferon, during the amplification phase because of KC HLA-DR expression. On cultured KCs, urushiol directly induced ICAM-1 expression but not HLA-DR. Thus, in addition to functioning as an antigenic hapten, urushiol directly induces KC ICAM-1 expression. The KC ICAM-1 expression may then alter the dynamic trafficking of memory T cells in the epidermis, so as to initiate cutaneous inflammation in a nonantigen specific manner. This initiation phase is followed by T-cell infiltration and consequent lymphokine production that significantly amplifies the original stimulus. Thus much can still be learned about the molecular pathophysiology of this common type of cutaneous inflammation. (+info)Gas plant (Dictamnus albus) phytophotodermatitis simulating poison ivy. (7/7)
A 48-year-old man presented with an itchy rash that resembled superficial burns or cane marks on his left forearm; similar lesions had appeared every summer for 5 years. Poison ivy dermatitis had been the initial diagnosis, but the patient knew that this plant was absent from his well tended garden. A visit to the garden revealed the gas plant Dictamnus albus, and occlusive patch testing with leaf cuttings produced a reaction after the skin was exposed to sunlight. Gas plant phytophotodermatitis was diagnosed. (+info)"Toxicodendron" is a genus of flowering plants in the family Anacardiaceae, also known as the cashew family. This genus includes several species that are well-known for causing allergic reactions in humans, particularly through contact with their sap or urushiol-containing parts of the plant. The most common and notorious species in this genus is Toxicodendron radicans, also known as poison ivy, poison oak, and poison sumac. These plants can cause an itchy, blistering rash upon contact with the skin, which is a type of allergic reaction called contact dermatitis. The severity of the reaction can vary from person to person, depending on their sensitivity to urushiol and the amount of exposure they have had to the plant.
Toxicodendron dermatitis is a type of contact dermatitis that results from exposure to plants belonging to the Toxicodendron genus, which includes poison ivy, poison oak, and poison sumac. The reaction is caused by an oily resin called urushiol found in these plants. When the oil comes into contact with the skin, it can cause an allergic reaction that leads to a red, itchy rash, often with blisters or weeping lesions.
The rash usually appears within 12-72 hours after exposure and can last for several weeks. The severity of the reaction varies from person to person, depending on their sensitivity to urushiol and the amount of contact they had with the plant. In addition to direct skin contact, urushiol can also be spread through secondary sources such as clothing, pets, or tools that have come into contact with the plant.
Prevention measures include avoiding contact with Toxicodendron plants, wearing protective clothing and gloves when working in areas where these plants may be present, and washing skin and clothing thoroughly with soap and water after exposure. In some cases, medical treatment may be necessary to manage symptoms and prevent complications.
Atopic dermatitis is a chronic, inflammatory skin condition that is commonly known as eczema. It is characterized by dry, itchy, and scaly patches on the skin that can become red, swollen, and cracked over time. The condition often affects the skin on the face, hands, feet, and behind the knees, and it can be triggered or worsened by exposure to certain allergens, irritants, stress, or changes in temperature and humidity. Atopic dermatitis is more common in people with a family history of allergies, such as asthma or hay fever, and it often begins in infancy or early childhood. The exact cause of atopic dermatitis is not fully understood, but it is thought to involve a combination of genetic and environmental factors that affect the immune system and the skin's ability to maintain a healthy barrier function.
Dermatitis is a general term that describes inflammation of the skin. It is often characterized by redness, swelling, itching, and tenderness. There are many different types of dermatitis, including atopic dermatitis (eczema), contact dermatitis, seborrheic dermatitis, and nummular dermatitis.
Atopic dermatitis is a chronic skin condition that often affects people with a family history of allergies, such as asthma or hay fever. It typically causes dry, scaly patches on the skin that can be extremely itchy.
Contact dermatitis occurs when the skin comes into contact with an irritant or allergen, such as poison ivy or certain chemicals. This type of dermatitis can cause redness, swelling, and blistering.
Seborrheic dermatitis is a common condition that causes a red, itchy rash, often on the scalp, face, or other areas of the body where oil glands are located. It is thought to be related to an overproduction of oil by the skin's sebaceous glands.
Nummular dermatitis is a type of eczema that causes round, coin-shaped patches of dry, scaly skin. It is more common in older adults and often occurs during the winter months.
Treatment for dermatitis depends on the underlying cause and severity of the condition. In some cases, over-the-counter creams or lotions may be sufficient to relieve symptoms. Prescription medications, such as corticosteroids or immunosuppressants, may be necessary in more severe cases. Avoiding triggers and irritants can also help prevent flare-ups of dermatitis.
Allergic contact dermatitis is a type of inflammatory skin reaction that occurs when the skin comes into contact with a substance (allergen) that the immune system recognizes as foreign and triggers an allergic response. This condition is characterized by redness, itching, swelling, blistering, and cracking of the skin, which usually develops within 24-48 hours after exposure to the allergen. Common allergens include metals (such as nickel), rubber, medications, fragrances, and cosmetics. It is important to note that a person must first be sensitized to the allergen before developing an allergic response upon subsequent exposures.
Dermatitis herpetiformis (DH) is a chronic, autoimmune blistering skin disorder that is characterized by the presence of symmetrical, pruritic (itchy), papulo-vesicular (papules and small fluid-filled blisters) eruptions on the extensor surfaces of the body, such as the elbows, knees, buttocks, and shoulders. It is often associated with gluten sensitivity or celiac disease, a condition that causes an abnormal immune response to gluten, a protein found in wheat, barley, and rye.
The exact cause of DH is not fully understood, but it is believed to result from the interaction between genetic, environmental, and immunological factors. The disorder is characterized by the presence of IgA antibodies in the skin, which trigger an immune response that leads to the formation of the characteristic rash.
DH is typically treated with a gluten-free diet, which can help to control the symptoms and prevent complications such as malabsorption and nutritional deficiencies. Medications such as dapsone may also be used to control the itching and blistering associated with the disorder. In some cases, topical corticosteroids or other anti-inflammatory medications may be prescribed to help manage symptoms.
It is important to note that DH is a chronic condition that requires ongoing management and monitoring. People with DH should work closely with their healthcare provider to develop an appropriate treatment plan and monitor their progress over time.
Seborrheic dermatitis is a common, inflammatory skin condition that mainly affects the scalp, face, and upper part of the body. It causes skin irritation, flaking, and redness, often in areas where the skin is oily or greasy. The exact cause of seborrheic dermatitis is not fully understood, but it appears to be related to a combination of genetic, environmental, and microbial factors.
The symptoms of seborrheic dermatitis can vary in severity and may include:
* Greasy or flaky scales on the scalp, eyebrows, eyelashes, ears, or beard
* Redness and inflammation of the skin
* Itching, burning, or stinging sensations
* Yellow or white crusty patches on the scalp or other affected areas
* Hair loss (in severe cases)
Seborrheic dermatitis is a chronic condition that tends to flare up and then subside over time. While there is no cure for seborrheic dermatitis, various treatments can help manage the symptoms and prevent complications. These may include medicated shampoos, topical creams or ointments, and lifestyle changes such as stress reduction and avoiding triggers that worsen symptoms.
It is important to note that seborrheic dermatitis should not be confused with other skin conditions, such as psoriasis or eczema, which may have similar symptoms. A healthcare professional can provide a proper diagnosis and recommend appropriate treatment options based on the individual's specific needs.
Occupational dermatitis is a specific type of contact dermatitis that results from exposure to certain substances or conditions in the workplace. It can be caused by direct contact with chemicals, irritants, or allergens present in the work environment. This condition typically affects the skin on the hands and forearms but can also involve other areas of the body, depending on the nature of the exposure.
There are two main types of occupational dermatitis:
1. Irritant contact dermatitis (ICD): This type occurs when the skin comes into direct contact with an irritating substance, leading to redness, swelling, itching, and sometimes blistering. Common irritants include solvents, detergents, oils, and other industrial chemicals.
2. Allergic contact dermatitis (ACD): This type is a result of an allergic reaction to a specific substance. The immune system identifies the allergen as harmful and mounts a response, causing skin inflammation. Common allergens include latex, metals (such as nickel), and certain plants (like poison ivy).
Prevention measures for occupational dermatitis include using appropriate personal protective equipment (PPE) like gloves, masks, and aprons, as well as practicing good hygiene, such as washing hands regularly and avoiding touching the face with contaminated hands. If you suspect you have developed occupational dermatitis, consult a healthcare professional for proper diagnosis and treatment.
Urushiol-induced contact dermatitis
Contact dermatitis
Poison ivy
Tecnu
Toxicodendron vernix
Lacquer dermatitis
Medicinal clay
Koebner phenomenon
Toxicodendron pubescens
Urushiol
Toxicodendron
Lacquer
Toxicodendron rydbergii
Toxicodendron diversilobum
Toxicodendron radicans
Impatiens
Occupational hazard
Allergic contact dermatitis
Brown recluse spider
List of MeSH codes (C20)
Toxicodendron orientale
Loxoscelism
List of MeSH codes (C17)
Zirconium granuloma
Hedera
Ginkgo biloba
Edema
Frangula californica
Symphyotrichum novae-angliae
Allergy
Toxicodendron Poisoning Medication: Corticosteroids, Antihistamines
Urushiol-induced contact dermatitis - Wikipedia
Poison ivy rash - Symptoms and causes - Mayo Clinic
Poison ivy - oak - sumac: MedlinePlus Medical Encyclopedia
Rosacea, Acne, Shingles, Covid-19 Rashes: Common Adult Skin Diseases
Update: Plant Dermatitis Among Active Component Service Members, U.S. Armed Forces, 2010-2020 | Health.mil
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Outdoor Workers and Poisonous Plant Exposures | Blogs | CDC
Scientists say most so-called spider bites are misdiagnosed - NaturalNews.com
Poison ivy - oak - sumac Information | Mount Sinai - New York
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Toxicodendron vernicifluum Lacquer Tree PFAF Plant Database
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Toxicodendron diversilobum | Landscape Plants | Oregon State University
Electronic Textbook of Dermatology--Botanical Dermatology , Occupational Plant Dermatoses
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Poison ivy-induced dermatitis on NativeSkin® | Genoskin
Contact dermatitis50
- Contact dermatitis caused by the sap of plants from the Anacardiaceae family, including poison ivy, oak, and sumac, is among the most common skin conditions in the United States. (jcadonline.com)
- The patient was diagnosed with Toxicodendron contact dermatitis caused by exposure to poison ivy. (jcadonline.com)
- Toxicodendron contact dermatitis (TCD) is a highly pruritic papulovesicular eruption that represents a Type-IV delayed hypersensitivity reaction triggered by contact with the sap of plants from the Anacardiaceae family, which includes poison ivy (the most common), oak, and sumac. (jcadonline.com)
- Toxicodendron dermatitis is an allergic contact dermatitis (allergic phytodermatitis) that occurs from exposure to urushiol, a skin-irritating oil produced by members of the plant genus Toxicodendron . (medscape.com)
- Toxicodendron dermatitis is the most common cause of contact dermatitis in the United States, exceeding all other causes combined. (medscape.com)
- Contact dermatitis from Rhus toxicodendron in a homeopathic remedy. (medscape.com)
- Botanical dermatology: allergic contact dermatitis. (medscape.com)
- The effectiveness of jewelweed, Impatiens capensis, the related cultivar I. balsamina and the component, lawsone in preventing post poison ivy exposure contact dermatitis. (medscape.com)
- Urushiol-induced contact dermatitis (also called Toxicodendron dermatitis or Rhus dermatitis) is a type of allergic contact dermatitis caused by the oil urushiol found in various plants, most notably sumac family species of the genus Toxicodendron: poison ivy, poison oak, poison sumac, and the Chinese lacquer tree. (wikipedia.org)
- As is the case with all contact dermatitis, urushiol-induced allergic rashes are a Type IV hypersensitivity reaction, also known as delayed-type hypersensitivity. (wikipedia.org)
- Urushiol-induced contact dermatitis is caused by contact with a plant or any other object containing urushiol oil. (wikipedia.org)
- Poison ivy and a related Toxicodendron species are considered a public health concern because they cause contact dermatitis, an inflammation of the skin caused by skin to plant contact (12). (cdc.gov)
- This makes up about 80% of all contact dermatitis cases and is the most common occupational skin disorder. (cdc.gov)
- Allergic contact dermatitis requires prior exposure to a plant chemical, like urushiol, which sensitizes the immune system (5). (cdc.gov)
- Poison ivy and its relatives, poison oak and poison sumac, are the most common cause of acute allergic contact dermatitis in the United States, estimated to affect 10-50 million Americans per year (1, 3). (cdc.gov)
- Here we provide a number of critical examples that range over various health concerns related to plant biology and climate change, including aerobiology, contact dermatitis, pharmacology, toxicology, and pesticide use. (nih.gov)
- However, if contact dermatitis is being considered, patch testing for other allergens might be considered. (medscape.com)
- And, like poison ivy, poison sumac will cause contact dermatitis as both plants contain urushiol in leaves, stems, and roots. (friendsofthefoxriver.org)
- For example, poison ivy ( Toxicodendron radicans ) poison oak ( Toxicodendron pubescens ) or poison sumac ( Toxicodendron vernix ) contain an oily organic allergen called urushiol that causes contact dermatitis. (botanical-online.com)
- Don't touch plants that contain products that can produce contact dermatitis. (botanical-online.com)
- Oleoresin, the allergen, causes a severe, delayed hypersensitivity or contact dermatitis 24 to 48 hours after contact with the plant. (pollenlibrary.com)
- What Is Contact Dermatitis? (allergystore.com)
- Contact dermatitis is the second most common dermatological diagnosis. (allergystore.com)
- A survey of medical professionals in 1995 found that contact dermatitis resulted in almost 8.4 million outpatient visits to medical facilities. (allergystore.com)
- With contact dermatitis, the resulting skin condition can last for days or weeks and will continue as long as the skin continues to be exposed to the irritant. (allergystore.com)
- Contact dermatitis can appear as a red rash. (allergystore.com)
- There are three major types of contact dermatitis: irritant, allergic, and photosensitive. (allergystore.com)
- As suggested by the name, irritant contact dermatitis occurs when a substance that causes irritation is exposed to the skin. (allergystore.com)
- In this type of contact dermatitis, the skin is actually injured. (allergystore.com)
- The hands are the most common site of irritant contact dermatitis. (allergystore.com)
- With this type of contact dermatitis, the immune system is involved and a true allergic reaction occurs. (allergystore.com)
- One of the most common causes of allergic contact dermatitis is the urushiol oil in the Toxicodendron family of plants. (allergystore.com)
- Clothing can also be a source of allergic contact dermatitis. (allergystore.com)
- Quaternium-15 is a widely used preservative as it is a formaldehyde releaser and is frequently the culprit in allergic contact dermatitis. (allergystore.com)
- This contact dermatitis occurs when a combination of ultraviolet light and a sensitizing agent causes a rash or blisters. (allergystore.com)
- Treatment of mild contact dermatitis can be done at home. (allergystore.com)
- If the contact dermatitis is caused by an allergic reaction oral antihistamines will help. (allergystore.com)
- Antihistamines will not help with an irritant or photosensitive contact dermatitis. (allergystore.com)
- An allergic contact dermatitis caused by exposure to plants of the genus Toxicodendron (formerly Rhus). (bvsalud.org)
- Exposure to plants of the Anacardiaceae family account for the majority of cases of allergic contact dermatitis. (orentreich.com)
- Contact dermatitis is inflammation of the skin caused by direct contact with irritants (irritant contact dermatitis) or allergens (allergic contact dermatitis). (msdmanuals.com)
- Contact dermatitis is caused by irritants or allergens. (msdmanuals.com)
- To cite this article: Martin SF, Esser PR, Weber FC, Jakob T, Freudenberg MA, Schmidt M, Goebeler M. Mechanisms of chemical-induced innate immunity in allergic contact dermatitis. (cdc.gov)
- Allergic contact dermatitis - hypersensitivity induced obvious consequences. (cdc.gov)
- Contact allergen-induced innate immunity contrast to irritant contact dermatitis (ICD), which evolves emollients may help reduce irritancy and stabilize the barrier as a consequence of direct toxic effects of physical or chemi- function of the skin avoiding the penetration of contact aller- cal agents resulting in keratinocyte damage and local inflam- gens. (cdc.gov)
- Coming into contact with urushiol causes contact dermatitis, which is a type of allergic skin reaction. (medicalnewstoday.com)
- The efficacy of orange jewelweed (I. capensis) and yellow jewelweed (I. pallida) in preventing poison ivy contact dermatitis has been studied, with conflicting results. (findmeacure.com)
- A 2012 study found that while an extract of orange jewelweed and garden jewelweed (I. balsamina) was not effective in reducing contact dermatitis, a mash of the plants applied topically decreased it. (findmeacure.com)
- The rash is considered considered to be contact dermatitis. (cloverleaffarmblog.com)
- but The hallmark symptoms of hair-dye related contact dermatitis are redness, itchiness, and rash on the face, eyelids, ears, and neck. (ikuttab.com)
Radicans7
- As the name implies, poison ivy (Toxicodendron radicans) usually grows as an ivy. (health.mil)
- They are also used after poison ivy ( Toxicodendron radicans ) contact to prevent a rash from developing. (findmeacure.com)
- That is not how poison oak ( Toxicodendron diversilobum ) looks like, that is how poison ivy ( Toxicodendron radicans ) looks like. (botanicalaccuracy.com)
- Poison ivy (Toxicodendron radicans) is a plant that contains the oil urushiol. (cloverleaffarmblog.com)
- Toxicodendron radicans, commonly known as poison ivy, affects more than 350,000 people annually in the U.S. A rash from poison ivy, poison oak, or poison sumac is caused by an oil found in these plants called urushiol (you-ROO-shee-all). (healthline.com)
- Poison ivy, (Toxicodendron radicans), also called eastern poison ivy, poisonous vine or shrub of the cashew family (Anacardiaceae), native to eastern North America. (alexanderbygg.com)
- Poison ivy Toxicodendron radicans. (alexanderbygg.com)
Allergic5
- Plant dermatitis is an allergic inflammatory skin reaction in response to the oils of poisonous plants. (health.mil)
- This is not to be confused with photosensitive allergic dermatitis that is caused purely by exposure to ultraviolet light and does not require contact with any substance. (allergystore.com)
- Treatment of toxicodendron dermatitis varies due to the severity of the allergic rash. (panamacitymfm.com)
- That's when you get that allergic dermatitis," said Waldorf. (healthline.com)
- When people come into contact with poison ivy, urushiol can cause an allergic skin reaction that results in a reddish, itchy and painful inflammation of the skin (dermatitis). (andalusian-usa.com)
Genus6
- Rapid detection of urushiol allergens of Toxicodendron genus using leaf spray mass spectrometry. (medscape.com)
- In the U.S., the most common dermatitis-causing plant genus is Toxicodendron (formerly Rhus ) (i.e., poison ivy, poison oak, and poison sumac). (health.mil)
- Poison sumac is from the same family as the other sumacs, but belongs to a different genus, Toxicodendron . (friendsofthefoxriver.org)
- Poison-Ivy, Poison-Oak, Poison-Sumac (Toxicodendron) is a genus of the ANACARDIACEAE family. (pollenlibrary.com)
- These include poison ivy, poison oak and poison sumac, which are members of the genus Toxicodendron. (orentreich.com)
- ETHNOPHARMACOLOGICAL RELEVANCE: Comprising of about 30 species, the genus Toxicodendron (Anacardiaceae) are mainly distributed in East Asia and North America. (bvsalud.org)
Poison oak5
- This article reviews the current fund of knowledge on poison ivy, poison oak, and poison sumac dermatitis. (nih.gov)
- Poison ivy ( Toxicodendron rydbergii ), poison oak ( Toxicodendron diversilobum ), and poison sumac ( Toxicodendron vernix ) are most common in North America. (medscape.com)
- Canavan D, Yarnell E. Successful treatment of poison oak dermatitis treated with Grindelia spp. (medscape.com)
- Poison sumac , or Toxicodendron vernix , is more closely related to poison ivy and poison oak than other sumac plants. (medicalnewstoday.com)
- Generally speaking, if you are on the west coast of North America you have to look out for poison oak, and if you are east of the Rocky Mountains and in the eastern part of the United States, you better learn quickly how poison ivy looks like.CDC has a good overview of the different toxic North American species in Toxicodendron . (botanicalaccuracy.com)
Irritant dermatitis2
- Irritant dermatitis can happen when an irritating chemical in the plant makes contact with the skin. (cdc.gov)
- Phytophotodermatitis , the most common form of irritant dermatitis, occurs when a chemical in a plant (called a phototoxin) gets on the skin and then reacts with ultraviolet (UV) light from the sun. (cdc.gov)
Rhus Dermatitis1
- Gach JE, Tucker W, Hill VA. Three cases of severe Rhus dermatitis in an English primary school. (medscape.com)
Species10
- Although technically not Toxicodendron species, mangoes and Japanese lacquer trees also contain urushiol and can incite a similar clinical picture. (medscape.com)
- Toxicodendron species contain oleoresins known collectively as urushiol. (medscape.com)
- Toxicodendron species are abundant throughout the United States except in desert areas, elevations above 4000 ft, Alaska, and Hawaii. (medscape.com)
- And the search terms "Toxicodendron" and "the names of 31 species and their synonyms" were used to search for information from electronic databases such as Web of Science, Scopus, Google Scholar, Science Direct, PubMed, Baidu Scholar, Springer, and Wiley Online Library. (bvsalud.org)
- RESULTS: These species on Toxicodendron are widely used in folkloric medicine and modern pharmacological activities. (bvsalud.org)
- CONCLUSIONS: Selected species of Toxicodendron have been used as herbal medicines in the Southeast Asian for a long time. (bvsalud.org)
- A study in 1958 found that Impatiens biflora was an effective alternative to standard treatment for dermatitis caused by contact with sumac, while later studies found that the species had no antipruritic effects after the rash has developed. (findmeacure.com)
- There are also several other species of Toxicodendron in North America, all of them toxic. (botanicalaccuracy.com)
- Both species are toxic and give horrible dermatitis, but they occur in different parts of the country. (botanicalaccuracy.com)
- The dermatitis caused by these species can be very severe, and this is a plant group we all should be able to identify. (botanicalaccuracy.com)
Diversilobum2
- Toxicodendron diversilobum, single grain. (pollenlibrary.com)
- Toxicodendron diversilobum, multiple grains. (pollenlibrary.com)
Eczema1
- Seborrheic Dermatitis or Seborrheic Eczema is an inflammatory disorder, particularly affecting the scalp, face and the ba. (ayurvediccure.com)
North America1
- Toxicodendron dermatitis occurs outside North America. (medscape.com)
Urushiol-induced1
- The American Academy of Dermatology estimates that there are up to 50 million cases of urushiol-induced dermatitis annually in the United States alone, accounting for 10% of all lost-time injuries in the United States Forest Service. (wikipedia.org)
Allergens1
- However, the most prevalent form of plant dermatitis worldwide occurs from exposure to the numerous members of the family Compositae and varied sesquiterpene lactone allergens from these plants. (medscape.com)
Homeopathic1
- Prevention of poison ivy dermatitis with oral homeopathic Rhus toxicodendron. (nih.gov)
Mass spectrometry1
- Urushiol compounds detected in Toxicodendron-labeled consumer products using mass spectrometry. (cdc.gov)
20231
- Therefore, this review aims to provide a reference for further research and development on medicinal purpose of Toxicodendron by summarizing the works (from 1980 to 2023), and focusing on its botany, traditional uses, phytochemistry and pharmacology. (bvsalud.org)
Occurs2
- Mechanical dermatitis occurs when the skin is cut, punctured, or abraded by thorns, spines, and hairy appendages of plants that result in secondary infections. (cdc.gov)
- Phototoxic dermatitis, therefore, occurs only in UV-exposed skin, typically with a sharp demarcation. (msdmanuals.com)
Rashes1
- Rashes and other symptoms that is caused by the plant Toxicodendron radicus, is commonly known as poison ivy. (ayurvediccure.com)
Rash1
- Once the urushiol touches your skin, it quickly penetrates and binds, causing a rash known as toxicodendron dermatitis. (panamacitymfm.com)
Exposure to poison ivy1
- Uncommonly, dermatitis from exposure to poison ivy may include black spots. (medscape.com)
20201
- Incidence of plant dermatitis remained stable between 2010 and 2015, increased in 2016 and 2017, then decreased in 2018 through 2020. (health.mil)
Plant3
- According to the 2018 Annual Report of the American Association of Poison Control Centers' National Poison Data System, skin irritation from Toxicodendron was the 6th most common plant exposure, accounting for 1015 cases. (medscape.com)
- Plant dermatitis can cause significant disability and lost duty time particularly in highly sensitized individuals. (health.mil)
- Plant dermatitis can cause significant disability, substantial medical costs, and lost duty time. (health.mil)
Dermatology1
- However, in clinical dermatology, dermatitis is used to describe a variety of different skin conditions that share the same. (msdmanuals.com)
Plants8
- The author describes symptoms, assessment methods, and differential diagnoses, as well as provides a brief review of pathologic and etiologic factors, epidemiology, and treatment approaches for cutaneous dermatitis caused by contact with poison ivy and related plants. (jcadonline.com)
- With increasing global travel and transport of plants, true toxicodendron dermatitis is being increasingly reported in Europe, although it is still case reportable. (medscape.com)
- Dermatitis (skin inflammation) caused by the oil of poisonous plants is an occupational hazard for U.S. military members who are assigned and train in endemic areas. (health.mil)
- The leaves, fruit, bark, or woody portion of plants and trees can touch the skin, resulting in mild to severe dermatitis. (cdc.gov)
- The classic allergenic dermatitis of these plants is caused by urushiol. (cdc.gov)
- Relatively few poisonous plants are largely responsible for many people's aversion/fear of nature for example Poison Ivy/Sumac/Oak ( Toxicodendron spp. (botanyeveryday.com)
- Among them, phenolic acids and flavonoids are the main compound classes that show pharmacological activities in Toxicodendron plants both in vitro and in vivo. (bvsalud.org)
- Therefore, in this review, the traditional medicinal, phytochemical and modern pharmacology of Toxicodendron plants are summarized to help future researchers to find new drug leads or to get a better understanding of structure-activity relationships. (bvsalud.org)
Severe1
- Milder cases of the dermatitis can last five to 12 days, while a more severe case may take several weeks to resolve. (panamacitymfm.com)
Susceptible2
- Exposure to unroasted cashew nut shells can cause a dermatitis often confused with toxicodendron dermatitis in susceptible individuals. (medscape.com)
- 1 Approximately 50%-75% of the U.S. adult population are susceptible to skin reactions upon exposure to Toxicodendron oil or oleoresin, called urushiol. (health.mil)
Pharmacology1
- The existing research on Toxicodendron has been reviewed, and the phytochemistry and pharmacology provide theoretical basis for some of the traditional medicinal uses. (bvsalud.org)
Occupational1
- Occupational poison ivy and oak dermatitis. (nih.gov)
Acute1
- A case of acute dermatitis caused by handling the Rhus toxicodendron. (nih.gov)
Skin3
- The affected cells then migrate to lymph nodes, where T cells become activated, then return to the skin where they stimulate an urushiol-activated dermatitis. (wikipedia.org)
- Definition of Dermatitis The meaning of the word "dermatitis" is inflammation of the skin. (msdmanuals.com)
- But even if we consider only one type, the atopic dermatitis , which is the most common type of skin condition, everyone is affected in different ways in different degrees, as I have seen in my practice. (homeopathyheal.co.uk)
Unusual1
- A case of poisoning by Rhus toxicodendron acquired in an unusual manner. (nih.gov)
Prevention2
- Post-exposure prevention of toxicodendron dermatitis with early forceful unidirectional washing. (medscape.com)
- Stibich AS, Yagan M, Sharma V, Herndon B, Montgomery C. Cost-effective post-exposure prevention of poison ivy dermatitis. (medscape.com)
Leaf1
- Ampelopsis trifolia - Three-leaf Woodbine - (Toxic dermatitis due to vegetable poisons - 30 and 200. (abchomeopathy.com)
Severity1
- Due to the severity of pruritus, the patient sought care at a local urgent care clinic and was diagnosed with dermatitis with secondary infection. (jcadonline.com)
Biology1
- Toxicodendron characteristics, morphology, and biology are reviewed. (nih.gov)
Common1
- Dermatitis from poison sumac is less common. (wikipedia.org)
Therapeutic1
- BACKGROUND: The purpose of the present research was to explore the therapeutic impact of raw lacquer extract from Toxicodendron vernicifluum on colorectal cancer cells and to investigate the outcome of raw lacquer extract and ONC201 co-treatment on the activity of colorectal cancer cells. (bvsalud.org)