Crohn Disease
Ileum
Differential inflammatory modulation of canine ileal longitudinal and circular muscle cells. (1/218)
The aim of this study was to identify the subtypes of muscarinic receptors that mediate in vivo and in vitro canine ileal longitudinal muscle contractions and whether their role is modulated by inflammation. Previous studies have reported that circular muscle contractions are suppressed in ileal inflammation induced by mucosal exposure to ethanol and acetic acid. We found that inflammation had no significant effect on in vivo and in vitro spontaneous or muscarinic receptor-mediated contractions of the longitudinal muscle. The longitudinal muscle contractions were mediated primarily by the M(3) receptor subtype. However, the IC(50) of the M(2) receptor antagonist methoctramine was only 10 times greater than that of the M(3) receptor antagonist 4-DAMP in the longitudinal muscle, whereas it was 224 times greater in the circular muscle. M(2) receptor-coupled decrease of intracellular cAMP occurred in the longitudinal but not in the circular muscle from the normal ileum. Inflammation did not alter this coupling in the longitudinal muscle but established it in the circular muscle. In conclusion, M(2) receptors may play a greater role in the mediation of longitudinal muscle contractions than circular muscle contractions. Inflammation does not alter the contractility or the relative role of muscarinic receptor subtypes in longitudinal muscle cells. However, it modulates the M(2) receptor coupling to adenylate cyclase in the circular muscle. (+info)Characterization of M cell development during indomethacin-induced ileitis in rats. (2/218)
BACKGROUND: M cells play an important role in the intestinal immune system as they have a high capacity for transcytosis of a wide range of microorganisms and macromolecules. However, little is known about the role of M cells during intestinal inflammation. AIM: We studied M cell development during indomethacin-induced intestinal inflammation in rats. METHODS: Ileitis in rats was induced by two subcutaneous injections with indomethacin (7.5 mg/kg) given 24 h apart. Rats were sacrificed after 14 days and tissue was analysed by fluorescence microscopy and electron microscopy. M cells could be visualized by using the FITC-labelled mAb anti-cytokeratin (CK)-8 (clone 4.1.18), which was recently identified as specific M cell marker in rats. The number of cytokeratin-8 positive M cells was related to the surface of the follicle associated epithelium. For morphological studies, we used both transmission electron microscopy (T.E.M.) and scanning electron microscopy (S.E.M.). RESULTS: In non-inflamed ileum M cells were scarce. Only 4% of the follicle associated epithelium were M cells, whereas an increase of M cells up to 11% was found in inflamed follicle associated epithelium (P < 0.001). The rate of M cell induction depended on the macroscopic degree of inflammation. T.E.M./S.E.M. studies showed that in inflamed tissue most M cells underwent apoptosis with typical morphological signs. In contrast to apoptotic M cells, the neighbouring enterocytes usually appeared intact. The number of mononuclear cells below the follicle associated epithelium was significantly increased. S.E.M. studies revealed that during induced ileitis mononuclear cells migrated from the lamina propria into the gut lumen by passing through apoptotic M cells. CONCLUSIONS: During indomethacin-induced ileitis in rats the increase in M cell number in association with apoptosis of M cells may alter the intestinal barrier function. These observations may play a pivotal role in the pathogenesis of chronic intestinal inflammation, e.g. in inflammatory bowel disease. (+info)Mechanism of glucocorticoid-mediated reversal of inhibition of Cl(-)/HCO(-)(3) exchange during chronic ileitis. (3/218)
In the normal ileum, coupled NaCl absorption occurs via the dual operation of Na(+)/H(+) and Cl(-)/HCO(-)(3) exchange on the brush-border membrane (BBM) of villus cells. In a rabbit model of chronic small intestinal inflammation we determined the cellular mechanism of inhibition of NaCl absorption and the effect of steroids on this inhibition. Cl(-)/HCO(-)(3) but not Na(+)/H(+) exchange was reduced in the BBM of villus cells during chronic ileitis. Cl(-)/HCO(-)(3) exchange was inhibited secondary to a decrease in the affinity for Cl(-) rather than an alteration in the maximal rate of uptake of Cl(-) (V(max)). Methylprednisolone (MP) stimulated Cl(-)/HCO(-)(3) exchange in the normal ileum by increasing the V(max) of Cl(-) uptake rather than altering affinity for Cl(-). MP reversed the inhibition of Cl(-)/HCO(-)(3) exchange in rabbits with chronic ileitis. However, MP alleviated the Cl(-)/HCO(-)(3) exchange inhibition by restoring the affinity for Cl(-) rather than altering the V(max) of Cl(-) uptake. These data suggest that glucocorticoids mediate the alleviation of Cl(-)/HCO(-)(3) exchange inhibition in chronically inflamed ileum by reversing the same mechanism that was responsible for inhibition of this transporter rather than exerting a direct effect on the transporter itself, as was the case in normal ileum. (+info)Effect of chronic inflammation on ileal short-chain fatty acid/bicarbonate exchange. (4/218)
Short-chain fatty acids (SCFA) have been demonstrated to at least partially ameliorate chronic intestinal inflammation. However, whether and how intestinal SCFA absorption may be altered during chronic intestinal inflammation is unknown. A rabbit model of chronic ileitis produced by coccidia was used to determine the effect of chronic inflammation on ileal SCFA/HCO(-)(3) exchange. SCFA/HCO(-)(3) exchange was present in the brush-border membrane (BBM) of villus but not crypt cells from normal rabbit ileum. An anion-exchange inhibitor, DIDS, significantly inhibited SCFA/HCO(-)(3) exchange. Extravesicular Cl(-) did not alter the uptake of SCFA, suggesting that SCFA/HCO(-)(3) exchange is a transport process distinct from Cl(-)/HCO(-)(3) exchange. In chronically inflamed ileum, SCFA/HCO(-)(3) exchange was also present only in BBM of villus cells. The exchanger was sensitive to DIDS and was unaffected by extravesicular Cl(-). However, SCFA/HCO(-)(3) exchange was significantly reduced in villus cell BBM vesicles (BBMV) from chronically inflamed ileum. Kinetic studies demonstrated that the maximal rate of uptake of SCFA, but not the affinity for SCFA, was reduced in chronically inflamed rabbit ileum. These data demonstrate that a distinct SCFA/HCO(-)(3) exchange is present on BBMV of villus but not crypt cells in normal rabbit ileum. SCFA/HCO(-)(3) exchange is inhibited in chronically inflamed rabbit ileum. The mechanism of inhibition is most likely secondary to a reduction in transporter numbers rather than altered affinity for SCFA. (+info)Lessons from genetically engineered animal models XI. Novel mouse models to study pathogenic mechanisms of Crohn's disease. (5/218)
Crohn's Disease (CD) affects more than 500,000 individuals in the United States and represents the second most common chronic inflammatory disorder after rheumatoid arthritis. Although major advances have been made in defining the basic mechanisms underlying chronic intestinal inflammation, the precise etiopathogenesis of CD remains unknown. We have recently characterized two novel mouse models of enteritis that express a CD-like phenotype, namely the TNF DeltaARE model of tumor necrosis factor (TNF) overexpression and the SAMP1/Yit model of spontaneous ileitis. The unique feature of these models is that they closely resemble CD for location and histopathology. These genetically manipulated new models of intestinal inflammation offer a powerful tool to investigate potential causes of human disease and may allow the development of novel disease-modifying therapeutic modalities for the treatment of CD. (+info)Advances in gastroenterology and hepatology. (6/218)
This is a review of some of the most important growing points in the specialties of gastroenterology and hepatology. It does not aim to be completely comprehensive but to pick out major areas of importance to examination candidates and doctors without special experience in the field. Topics covered include: upper gastrointestinal haemorrhage; Barrett's oesophagus; carcinoma of the oesophagus; achalasia; Helicobacter pylori; duodenal ulcer prevention; coeliac disease; dermatitis herpetiformis; Crohn's disease; small bowel overgrowth; ulcerative colitis; carcinoma of the large bowel; obesity; endoscope sterilisation; gall stones; liver transplantation; autoimmune liver disease; viral hepatitis; metabolic liver diseases; and pancreatic insufficiency. (+info)Focal up-regulation of E-cadherin-catenin complex in inflamed bowel mucosa but reduced expression in ulcer-associated cell lineage. (7/218)
The E-cadherin-catenin complex is important for the maintenance of epithelial architecture. We studied its expression in Crohn disease, ulcerative colitis, acute ileitis, and controls. Immunohistochemical stainings for E-cadherin, alpha-catenin, beta-catenin and gamma-catenin were performed. E-cadherin messenger RNA (mRNA) was detected using riboprobes. In active inflammation, there was up-regulation of the complex. In particular, epithelium adjacent to ulcers showed increased expression of protein and mRNA, but in ulcer-associated cell lineage, the intensity of staining was weak to negative. In focal inflammation, up-regulation was found in affected areas. Reparative epithelium growing over denuded areas showed weaker expression. Since structural or functional perturbation in any of the molecules of the E-cadherin-catenin complex results in loss of intercellular adhesion, the preexistent epithelium may benefit from up-regulation to try to maintain its normal architecture under inflammatory conditions. Reduced expression in reparative epithelium and ulcer-associated cell lineage could facilitate the motility of these cells. (+info)Consequences of intestinal inflammation on the enteric nervous system: neuronal activation induced by inflammatory mediators. (8/218)
The ENS is responsible for the regulation and control of all gastrointestinal functions. Because of this critical role, and probably as a consequence of its remarkable plasticity, the ENS is often relatively well preserved in conditions where the architecture of the intestine is seriously disrupted, such as in IBD. There are structural and functional changes in the enteric innervation in animal models of experimental intestinal inflammation and in IBD. These include both up and down regulation of transmitter expression and the induction of new genes in enteric neurons. Using Fos expression as a surrogate marker of neuronal activation it is now well established that enteric neurons (and also enteric glia) respond to inflammation. Whether this "activation" is limited to a short-term functional response, such as increased neuronal excitability, or reflects a long-term change in some aspect of the neuronal phenotype (or both) has yet to be firmly established, but it appears that enteric neurons are highly plastic in their response to inflammation. (+info)Ileitis is a medical term that refers to inflammation of the ileum, which is the last part of the small intestine. The condition can have various causes, including infections, autoimmune disorders, and inflammatory bowel diseases such as Crohn's disease.
The symptoms of ileitis may include abdominal pain, diarrhea, fever, weight loss, and nausea or vomiting. The diagnosis of ileitis typically involves a combination of medical history, physical examination, laboratory tests, and imaging studies such as CT scans or MRI.
Treatment for ileitis depends on the underlying cause of the inflammation. In cases of infectious ileitis, antibiotics may be used to treat the infection. For autoimmune or inflammatory causes, medications that suppress the immune system may be necessary to reduce inflammation and manage symptoms.
In severe cases of ileitis, surgery may be required to remove damaged portions of the intestine or to drain abscesses. It is important to seek medical attention if you experience symptoms of ileitis, as early diagnosis and treatment can help prevent complications and improve outcomes.
Crohn's disease is a type of inflammatory bowel disease (IBD) that can affect any part of the gastrointestinal tract, from the mouth to the anus. It is characterized by chronic inflammation of the digestive tract, which can lead to symptoms such as abdominal pain, diarrhea, fatigue, weight loss, and malnutrition.
The specific causes of Crohn's disease are not fully understood, but it is believed to be related to a combination of genetic, environmental, and immune system factors. The disease can affect people of any age, but it is most commonly diagnosed in young adults between the ages of 15 and 35.
There is no cure for Crohn's disease, but treatments such as medications, lifestyle changes, and surgery can help manage symptoms and prevent complications. Treatment options depend on the severity and location of the disease, as well as the individual patient's needs and preferences.
The ileum is the third and final segment of the small intestine, located between the jejunum and the cecum (the beginning of the large intestine). It plays a crucial role in nutrient absorption, particularly for vitamin B12 and bile salts. The ileum is characterized by its thin, lined walls and the presence of Peyer's patches, which are part of the immune system and help surveil for pathogens.
I'm sorry for any confusion, but "Trinitrobenzenesulfonic Acid" is not a medical term. It is an organic compound used in industrial and research applications, such as a reagent in chemical reactions. Its formula is C6H3N3O9S. If you have any questions about chemical compounds or scientific terms, I'd be happy to try to help with those!
Mesenteric lymphadenitis is a condition characterized by inflammation of the lymph nodes in the mesentery, which is the membrane that attaches the intestine to the abdominal wall. These lymph nodes are part of the immune system and help fight infection.
Mesenteric lymphadenitis can be caused by a variety of factors, including bacterial or viral infections, inflammatory bowel disease, or autoimmune disorders. In many cases, however, a specific cause cannot be identified. Symptoms may include abdominal pain, fever, nausea, vomiting, and diarrhea.
In most cases, mesenteric lymphadenitis is a self-limiting condition, which means that it will resolve on its own without treatment. However, in some cases, antibiotics may be necessary to treat an underlying infection. In rare cases, surgery may be required to remove severely inflamed or infected lymph nodes.
Paneth cells are specialized epithelial cells located in the small intestine, specifically in the crypts of Lieberkühn. They play an essential role in the immune function and maintenance of the intestinal environment. Paneth cells are characterized by their large, granulated secretory vesicles that contain antimicrobial peptides and proteins, such as defensins and lysozyme. These substances help to control the growth of bacteria in the small intestine and maintain a balanced microbiota. Additionally, Paneth cells secrete other factors that support the function and survival of stem cells located in the crypts. They are also involved in the inflammatory response by producing cytokines and chemokines, which help to recruit immune cells to the site of infection or injury.
Ileitis
James S. Jeffrey
Burrill Bernard Crohn
Inflammatory bowel disease
Dwight D. Eisenhower
1932 in science
Ulcerative colitis
Pernik
Wet-tail
Necroptosis
Campylobacter hyointestinalis
Fulminant
Martin Hume Johnson
Gastrointestinal wall
Chlamydia muridarum
Lloyd Mayer
Gastrointestinal tract
CircoFLEX
PLA2G1B
Pattern recognition receptor
Leon Ginzburg
Yersinia enterocolitica
Enteritis
Valnemulin
Equine proximal enteritis
Gastrointestinal disease
Crohn's disease
Nutritional neuroscience
Nils Kock
Anna Mae Hays
Ileitis - Wikipedia
Ileitis strikes high performing herds | The Pig Site
Ileitis - causes, side effects and treatments at NaturalPedia.com
Necrotic Enteritis (NE) / Necrotic Ileitis - Pig Progress
Dysbiotic gut microbiota causes transmissible Crohn's disease-like ileitis independent of failure in antimicrobial defence | Gut
Image: Proliferative ileitis, gross lesions, hamster - MSD Veterinary Manual
Medical, Ileitis and Colitis Foundation?
History and Etiology of ileitis - Lawsonia
Ultrasound assessment of acute appendicitis in paediatric patients: methodology and pictorial overview of findings seen |...
5 Types of Crohn's Disease: Ileocolitis, Jejunoileitis, and More
Backwash Ileitis Medical Definition | Day of Difference
Improving Ileitis and Swine Dysentery management in fattening pigs
Pouchitis - Medical Dictionary online-medical-dictionary.org
View of A case of terminal ileitis that was not Crohn's disease
Erythrocyte sedimentation as a measure of crohn's disease activity: Opposite trends in ileitis versus colitis<...
Crohn disease: MedlinePlus Medical Encyclopedia
SAMP1/YitFc mice develop ileitis via loss of CCL21 and defects in dendritic cell migration
Crohn's Disease: Symptoms, causes, diagnosis, and more
British Medical Journal (Clinical research ed.): 283 (6305) | The BMJ
Dermatologic Manifestations of Pellagra: Practice Essentials, Pathophysiology, Etiology
Nutrients | Free Full-Text | The Effects of Commonly Consumed Dietary Fibres on the Gut Microbiome and Its Fibre Fermentative...
Gastroduodenal Crohn's Disease: Causes and Treatment
Bone mineral density in patients with Crohn's disease during long-term treatment with azathioprine
Table 2 - Isolation of Waddlia malaysiensis, A Novel Intracellular Bacterium, from Fruit Bat (Eonycteris spelaea) - Volume 11,...
Michael V. Chiorean, MD | Issaquah, WA
Volume 16(7) 2010 (23)
Laparoscopic ileocaecal resection versus infliximab for terminal ileitis in Crohn's disease: a randomised controlled, open...
Javier Herrera, MD | Edmonds, WA
products - animal-health-online.com
Crohn's8
- Crohn's ileitis is a type of Crohn's disease affecting the ileum. (wikipedia.org)
- If the inflammation is more severe and affects both the small and large intestines, it is known as regional ileitis or Crohn's disease. (naturalpedia.com)
- Backwash ileitis is caused by reflux of colonic contents, and when present, may raise the differential diagnosis of Crohn's disease. (dayofdifference.org.au)
- We demonstrate that CCL21 is almost completely absent from SAMP1/YitFc (SAMP) mice, which spontaneously develop chronic ileitis that resembles Crohn's disease, and that DC migration is severely impaired in these mice compared with AKR mice (controls). (iucc.ac.il)
- Laparoscopic ileocaecal resection versus infliximab for terminal ileitis in Crohn's disease: a randomised controlled, open-label, multicentre trial. (bvsalud.org)
- Crohn's disease, also called regional enteritis or ileitis, is a lifelong form of inflammatory bowel disease (IBD) . (clevelandclinic.org)
- Man who underwent Roux-en-Y gastric bypass at age 26, subsequently diagnosed with Crohn's disease, involving jejunum, ileum and colon, at age 42 when he presented with large volume diarrhoea and rectal bleeding. (medscape.com)
- Innate dysfunction promotes linear growth failure in pediatric Crohn's disease and growth hormone resistance in murine ileitis. (cdc.gov)
Ileum5
- Ileitis is an inflammation of the ileum, a portion of the small intestine. (wikipedia.org)
- Ileitis is a chronic inflammation of one or more sections of the intestine , particularly in the lower section of the small intestine known as ileum. (naturalpedia.com)
- Like ileocolitis, ileitis causes inflammation and irritation of the ileum, but it doesn't affect the colon. (healthline.com)
- Backwash ileitis refers to inflammation in the distal few centimeters of terminal ileum in patients with ulcerative colitis 13). (dayofdifference.org.au)
- Inflammation of the ileum is called ileitis. (msdmanuals.com)
Proliferative Enteropathy3
- Most cases are a stage in Proliferative Enteropathy (PE, ileitis), which can also cause regional ileitis (RI), porcine intestinal adenopathy (PIA) and proliferative haemorrhagic enteropathy (PHE). (pigprogress.net)
- Porcine proliferative enteropathy (PPE), also known as ileitis, is an infectious enteric disease caused by the obligate intracellular bacterium Lawsonia intracellularis . (lawsonia.net)
- Also called Porcine Proliferative Enteropathy (PPE), ileitis is an infectious enteric disease. (norfeed.net)
Inflammatory2
- The known side effects of ileitis include diarrhea, weight loss, cramping or pain in the middle or lower right part of the abdomen, and inflammatory abscesses and fistulas in the lower right part of the abdomen. (naturalpedia.com)
- But people with ileitis may also develop fistulas (inflammatory abscesses) in the lower-right section of the abdomen. (healthline.com)
Ileocolitis1
- The symptoms of ileitis are the same as those of ileocolitis. (healthline.com)
Terminal2
- I have erosive terminal ileitis along with other conditions. (crohnsforum.com)
- Abdominal tomography (CT) was performed and showed terminal ileitis (Figure 1(a) ). (hindawi.com)
Prevalence1
- However, a systematic analysis of ileal changes in UC has never been performed, and the prevalence and criteria for 'backwash' ileitis have not been defined. (dayofdifference.org.au)
Acute2
- The symptoms of ileitis in pigs are diverse, and include hemorrhagic or acute, chronic and subclinical. (lawsonia.net)
- 6011' 0138 1t+ showed histologic signs of an acute process unlike changes characteristic for proliferative ileitis. (utlib.ee)
Colitis3
- Medical, Ileitis and Colitis Foundation? (science-bbs.com)
- National Foundation for Ileitis and Colitis, Inc. (science-bbs.com)
- Backwash ileitis is seen in patients with ulcerative colitis (UC), where the entire colon is involved. (dayofdifference.org.au)
Symptoms1
- Nonetheless, there are some foods to avoid when a person has ileitis to avoid worsening its symptoms. (naturalpedia.com)
Diagnosis1
- Most cases were women, had Roux-en-Y surgery years prior to diagnosis and few IBD-related complications. (medscape.com)
Ileal2
- Backwash ileitis refers to ileal involvement of UC secondary to incompetence of the ileocecal valve, usually in the context of pancolitis. (dayofdifference.org.au)
- Structural lesions leading to vitamin B 12 deficiency include regional ileitis, ileal resections, malignancies, etc. (drugs.com)
Lawsonia1
- Ileitis is caused by the bacterium Lawsonia intracellularis. (wikipedia.org)
Mice5
- Mice were fed R848 to determine its effects on migration of DCs and development of ileitis in SAMP mice. (iucc.ac.il)
- Young SAMP mice had reduced CCL21 expression and decreased DC migration before developing ileitis. (iucc.ac.il)
- Administration of R848 to adult SAMP mice increased migration of DC to the MLN and development of regulatory T cells there, and reduced the severity of ileitis. (iucc.ac.il)
- Conclusions Loss of CCL21 signaling and DC migration is required for development of ileitis in SAMP mice. (iucc.ac.il)
- Experimental results indicate that luminal iron deprivation in combination with systemic iron repletion inhibited the development of inflammation in TNFΔARE/WT mice, resembling human CD-like ileitis. (tum.de)
Disease1
- The disease is caused by Brachyspira hyodyse nteriae and Brachyspira pilosicoli and, like Ileitis, contamination is known to happen orally and spread to the environment via the infected feces. (norfeed.net)
Digestive2
- The main body system harmed by ileitis is the digestive system as it is a problem in the intestines. (naturalpedia.com)
- Ileitis harms the digestive system. (naturalpedia.com)
Pathogenesis1
- The aim of the present work was the characterization of iron as environmental factor in the pathogenesis of CD-like ileitis. (tum.de)
Generally1
- Generally, ileitis can affect anyone regardless of age and gender. (naturalpedia.com)
Medical3
- At dayofdifference.org.au you will find all the information about Backwash Ileitis Medical Definition. (dayofdifference.org.au)
- Found info on Backwash Ileitis Medical Definition? (dayofdifference.org.au)
- We hope that you have found the information about Backwash Ileitis Medical Definition that interests you. (dayofdifference.org.au)
Skip1
- Backwash ileitis extends contiguously backward from the cecum without skip regions. (dayofdifference.org.au)
Loss1
- However, ileitis often strikes well managed operations causing production variation, loss of performance and lower output. (thepigsite.com)
Results1
- The results suggest that simultaneous infection with C. jejuni and E. coli is not the cause of pro: literative ileitis in hamsters. (utlib.ee)
Terminal ileitis9
- Yersinia enterocolitica (see the image below) is a bacterial species in the family Enterobacteriaceae that most often causes enterocolitis, acute diarrhea, terminal ileitis, mesenteric lymphadenitis, and pseudoappendicitis but, if it spreads systemically, can also result in fatal sepsis. (medscape.com)
- As a human pathogen, Y enterocolitica is most frequently associated with enterocolitis, acute diarrhea, terminal ileitis, mesenteric lymphadenitis, and pseudoappendicitis,[1] with the spectrum of disease ranging from asymptomatic to life-threatening sepsis, especially in infants. (medscape.com)
- Prevalence and Progression of Incidental Terminal Ileitis on Non-diagnostic Colonoscopy: A Systematic Review and Meta-analysis. (bvsalud.org)
- Incidentally diagnosed terminal ileitis [IDTI] has been reported among asymptomatic persons undergoing non-diagnostic colonoscopy . (bvsalud.org)
- The purpose of our study was to determine the prevalence and long-term outcomes of asymptomatic terminal ileitis . (bvsalud.org)
- Further long-term follow-up studies are needed to inform the natural history of incidental terminal ileitis , factors that predict progression to CD, and therapeutic implications. (bvsalud.org)
- Revised Algorithmic Approach to Differentiate Between Nonspecific and Specific Etiologies of Chronic Terminal Ileitis. (amedeo.com)
- Accuracy values of CEUS and MRE for the presence of active terminal ileitis were evaluated using the Receiver Operating Characteristic method, using endoscopic findings as a reference standard. (plos.org)
- Silverstein J, Grand D, Kawatu D, Shah SA, Steinkeler J, LeLeiko N. Feasibility of using MR enterography for the assessment of terminal ileitis and inflammatory activity in children with Crohn disease. (uamshealth.com)
Backwash ileitis2
- However, a systematic analysis of ileal changes in UC has never been performed, and the prevalence and criteria for "backwash" ileitis have not been defined. (nih.gov)
- If this does develop, due to pancolitis (the so-called backwash ileitis), differentiation becomes difficult. (diagnosticimaging.com)
Granulomatous ileitis2
- Firstly, this case is important as a reminder that there is a wide differential diagnosis for granulomatous ileitis, even though the vast majority of such cases will be associated with Crohn's disease. (medscape.com)
- Cite this: Granulomatous Ileitis in a Patient with Ankylosing Spondylitis - Medscape - Jun 01, 2007. (medscape.com)
Inflammation2
- SALOFALK tablets contain the active ingredient called mesalazine (5-aminosalicylic acid), which is used to treat and prevent relapse of mild to moderate attacks of ulcerative colitis (inflammation of the large bowel) and Crohn's ileitis and colitis (inflammation of the large bowel and last section of the small bowel). (mydr.com.au)
- Research from Ann & Robert H. Lurie Children's Hospital of Chicago found that direct injection of neonatal mesenchymal stem cells, derived from heart tissue discarded during surgery, reduces intestinal inflammation and promotes wound healing in a mouse model of Crohn's disease-like ileitis, an illness marked by chronic intestinal inflammation and progressive tissue damage. (news-medical.net)
Intestinal2
- We report a continuative application of infliximab as maintenance therapy for recurrent chronic ulcerative ileitis in a recipient of isolated intestinal transplantation (ITx). (elsevierpure.com)
- The number of enteric neurons expressing MrgE and MrgF receptors was significantly reduced during intestinal schistosomiasis and ileitis. (genscript.com)
Swine1
- ileitis, swine dysentery). (thepigsite.com)
Small intestine1
- The group implanted the cells into perforations in the small intestine of the mouse model of ileitis like Crohn's disease using neonatal mesenchymal stem cells (nMSC) derived from discarded heart tissue. (marketresearchfuture.com)
Crohn's Disease1
- Lastly, there is emerging evidence that ileitis of spondylarthropathy might represent subclinical Crohn's disease. (medscape.com)
Pigs1
- During fattening, pigs, which were simultaneously immunized with Enterisol Ileitis and Ingelvac CircoFLEX vaccine, gained significantly more weight (792 g/day) when compared to piglet groups mono-vaccinated with Ingelvac® CircoFLEXTM (772 g/day) or either with Enterisol® Ileitis (774 g/day). (gstsvs.ch)
Involves3
- Diagnosing ileitis involves thoroughly reviewing the patient's medical history and a comprehensive physical examination. (belmarrahealth.com)
- Managing ileitis involves more than just medical treatment. (belmarrahealth.com)
- Furthermore, we show that established but not early ileitis in Tnf ΔARE/+ mice involves defective expression of antimicrobials and dysbiosis, characterized by Firmicutes expansion, including epithelial-attaching segmented filamentous bacteria, and decreased abundance of Bacteroidetes. (mssm.edu)
Colonoscopy1
- Colonoscopy shows ileitis. (medscape.com)
Chronic1
- CONCLUSION Infliximab is effective as maintenance therapy for recurrent chronic ulcerative ileitis in an isolated ITx patient. (elsevierpure.com)
Regional1
- 18. Colorectal cancer in regional ileitis. (nih.gov)
Treatment1
- Microbiota modulation by antibiotic treatment at an early disease stage rescues ileitis. (mssm.edu)