A condition characterized by a reticular or fishnet pattern on the skin of lower extremities and other parts of the body. This red and blue pattern is due to deoxygenated blood in unstable dermal blood vessels. The condition is intensified by cold exposure and relieved by rewarming.
Skin diseases affecting or involving the cutaneous blood vessels and generally manifested as inflammation, swelling, erythema, or necrosis in the affected area.
A systemic non-inflammatory arteriopathy primarily of middle-aged females characterized by the association of livedo reticularis, multiple thrombotic CEREBRAL INFARCTION; CORONARY DISEASE, and HYPERTENSION. Elevation of antiphospholipid antibody titers (see also ANTIPHOSPHOLIPID SYNDROME), cardiac valvulopathy, ISCHEMIC ATTACK, TRANSIENT; SEIZURES; DEMENTIA; and chronic ischemia of the extremities may also occur. Pathologic examination of affected arteries reveals non-inflammatory adventitial fibrosis, thrombosis, and changes in the media. (From Jablonski, Dictionary of Syndromes & Eponymic Diseases, 2d ed; Adams et al., Principles of Neurology, 6th ed, p861; Arch Neurol 1997 Jan;54(1):53-60)
The inner zone of the adrenal cortex. This zone produces the enzymes that convert PREGNENOLONE, a 21-carbon steroid, to 19-carbon steroids (DEHYDROEPIANDROSTERONE; and ANDROSTENEDIONE) via 17-ALPHA-HYDROXYPREGNENOLONE.
Blocking of a blood vessel by CHOLESTEROL-rich atheromatous deposits, generally occurring in the flow from a large artery to small arterial branches. It is also called arterial-arterial embolization or atheroembolism which may be spontaneous or iatrogenic. Patients with spontaneous atheroembolism often have painful, cyanotic digits of acute onset.
Autoantibodies directed against phospholipids. These antibodies are characteristically found in patients with systemic lupus erythematosus (LUPUS ERYTHEMATOSUS, SYSTEMIC;), ANTIPHOSPHOLIPID SYNDROME; related autoimmune diseases, some non-autoimmune diseases, and also in healthy individuals.
Antiphospholipid antibodies found in association with systemic lupus erythematosus (LUPUS ERYTHEMATOSUS, SYSTEMIC;), ANTIPHOSPHOLIPID SYNDROME; and in a variety of other diseases as well as in healthy individuals. The antibodies are detected by solid-phase IMMUNOASSAY employing the purified phospholipid antigen CARDIOLIPIN.
The presence of antibodies directed against phospholipids (ANTIBODIES, ANTIPHOSPHOLIPID). The condition is associated with a variety of diseases, notably systemic lupus erythematosus and other connective tissue diseases, thrombopenia, and arterial or venous thromboses. In pregnancy it can cause abortion. Of the phospholipids, the cardiolipins show markedly elevated levels of anticardiolipin antibodies (ANTIBODIES, ANTICARDIOLIPIN). Present also are high levels of lupus anticoagulant (LUPUS COAGULATION INHIBITOR).
A region extending from the PONS & MEDULLA OBLONGATA through the MESENCEPHALON, characterized by a diversity of neurons of various sizes and shapes, arranged in different aggregations and enmeshed in a complicated fiber network.
A stage of development at which the ADRENAL GLANDS undergo maturation leading to the capability of producing increasing amounts of adrenal androgens, DEHYDROEPIANDROSTERONE and ANDROSTENEDIONE. Adrenarche usually begins at about 7 or 8 years of age before the signs of PUBERTY and continues throughout puberty.
The wide middle zone of the adrenal cortex. This zone produces a series of enzymes that convert PREGNENOLONE to cortisol (HYDROCORTISONE) via 17-ALPHA-HYDROXYPROGESTERONE.
The outer layer of the adrenal gland. It is derived from MESODERM and comprised of three zones (outer ZONA GLOMERULOSA, middle ZONA FASCICULATA, and inner ZONA RETICULARIS) with each producing various steroids preferentially, such as ALDOSTERONE; HYDROCORTISONE; DEHYDROEPIANDROSTERONE; and ANDROSTENEDIONE. Adrenal cortex function is regulated by pituitary ADRENOCORTICOTROPIN.

Buerger's disease manifesting nodular erythema with livedo reticularis. (1/17)

We report a patient with Buerger's disease (BD) who showed painful nodular erythema with livedo reticularis as an initial symptom. The patient developed this cutaneous manifestation in both lower extremities, and a skin biopsy demonstrated perivascular infiltration of mononuclear cells in the border zone between the dermis and subcutaneous tissue. Both nodular erythema and livedo reticularis were successfully treated with oral prednisolone, but both feet developed necrosis with ulcerations and had to be amputated 1.5 years later because of acute gangrene. Histopathology of the amputated tissue showed acute inflammation and multiple thrombi with recanalization in the posterior tibial arteries, leading to a diagnosis of BD. This disease should be considered as a possible diagnosis in refractory patients with nodular erythema and livedo reticularis, particularly when ulcerations and necrosis rapidly worsen.  (+info)

Microembolization from an abdominal aortic aneurysm after thoracic aortic replacement. (2/17)

A 66-year-old man with thoracic and abdominal aortic aneurysm suffered from microembolism in the lower extremities after total arch replacement. He presented with livedo reticularis with palpable peripheral pulses, and the serum creatinine kinase level elevated up to 7,695. The abdominal aortic aneurysm, but not the thoracic aorta, was the origin of this complication. The morphological change of thrombus in the abdominal aorta detected by ultrasonography was the key to the diagnosis. Graft replacement of the abdominal aorta finally resolved his problem.  (+info)

Homocystinuria due to cystathionine beta synthase deficiency. (3/17)

A two year-old male child presented with cutis marmorata congenita universalis, brittle hair, mild mental retardation, and finger spasms. Biochemical findings include increased levels of homocysteine in the blood-106.62 micromol/L (normal levels: 5.90-16 micromol/L). Biochemical tests such as the silver nitroprusside and nitroprusside tests were positive suggesting homocystinuria. The patient was treated with oral pyridoxine therapy for three months. The child responded well to this therapy and the muscle spasms as well as skin manifestations such as cutis marmorata subsided. The treatment is being continued; the case is reported here because of its rarity. Homocysteinuria arising due to cystathionine beta-synthase (CBS) deficiency is an autosomal recessive disorder of methionine metabolism that produces increased levels of urinary homocysteine and methionine It manifests itself in vascular, central nervous system, cutaneous, and connective tissue disturbances and phenotypically resembles Marfan's syndrome. Skin manifestations include malar flush, thin hair, and cutis reticulata / marmorata.  (+info)

Differences in anti-phosphatidylserine-prothrombin complex antibodies and cutaneous vasculitis between regular livedo reticularis and livedo racemosa. (4/17)

 (+info)

Livedo racemosa, secondary to drug-induced systemic lupus erythematosus. (5/17)

We present a 40-year-old man with erythematous-to-violaceous, broken, reticulated patches on the upper chest, back, and extremities, which is consistent with livedo racemosa. The cutaneous findings appeared after an increase in dilantin dose and subsequently improved after a reduction in dilantin dose. Furthermore, antinuclear antibodies and antihistone antibodies were detected. We therefore believe that the livedo racemosa is a cutaneous manifestation of a drug-induced systemic lupus erythematosus. We review the distinctive features of livedo racemosa as well as its associations with several disorders. Although there are no effective treatments for livedo racemosa, patients often are placed on low-dose aspirin and counseled to avoid smoking in an effort to protect against their increased risk of stroke and arterial thrombosis.  (+info)

A patient with idiopathic cholesterol crystal embolization: effectiveness of early detection and treatment. (6/17)

A 72-year-old man was admitted to our hospital because of progressive renal dysfunction persisting for 1.5 months. Physical examination showed livedo reticularis of the toes of both feet, peripheral edema, and gait disturbance due to the toe pain. The levels of blood urea nitrogen (50.0 mg/dL) and creatinine (2.81 mg/dL) were elevated, and eosinophilia (10%, 870/microL) was noted. A biopsy of the area of livedo reticularis revealed cholesterin crystals. The patient had not undergone angiography, anticoagulation therapy, or antithrombotic treatment. Idiopathic cholesterol crystal embolization was diagnosed. Transesophageal echocardiography revealed intimal thickening of the aorta and plaque. Oral steroid therapy was started because of the progressive renal dysfunction. After steroid therapy, the symptoms improved. Early diagnosis and treatment are important. Renal dysfunction is a common symptom in elderly patients. Cholesterol crystal embolization should also be considered as a cause of unexplained renal dysfunction, especially in such patients.  (+info)

Livedoid vasculopathy: an intringuing cutaneous disease. (7/17)

 (+info)

Livedo reticularis associated with autoimmune hemolytic anemia: prolonged remission induced by peripheral blood stem cell transplantation relapse after 10 years and restoration of hemoglobin levels by rituximab. (8/17)

 (+info)

Livedo reticularis is a cutaneous manifestation characterized by a bluish-purple, netlike pattern of discoloration on the skin. It is caused by the abnormal dilation and constriction of blood vessels near the skin's surface, leading to impaired circulation in the affected areas.

The condition can be idiopathic (primary) or secondary to various underlying disorders such as autoimmune diseases, vasculitis, hematologic disorders, infections, or medications that affect the blood vessels. In some cases, livedo reticularis may be a sign of an underlying medical condition requiring further evaluation and treatment.

It is essential to differentiate livedo reticularis from other related conditions like livedo racemosa, which presents with more irregular and diffuse patterns, and is typically associated with vasculitis or severe systemic disorders. Additionally, livedo reticularis should not be confused with cutis marmorata, a physiological response to cold temperatures that resolves upon warming the affected area.

Vascular skin diseases are a group of medical conditions that affect the blood vessels in the skin. These disorders can be caused by problems with the structure or function of the blood vessels, which can lead to various symptoms such as redness, discoloration, pain, itching, and ulcerations. Some examples of vascular skin diseases include:

1. Rosacea: a chronic skin condition that causes redness, flushing, and visible blood vessels in the face.
2. Eczema: a group of inflammatory skin conditions that can cause redness, itching, and dryness. Some types of eczema, such as varicose eczema, are associated with problems with the veins.
3. Psoriasis: an autoimmune condition that causes red, scaly patches on the skin. Some people with psoriasis may also develop psoriatic arthritis, which can affect the blood vessels in the skin and joints.
4. Vasculitis: a group of conditions that cause inflammation of the blood vessels. This can lead to symptoms such as redness, pain, and ulcerations.
5. Livedo reticularis: a condition that causes a net-like pattern of discoloration on the skin, usually on the legs. It is caused by abnormalities in the small blood vessels.
6. Henoch-Schönlein purpura: a rare condition that causes inflammation of the small blood vessels, leading to purple spots on the skin and joint pain.
7. Raynaud's phenomenon: a condition that affects the blood vessels in the fingers and toes, causing them to become narrow and restrict blood flow in response to cold temperatures or stress.

Treatment for vascular skin diseases depends on the specific condition and its severity. It may include medications, lifestyle changes, and in some cases, surgery.

Sneddon syndrome is a rare medical condition characterized by the concurrence of livedo reticularis (a purplish, net-like discoloration of the skin) and recurrent strokes or transient ischemic attacks (TIAs). It primarily affects young to middle-aged women. The exact cause of Sneddon syndrome remains unknown, but it's thought to be an autoimmune disorder with potential involvement of the coagulation system.

The main diagnostic criteria for Sneddon syndrome are:

1. Livedo reticularis (fixed, persistent form)
2. One or more cerebrovascular events (strokes or TIAs)

Additional features may include cognitive impairment, migraine-like headaches, seizures, and other neurological symptoms. Diagnosis is often challenging due to its rarity and the need to exclude other conditions that can present with similar symptoms. Treatment typically involves anticoagulation therapy, antiplatelet agents, or immunosuppressive medications to manage symptoms and prevent further cerebrovascular events.

The zona reticularis is a layer of the adrenal cortex, which is the outer part of the adrenal gland. These glands are located on top of the kidneys and are responsible for producing several important hormones. The adrenal cortex itself has three distinct layers: the zona glomerulosa, the zona fasciculata, and the zona reticularis.

The zona reticularis is the innermost layer of the adrenal cortex. It is responsible for producing and releasing certain steroid hormones, particularly androgens such as dehydroepiandrosterone (DHEA) and its sulfate (DHEAS). These androgens are precursor hormones that can be converted into more potent androgens or estrogens in other parts of the body. The zona reticularis plays a crucial role in sexual development and function, as well as maintaining overall health and well-being.

Disorders related to the zona reticularis may result in abnormal hormone production, leading to conditions such as congenital adrenal hyperplasia, Cushing's syndrome, or Addison's disease. Proper diagnosis and treatment of these disorders typically involve endocrinologists, healthcare professionals specializing in hormonal and metabolic disorders.

Cholesterol embolism is a medical condition that occurs when cholesteral crystals or plaque debris from an atherosclerotic lesion in the aorta or its major branches dislodge and travel to smaller vessels, where they obstruct blood flow. This can lead to tissue damage or infarction in various organs, depending on the location of the embolism.

Cholesterol emboli are typically small, crystalline, and composed of cholesterol, calcium, and other debris from atherosclerotic plaques. They can cause inflammation and damage to the endothelial cells lining the blood vessels, leading to further narrowing or occlusion of the vessel lumen.

Symptoms of cholesterol embolism depend on the location and extent of the obstruction. Common sites for embolization include the kidneys, brain, eyes, skin, and extremities. Symptoms can range from mild to severe and may include sudden pain, weakness, or numbness in the affected area; skin discoloration or ulcerations; vision changes; kidney dysfunction; and stroke-like symptoms.

Cholesterol embolism is often a complication of invasive procedures such as angiography, coronary artery bypass grafting, or aortic surgery. It can also occur spontaneously in patients with advanced atherosclerosis or those who have recently undergone anticoagulation therapy.

Diagnosis of cholesterol embolism is often challenging due to its nonspecific symptoms and variable presentation. Imaging studies, such as angiography or CT scans, may be used to visualize the location and extent of the obstruction. Blood tests and biopsy of affected tissues can also provide diagnostic clues.

Treatment of cholesterol embolism is primarily supportive and aimed at managing symptoms and preventing further complications. Antiplatelet therapy, statins, and anti-inflammatory agents may be used to reduce the risk of recurrent embolization and improve outcomes. In severe cases, surgical intervention or endovascular procedures may be necessary to remove the obstruction or restore blood flow.

Antiphospholipid antibodies are a type of autoantibody that targets and binds to certain proteins found in the blood that attach to phospholipids (a type of fat molecule). These antibodies are associated with an increased risk of developing antiphospholipid syndrome, a disorder characterized by abnormal blood clotting.

There are several types of antiphospholipid antibodies, including:

1. Lupus anticoagulant: This type of antiphospholipid antibody can interfere with blood clotting tests and may increase the risk of thrombosis (blood clots) in both arteries and veins.
2. Anticardiolipin antibodies: These antibodies target a specific phospholipid called cardiolipin, which is found in the inner membrane of mitochondria. High levels of anticardiolipin antibodies are associated with an increased risk of thrombosis and pregnancy complications such as recurrent miscarriage.
3. Anti-β2 glycoprotein I antibodies: These antibodies target a protein called β2 glycoprotein I, which binds to negatively charged phospholipids on the surface of cells. High levels of anti-β2 glycoprotein I antibodies are associated with an increased risk of thrombosis and pregnancy complications.

The exact mechanism by which antiphospholipid antibodies cause blood clotting is not fully understood, but it is thought to involve the activation of platelets, the inhibition of natural anticoagulants, and the promotion of inflammation. Antiphospholipid syndrome can be treated with medications that thin the blood or prevent clots from forming, such as aspirin, warfarin, or heparin.

Anticardiolipin antibodies are a type of autoantibody that targets and binds to cardiolipin, a phospholipid component found in the inner mitochondrial membrane of cells. These antibodies are clinically significant because they have been associated with a variety of autoimmune disorders, including antiphospholipid syndrome (APS).

APS is a condition characterized by recurrent blood clots, pregnancy losses, and thrombocytopenia (low platelet count). Anticardiolipin antibodies are one of the three main types of autoantibodies found in APS, along with lupus anticoagulant and anti-β2 glycoprotein I antibodies.

The presence of high levels of anticardiolipin antibodies in the blood can lead to abnormal blood clotting, which can cause serious complications such as deep vein thrombosis, pulmonary embolism, and stroke. Anticardiolipin antibodies can also contribute to pregnancy losses by causing placental insufficiency or abnormal blood clotting in the placenta.

Anticardiolipin antibodies are typically detected through a blood test that measures their levels in the serum. A positive result is usually confirmed with a second test performed at least 12 weeks later to establish persistence. Treatment for anticardiolipin antibody-related disorders typically involves anticoagulation therapy to prevent blood clots and other complications.

Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by the presence of antiphospholipid antibodies in the blood. These antibodies are directed against phospholipids, a type of fat molecule found in cell membranes and plasma lipoproteins. The presence of these antibodies can lead to abnormal blood clotting, which can cause serious complications such as stroke, heart attack, deep vein thrombosis, and pulmonary embolism.

APS can occur either on its own (primary APS) or in conjunction with other autoimmune disorders, such as systemic lupus erythematosus (secondary APS). The exact cause of APS is not fully understood, but it is believed to involve a combination of genetic and environmental factors.

Symptoms of APS can vary widely depending on the location and severity of the blood clots. They may include:

* Recurrent miscarriages or stillbirths
* Blood clots in the legs, lungs, or other parts of the body
* Skin ulcers or lesions
* Headaches, seizures, or stroke-like symptoms
* Kidney problems
* Heart valve abnormalities

Diagnosis of APS typically involves blood tests to detect the presence of antiphospholipid antibodies. Treatment may include medications to prevent blood clots, such as anticoagulants and antiplatelet agents, as well as management of any underlying autoimmune disorders.

The reticular formation is not a single structure but rather a complex network of interconnected neurons located in the brainstem, extending from the medulla oblongata through the pons and mesencephalon (midbrain) up to the diencephalon (thalamus and hypothalamus). It forms part of the reticular activating system, which is involved in regulating arousal, awareness, and sleep-wake cycles.

The reticular formation plays a crucial role in various functions such as:

1. Modulation of sensory input: The neurons in the reticular formation receive inputs from all senses (visual, auditory, tactile, etc.) and help filter and prioritize this information before it reaches higher cognitive areas.

2. Control of motor function: The reticular formation contributes to the regulation of muscle tone, posture, and locomotion by modulating the activity of motor neurons in the spinal cord.

3. Regulation of autonomic functions: The reticular formation is involved in controlling heart rate, blood pressure, respiration, and other visceral functions through its connections with the autonomic nervous system.

4. Consciousness and arousal: The ascending reticular activating system (ARAS) originates from the reticular formation and projects to the thalamus and cerebral cortex, where it helps maintain wakefulness and arousal. Damage to the ARAS can lead to coma or other states of altered consciousness.

5. Sleep-wake cycle regulation: The reticular formation contains cells that release neurotransmitters like histamine, serotonin, and orexin/hypocretin, which are essential for sleep-wake regulation. Dysfunction in these circuits has been implicated in various sleep disorders, such as narcolepsy and insomnia.

Adrenarche is a phase of development in which the adrenal glands begin to produce androgens, specifically DHEA (dehydroepiandrosterone) and its sulfate form DHEAS. This process usually begins between the ages of 6-8 in children, although it can vary. The androgens produced during adrenarche contribute to the development of secondary sexual characteristics such as pubic and underarm hair, but do not play a significant role in the growth spurt or reproductive function. It is important to note that adrenarche is separate from puberty, which is initiated by the hypothalamus and pituitary gland and involves the release of gonadotropins that stimulate the gonads to produce sex steroids.

The Zona Fasciculata is a region within the adrenal gland, which is a small gland located on top of the kidneys. It plays an essential role in endocrine function. The adrenal gland is divided into two main parts: the outer cortex and the inner medulla. The cortex itself is further divided into three zones: the Zona Glomerulosa, the Zona Fasciculata, and the Zona Reticularis.

The Zona Fasciculata is the middle layer of the adrenal cortex. It is primarily responsible for producing and releasing steroid hormones, particularly glucocorticoids such as cortisol. Cortisol helps regulate metabolism, immune response, and stress response, among other functions. The Zona Fasciculata contains large, column-shaped cells called fasciculated cells that contain lipid droplets filled with cholesterol esters. These cells convert cholesterol into pregnenolone, which is then converted into cortisol through a series of enzymatic reactions.

In summary, the Zona Fasciculata is a crucial region within the adrenal gland that produces and releases cortisol, a vital glucocorticoid hormone involved in various physiological processes.

The adrenal cortex is the outer portion of the adrenal gland, which is located on top of the kidneys. It plays a crucial role in producing hormones that are essential for various bodily functions. The adrenal cortex is divided into three zones:

1. Zona glomerulosa: This outermost zone produces mineralocorticoids, primarily aldosterone. Aldosterone helps regulate sodium and potassium balance and thus influences blood pressure by controlling the amount of fluid in the body.
2. Zona fasciculata: The middle layer is responsible for producing glucocorticoids, with cortisol being the most important one. Cortisol regulates metabolism, helps manage stress responses, and has anti-inflammatory properties. It also plays a role in blood sugar regulation and maintaining the body's response to injury and illness.
3. Zona reticularis: The innermost zone produces androgens, primarily dehydroepiandrosterone (DHEA) and its sulfate form (DHEAS). These androgens are weak compared to those produced by the gonads (ovaries or testes), but they can be converted into more potent androgens or estrogens in peripheral tissues.

Disorders related to the adrenal cortex can lead to hormonal imbalances, affecting various bodily functions. Examples include Addison's disease (insufficient adrenal cortical hormone production) and Cushing's syndrome (excessive glucocorticoid levels).

"Livedo reticularis durch Kältegelierung des Blutes bei γ-Plasmocytom" [Livedo reticularis due to cold gelation of the blood by ... "Oxalosis y livedo reticularis" [Oxalosis and Livedo Reticularis]. Actas Dermo-Sifiliográficas (in Spanish). 104 (9): 815-8. doi ... "Livedo reticularis". DermNet NZ. Retrieved February 2, 2018. Fleischer A, Resnick S (1990). "Livedo reticularis". Dermatol Clin ... due to underlying genetic cause Idiopathic livedo reticularis - the most common form of livedo reticularis, completely benign ...
... livedo reticularis). Livedo reticularis appears as a bluish-purple, netlike mottling of the skin. Sneddon's syndrome may ... transient neurological symptoms or stroke Livedo reticularis, or livedo racemosa Sneddon's syndrome generally manifests with ... synd/1732 at Who Named It? Sneddon IB (April 1965). "Cerebrovascular lesions and livedo reticularis". British Journal of ... Generally, Livedo precedes cerebrovascular involvement by roughly ten years, and many years of cerebrovascular involvement ...
Rare cases of skin rashes, such as Stevens-Johnson syndrome and livedo reticularis have also been reported in patients treated ... Vollum DI, Parkes JD, Doyle D (June 1971). "Livedo reticularis during amantadine treatment". British Medical Journal. 2 (5762 ...
Livedo reticularis Vasculitis Discontinuing contact with the heat source is the initial treatment of erythema ab igne. If the ... Riahi RR, Cohen PR, Robinson FW, Gray JM (Nov 2010). "Erythema ab igne mimicking livedo reticularis". International Journal of ...
Sangle S, D'Cruz DP, Hughes GR (January 2005). "Livedo reticularis and pregnancy morbidity in patients negative for ...
Weight loss greater than/equal to 4.5 kg Livedo reticularis (a mottled purplish skin discoloration over the extremities or ... Skin manifestations of PAN include palpable purpura and livedo reticularis in some individuals. Neurologic system: Nerve ...
343 List of cutaneous conditions Livedo Livedo reticularis Livedoid dermatitis William D. James; Timothy G. Berger; Dirk M. ...
Livedo racemosa and livedo reticularis are the most common manifestations in skin, although other symptoms such as digital ... "Deficiency of ADA2 mimicking autoimmune lymphoproliferative syndrome in the absence of livedo reticularis and vasculitis". ... Manifestations of the disease include but are not limited to recurrent fever, livedoid rash (reticularis or racemosa), various ...
Other skin abnormalities include thickening, hypertrichosis, acquired facial lipoatrophy, and infiltrating livedo reticularis ...
... is a cutaneous condition characterized by livedo reticularis on the legs and hyperesthesia. ...
Most commonly this causes skin symptoms (usually livedo reticularis), gangrene of the extremities and sometimes kidney failure ... and areas of the skin that assume a marbled pattern known as livedo reticularis. The pain is usually severe and requires ...
... and/or livedo reticularis. This disorder is caused by mutations in one or more of the following genes: FOXE3 (located on 1p33) ...
Livedo reticularis refers to slowing of blood flow, leading to desaturation of blood and bluish discolouration of the skin. ... livedo reticularis and purpuric "vasculitic" pattern. Chilblains and Multisystem inflammatory syndrome in children are also ...
The first skin changes in calciphylaxis lesions are mottling of the skin and induration in a livedo reticularis pattern. As ...
... livedo telangiectatica, congenital livedo reticularis and Van Lohuizen syndrome. It should not be confused with the more ... which refers to livedo reticularis caused by cold. For a full and up-to-date description visit the CMTC webpages of the global ... CMTC is also used synonymously with congenital generalized phlebectasia, nevus vascularis reticularis, congenital phlebectasia ...
Livedo reticularis Livedoid dermatitis List of cutaneous conditions "Livedo racemosa: Clinical, laboratory and ... Livedo racemosa is a skin condition with persistent red or violet discoloration, characterised by a broken, branched, ...
... and livedo reticularis. There are also associations between antiphospholipid antibodies and different neurologic manifestations ...
... livedo reticularis, and piezogenic papules are less common. In vascular EDS, skin can also be thin and translucent. In ...
... livedo reticularis). In people with secondary CAD (associated with another underlying condition), there may be additional signs ...
4 kg Livedo reticularis Testicular pain Myalgias, weakness Abdominal pain, diarrhea, and GI bleeding Mononeuropathy or ...
It is also more strongly associated with miscarriage, and can cause a number of other symptoms (such as livedo reticularis of ...
... and livedo reticularis; spontaneous nose bleeds, joint pain, membranoproliferative glomerulonephritis; and cardiovascular ...
... refers to a form of skin discoloration. Livedo reticularis Livedo racemosa Livedoid dermatitis Livedoid vasculitis " ... "livedo" at Dorland's Medical Dictionary v t e (Articles with short description, Short description matches Wikidata, ...
Livedo racemosa Livedo reticularis Livedoid dermatitis (embolia cutis medicamentosa, Nicolau syndrome) Livedoid vasculopathy ( ... idiopathic livedo reticularis with cerebrovascular accidents) Solar purpura (actinic purpura, senile purpura) Stasis dermatitis ... atrophie blanche, livedo reticularis with summer ulceration, livedoid vasculitis, PURPLE syndrome, segmental hyalinizing ... dermatopathia pigmentosa reticularis hyperkeratotica et mutilans, dermatopathia pigmentosa reticularis hypohidotica et ...
... livedo reticularis Muscles and joints: muscle pain or inflammation, joint pain or joint swelling Nervous system: mononeuritis ...
... livedo reticularis), especially extremities, due to insufficient perfusion of the skin The severity of hemorrhagic shock can be ...
... livedo reticularis, hives, and erythema multiforme. Cardiovascular system: Relapsing polychrondritis may cause inflammation of ...
... livedo reticularis) Testicular pain Pain in a muscle or group of muscles (myalgias) Damage to peripheral nerves Elevation of ...
"Livedo reticularis durch Kältegelierung des Blutes bei γ-Plasmocytom" [Livedo reticularis due to cold gelation of the blood by ... "Oxalosis y livedo reticularis" [Oxalosis and Livedo Reticularis]. Actas Dermo-Sifiliográficas (in Spanish). 104 (9): 815-8. doi ... "Livedo reticularis". DermNet NZ. Retrieved February 2, 2018. Fleischer A, Resnick S (1990). "Livedo reticularis". Dermatol Clin ... due to underlying genetic cause Idiopathic livedo reticularis - the most common form of livedo reticularis, completely benign ...
... has many causes. It is thought to be due to spasms of the blood vessels or an abnormality of the local ... Livedo reticularis can be a normal condition that is simply more obvious when a person is exposed to the cold; however, over ...
Livedo reticularis (LR) is a skin symptom. It refers to a netlike pattern of reddish-blue skin discoloration. The legs are ... Livedo reticularis (LR) is a skin symptom. It refers to a netlike pattern of reddish-blue skin discoloration. The legs are ... Cutis marmorata; Livedo reticularis - idiopathic; Sneddon syndrome - idiopathic livedo reticularis; Livedo racemosa ... Livedo reticularis (LR) is a skin symptom. It refers to a netlike pattern of reddish-blue skin discoloration. The legs are ...
Classic presentation includes livedo reticularis, vasculitis, and stroke. However, the phenotype and disease severity are ... Deficiency of ADA2 mimicking autoimmune lymphoproliferative syndrome in the absence of livedo reticularis and vasculitis ... Classic presentation includes livedo reticularis, vasculitis, and stroke. However, the phenotype and disease severity are ... Deficiency of ADA2 mimicking autoimmune lymphoproliferative syndrome in the absence of livedo reticularis and vasculitis ...
Livedo reticularis is a purplish-colored lace pattern under the skin. There is no ... Does livedo reticularis have anything to do with Rheumatoid disease? ... Does livedo reticularis have anything to do with Rheumatoid disease?. Livedo reticularis is a purplish-colored lace pattern ... Livedo Reticularis & Rheumatoid Arthritis: Nothing Rash about It January 15, 2013. May 14, 2018. Kelly ONeill 86 Comments ...
Livedo Reticularis Rash. 7/11. This purplish, marbled rash looks webbed like lace. Inflammatory conditions (like lupus and ... Medscape: "Livedo Reticularis.". National Institute of Diabetes and Digestive and Kidney Diseases: "Insulin Resistance & ... Mayo Clinic: "Livedo reticularis: When is it a concern?". ...
Hardin J. Hardin J Hardin, J. Matthew. "Livedo Reticularis." The Atlas of Emergency Medicine, 5e Knoop KJ, Stack LB, Storrow AB ... Hardin J. Hardin J Hardin, J. Matthew.Livedo Reticularis. In: Knoop KJ, Stack LB, Storrow AB, Thurman R. Knoop K.J., & Stack L. ... Physiologic livedo reticularis is a response to cold temperatures and is a common finding in infants, children, and adults ... Hardin J. Hardin J Hardin, J. Matthew. (2021). Livedo reticularis. Knoop KJ, Stack LB, Storrow AB, Thurman R. Knoop K.J., & ...
Dive into the research topics of Painful subcutaneous nodules in a patch of livedo reticularis. Together they form a unique ...
Cutaneous lesions are reported to occur in 25-50% of patients with systemic PAN and include livedo reticularis and ulceration ... The cutaneous signs of cholesterol emboli include livedo reticularis, petechiae, purpura, and blue toes. Because aortic ...
Livedo reticularis. * Antiphospholipid syndrome. Arterial thrombosis resulting in ischemia and necrosis of the foot. ...
Livedo reticularis. Antiphospholipid-antibody syndrome, vasculitis, cholesterol emboli. Lesions. Keratoderma blennorrhagicum. ...
Vascular Disorders: 1%-,10% - flushing, hypertension; 0.1%-,1% - hypotension, thrombophlebitis; ,0.1% - livedo reticularis. ...
Nonspecific, firm, tender subcutaneous nodules without livedo reticularis and/or systemic involvement may be the first sign of ... Tender, hyperpigmented, firm subcutaneous nodules with a background of livedo reticularis common in cutaneous polyarteritis ...
Livedo reticularis ... As a reaction to certain medicines such as amantadine or interferon Other blood vessel diseases such as ...
Physical examination demonstrated new hepatomegaly and lower extremity livedo reticularis. Results of broad testing for ...
Skin becomes cool and hyperhidrotic with livedo reticularis or cyanosis. Hair may be lost, and nails become ridged, cracked, ...
Livedo reticularis, cutaneous ulcerations, or skin nodules. *Testicular pain or tenderness. *Myalgias ...
5. Erythema ab igne mimicking livedo reticularis.. Riahi RR; Cohen PR; Robinson FW; Gray JM. Int J Dermatol; 2010 Nov; 49(11): ...
Ehrmann-Sneddon syndrome; Livedo racemosa-cerebrovascular accident syndrome; Livedo reticularis-cerebrovascular accident ... syndromeEhrmann-Sneddon syndrome; Livedo racemosa-cerebrovascular accident syndrome; Livedo reticularis-cerebrovascular ...
Livedo Reticularis Actions. * Search in PubMed * Search in MeSH * Add to Search ...
Livedo reticularis (6%). *Contusion (6%). *Depression/depressed mood (6%). *Headache (6%). *Benign prostate hyperplasia (6%) ...
Livedo reticularis: a lacey, purplish discoloration of the skin on the legs with some leg swelling. This occurs in less than 1 ...
Livedo reticularis, leg/foot pain, and good foot pulses. *Livedo reticularis, kidney impairment, and eosinophilia (high numbers ... The skin is affected in over one-third of patients resulting in livedo reticularis, gangrene, cyanosis (blue toe syndrome ...
Cutis marmorata, livedo reticularis, livedo racemosa + Cryoglobulinemia + Cryoglobulinemia in monoclonal gamapathy + Mixed ...
Livedo reticularis is the most common skin disorder (49%), followed by gangrene (35%), cyanosis (28%), and ulceration (17%).2 ... The triad of thigh and foot pain, livedo reticularis, and intact peripheral pulses is considered to be pathognomonic for ... The triad of pain, livedo reticularis, and intact peripheral pulses is pathognomonic for cholesterol embolisation. The ...
In addition, interest on cardiovascular manifestations, livedo reticularis, atherosclerosis, valvulopathy, and neurological and ...
Livedo reticularis: an update. J Am Acad Dermatol. 2005;52:1009-1019.. 21. Arnold AC, Okamoto LE, Diedrich A, et al. Low-dose ... The two main causes of livedo reticularis are venodilation of vessels and deoxygenation of blood in the subpapillary venous ... These include livedo reticularis, koilonychia, acrocyanosis, telogen effluvium, madarosis, vascular cutaneous abnormalities, ... and livedo reticularis. The treatment of this disease poses a great challenge. The author reports the unprecedented use of an ...
Livedo reticularis (a blue or purple pattern that showing through the skin that looks like lace or fishnet stockings) ...
  • Sneddon's syndrome is an uncommon cause of stroke in young people, characterised by livedo reticularis and cerebrovascular disease. (bmj.com)
  • Livedoid dermatitis Livedo racemosa Perinatal gangrene of the buttock erythema ab igne (AKA livedo recticularis e calore) - a rash caused by prolonged heat exposure (e.g. hot water bottle or heat pad) List of cutaneous conditions Rapini, Ronald P. (wikipedia.org)
  • Livedo reticularis is a rash that occurs when the blood supply is disrupted. (drtimpearce.com)
  • WOMACK: When I got the vaccine, within a few minutes, I got something called Livedo Reticularis, which is basically like a rash - a red rash on my arms, intense facial flushing like a tomato, very, very bad headache. (ketr.org)
  • They found that two-thirds of the children experienced additional symptoms not formally associated with antiphospholipid syndrome in adults , including low platelet counts, hemolytic anemia and livedo reticularis, a rash indicating abnormal blood flow to the skin. (michiganmedicine.org)
  • No further test or examination confirms idiopathic livedo reticularis. (wikipedia.org)
  • Other than identifying and treating any underlying conditions in secondary livedo, idiopathic livedo reticularis may improve with warming the area. (wikipedia.org)
  • The skin is affected in over one-third of patients resulting in livedo reticularis , gangrene , cyanosis (' blue toe syndrome ' secondary to lack of oxygenated blood supply), ulceration , painful red nodules , and purpura (purple patches). (dermnetnz.org)
  • Livedo reticularis is a common skin finding consisting of a mottled reticulated vascular pattern that appears as a lace-like purplish discoloration of the skin. (wikipedia.org)
  • Livedo reticularis is a purplish-colored lace pattern under the skin. (rawarrior.com)
  • Livedo reticularis: a lacey, purplish discoloration of the skin on the legs with some leg swelling. (parkinson.org)
  • Livedo reticularis - a purplish pattern on the skin of the hands and feet over the vascular reticular system. (bvfootclinic.com)
  • It was the discoloration and the livedo reticularis pattern albeit a fleeting example, which confirmed their suspicions. (drtimpearce.com)
  • 5. Erythema ab igne mimicking livedo reticularis. (nih.gov)
  • Sneddon's syndrome is characterized by livedo reticularis and cerebrovascular lesions. (cumhuriyet.edu.tr)
  • 10. Dexrazoxane for rapid extended livedo reticularis-like skin reaction due to systemic epirubicin diffusion during transcatheter arterial chemoembolization procedure for hepatocellular carcinoma. (nih.gov)
  • The triad of pain, livedo reticularis, and intact peripheral pulses is pathognomonic for cholesterol embolisation. (bmj.com)
  • The triad of thigh and foot pain, livedo reticularis, and intact peripheral pulses is considered to be pathognomonic for cholesterol embolisation. (bmj.com)
  • Peripheral edema, livedo reticularis, ataxia. (psychdb.com)
  • Classic presentation includes livedo reticularis, vasculitis, and stroke. (nih.gov)
  • And I immediately found pictures of livedo reticularis caused by vasculitis related to Rheumatoid disease (or Lupus). (rawarrior.com)
  • Livedo reticularis , kidney impairment, and eosinophilia (high numbers of circulating eosinophils in the blood). (dermnetnz.org)
  • Although not included in the diagnostic criteria, other clinical symptoms, such as livedo reticularis, nephropathy, thrombocytopenia, cardiac valvular disease, and neurological symptoms are commonly associated comorbidities. (medscape.com)
  • aCL assay is also indicated in cutaneous circulation disturbances without clear explanations, such as blue toe syndrome, ulcers resembling pyoderma gangrenosum, and livedo reticularis. (medscape.com)
  • Physiologic livedo reticularis improves or disappears with warming, whereas secondary causes usually do not. (mhmedical.com)
  • Afección caracterizada por la presencia de un patrón reticular en la piel de las extremidades inferiores y otras partes del cuerpo. (bvsalud.org)
  • Livedo reticularis is diagnosed by its clinical appearance and history. (wikipedia.org)
  • The causes and treatments for livedo reticularis are not fully understood, however it is believed that when some small blood vessels are blocked or no longer feed surface tissues, other vessels enlarge to compensate, creating the netted pattern. (rawarrior.com)
  • Livedo Reticularis or "What is the Weird Blue Pattern on my Feet? (bvfootclinic.com)
  • Does livedo reticularis have anything to do with Rheumatoid disease? (rawarrior.com)
  • Physiologic livedo reticularis is a response to cold temperatures and is a common finding in infants, children, and adults prone to acrocyanosis. (mhmedical.com)
  • Livedo reticularis is a reactive macular, reticulated (net-like) patch of nonpalpable cutaneous vasodilatation due to normal physiologic variation, vasospasm, vessel wall damage, or intraluminal pathology. (mhmedical.com)
  • Livedo reticularis was confirmed by biopsy (see Figure 1). (medscape.com)