SCHISTOSOMIASIS of the brain, spinal cord, or meninges caused by infections with trematodes of the genus SCHISTOSOMA (primarily SCHISTOSOMA JAPONICUM; SCHISTOSOMA MANSONI; and SCHISTOSOMA HAEMATOBIUM in humans). S. japonicum infections of the nervous system may cause an acute meningoencephalitis or a chronic encephalopathy. S. mansoni and S. haematobium nervous system infections are associated with acute transverse myelitis involving the lower portions of the spinal cord. (From Joynt, Clinical Neurology, 1998, Ch27, pp61-2)
Clinical or subclinical disturbances of cortical function due to a sudden, abnormal, excessive, and disorganized discharge of brain cells. Clinical manifestations include abnormal motor, sensory and psychic phenomena. Recurrent seizures are usually referred to as EPILEPSY or "seizure disorder."
A nutritional condition produced by a deficiency of THIAMINE in the diet, characterized by anorexia, irritability, and weight loss. Later, patients experience weakness, peripheral neuropathy, headache, and tachycardia. In addition to being caused by a poor diet, thiamine deficiency in the United States most commonly occurs as a result of alcoholism, since ethanol interferes with thiamine absorption. In countries relying on polished rice as a dietary staple, BERIBERI prevalence is very high. (From Cecil Textbook of Medicine, 19th ed, p1171)
A nutritional condition produced by a deficiency of VITAMIN B 6 in the diet, characterized by dermatitis, glossitis, cheilosis, and stomatitis. Marked deficiency causes irritability, weakness, depression, dizziness, peripheral neuropathy, and seizures. In infants and children typical manifestations are diarrhea, anemia, and seizures. Deficiency can be caused by certain medications, such as isoniazid.
A group of inherited metabolic disorders which have in common elevations of serum LYSINE levels. Enzyme deficiencies of alpha-aminoadipic semialdehyde dehydrogenase and the SACCHAROPINE DEHYDROGENASES have been associated with hyperlysinemia. Clinical manifestations include mental retardation, recurrent emesis, hypotonia, lethargy, diarrhea, and developmental delay. (From Menkes, Textbook of Child Neurology, 5th ed, p56)
Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)
The 4-methanol form of VITAMIN B 6 which is converted to PYRIDOXAL PHOSPHATE which is a coenzyme for synthesis of amino acids, neurotransmitters (serotonin, norepinephrine), sphingolipids, aminolevulinic acid. Although pyridoxine and Vitamin B 6 are still frequently used as synonyms, especially by medical researchers, this practice is erroneous and sometimes misleading (EE Snell; Ann NY Acad Sci, vol 585 pg 1, 1990).
A syndrome characterized by multiple abnormalities, MENTAL RETARDATION, and movement disorders. Present usually are skull and other abnormalities, frequent infantile spasms (SPASMS, INFANTILE); easily provoked and prolonged paroxysms of laughter (hence "happy"); jerky puppetlike movements (hence "puppet"); continuous tongue protrusion; motor retardation; ATAXIA; MUSCLE HYPOTONIA; and a peculiar facies. It is associated with maternal deletions of chromosome 15q11-13 and other genetic abnormalities. (From Am J Med Genet 1998 Dec 4;80(4):385-90; Hum Mol Genet 1999 Jan;8(1):129-35)

Schistosomal myeloradiculopathy due to Schistosoma mansoni: report on 23 cases. (1/26)

Schistosoma mansoni infection is likely to be responsible for a significant proportion of cases of myelopathy occurring in areas where schistosomiasis is endemic. The aim of this study is to describe the clinical, laboratory and therapeutic data of 23 patients with schistosomal myeloradiculopathy. The medical records of 23 patients with schistosomal myelopathy admitted to two general hospitals of Belo Horizonte (MG), in Brazil, from 1995 to 1999, were reviewed retrospectively. Seventeen patients were male (74%). The mean age for the whole group was 27 years. Lower limb weakness and associated lumbar and/or lower limb pain were reported by 20 patients (87%), and 16 (70%) were unable to walk. All individuals presented urinary retention and 19 (83%) complained of intestinal dysfunction. The treatment was based on the association of antischistosomal drugs and corticosteroids. Five patients (22%) presented a full response to treatment, 13 (57%) partial response without functional limitations and 4 (17%) partial improvement with limitations or no response. Three out of the 4 patients who stopped steroids before 45 days of treatment developed recurrence of the symptoms and signs of myelopathy. Our cases demonstrate the severe presentation of the disease and the data disclosed here suggest that treatment with steroids should be kept for months after clinical improvement.  (+info)

Experimental neuroschistosomiasis: inadequacy of the murine model. (2/26)

Neuroschistosomiasis is rarely observed in human pathology, but it is of considerable importance. To investigate its pathogenesis, consequences and response to treatment, an experimental model would be desirable, but is not yet available, in spite of a few indications of a suitable mouse model in the literature. Severe, recent and late Schistosoma mansoni infections in outbred and inbred strains of mice revealed widespread distribution of parasite eggs in several organs, but only exceptionally did eggs reach the encephalus, thus revealing the inadequacy of the mouse as an experimental model for neuroschistosomiasis.  (+info)

Clinical and cerebrospinal fluid (CSF) profile and CSF criteria for the diagnosis of spinal cord schistosomiasis. (3/26)

OBJECTIVES: To describe the clinical and CSF findings among patients with presumptive neuroschistosomiasis (NS) and to suggest a classification for the CSF diagnosis of presumptive NS. METHOD: The charts of all patients whose CSF exam was performed at the CSF Lab, Jos Silveira Foundation, Salvador, Brazil, from 1988 to 2002 were reviewed. Those with clinically suspected NS whose indirect fluorescent antibody test (IFA) and or hemagglutination-inhibiting antibodies test (HAI) were positive to S. mansoni were identified. RESULTS: Of 377 patients, 67.9% were males; the median age was 36 years (mean 37 + 16 yrs, range 3-82 yrs). The most frequent complaints were paraparesis (59.9%), urinary retention (36.2%), lower limb pain (22.8%). WBC of CSF (count/mm ) was > 4 in 66.0% (mean 83 + 124, median 40, range 4.3-1,100), protein (mg/dl) was > 40 in 84.6% (mean 185 + 519, median 81, range 41-6,800) and eosinophils were present in 46.9%. IFA and HAI were positive in 75.3%. WBC > 4 and presence of eosinophils were associated with IFA and HAI positive (67.3% versus 51.4%, p 0.014; 49.1% versus 23.0%, p 0.0001, respectively) and protein > 40 was not (85.4% versus 77.0%, p 0.09). Presence of WBC > 4, protein > 40 and eosinophils was associated with IFA and HAI positive (71.6% versus 38.2%, p 0.0003) but presence of eosinophils and any other combination of WBC and protein were not. CONCLUSION: NS should be considered as a possible diagnosis in patients who had had contact with schistosome-infected water and present with spinal cord compromising. Presence of IFA and HAI positive to S. mansoni, WBC > 4, protein > 40 and presence of eosinophils in the CSF may be considered as a criterium of highly probable presumptive diagnosis.  (+info)

Involvement of central nervous system in the schistosomiasis. (4/26)

The involvement of the central nervous system (CNS) by schistosomes may or may not determine clinical manifestations. When symptomatic, neuroschistosomiasis (NS) is one of the most severe presentations of schistosomal infection. Considering the symptomatic form, cerebral involvement is almost always due to Schistosoma japonicum and the spinal cord disease, caused by S. mansoni or S. haematobium. Available evidence suggests that NS depends basically on the presence of parasite eggs in the nervous tissue and on the host immune response. The patients with cerebral NS usually have the clinical manifestations of increased intracranial pressure associated with focal neurological signs; and those with schistosomal myeloradiculopathy (SMR) present rapidly progressing symptoms of myelitis involving the lower cord, usually in association with the involvement of the cauda esquina roots. The diagnosis of cerebral NS is established by biopsy of the nervous tissue and SMR is usually diagnosed according to a clinical criterion. Antischistosomal drugs, corticosteroids and surgery are the resources available for treating NS. The outcome is variable and is better in cerebral disease.  (+info)

Treatment of schistosomal myeloradiculopathy with praziquantel and corticosteroids and evaluation by magnetic resonance imaging: a longitudinal study. (5/26)

BACKGROUND: The best treatment for schistosomal myeloradiculopathy (SMR) remains undefined. There is also no longitudinal study to estimate the value of magnetic resonance imaging (MRI) in the diagnosis and follow-up of this disease. METHODS: Patients with the following presentation were considered for study: lumbar and/or lower limb pain; lower limb weakness; anesthesia, hypoesthesia, or paresthesia; bladder and/or intestinal dysfunction; and sexual impotence. Sixteen patients with SMR were treated with oral praziquantel (50 mg/kg in a single dose) and methylprednisolone (15 mg/kg/day intravenously for 5 days) followed by prednisone (1 mg/kg/day orally for 6 months). Clinical outcome was prospectively evaluated in months 2 and 6 of treatment. RESULTS: Image alterations were detected by MRI at diagnosis for all patients, and normalization or improvement was reported at the end of treatment. There was statistically significant clinical melioration at both the second and sixth months of therapy for most neurological alterations. However, the best clinical outcome was achieved when the steroid was given for >2 months. CONCLUSIONS: Treatment with praziquantel associated with corticosteroids was successful in all cases. MRI proved to be a good method for the diagnosis of SMR and helpful in the evaluation of response to treatment.  (+info)

Spinal cord schistosomiasis: MR imaging appearance with surgical and pathologic correlation. (6/26)

BACKGROUND AND PURPOSE: Spinal cord involvement is a rare manifestation of schistosomiasis. We describe the MR imaging findings of spinal cord schistosomiasis in correlation with surgery and pathology. METHODS: We report eight cases of spinal cord schistosomiasis. All patients were men (mean age, 16.7 years) with neurologic manifestations who had been referred for spinal MR imaging. In all cases, spinal masses were surgically removed. MR imaging findings were correlated with surgery and pathology. RESULTS: MR imaging showed moderate expansion of the distal spinal cord in all cases. Abnormalities were isointense to cord in T1 and patchy hyperintense in T2-weighted spin-echo images (n = 8). Three forms of contrast enhancement were recognized: (1) intramedullary nodular (n = 8); (2) peripheral (n = 8); and (3) linear radicular (n = 4). Total gross surgical removal of masses by using the Cavitron ultrasonic surgical aspirator was possible in six cases. Diagnosis was established by identification of ova in histopathologic studies: Schistosoma mansoni (n = 3), S. hematobium (n = 1), and uncertain species (n = 4). Intramedullary nodular enhancement was correlated to multiple schistosomiasis microtubercles. Peripheral enhancing lesions correlated to thickened leptomeninges infested by chronic granulomatous inflammatory cells and schistosoma eggs. Linear radicular enhancement correlated with thickened resected nervous roots infested by granulomatous cells and schistosoma eggs. CONCLUSION: Multinodular intramedullary contrast enhancement of the distal cord enabled correct presumptive preoperative MR imaging diagnosis of spinal schistosomiasis in three cases. Accurate diagnosis, through recognition of its MR imaging appearance, allows early treatment and better prognosis of spinal cord schistosomiasis.  (+info)

Neuroschistosomiasis due to Schistosoma mansoni: a review of pathogenesis, clinical syndromes and diagnostic approaches. (7/26)

Neuroschistosomiasis (NS) is the second most common form of presentation of infection by the trematode, Schistosoma mansoni. Granulomatous inflammatory reaction occurs as a result of schistosome eggs being transmitted to spinal cord or brain via the vascular system, or by inadvertent adult worm migration to these organs. The two main clinical syndromes are spinal cord neuroschistosomiasis (acute or subacute myelopathy) and localized cerebral or cerebellar neuroschistosomiasis (focal CNS impairment, seizures, increased intracranial pressure). Presumptive diagnosis of NS requires confirming the presence of S. mansoni infection by stool microscopy or rectal biopsy for trematode eggs, and serologic testing of blood and spinal fluid. The localized lesions are identified by signs and symptoms, and confirmed by imaging techniques (contrast myelography, CT and MRI). Algorithms are presented to allow a stepwise approach to diagnosis.  (+info)

Morbidity of Schistosomiasis mansoni in the state of Minas Gerais, Brazil. (8/26)

From 2002 to 2005, a program of active search for patients with hepatosplenic schistosomiasis and schistosomal myeloradiculopathy has been implemented in the state of Minas Gerais by the local Health Department. The state was divided in 28 regional health centers and the local representatives have been trained to identify and direct patients with hepatosplenic schistosomiasis and neuroschistosomiasis to a reference center in Belo Horizonte, the capital of the state of Minas Gerais. Seventy five patients with hepatosplenic schistosomiasis and 54 with schistosomal myeloradiculopathy have been referred and examined in the reference center in a period of time of 3 years. Schistosomal myeloradiculopathy should be emphasized because the number of cases reported is increasing rapidly and when timely diagnosed and treated, they respond promptly to treatment. Left untreated, they die or become invalid for life. In our view, the time has come for more active investigation of the different aspects of morbidity caused by schistosomiasis mansoni in Brazil.  (+info)

Neuroschistosomiasis is a form of schistosomiasis, which is a parasitic infection caused by Schistosoma species. It is characterized by the invasion and inflammation of the central nervous system (CNS) by the parasite's eggs or larvae. This can lead to various neurological symptoms such as seizures, headaches, visual disturbances, and motor or sensory deficits. Neuroschistosomiasis is a serious and potentially life-threatening condition that requires prompt diagnosis and treatment.

The two Schistosoma species most commonly associated with neuroschistosomiasis are S. japonicum and S. mansoni. The parasites typically enter the human body through skin contact with contaminated water, where they mature into adult worms in the bloodstream. Female worms then lay eggs, some of which may be carried to the CNS by the circulatory system.

Neuroschistosomiasis can occur in both acute and chronic forms. Acute neuroschistosomiasis is characterized by an inflammatory response to the parasite's eggs or larvae, which can cause eosinophilic meningitis or encephalitis. Chronic neuroschistosomiasis may result in the formation of granulomas around the eggs, leading to various neurological symptoms depending on the location and extent of the damage.

Diagnosis of neuroschistosomiasis typically involves a combination of clinical evaluation, imaging studies (such as MRI or CT scans), and laboratory tests (such as serology or CSF analysis). Treatment usually consists of anti-parasitic drugs such as praziquantel, combined with corticosteroids to manage the inflammatory response. In severe cases, surgical intervention may be necessary to alleviate symptoms or prevent further damage.

A seizure is an uncontrolled, abnormal firing of neurons (brain cells) that can cause various symptoms such as convulsions, loss of consciousness, altered awareness, or changes in behavior. Seizures can be caused by a variety of factors including epilepsy, brain injury, infection, toxic substances, or genetic disorders. They can also occur without any identifiable cause, known as idiopathic seizures. Seizures are a medical emergency and require immediate attention.

Thiamine deficiency, also known as beriberi, is a condition that results from inadequate intake or impaired absorption of thiamine (vitamin B1), which is essential for energy metabolism and nerve function. This deficiency can lead to various symptoms such as peripheral neuropathy, muscle weakness, heart failure, and in severe cases, Wernicke-Korsakoff syndrome, a neurological disorder associated with alcoholism. Thiamine deficiency is commonly found in populations with poor nutrition, alcohol dependence, and gastrointestinal disorders affecting nutrient absorption.

Vitamin B6 deficiency refers to the condition in which there is an insufficient amount of vitamin B6 (pyridoxine) in the body. Vitamin B6 is an essential nutrient that plays a crucial role in various bodily functions, including protein metabolism, neurotransmitter synthesis, hemoglobin production, and immune function.

A deficiency in vitamin B6 can lead to several health issues, such as:

1. Anemia: Vitamin B6 is essential for the production of hemoglobin, a protein in red blood cells that carries oxygen throughout the body. A deficiency in this nutrient can lead to anemia, characterized by fatigue, weakness, and shortness of breath.
2. Peripheral neuropathy: Vitamin B6 deficiency can cause nerve damage, leading to symptoms such as numbness, tingling, and pain in the hands and feet.
3. Depression and cognitive impairment: Pyridoxine is necessary for the synthesis of neurotransmitters like serotonin and dopamine, which are involved in mood regulation. A deficiency in vitamin B6 can lead to depression, irritability, and cognitive decline.
4. Seizures: In severe cases, vitamin B6 deficiency can cause seizures due to the impaired synthesis of gamma-aminobutyric acid (GABA), an inhibitory neurotransmitter that helps regulate brain activity.
5. Skin changes: A deficiency in this nutrient can also lead to skin changes, such as dryness, scaling, and cracks around the mouth.

Vitamin B6 deficiency is relatively uncommon in developed countries but can occur in individuals with certain medical conditions, such as malabsorption syndromes, alcoholism, kidney disease, or those taking medications that interfere with vitamin B6 metabolism. Additionally, older adults, pregnant women, and breastfeeding mothers may have an increased need for this nutrient, making them more susceptible to deficiency.

Hyperlysinemias are inherited metabolic disorders characterized by an elevated level of the amino acid lysine in the blood. There are two major types: primary hyperlysinemia (also known as Lysinuric protein intolerance) and secondary hyperlysinemia.

Primary hyperlysinemia is caused by a deficiency of the enzyme responsible for breaking down lysine, arginine, and ornithine in the body. This leads to an accumulation of these amino acids in the blood and can cause various symptoms such as vomiting, seizures, developmental delay, and kidney problems.

Secondary hyperlysinemia, on the other hand, is not a result of a specific enzyme deficiency but rather due to an excess intake of lysine or a disorder that affects lysine metabolism. Symptoms may vary depending on the underlying cause and can range from mild to severe.

It's important to note that hyperlysinemias are rare disorders, and their diagnosis typically requires specialized testing. Treatment may involve dietary modifications, supplementation of missing enzymes or cofactors, and management of symptoms as they arise.

An encyclopedia is a comprehensive reference work containing articles on various topics, usually arranged in alphabetical order. In the context of medicine, a medical encyclopedia is a collection of articles that provide information about a wide range of medical topics, including diseases and conditions, treatments, tests, procedures, and anatomy and physiology. Medical encyclopedias may be published in print or electronic formats and are often used as a starting point for researching medical topics. They can provide reliable and accurate information on medical subjects, making them useful resources for healthcare professionals, students, and patients alike. Some well-known examples of medical encyclopedias include the Merck Manual and the Stedman's Medical Dictionary.

Pyridoxine is the chemical name for Vitamin B6. According to the medical definition, Pyridoxine is a water-soluble vitamin that is part of the B-vitamin complex and is essential for the metabolism of proteins, carbohydrates, and fats. It plays a vital role in the regulation of homocysteine levels in the body, the formation of neurotransmitters such as serotonin and dopamine, and the synthesis of hemoglobin.

Pyridoxine can be found naturally in various foods, including whole grains, legumes, vegetables, nuts, seeds, meat, poultry, and fish. It is also available as a dietary supplement and may be prescribed by healthcare providers to treat or prevent certain medical conditions, such as vitamin B6 deficiency, anemia, seizures, and carpal tunnel syndrome.

Like other water-soluble vitamins, Pyridoxine cannot be stored in the body and must be replenished regularly through diet or supplementation. Excessive intake of Pyridoxine can lead to toxicity symptoms such as nerve damage, skin lesions, and light sensitivity.

Angelman Syndrome is a genetic disorder that affects the nervous system and is characterized by intellectual disability, developmental delay, lack of speech or limited speech, movement and balance disorders, and a happy, excitable demeanor. Individuals with Angelman Syndrome often have a distinctive facial appearance, including widely spaced teeth, a wide mouth, and protruding tongue. Seizures are also common in individuals with this condition.

The disorder is caused by the absence or malfunction of a gene called UBE3A, which is located on chromosome 15. In about 70% of cases, the deletion of a portion of chromosome 15 that includes the UBE3A gene is responsible for the syndrome. In other cases, mutations in the UBE3A gene or inheritance of two copies of chromosome 15 from the father (uniparental disomy) can cause the disorder.

There is no cure for Angelman Syndrome, but early intervention with physical therapy, speech therapy, and other supportive therapies can help improve outcomes. Anticonvulsant medications may be used to manage seizures. The prognosis for individuals with Angelman Syndrome varies, but most are able to live active, fulfilling lives with appropriate support and care.

Symptomatic epilepsy can be the first manifestation of neuroschistosomiasis in patients without any systemic symptoms. The ...
Neuroschistosomiasis due to portosystemic anastomoses from portal hypertension Pulmonary Schistosomiasis due to portosystemic ... large granulomas in circumscribed areas of the brain characterize the pseudotumoral form of neuroschistosomiasis and are ...
... neuroschistosomiasis MeSH C10.228.228.205.300 - central nervous system protozoal infections MeSH C10.228.228.205.300.500 - ...
MeSH C03.105.250.550 - neurocysticercosis MeSH C03.105.250.600 - neuroschistosomiasis MeSH C03.105.300.500 - malaria, cerebral ... neuroschistosomiasis MeSH C03.335.865.859.427 - schistosomiasis haematobia MeSH C03.335.865.859.521 - schistosomiasis japonica ...
Neuroschistosomiasis has been reported in persons living near Lake Malawi, in Malawi (6). Four members of a Belgian expatriate ... Early Neuroschistosomiasis Complicating Katayama Syndrome. Volume 12, Number 9-September 2006. Article Views: 869. Data is ... Early Neuroschistosomiasis Complicating Katayama Syndrome. Emerging Infectious Diseases. 2006;12(9):1465-1466. doi:10.3201/ ... Clerinx J, van Gompel A, Lynen L, Ceulemans B. Early Neuroschistosomiasis Complicating Katayama Syndrome. Emerg Infect Dis. ...
"Early Neuroschistosomiasis Complicating Katayama Syndrome" 12, no. 9 (2006). Clerinx, Jan et al. "Early Neuroschistosomiasis ... Animals Brain Child Cough Eosinophilia Female Fever Humans Male Neuroschistosomiasis Schistosoma Haematobium Schistosomiasis ... Title : Early Neuroschistosomiasis Complicating Katayama Syndrome Personal Author(s) : Clerinx, Jan;van Gompel, Alfons;Lynen, ... 2006). Early Neuroschistosomiasis Complicating Katayama Syndrome. 12(9). Clerinx, Jan et al. " ...
Humans, Neuroschistosomiasis, Myelitis, Transverse, Fecal Incontinence, Neuralgia, Paraplegia, Urinary Incontinence, ...
Neuroschistosomiasis. Expert Rev Anti Infect Ther. 2010 Nov. 8(11):1307-18. [QxMD MEDLINE Link]. ...
Symptomatic epilepsy can be the first manifestation of neuroschistosomiasis in patients without any systemic symptoms. The ...
Categories: Neuroschistosomiasis Image Types: Photo, Illustrations, Video, Color, Black&White, PublicDomain, ...
Neuroschistosomiasis due to Schistosoma mansoni: A review of pathogenesis, clinical syndrome and diagnostic approaches. Revista ...
Neuroschistosomiasis is diagnosed if there is infection at an extraneural site along with clinical and radiographic evidence of ...
SPINAL NEUROSCHISTOSOMIASIS CAUSED BY SCHISTOMA MANSONI: CASES REPORTED IN TWO BROTHERS. Qualis A1 , BMC INFECTIOUS DISEASES ( ...
Neuroschistosomiasis presenting as Conus Medullaris Lesion; A Case Report. PDF , Full-text , doi: 10.15744/2454-4981.4.204 ...
NEUROSCHISTOSOMIASIS. NEUROESQUISTOSSOMOSE. NEURONITIS VESTIBULAR. VESTIBULAR NEURONITIS. NEUROTONITE VESTIBULAR. NEUROPATIA ...
NEUROSCHISTOSOMIASIS. NEUROESQUISTOSSOMOSE. NEURONITIS VESTIBULAR. VESTIBULAR NEURONITIS. NEUROTONITE VESTIBULAR. NEUROPATIA ...
NEUROSCHISTOSOMIASIS. NEUROESQUISTOSSOMOSE. NEURONITIS VESTIBULAR. VESTIBULAR NEURONITIS. NEUROTONITE VESTIBULAR. NEUROPATIA ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROESQUISTOSSOMOSE NEUROTOXICITY SYNDROMES SINDROMES DE NEUROTOXICIDAD SÍNDROMES ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROESQUISTOSSOMOSE NEUROTOXICITY SYNDROMES SINDROMES DE NEUROTOXICIDAD SÍNDROMES ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROPATIA ALCOÓLICA ALCOHOLIC NEUROPATHY NEUROPATIA ALCOHOLICA NEUROPATIA CIÁTICA ...
NEUROSCHISTOSOMIASIS. NEUROESQUISTOSSOMOSE. NEURONITIS VESTIBULAR. VESTIBULAR NEURONITIS. NEUROTONITE VESTIBULAR. NEUROPATIA ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROESQUISTOSSOMOSE NEUROTOXICITY SYNDROMES SINDROMES DE NEUROTOXICIDAD SÍNDROMES ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROPATIA ALCOÓLICA ALCOHOLIC NEUROPATHY NEUROPATIA ALCOHOLICA NEUROPATIA CIÁTICA ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROESQUISTOSSOMOSE NEUROTOXICITY SYNDROMES SINDROMES DE NEUROTOXICIDAD SÍNDROMES ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROESQUISTOSSOMOSE NEUROTOXICITY SYNDROMES SINDROMES DE NEUROTOXICIDAD SÍNDROMES ...
NEUROSCHISTOSOMIASIS NEUROESQUISTOSOMIASIS NEUROPATIA ALCOÓLICA ALCOHOLIC NEUROPATHY NEUROPATIA ALCOHOLICA NEUROPATIA CIÁTICA ...
Neuroschistosomiasis; an unexpected finding in a Dutch woman].. *[Schistosomiasis imported by Spanish travelers: clinico- ... Two patients with imported acute neuroschistosomiasis due to Schistosoma mansoni.. *Imported Katayama fever: clinical and ...
Volume 76 (2007): Issue 5 (May 2007) published on May 2007 by American Society of Tropical Medicine and Hygiene.
Neuroschistosomiasis is a severe form of presentation of schistosomiasis in which Schistosoma spp. affects the central nervous ... Characterisation of ocular involvement in an experimental model of neuroschistosomiasis mansoni. Fidelis, Thiago Andre Alves; ... Characterisation of ocular involvement in an experimental model of neuroschistosomiasis mansoni Departamento de Doenças ... However, the behavioral tests did not show any clinical symptoms of neuroschistosomiasis in animals submitted to physical ...
Neuroschistosomiasis is diagnosed if there is infection at an extraneural site along with clinical and radiographic evidence of ...
... large granulomas in circumscribed areas of the brain characterize the pseudotumoral form of neuroschistosomiasis and (ancestry ...
Thousands of medical professionals trust MedLink for current, authoritative information spanning the full spectrum of neurology.
Thousands of medical professionals trust MedLink for current, authoritative information spanning the full spectrum of neurology.
Neuroschistosomiasis. *Cysticercosis. *Neurocysticercosis. Below are MeSH descriptors whose meaning is more specific than " ...
  • We conclude that in areas of the world where infection by S. mansoni is common, neuroschistosomiasis should be an important diagnostic consideration in children with neurological signs and symptoms. (nih.gov)
  • Characterisation of ocular involvement in an experimental model of neuroschistosomiasis mansoni. (shengsci.com)
  • This short communication reports ocular histolopathological findings in a murine model of neuroschistosomiasis mansoni. (shengsci.com)
  • Les biopsies ont mis en évidence des œufs de S. mansoni encapsulés par une réaction inflammatoire. (who.int)
  • A tentative diagnosis of acute neuroschistosomiasis was made, and the patient was given corticosteroids with praziquantel, 750 mg twice a day for 14 days. (cdc.gov)