Loss or destruction of periodontal tissue caused by periodontitis or other destructive periodontal diseases or by injury during instrumentation. Attachment refers to the periodontal ligament which attaches to the alveolar bone. It has been hypothesized that treatment of the underlying periodontal disease and the seeding of periodontal ligament cells enable the creating of new attachment.
An abnormal extension of a gingival sulcus accompanied by the apical migration of the epithelial attachment and bone resorption.
A numerical rating scale for classifying the periodontal status of a person or population with a single figure which takes into consideration prevalence as well as severity of the condition. It is based upon probe measurement of periodontal pockets and on gingival tissue status.
An index which scores the degree of dental plaque accumulation.
Pathological processes involving the PERIODONTIUM including the gum (GINGIVA), the alveolar bone (ALVEOLAR PROCESS), the DENTAL CEMENTUM, and the PERIODONTAL LIGAMENT.
Inflammation of gum tissue (GINGIVA) without loss of connective tissue.
Devices used in the home by persons to maintain dental and periodontal health. The devices include toothbrushes, dental flosses, water irrigators, gingival stimulators, etc.
Inflammation and loss of connective tissues supporting or surrounding the teeth. This may involve any part of the PERIODONTIUM. Periodontitis is currently classified by disease progression (CHRONIC PERIODONTITIS; AGGRESSIVE PERIODONTITIS) instead of age of onset. (From 1999 International Workshop for a Classification of Periodontal Diseases and Conditions, American Academy of Periodontology)
Horizontal and, to a lesser degree, axial movement of a tooth in response to normal forces, as in occlusion. It refers also to the movability of a tooth resulting from loss of all or a portion of its attachment and supportive apparatus, as seen in periodontitis, occlusal trauma, and periodontosis. (From Jablonski, Dictionary of Dentistry, 1992, p507 & Boucher's Clinical Dental Terminology, 4th ed, p313)
The movement of teeth into altered positions in relationship to the basal bone of the ALVEOLAR PROCESS and to adjoining and opposing teeth as a result of loss of approximating or opposing teeth, occlusal interferences, habits, inflammatory and dystrophic disease of the attaching and supporting structures of the teeth. (From Boucher's Clinical Dental Terminology, 4th ed)
The flowing of blood from the marginal gingival area, particularly the sulcus, seen in such conditions as GINGIVITIS, marginal PERIODONTITIS, injury, and ASCORBIC ACID DEFICIENCY.
Chronic inflammation and loss of PERIODONTIUM that is associated with the amount of DENTAL PLAQUE or DENTAL CALCULUS present. Chronic periodontitis occurs mostly in adults and was called adult periodontitis, but this disease can appear in young people.
Exposure of the root surface when the edge of the gum (GINGIVA) moves apically away from the crown of the tooth. This is common with advancing age, vigorous tooth brushing, diseases, or tissue loss of the gingiva, the PERIODONTAL LIGAMENT and the supporting bone (ALVEOLAR PROCESS).
Removal of dental plaque and dental calculus from the surface of a tooth, from the surface of a tooth apical to the gingival margin accumulated in periodontal pockets, or from the surface coronal to the gingival margin.
The practice of personal hygiene of the mouth. It includes the maintenance of oral cleanliness, tissue tone, and general preservation of oral health.
A film that attaches to teeth, often causing DENTAL CARIES and GINGIVITIS. It is composed of MUCINS, secreted from salivary glands, and microorganisms.
A fluid occurring in minute amounts in the gingival crevice, believed by some authorities to be an inflammatory exudate and by others to cleanse material from the crevice, containing sticky plasma proteins which improve adhesions of the epithelial attachment, have antimicrobial properties, and exert antibody activity. (From Jablonski, Illustrated Dictionary of Dentistry, 1982)
Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)
Inflammation and loss of PERIODONTIUM that is characterized by rapid attachment loss and bone destruction in the presence of little local factors such as DENTAL PLAQUE and DENTAL CALCULUS. This highly destructive form of periodontitis often occurs in young people and was called early-onset periodontitis, but this disease also appears in old people.

The antimicrobial treatment of periodontal disease: changing the treatment paradigm. (1/161)

Over the last 100 years, methods of surgical periodontal treatment have enjoyed a history of success in improving oral health. The paradigm of care is based on the "non-specific plaque hypothesis"--that is, the overgrowth of bacterial plaques cause periodontal disease, and the suppression of this overgrowth reduces disease risk. The central feature of this approach to care is the removal of inflamed gingival tissue around the teeth to reduce periodontal pocket depth, thereby facilitating plaque removal by the dentist and by the patient at home. Over the last 30 years, with the recognition that periodontal disease(s) is caused by specific bacteria and that specific antimicrobial agents can reduce or eliminate the infection, a second paradigm has developed. This new paradigm, the "specific plaque hypothesis", focuses on reducing the specific bacteria that cause periodontal attachment loss. The contrast between the two paradigms can be succinctly stated as follows: The antimicrobial therapy reduces the cause, while the surgical therapy reduces the result of the periodontal infection. The specific plaque hypothesis has two important implications. First, with the increasing attention to evidence-based models for prevention, treatment, outcome assessment, and reimbursement of care, increasing attention and financial effort will be channeled into effective preventive and treatment methods. Second, the recent observations that periodontal infections increase the risk of specific systemic health problems, such as cardiovascular disease, argue for the prevention and elimination of these periodontal infections. This review highlights some of the evidence for the specific plaque hypothesis, and the questions that should be addressed if antimicrobial agents are to be used responsively and effectively.  (+info)

Tooth loss in periodontal patients. (2/161)

OBJECTIVE: To compare tooth loss between patients who received surgical therapy for chronic periodontitis and those who received nonsurgical therapy alone. METHODS: A retrospective chart study was conducted at Dalhousie University. All patients who had periodontal treatment and were active cases for at least 10 years were included (n = 335). The sample consisted of 120 males (35.8%) and 215 females (64.2%). Ages ranged from 16 to 77 (mean = 46.1 +/- 12.0 years). All patients received nonsurgical therapy; 44.8% received periodontal surgery as well. Variables recorded were demographics, initial attachment loss, treatment type, recall frequency, patient compliance and history of extracted teeth. Independent t-tests or chi-squared tests were used to compare these for surgical and nonsurgical patients. ANOVA was used to test for interactions between initial attachment loss, age, gender, compliance and type of therapy a patient received as reasons for tooth loss. RESULTS: 521 teeth were lost in 69 patients (20.6% of sample). Of teeth lost, 61.8% were due to periodontal disease; 24.8% to caries; 13.2% to other reasons. Patients initially diagnosed with early attachment loss lost an average of 0.37 (+/- 1.33) teeth. Patients diagnosed with moderate attachment loss lost an average of 1.50 (+/- 2.54) teeth and those diagnosed with advanced attachment loss lost an average of 3.11 (+/- 3.01) teeth. Those who received surgical therapy lost more teeth (mean = 1.31 +/- 2.36) than those who received nonsurgical treatment (mean = 0.68 +/- 1.87; p = 0.001). However, initial attachment loss was the only factor that could predict tooth loss. The type of therapy (surgical or nonsurgical) was not statistically significant. CONCLUSIONS: Most periodontal patients (79.4%) who received treatment at this dental school clinic did not lose any teeth due to periodontal disease over at least 10 years. Although patients who had surgical therapy lost more teeth than those who had nonsurgical therapy alone, this was not an important predictor of tooth loss.  (+info)

C-telopeptide pyridinoline cross-links (ICTP) and periodontal pathogens associated with endosseous oral implants. (3/161)

Detection of periodontal or peri-implant sites exhibiting progressing disease or those at risk of deterioration has proven difficult. Pyridinoline cross-linked carboxyterminal telopeptide of type I collagen (ICTP), a marker specific for bone degradation found in gingival crevicular fluid (GCF), has been associated with both bone and attachment loss in periodontitis and may be useful for predicting disease activity. The aim of this cross-sectional study was to examine the relationship between ICTP levels and subgingival species around implants and teeth from 20 partially and 2 fully edentulous patients. GCF and plaque samples were collected from the mesiobuccal site of each implant and tooth. Radioimmunoassay techniques were utilized to determine GCF ICTP levels. Plaque samples were analyzed utilizing checkerboard DNA-DNA hybridization. Traditional clinical parameters were assessed. Seventy-one implants and 370 teeth from 22 subjects were examined. ICTP levels and subgingival plaque composition were not significantly different between implants and teeth. Implant sites colonized by Prevotella intermedia, Capnocytophaga gingivalis, Fusobacterium nucleatum ss vincentii, and Streptococcus gordonii exhibited odds ratios of 12.4, 9.3, 8.1, and 6.7, respectively of detecting ICTP. These results suggest a relationship between elevated ICTP levels at implant sites and some species associated with disease progression. Longitudinal studies are necessary to determine whether elevated ICTP levels may predict the development of peri-implant bone loss.  (+info)

The oral cleanliness and periodontal health of UK adults in 1998. (4/161)

Periodontal disease continues to be a major concern for dentists and patients. This paper reports the findings of the 1998 UK Adult Dental Health survey in relation to plaque, calculus, periodontal pocketing and loss of attachment. It is apparent from this study that moderate periodontal disease remains commonplace amongst UK adults and that the associated risk factors of plaque and calculus are in abundance, even amongst those who profess to be motivated about their oral health and attend the dentist regularly. The continued high prevalence of disease needs to be seen in the context of the far larger number of people who are now potentially at some risk, particularly in the older age groups, because of improvements in tooth retention. However, the cumulative effect of disease means that control of the periodontal diseases, even mild and slowly progressing disease, will be a key issue if large numbers of teeth are to be retained into old age. If that level of control is to be achieved we need a widespread improvement in our management of the disease, particularly in our ability to improve the oral cleanliness of the UK population.  (+info)

Tissue reaction to orthodontic tooth movement--a new paradigm. (5/161)

Direct or indirect resorption are both perceived as a reaction to an applied force. This is in contrast to orthopaedic surgeons who describe apposition as 'the reaction to loading of bone'. The article reviews the literature on intrusion of teeth with periodontal breakdown, and on the basis of clinical and experimental studies. The conclusion is reached that intrusion can lead to an improved attachment level, and that forces have to be to low and continuous. The tissue reaction to a force system generating translation of premolars and molars in the five Macaca fascicularis monkeys is described. Three force levels, 100, 200, and 300 cN were applied for a period of 11 weeks. Undecalcified serial sections were cut parallel to the occlusal plane and a grid consisting of three concentric outlines of the root intersected by six radii was placed on each section so that areas anticipated to be subject to differing stress/strain distributions were isolated. A posteriori tests were utilized in order to separate areas that differed with regard to parameters reflecting bone turnover. Based on these results and a finite element model simulating the loading, a new hypothesis regarding tissue reaction to change in the stress strain distribution generated by orthodontic forces is suggested. The direct resorption could be perceived as a result of lowering of the normal strain from the functioning periodontal ligament (PDL) and as such as a start of remodelling, in the bone biological sense of the word. Indirect remodelling could be perceived as sterile inflammation attempting to remove ischaemic bone under the hyalinized tissue. At a distance from the alveolus, dense woven bone was observed as a sign of a regional acceleratory phenomena (RAP). The results of the intrusion could, according to the new hypothesis, be perceived as bending of the alveolar wall produced by the pull from Sharpey's fibres.  (+info)

Inflammation and tissue loss caused by periodontal pathogens is reduced by interleukin-1 antagonists. (6/161)

Periodontal disease is a significant cause of tooth loss among adults. It is initiated by pathogenic bacteria, which trigger an inflammatory response that is effective in preventing significant microbial colonization of the gingival tissues. In some individuals, the reaction to bacteria may lead to an excessive host response, resulting in periodontal tissue destruction. Recent developments suggest that interleukin (IL)-1 genetic polymorphisms may identify certain individuals who have a predisposed susceptibility to periodontal breakdown and that elevated levels of IL-1 are found in individuals with periodontal disease. However, there is no direct evidence that IL-1 per se is responsible for the critical events that occur in periodontitis. We investigated the role of IL-1 in periodontal disease in a Macaca fascicularis primate model, using human soluble IL-1 receptor type I as a specific inhibitor. The results indicate that inhibition of IL-1 alone significantly reduces inflammation, connective tissue attachment loss, and bone resorption that are induced by periodontal pathogens.  (+info)

Antibody-based diagnostic for 'refractory' periodontitis. (7/161)

OBJECTIVE: About 10-15% of US adults are 'refractory' to therapy for chronic periodontitis. Recently, studies suggest that these patients have elevated lysine decarboxylase activity in the sulcular microbiota. The aim of this study was to determine whether an elevated IgG antibody response to lysine decarboxylase, alone or with antibody to other bacterial antigens and baseline clinical measurements, would predict 'refractory' patients with high accuracy. METHODS: Chronic periodontitis patients were treated using scaling and root planing (SRP) followed by maintenance SRP and 3-monthly re-examinations. If there was a loss of mean full mouth attachment or more than three sites appeared with > 2.5 mm new loss within a year, the subjects were re-treated (modified Widman flap surgery and systemically administered tetracycline). If attachment loss as above recurred, the subjects were 'refractory'. Baseline clinical measurements and specific antibody responses were used in a logistic regression model to predict 'refractory' subjects. RESULTS: Antibody to a peptide portion of lysine decarboxylase (HKL-Ab) and baseline bleeding on probing (BOP) prevalence measurements predicted attachment loss 3 months after initial therapy [pIAL = loss (0) or gain (1)]. IgG antibody contents to a purified antigen from Actinomyces spp. (A-Ab) and streptococcal d-alanyl glycerol lipoteichoic acid (S-Ab) were related in 'refractory' patients (R2 = 0.37, p < 0.01). From the regression equation, the relationship between the antibodies was defined as linear (pLA/S-Ab = 0) or non-linear pLA/S-Ab = 1). Using pLA/S-Ab, pIAL and age, a logistic regression equation was derived from 48 of the patients. Of 59 subjects, 37 had 2-4 mm attachment loss and were assigned as 'refractory' or successfully treated with 86% accuracy. CONCLUSION: HKL-Ab facilitated an accurate prediction of therapeutic outcome in subjects with moderate periodontitis.  (+info)

Periodontal disease as a risk factor for ischemic stroke. (8/161)

BACKGROUND AND PURPOSE: Chronic infectious diseases may increase the risk of stroke. We investigated whether periodontal disease, including periodontitis and gingivitis, is a risk factor for cerebral ischemia. METHODS: We performed a case-control study with 303 patients examined within 7 days after acute ischemic stroke or transient ischemic attack, 300 population controls, and 168 hospital controls with nonvascular and noninflammatory neurological diseases. All subjects received a complete clinical and radiographic dental examination. The individual mean clinical attachment loss measured at 4 sites per tooth served as the main indicator for periodontitis. RESULTS: Patients had higher clinical attachment loss than population (P<0.001) and hospital (P=0.010) controls. After adjustment for age, sex, number of teeth, vascular risk factors and diseases, childhood and adult socioeconomic conditions, and lifestyle factors, the risk of cerebral ischemia increased with more severe periodontitis. Subjects with severe periodontitis (mean clinical attachment loss >6 mm) had a 4.3-times-higher (95% confidence interval, 1.85 to 10.2) risk of cerebral ischemia than subjects with mild or without periodontitis (+info)

Periodontal attachment loss (PAL) is a clinical measurement in dentistry that refers to the amount of connective tissue attachment between the tooth and its surrounding supportive structures (including the gingiva, periodontal ligament, and alveolar bone) that has been lost due to periodontal disease. It is typically expressed in millimeters and represents the distance from the cementoenamel junction (CEJ), which is the point where the tooth's crown meets the root, to the bottom of the periodontal pocket.

Periodontal pockets are formed when the gums detach from the tooth due to inflammation and infection caused by bacterial biofilms accumulating on the teeth. As the disease progresses, more and more of the supporting structures are destroyed, leading to increased pocket depths and attachment loss. This can eventually result in loose teeth and even tooth loss if left untreated.

Therefore, periodontal attachment loss is an important indicator of the severity and progression of periodontal disease, and its measurement helps dental professionals assess the effectiveness of treatment interventions and monitor disease status over time.

A periodontal pocket is a pathological space or gap that develops between the tooth and the surrounding gum tissue (gingiva) as a result of periodontal disease. This condition is also known as a "periodontal depth" or "probing depth." It is measured in millimeters using a dental probe, and it indicates the level of attachment loss of the gingival tissue to the tooth.

In a healthy periodontium, the sulcus (the normal space between the tooth and gum) measures 1-3 mm in depth. However, when there is inflammation due to bacterial accumulation, the gums may become red, swollen, and bleed easily. As the disease progresses, the sulcus deepens, forming a periodontal pocket, which can extend deeper than 3 mm.

Periodontal pockets provide an environment that is conducive to the growth of harmful bacteria, leading to further tissue destruction and bone loss around the tooth. If left untreated, periodontal disease can result in loose teeth and eventually tooth loss. Regular dental check-ups and professional cleanings are essential for maintaining healthy gums and preventing periodontal pockets from developing or worsening.

The Periodontal Index (PI) is not a current or widely used medical/dental term. However, in the past, it was used to describe a method for assessing and measuring the severity of periodontal disease, also known as gum disease.

Developed by Henry H. Klein and colleagues in 1978, the Periodontal Index was a scoring system that evaluated four parameters: gingival inflammation, gingival bleeding, calculus (tartar) presence, and periodontal pocket depths. The scores for each parameter ranged from 0 to 3, with higher scores indicating worse periodontal health. The overall PI score was the sum of the individual parameter scores, ranging from 0 to 12.

However, due to its limited ability to predict future disease progression and the introduction of more comprehensive assessment methods like the Community Periodontal Index (CPI) and the Basic Periodontal Examination (BPE), the use of the Periodontal Index has become less common in dental practice and research.

The dental plaque index (DPI) is a clinical measurement used in dentistry to assess the amount of dental plaque accumulation on a person's teeth. It was first introduced by Silness and Löe in 1964 as a method to standardize the assessment of oral hygiene and the effectiveness of oral hygiene interventions.

The DPI is based on a visual examination of the amount of plaque present on four surfaces of the teeth, including the buccal (cheek-facing) and lingual (tongue-facing) surfaces of both upper and lower first molars and upper and lower incisors. The examiner assigns a score from 0 to 3 for each surface, with higher scores indicating greater plaque accumulation:

* Score 0: No plaque detected, even after probing the area with a dental explorer.
* Score 1: Plaque detected by visual examination and/or probing but is not visible when the area is gently dried with air.
* Score 2: Moderate accumulation of soft deposits that are visible upon visual examination before air drying, but which can be removed by scraping with a dental explorer.
* Score 3: Abundant soft matter, visible upon visual examination before air drying and not easily removable with a dental explorer.

The DPI is calculated as the average score of all surfaces examined, providing an overall measure of plaque accumulation in the mouth. It can be used to monitor changes in oral hygiene over time or to evaluate the effectiveness of different oral hygiene interventions. However, it should be noted that the DPI has limitations and may not accurately reflect the presence of bacterial biofilms or the risk of dental caries and gum disease.

According to the American Academy of Periodontology, periodontal diseases are chronic inflammatory conditions that affect the tissues surrounding and supporting the teeth. These tissues include the gums, periodontal ligament, and alveolar bone. The primary cause of periodontal disease is bacterial plaque, a sticky film that constantly forms on our teeth.

There are two major stages of periodontal disease:

1. Gingivitis: This is the milder form of periodontal disease, characterized by inflammation of the gums (gingiva) without loss of attachment to the teeth. The gums may appear red, swollen, and bleed easily during brushing or flossing. At this stage, the damage can be reversed with proper dental care and improved oral hygiene.
2. Periodontitis: If left untreated, gingivitis can progress to periodontitis, a more severe form of periodontal disease. In periodontitis, the inflammation extends beyond the gums and affects the deeper periodontal tissues, leading to loss of bone support around the teeth. Pockets filled with infection-causing bacteria form between the teeth and gums, causing further damage and potential tooth loss if not treated promptly.

Risk factors for developing periodontal disease include poor oral hygiene, smoking or using smokeless tobacco, genetic predisposition, diabetes, hormonal changes (such as pregnancy or menopause), certain medications, and systemic diseases like AIDS or cancer. Regular dental check-ups and good oral hygiene practices are crucial for preventing periodontal disease and maintaining overall oral health.

Gingivitis is a mild form of gum disease (periodontal disease) that causes irritation, redness, swelling and bleeding of the gingiva, or gums. It's important to note that it is reversible with good oral hygiene and professional dental treatment. If left untreated, however, gingivitis can progress to a more severe form of gum disease known as periodontitis, which can result in tissue damage and eventual tooth loss.

Gingivitis is most commonly caused by the buildup of plaque, a sticky film of bacteria that constantly forms on our teeth. When not removed regularly through brushing and flossing, this plaque can harden into tartar, which is more difficult to remove and contributes to gum inflammation. Other factors like hormonal changes, poor nutrition, certain medications, smoking or a weakened immune system may also increase the risk of developing gingivitis.

Dental devices for home care are products designed for use by individuals or their caregivers in a home setting to maintain oral hygiene, manage dental health issues, and promote overall oral health. These devices can include:

1. Toothbrushes: Manual, electric, or battery-operated toothbrushes used to clean teeth and remove plaque and food debris.
2. Dental floss: A thin string used to remove food particles and plaque from between the teeth and under the gum line.
3. Interdental brushes: Small brushes designed to clean between the teeth and around dental appliances, such as braces or implants.
4. Water flossers/oral irrigators: Devices that use a stream of water to remove food particles and plaque from between the teeth and under the gum line.
5. Tongue scrapers: Tools used to clean the tongue's surface, removing bacteria and reducing bad breath.
6. Rubber tips/gum stimulators: Devices used to massage and stimulate the gums, promoting blood circulation and helping to maintain gum health.
7. Dental picks/sticks: Pointed tools used to remove food particles and plaque from between the teeth and under the gum line.
8. Mouthguards: Protective devices worn over the teeth to prevent damage from grinding, clenching, or sports-related injuries.
9. Night guards: Similar to mouthguards, these are designed to protect the teeth from damage caused by nighttime teeth grinding (bruxism).
10. Dental retainers: Devices used to maintain the alignment of teeth after orthodontic treatment.
11. Whitening trays and strips: At-home products used to whiten teeth by applying a bleaching agent to the tooth surface.
12. Fluoride mouth rinses: Anticavity rinses containing fluoride, which help strengthen tooth enamel and prevent decay.
13. Oral pain relievers: Topical gels or creams used to alleviate oral pain, such as canker sores or denture irritation.

Proper use of these dental devices, along with regular dental check-ups and professional cleanings, can help maintain good oral health and prevent dental issues.

Periodontitis is a severe form of gum disease that damages the soft tissue and destroys the bone supporting your teeth. If left untreated, it can lead to tooth loss. It is caused by the buildup of plaque, a sticky film of bacteria that constantly forms on our teeth. The body's immune system fights the bacterial infection, which causes an inflammatory response. If the inflammation continues for a long time, it can damage the tissues and bones that support the teeth.

The early stage of periodontitis is called gingivitis, which is characterized by red, swollen gums that bleed easily when brushed or flossed. When gingivitis is not treated, it can advance to periodontitis. In addition to plaque, other factors that increase the risk of developing periodontitis include smoking or using tobacco products, poor oral hygiene, diabetes, a weakened immune system, and genetic factors.

Regular dental checkups and good oral hygiene practices, such as brushing twice a day, flossing daily, and using an antimicrobial mouth rinse, can help prevent periodontitis. Treatment for periodontitis may include deep cleaning procedures, medications, or surgery in severe cases.

Tooth mobility, also known as loose teeth, refers to the degree of movement or displacement of a tooth in its socket when lateral forces are applied. It is often described in terms of grades:

* Grade 1: Tooth can be moved slightly (up to 1 mm) with finger pressure.
* Grade 2: Tooth can be moved up to 2 mm with finger pressure.
* Grade 3: Tooth can be moved more than 2 mm or can be removed from its socket with manual pressure.

Increased tooth mobility can be a sign of periodontal disease, trauma, or other dental conditions and should be evaluated by a dentist. Treatment may include deep cleaning, splinting, or surgery to restore stability to the affected teeth.

Tooth migration, in a dental or medical context, refers to the movement or shifting of teeth from their normal position within the dental arch. This phenomenon can occur due to various reasons such as:

1. Loss of adjacent teeth: When a tooth is lost, the surrounding teeth may drift or tilt into the empty space, causing other teeth to migrate out of their original positions.
2. Periodontal disease: Advanced periodontitis (severe gum disease) can lead to bone loss and ligament damage around the teeth, allowing them to move and potentially migrate.
3. Orthodontic treatment: Although controlled tooth movement is the goal of orthodontics, improper or unfinished treatment may result in undesirable tooth migration.
4. Aging: As people age, the supportive structures around teeth (bone and ligaments) can weaken, leading to tooth mobility and potential migration.
5. Tooth wear: Excessive tooth wear due to bruxism (grinding) or abrasion may alter the vertical dimension of the mouth, causing tooth migration over time.

It is essential to address tooth migration promptly to prevent further complications such as difficulty in chewing, speaking, and maintaining oral hygiene, which could lead to additional dental issues like decay and periodontal disease. Dental professionals may recommend various treatments, including orthodontic therapy, dental restorations, or even implants, depending on the cause and severity of tooth migration.

Gingival hemorrhage is the medical term for bleeding of the gingiva, or gums. It refers to the condition where the gums bleed, often as a result of trauma or injury, but also can be caused by various systemic conditions such as disorders of coagulation, leukemia, or scurvy.

Gingival hemorrhage is commonly seen in individuals with poor oral hygiene and periodontal disease, which can cause inflammation and damage to the gums. This can lead to increased susceptibility to bleeding, even during routine activities such as brushing or flossing. It's important to address any underlying causes of gingival hemorrhage to prevent further complications.

Chronic periodontitis is a type of gum disease that is characterized by the inflammation and infection of the tissues surrounding and supporting the teeth. It is a slow-progressing condition that can lead to the destruction of the periodontal ligament and alveolar bone, which can result in loose teeth or tooth loss if left untreated.

Chronic periodontitis is caused by the buildup of dental plaque and calculus (tartar) on the teeth, which harbor bacteria that release toxins that irritate and inflame the gums. Over time, this chronic inflammation can lead to the destruction of the periodontal tissues, including the gingiva, periodontal ligament, and alveolar bone.

The signs and symptoms of chronic periodontitis include:

* Red, swollen, or tender gums
* Bleeding gums during brushing or flossing
* Persistent bad breath (halitosis)
* Receding gums (exposure of the tooth root)
* Loose teeth or changes in bite alignment
* Deep periodontal pockets (spaces between the teeth and gums)

Risk factors for chronic periodontitis include poor oral hygiene, smoking, diabetes, genetics, and certain medications. Treatment typically involves a thorough dental cleaning to remove plaque and calculus, followed by additional procedures such as scaling and root planing or surgery to eliminate infection and promote healing of the periodontal tissues. Good oral hygiene practices, regular dental checkups, and quitting smoking are essential for preventing chronic periodontitis and maintaining good oral health.

Gingival recession is the term used to describe the exposure of the root surface of a tooth as a result of the loss of gum tissue (gingiva) due to periodontal disease or improper oral hygiene practices. It can also occur due to other factors such as aggressive brushing, grinding or clenching of teeth, and misaligned teeth. Gingival recession is often characterized by red, swollen, or sensitive gums, and can lead to tooth sensitivity, decay, and even tooth loss if left untreated.

Dental scaling is a professional dental cleaning procedure that involves the removal of plaque, tartar (calculus), and stains from the tooth surfaces. This is typically performed by a dentist or dental hygienist using specialized instruments called scalers and curettes. The procedure helps to prevent gum disease and tooth decay by removing bacterial deposits that can cause inflammation and infection of the gums. Dental scaling may be recommended as part of a routine dental check-up or if there are signs of periodontal disease, such as red, swollen, or bleeding gums. In some cases, local anesthesia may be used to numb the area and make the procedure more comfortable for the patient.

Oral hygiene is the practice of keeping the mouth and teeth clean to prevent dental issues such as cavities, gum disease, bad breath, and other oral health problems. It involves regular brushing, flossing, and using mouthwash to remove plaque and food particles that can lead to tooth decay and gum disease. Regular dental check-ups and cleanings are also an essential part of maintaining good oral hygiene. Poor oral hygiene can lead to a range of health problems, including heart disease, diabetes, and respiratory infections, so it is important to prioritize oral health as part of overall health and wellbeing.

Dental plaque is a biofilm or mass of bacteria that accumulates on the surface of the teeth, restorative materials, and prosthetic devices such as dentures. It is initiated when bacterial colonizers attach to the smooth surfaces of teeth through van der Waals forces and specific molecular adhesion mechanisms.

The microorganisms within the dental plaque produce extracellular polysaccharides that help to stabilize and strengthen the biofilm, making it resistant to removal by simple brushing or rinsing. Over time, if not regularly removed through oral hygiene practices such as brushing and flossing, dental plaque can mineralize and harden into tartar or calculus.

The bacteria in dental plaque can cause tooth decay (dental caries) by metabolizing sugars and producing acid that demineralizes the tooth enamel. Additionally, certain types of bacteria in dental plaque can cause periodontal disease, an inflammation of the gums that can lead to tissue damage and bone loss around the teeth. Regular professional dental cleanings and good oral hygiene practices are essential for preventing the buildup of dental plaque and maintaining good oral health.

Gingival crevicular fluid (GCF) is defined as the serum transudate or inflammatory exudate that flows from the gingival sulcus or periodontal pocket. It is a physiological fluid found in the narrow space between the tooth and the surrounding gum tissue, which deepens during periodontal disease. The analysis of GCF has been used as a non-invasive method to assess the status of periodontal health and disease since it contains various markers of inflammation, host response, and bacterial products.

An encyclopedia is a comprehensive reference work containing articles on various topics, usually arranged in alphabetical order. In the context of medicine, a medical encyclopedia is a collection of articles that provide information about a wide range of medical topics, including diseases and conditions, treatments, tests, procedures, and anatomy and physiology. Medical encyclopedias may be published in print or electronic formats and are often used as a starting point for researching medical topics. They can provide reliable and accurate information on medical subjects, making them useful resources for healthcare professionals, students, and patients alike. Some well-known examples of medical encyclopedias include the Merck Manual and the Stedman's Medical Dictionary.

Aggressive periodontitis is a severe form of periodontal disease that affects the tissues surrounding and supporting the teeth, including the gums, periodontal ligament, and alveolar bone. It is characterized by rapid destruction of the periodontal tissues and can result in significant tooth loss if left untreated.

Aggressive periodontitis typically affects younger individuals, often before the age of 30, and can progress rapidly, even in the absence of obvious dental plaque or calculus accumulation. It is often associated with a genetic predisposition and may cluster in families.

The disease is classified as localized or generalized based on the distribution of affected sites. Localized aggressive periodontitis typically affects no more than two teeth next to each other, while generalized aggressive periodontitis involves at least three or four teeth in different areas of the mouth.

In addition to genetic factors, other risk factors for aggressive periodontitis include smoking, diabetes, and hormonal changes. Treatment typically involves a combination of thorough dental cleanings, antibiotics, and sometimes surgical intervention to remove damaged tissue and promote healing. Regular maintenance care is essential to prevent recurrence and further progression of the disease.

Results: Smokers had greater overall mean PD (P = 0.001) and attachment loss (P = 0.006) and fewer bleeding on probing sites (P ... A full-mouth periodontal examination was performed, and a pooled subgingival plaque sample was collected from the deepest site ... Background: The purpose of the present investigation is to compare the presence and number of periodontal pathogens in the ... Detection and quantification of periodontal pathogens in smokers and never-smokers with chronic periodontitis by real-time ...
"A 5-year study of attachment loss and tooth loss in community-dwelling older adults". Journal of Periodontal Research. 32 (6): ... Susin C, Dalla Vecchia CF, Oppermann RV, Haugejorden O, Albandar JM (July 2004). "Periodontal attachment loss in an urban ... He found that the best baseline predictors of tooth loss of all remaining teeth was that at least one tooth had attachment loss ... Epidemiology of periodontal disease is the study of patterns, causes, and effects of periodontal diseases. Periodontal disease ...
... www.ada.org/en/science-research/dental-practice-parameters/gingival-inflammation-without-loss-of-periodontal-attachment. ... Gingival inflammation without loss of periodontal attachment (gingivitis). American Dental Association. http:// ... Gingivitis is a common and mild form of gum disease (periodontal disease) that causes irritation, redness and swelling ( ... Gingivitis can lead to much more serious gum disease called periodontitis and tooth loss. ...
Categories: Periodontal Attachment Loss Image Types: Photo, Illustrations, Video, Color, Black&White, PublicDomain, ...
The primary outcome was probing pocket depth (PPD). Secondary outcomes were bleeding on probing, clinical attachment loss, and ... "attachment loss"[All Fields] OR "clinical attachment"[All Fields] OR "bleeding on probing"[All Fields] OR "Periodontal Diseases ... periodontal probing depth, PPD [33]; secondary outcomes: bleeding on probing, BOP [34], and clinical attachment loss, CAL [33 ... Anwaristi, A.Y. Effect of coenzyme-q10 in the post-curettage against probing depth, relative attachment loss, and bleeding on ...
... and its universal characteristics are attachment loss, pocket formation, and ultimate tooth loss. Researches show that 80% of ... Once attachment is lost, it is almost impossible to regain it. In other word, periodontal disease is irreversible and incurable ... There is loss of integrity of the sulcular epithelium. Bacteria and their byproducts migrate into the periodontal ligament ... Pseudopockets, as the name implies, are not due to attachment loss but rather to gingival hyperplasia creeping up the side of ...
Attachment loss (HMDB: HMDB0000718). *Periodontal Probing Depth (HMDB: HMDB0000718). *Supragingival Plaque (HMDB: HMDB0000718) ... Attachment loss. *Liebsch C, Pitchika V, Pink C, Samietz S, Kastenmuller G, Artati A, Suhre K, Adamski J, Nauck M, Volzke H, ... Periodontal Probing Depth. *Liebsch C, Pitchika V, Pink C, Samietz S, Kastenmuller G, Artati A, Suhre K, Adamski J, Nauck M, ...
... including loss of teeth, compared with controls. Antiinflammatory treatment interferes with periodontal disease and might have ... Patients with longstanding active RA have a substantially increased frequency of periodontal disease, ... Correlations between indices of chronic destruction in periodontal disease (gingival attachment loss) and in RA (Larsen ... greater attachment loss (increased by 173%), and higher number of missing teeth (increased by 29%) compared with controls. No ...
... and its universal characteristics are attachment loss, pocket formation, and ultimate tooth loss. Researches show that 80% of ... Once attachment is lost it is almost impossible to regain it. In other word, periodontal disease is irreversible and incurable ... There is loss of integrity of the sulcular epithelium. Bacteria and their by-products migrate into the periodontal ligament ... Pseudopockets, as the name implies, are not due to attachment loss but rather to gingival hyperplasia creeping up the side of ...
PRNewswire-USNewswire/ -- Injectable progesterone contraceptives may be associated with poor periodontal health, according to ... All participants received a dental examination that noted clinical attachment (CA) loss, periodontal pocket assessment at two ... Periodontal disease is a chronic inflammatory disease that affects the gum tissue and bone that supports the teeth. Gingivitis ... "Hormones can play a role in womans periodontal health. These findings suggest that women that use, or have used, a hormone- ...
The gingiva progressively loses its attachment to the teeth, bone loss begins, and periodontal pockets deepen. With progressive ... This photo shows a close-up of tissue loss in a person with periodontal disease. Periodontal disease has caused a loss of gum ... Dental x-rays reveal alveolar bone loss adjacent to the periodontal pockets. ... Other periodontal conditions included in the AAP designations are periodontal abscesses, periodontitis associated with ...
... or more attachment loss.. Periodontal disease can be reversible in its earliest form. This form of periodontal disease is ... Periodontal disease is progressive and without periodontal disease treatment will lead to tooth loss and infections throughout ... Stages of Periodontal Disease. Periodontal disease is said to affect over 50% of all Americans. Periodontal disease advances in ... These support tissues include, gums, bone, cementum, and ligament attachments.. The main area in which periodontal disease is ...
Periodontal Attachment Loss/epidemiology, Periodontal Diseases/epidemiology, Periodontal Index, Periodontitis/epidemiology, ... Cigar, pipe, and cigarette smoking as risk factors for periodontal disease and tooth loss 2000 Department of Periodontology, ... We also investigated whether a history of smoking habits cessation may affect the risk of periodontal disease and tooth loss. ... Smoking cessation efforts should be considered as a means of improving periodontal health and reducing tooth loss in heavy ...
Find out about how common periodontal disease is with dogs, how it develops and how to spot it, and what you can do to prevent ... At this stage in the disease a dog can suffer a 25-30% loss of attachment in the affected teeth making them loose. When this ... In this case, there is at least a 50% loss of attachment in the affected teeth, and the gum tissue is usually receding and ... What is periodontal disease?. Periodontal disease is the inflammation of the structures of the mouth as a result of a bacterial ...
Study found people with deep periodontal pockets with a mean value greater than two millimeters had an increased risk for ECG ... And, people with severe attachment loss with a mean value greater than 2.5 millimeters had a significant risk for ECG ... People with deep periodontal pockets had an increased risk for electrocardiographic abnormalities (ECG) according to a recent ... We found that people with deep periodontal pockets with a mean value greater than two millimeters had an increased risk for ECG ...
... and clinical attachment loss (AL). All clinical parameters were measured with a Williams periodontal probe (Hu-Friedy). ... Although there were no significant differences among the groups for mean attachment loss (AL), probing depth (PD), and plaque ... iv). Patients with any disease that can affect periodontal health.. v). Patients who had received any periodontal treatment in ... Clinical periodontal measurements were obtained from six points around each tooth except third molars. The patients were ...
... clinical attachment loss (CAL), and periodontal description. Immediately after writing all her notes she turned her light on ... From there, she took periodontal probing depths (PPD), gingival margin to cementoenamel junction readings (GM to CEJ), ...
The researchers also measured probing pocket depth and clinical attachment loss to reflect periodontal status on a half-mouth ... a 0.23 mm greater increase in mean clinical attachment loss, and a relative risk of 1.57 for tooth loss compared to subjects in ... mean clinical attachment loss, and risk ratios of tooth loss over time. ... Gum disease can lead to deeper spaces around the teeth called periodontal pockets, and if untreated, these gum pockets could ...
... influencing patient compliance in a periodontal maintenance program and its influence on tooth loss and clinical attachment ... Patient-level risk factors and clinical parameters, including tooth loss throughout periodontal maintenance therapy, were ... program influences tooth loss and clinical attachment level. Data from 759 patients who were treated by a periodontist between ... Subjects were at least 35 years old, underwent active periodontal therapy and were maintained for a minimum of 5 years. ...
Loss of attachment score was significantly higher in smokers before treatment and remained higher after treatment. Smokers ... Both groups were examined periodontally for plaque, bleeding and loss of attachment, before and after a course of treatment ... with oral hygiene instructions, scaling, root planning periodontal and polishing. Before treatment, mean bleeding index score ... showed more signs of periodontal disease, and treatment did not reverse this fully. ...
People were classified as having periodontal disease if the measurements on their clinical attachment loss and periodontal ... Periodontitis is an inflammatory chronic gum disease characterized by the formation of periodontal pockets, loss of the ... Periodontal examinations were conducted in adults aged 30 and older. We compared the weighted prevalence of periodontal disease ... Diabetes mellitus and periodontal disease. Periodontol 2000 2007;44(1):127-53. CrossRefexternal icon PubMedexternal icon ...
These results suggest that H2S can be used as a biomarker of halitosis in patients with periodontal disease. ... Multivariate logistic regression analysis to predict the presence of halitosis, found periodontal disease was a significant ... Additionally, the presence of periodontal disease increased the probability of halitosis by 3.607 times. ... Our results emphasize the close and strong relationship between periodontal disease and halitosis through human clinical ...
... newly released Journal of Periodontology found that people who get enough calcium have significantly lower rates of periodontal ... were almost twice as likely to have periodontal disease, as measured by the loss of attachment of the gums from the teeth. The ... people should ask their dentist or periodontist about the state of their periodontal health to help prevent tooth loss and ... a leading cause of tooth loss. Researchers analyzed government data on calcium consumption and periodontal disease indicators ...
Bone loss from advancing periodontal disease also exposes more tooth length that was previously embedded in the bone for added ... These suspended attachments allow teeth to withstand the forces of biting and chewing. As you age, you naturally lose some of ... Some of the reasons you may experience gradual bone loss in the jaws include:. *Periodontal disease - Chronic bacterial ... X-rays, periodontal charting, and impressions can also diagnose progression of gum recession or bone loss. ...
This inflammation leads to subsequent loss of periodontal ligament, cementum, and alveolar bone, the attachment apparatus of ... Periodontal Status of adult patients treated with fixed buccal appliances and removable aligners over one year of active ... J Periodontal Implant Sci. 2015;45:193-204. doi: 10.5051/jpis.2015.45.6.193. [PMC free article] [PubMed][CrossRef] [Google ... Periodontal health status in patients treated with the Invisalign (®) system and fixed orthodontic appliances: A 3 months ...
Several clinical parameters of periodontitis, such as periodontal probing depth, clinical loss of attachment, or radiographic ... Radiographically evident severe alveolar bone loss and periodontitis in a 72-year-old man with severe periodontal disease. View ... Radiographically evident severe alveolar bone loss and periodontitis in a 72-year-old man with severe periodontal disease. ... 35] Appropriate periodontal treatment leads to a 0.4-0.6% improvement in HbA1c levels, meaning that periodontal treatment is ...
4 and periodontal status was measured by the percentage of periodontal sites with attachment loss ≥3 mm. (see Data S1 for ... and percentage of periodontal sites with attachment loss ≥3 mm (n=280). Model 5: adjusted for age, sex, race, education, BMI, ... and percentage of periodontal sites with attachment loss ≥3 mm (n=280; n=187). Model 5: adjusted for age, sex, race, education ... smoking, percentage of periodontal sites with attachment loss ≥3 mm, and dietary pattern (n=253). Model 6: adjusted for age, ...
You must demonstrate the amount of clinical attachment loss. Does your periodontal charting show recession along with pocket ... and this will give you the bone level loss. Once you have the bone level loss number, divide it by the healthy bone level ... This will give you a percentage of bone loss for staging severity when using RBL as a determinant (i.e., Stage I ,15%, Stage II ... Question: How do you appeal a claim denial for D4341/D4342 (SRP) when the reason stated is "inadequate bone loss"? The patient ...
Vitamin D can play a positive role in supporting and improving periodontal health in postmenopausal adults and pregnant women, ... at least two interproximal sites with at least 3 mm of clinical attachment loss) and were enrolled in maintenance programs at ... Researchers create algorithms to track periodontal disease change. Researchers have developed algorithms to track periodontal ... "This study suggests that periodontal health improves in patients attending regular periodontal care programs, regardless of ...

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