A type of cardiac arrhythmia with premature contractions of the HEART VENTRICLES. It is characterized by the premature QRS complex on ECG that is of abnormal shape and great duration (generally >129 msec). It is the most common form of all cardiac arrhythmias. Premature ventricular complexes have no clinical significance except in concurrence with heart diseases.
A type of cardiac arrhythmia with premature atrial contractions or beats caused by signals originating from ectopic atrial sites. The ectopic signals may or may not conduct to the HEART VENTRICLES. Atrial premature complexes are characterized by premature P waves on ECG which are different in configuration from the P waves generated by the normal pacemaker complex in the SINOATRIAL NODE.
An antiarrhythmia agent used primarily for ventricular rhythm disturbances.
Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.
Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
Modified cardiac muscle fibers composing the terminal portion of the heart conduction system.

Detection of abnormal high-frequency components in the QRS complex by the wavelet transform in patients with idiopathic dilated cardiomyopathy. (1/286)

In order to investigate whether increased fine, fractionated signals within the QRS complex can detect arrhythmogenic substrates and how these fine signals link with ventricular mechanical dysfunction, wavelet analysis was performed on averaged QRS complexes obtained from the left precordial lead in 26 patients with idiopatic dilated cardiomyopathy (IDCM) and in 12 normal subjects. The number of local maxima and the duration of the wavelet transform were significantly greater in patients with IDCM than in normal subjects; the number at 100 Hz was 8.8+/-3.1 vs 6.0+/-1.1 (p<0.01), and the duration at 100Hz was 93+/-15 vs 75+/-7ms (p<0.01). Both of these indices were greater in the patients with than in those without late potentials, repetitive ventricular premature beats or cardiac death. In addition, significant inverse curvilinear relationships were observed between the left ventricular ejection fraction and both the number of local maxima and the duration of the wavelet transform. In conclusion, fine fragmented signals in the QRS complex detected by wavelet analysis would be an important marker for potentially arrhythmogenic substrates and seemed to progress in parallel with left ventricular mechanical dysfunction in IDCM.  (+info)

Correlation of ventricular mechanosensory neurite activity with myocardial sensory field deformation. (2/286)

The mechanosensory activity generated by ventricular epicardial sensory neurites associated with afferent axons in thoracic sympathetic nerves was correlated with sensory field deformation (long axis, short axis, and transmural dimension changes), regional intramyocardial pressure, and ventricular chamber pressure in anesthetized dogs. Ventricular mechanosensory neurites generated activity that correlated best with strain developed along either the long or short axis of their epicardial sensory fields in most instances. Activity did not correlate normally to local wall thickness or to regional wall or chamber pressure development in most cases. During premature ventricular contractions, the activity generated by these sensory neurites correlated best with maximum strain developed along at least one sensory field epicardial vector. Identified sensory neurites were also activated by local application of the chemical bradykinin (10 microM) or by local ischemia. These data indicate that the activity generated by most ischemia-sensitive ventricular epicardial sensory neurites associated with afferent axons in sympathetic nerves is dependent on not only their local chemical milieu but on local mechanical deformation along at least one epicardial vector of their sensory fields.  (+info)

Possible theophylline toxicity during anesthesia. (3/286)

Asthmatic patients who undergo outpatient anesthesia are typically prescribed one or more drugs for treatment. Some of these agents have narrow therapeutic ranges and are associated with potentially serious adverse reactions, toxic effects, or drug interactions. Various clinical signs of toxicity may be first uncovered during routine monitoring of an office anesthetic. The case reported here demonstrates the need for proper understanding of the asthmatic patient's medical history and an appreciation for the medications used to control the disease. A sudden cardiovascular event possibly related to drug toxicity is witnessed and treated in an asthmatic patient during intravenous sedation. A possible drug interaction with a non-asthmatic medication taken concomitantly by the patient is implicated and discussed. In addition to the case report, the broad classification of drugs employed for bronchial asthma and their effects is reviewed.  (+info)

Dental anesthetic management of a patient with ventricular arrhythmias. (4/286)

During routine deep sedation for endodontic therapy, a dentist-anesthesiologist observed premature ventricular contractions (PVCs) on a 62-yr-old woman's electrocardiogram (EKG) tracing. The dentist was able to complete the root canal procedure under intravenous (i.v.) sedation without any problems. The dentist-anesthesiologist referred the patient for medical evaluation. She was found to be free from ischemic cardiac disease with normal ventricular function. The patient was cleared to continue her dental treatment with deep sedation. She subsequently continued to undergo dental treatment with deep intravenous sedation without incident, although her EKG exhibited frequent PVCs, up to 20 per minute, including couplets and episodes of trigeminy. This article will review indications for medical intervention, antiarrhythmic medications, and anesthetic interventions for perioperative PVCs.  (+info)

Unsuitability of corrected QT dispersion as a marker for ventricular arrhythmias and cardiac sudden death after acute myocardial infarction. (5/286)

The present study investigated whether corrected QT (QTc) dispersion could play a role as a marker of ventricular arrhythmias and sudden cardiac death after acute myocardial infarction (MI). The study included 76 males and 24 females with a mean age of 60+/-11 years. Standard 12-lead ECGs were recorded during the recovery phase (15+/-9 days) after the onset of MI. The QTc was calculated according to Bazett's formula and QTc dispersion was calculated as the difference between the maximum and minimum QTc intervals. Patients were divided into 2 groups: 21 patients (group A) had a QTc dispersion of > or =80ms, and the other 79 patients (group B) had a QTc dispersion of <80ms in the recovery stage (15+/-9 days). Clinical, angiographical, and Holter monitoring data, and prognosis (mean follow-up period 29+/-18 months) were compared between these 2 groups. The frequencies of early coronary reperfusion and recanalization of infarct-related vessels during the recovery phase were significantly higher in group B than group A. The left ventricular ejection fraction was also higher in group B than group A (51+/-12 vs 43+/-12%, p=0.0029). There were no significant differences in the number of premature ventricular contractions, the percentage of patients with repetitive ventricular arrhythmias, or in the frequency of sudden cardiac death during the follow-up period between the 2 groups. In summary, QTc dispersion in the recovery stage is not a useful marker for ventricular arrhythmias or sudden cardiac death after acute MI, although increased QTc dispersion may correlate with an ineffective early coronary reperfusion and with the degree of depressed left ventricular function.  (+info)

Cyclic bursts of ventricular premature contractions of more than one minute intervals. (6/286)

Ventricular premature contractions (VPCs) occasionally appear successively in the form of bigeminy, trigeminy or quadrigeminy associated with quiescent periods. However, details of these rhythmic VPC bursts have not been well documented. We analyzed the incidence, periodicity and interval of VPC bursts exhibiting bigeminy or trigeminy using ambulatory ECG monitoring and computer analysis. We defined VPC bursts as more than 5 successive groups of VPCs each containing more than 20 VPCs in the form of bigeminy or trigeminy that were interrupted by normal sinus rhythm lasting for more than 60 seconds. Bursts thus defined were observed transiently or continuously in 78 out of 500 consecutive patients showing > 3000 VPCs a day. Their age ranged from 14 to 76 years (mean 48). Forty patients were men and 38 were women. We could discriminate between two types of bursts on the instantaneous heart rate tachograms. Dome type bursts (n = 48) showed gradual shortening of the VPC coupling intervals whereas horizontal type bursts (n = 30) demonstrated fixed coupling intervals during the bursts. Cycle length of the dome type burst was 185 +/- 40 seconds and regular, whereas it was 210 +/- 63 seconds and irregular in the horizontal type (NS). Duration of the VPC bursts was 101 +/- 31 seconds in the dome type and 98 +/- 41 seconds in the horizontal type. Both burst types were associated with transient increases in sinus rate and abbreviated VPC-VPC intervals. We suspect ventricular parasystole to be the mechanism of these bursts especially in the dome type. Recognition of these two burst types from heart rate tachograms may be of value in the suppression of VPCs.  (+info)

Pacing-induced delayed protection against arrhythmias is attenuated by aminoguanidine, an inhibitor of nitric oxide synthase. (7/286)

1. Cardiac pacing, in anaesthetized dogs, protects against ischaemia and reperfusion-induced ventricular arrhythmias when this is initiated 24 h after the pacing stimulus. Now we have examined whether this delayed cardioprotection afforded by cardiac pacing is mediated through nitric oxide. 2. Twenty-two dogs were paced (4 x 5 min periods at 220 beats min(-1)) by way of the right ventricle, 24 h prior to a 25 min period of coronary artery occlusion. Nine of these dogs were given the inhibitor of induced nitric oxide synthase, aminoguanidine (50 mg kg(-1) i.v.), 0.5 h prior to coronary artery occlusion. Sham-operated non-paced dogs with and without aminoguanidine treatment served as controls. 3. Pacing markedly (P<0. 05) reduced arrhythmia severity (ventricular fibrillation, VF, during occlusion 15%; survival from the combined ischaemia-reperfusion insult 62%) compared to control, sham-operated, unpaced dogs (VF during occlusion 58%; survival 17%). This protection was attenuated by the administration of aminoguanidine prior to coronary artery occlusion (survival from the combined ischaemia-reperfusion insult 11%, which was significantly (P<0.05) less than in the paced dogs not given aminoguanidine and similar to the controls). Aminoguanidine had no significant effects on coronary artery occlusion when given to dogs that had not been paced. In the dose used aminoguanadine transiently elevated systemic arterial pressure by a mean of 20 mmHg and reduced heart rate by a mean of 22 beats min(-1). 4. These results suggest that nitric oxide, probably derived from induced nitric oxide synthase, contributes significantly to the delayed cardioprotection afforded by cardiac pacing.  (+info)

Heart rate variability analysis of patients with idiopathic left ventricular outflow tract tachycardia: role of triggered activity. (8/286)

There have been several reports with respect to idiopathic ventricular tachycardias (VTs) originating from the left ventricular outflow tract (LVOT). A previous report suggested that triggered activity plays a partial role in idiopathic LVOT tachycardia from the electrophysiological as well as the electropharmacological viewpoint. However, the exact role of triggered activity in this type of VT remains unknown. In the present study the relationship of the frequency of premature ventricular contractions (PVCs) and heart rate was examined and heart rate variability (HRV) was analyzed in 2 cases of LVOT tachycardia using 24-h Holter electrocardiographic (ECG) monitoring. The relation between the PVCs frequency and heart rate showed a persistently positive correlation, indicating frequent PVCs as heart rate increased. In HRV analysis, NN50(%), a time-domain variable of parasympathetic activity, showed no change prior to ventricular arrhythmias. In frequency-domain analysis of HRV, the high frequency (HF) component tended to fall prior to repetitive PVCs and VTs. The ratio of the low frequency to high frequency (LF/HF) components increased prior to single PVCs, repetitive PVCs and VTs. Sympathetic predominance predisposes the genesis of these kinds of arrhythmias originating from the LVOT and it is suggested that triggered activity plays an important role in LVOT tachycardia, at least in its initiation.  (+info)

Ventricular Premature Complexes (VPCs), also known as Ventricular Extrasystoles or Premature Ventricular Contractions (PVCs), are extra heartbeats that originate in the ventricles, the lower chambers of the heart. These premature beats disrupt the normal sequence of electrical impulses in the heart and cause the ventricles to contract earlier than they should.

VPCs can result in a noticeable "skipped" or "extra" beat sensation, often followed by a stronger beat as the heart returns to its regular rhythm. They may occur occasionally in healthy individuals with no underlying heart condition, but frequent VPCs could indicate an underlying issue such as heart disease, electrolyte imbalance, or digitalis toxicity. In some cases, VPCs can be harmless and require no treatment; however, if they are frequent or associated with structural heart problems, further evaluation and management may be necessary to prevent potential complications like reduced cardiac output or heart failure.

Atrial premature complexes (APCs or APCTs) are extra heartbeats that originate in the atria, which are the upper chambers of the heart. These early beats disrupt the normal rhythm and cause a premature contraction before the next scheduled beat. APCs can sometimes be felt as a "skipped" beat or palpitation. They are usually benign and do not require treatment unless they occur frequently or are associated with underlying heart disease.

Moricizine is an antiarrhythmic medication that belongs to the class IC. It works by stabilizing the heart's electrical activity and correcting irregular heart rhythms (arrhythmias). Moricizine is used to treat certain types of serious, life-threatening ventricular arrhythmias.

It's important to note that moricizine has been discontinued in many countries due to the availability of safer and more effective antiarrhythmic medications. The use of moricizine should be under the close supervision of a healthcare professional, and it is usually reserved for situations where other treatments have not been effective.

The medical definition of 'Moricizine' is:

A class IC antiarrhythmic drug that prolongs the refractory period of the ventricles by selectively blocking sodium channels during phase 0 of the action potential, thereby stabilizing cardiac membranes and suppressing ectopic pacemaker activity. Moricizine is used in the treatment of certain types of serious, life-threatening ventricular arrhythmias.

Ambulatory electrocardiography, also known as ambulatory ECG or Holter monitoring, is a non-invasive method of recording the electrical activity of the heart over an extended period of time (typically 24 hours or more) while the patient goes about their daily activities. The device used to record the ECG is called a Holter monitor, which consists of a small, portable recorder that is attached to the patient's chest with electrodes.

The recorded data provides information on any abnormalities in the heart's rhythm or electrical activity during different stages of activity and rest, allowing healthcare providers to diagnose and evaluate various cardiac conditions such as arrhythmias, ischemia, and infarction. The ability to monitor the heart's activity over an extended period while the patient performs their normal activities provides valuable information that may not be captured during a standard ECG, which only records the heart's electrical activity for a few seconds.

In summary, ambulatory electrocardiography is a diagnostic tool used to evaluate the electrical activity of the heart over an extended period, allowing healthcare providers to diagnose and manage various cardiac conditions.

Cardiac arrhythmias are abnormal heart rhythms that result from disturbances in the electrical conduction system of the heart. The heart's normal rhythm is controlled by an electrical signal that originates in the sinoatrial (SA) node, located in the right atrium. This signal travels through the atrioventricular (AV) node and into the ventricles, causing them to contract and pump blood throughout the body.

An arrhythmia occurs when there is a disruption in this electrical pathway or when the heart's natural pacemaker produces an abnormal rhythm. This can cause the heart to beat too fast (tachycardia), too slow (bradycardia), or irregularly.

There are several types of cardiac arrhythmias, including:

1. Atrial fibrillation: A rapid and irregular heartbeat that starts in the atria (the upper chambers of the heart).
2. Atrial flutter: A rapid but regular heartbeat that starts in the atria.
3. Supraventricular tachycardia (SVT): A rapid heartbeat that starts above the ventricles, usually in the atria or AV node.
4. Ventricular tachycardia: A rapid and potentially life-threatening heart rhythm that originates in the ventricles.
5. Ventricular fibrillation: A chaotic and disorganized electrical activity in the ventricles, which can be fatal if not treated immediately.
6. Heart block: A delay or interruption in the conduction of electrical signals from the atria to the ventricles.

Cardiac arrhythmias can cause various symptoms, such as palpitations, dizziness, shortness of breath, chest pain, and fatigue. In some cases, they may not cause any symptoms and go unnoticed. However, if left untreated, certain types of arrhythmias can lead to serious complications, including stroke, heart failure, or even sudden cardiac death.

Treatment for cardiac arrhythmias depends on the type, severity, and underlying causes. Options may include lifestyle changes, medications, cardioversion (electrical shock therapy), catheter ablation, implantable devices such as pacemakers or defibrillators, and surgery. It is essential to consult a healthcare professional for proper evaluation and management of cardiac arrhythmias.

Electrocardiography (ECG or EKG) is a medical procedure that records the electrical activity of the heart. It provides a graphic representation of the electrical changes that occur during each heartbeat. The resulting tracing, called an electrocardiogram, can reveal information about the heart's rate and rhythm, as well as any damage to its cells or abnormalities in its conduction system.

During an ECG, small electrodes are placed on the skin of the chest, arms, and legs. These electrodes detect the electrical signals produced by the heart and transmit them to a machine that amplifies and records them. The procedure is non-invasive, painless, and quick, usually taking only a few minutes.

ECGs are commonly used to diagnose and monitor various heart conditions, including arrhythmias, coronary artery disease, heart attacks, and electrolyte imbalances. They can also be used to evaluate the effectiveness of certain medications or treatments.

Purkinje fibers are specialized cardiac muscle fibers that are located in the subendocardial region of the inner ventricular walls of the heart. They play a crucial role in the electrical conduction system of the heart, transmitting electrical impulses from the bundle branches to the ventricular myocardium, which enables the coordinated contraction of the ventricles during each heartbeat.

These fibers have a unique structure that allows for rapid and efficient conduction of electrical signals. They are larger in diameter than regular cardiac muscle fibers, have fewer branching points, and possess more numerous mitochondria and a richer blood supply. These features enable Purkinje fibers to conduct electrical impulses at faster speeds, ensuring that the ventricles contract simultaneously and forcefully, promoting efficient pumping of blood throughout the body.

Ventricular premature complexes (VPCs) are ectopic impulses originating from an area distal to the His Purkinje system. VPCs ... encoded search term (Ventricular Premature Complexes) and Ventricular Premature Complexes What to Read Next on Medscape ... Latchamsetty R, Bogun F. Premature ventricular complexes and premature ventricular complex induced cardiomyopathy. Curr Probl ... Ventricular premature complexes (VPCs). Ventricular trigeminy is present. Note that the VPCs are unimorphic and that a ...
Mechanical suppression of premature ventricular complexes during catheter ablation procedures. Rahul Jain, Mamta Barmeda, Rohit ... Mechanical suppression of premature ventricular complexes during catheter ablation procedures. / Jain, Rahul; Barmeda, Mamta; ... Mechanical suppression of premature ventricular complexes during catheter ablation procedures. In: Indian Pacing and ... Jain, R., Barmeda, M., Jain, R., Shirazi, J., & Miller, J. M. (2021). Mechanical suppression of premature ventricular complexes ...
Diagnosis Sinus rhythm with premature ventricular complexes in a pattern of bigeminy. Ventricular rate 92/min Left ventricular ... Diagnosis Atrial fibrillation with rapid ventricular response (ventricular rate 206/min) Two premature ventricular complexes ST ... 85/min with frequent premature ventricular complexes in a pattern of bigeminy Left ventricular hypertrophy … ... Diagnosis Sinus rhythm with sinus arrhythmia Occasional premature ventricular complexes Otherwise, …. Case 11: A 26-Year-Old ...
November 2002). "A case of cardiomyopathy induced by premature ventricular complexes". Circulation Journal. 66 (11): 1065-7. ... of premature ventricular contractions (extrasystole) can develop dilated cardiomyopathy. In these cases, if the extrasystole ... Echocardiogram shows left ventricular dilatation with normal or thinned walls and reduced ejection fraction. Cardiac ... In addition, the ventricular walls maintain the same thickness, characteristic of pathophysiological cardiac dilation.[citation ...
Obesity is independently associated with increased likelihood of ectopic ventricular arrhythmia during exercise. ... Ventricular Premature Complexes / diagnosis * Ventricular Premature Complexes / etiology* * Ventricular Premature Complexes / ... defined as multiple ventricular premature beats (≥3), ventricular bigeminy, nonsustained ventricular tachycardia or sustained ... Obesity and exercise-induced ectopic ventricular arrhythmias in apparently healthy middle aged adults Eur J Prev Cardiol. 2016 ...
Characteristics of diurnal ventricular premature complex variation in right ventricular outflow tract arrhythmias after ... Characteristics of diurnal ventricular premature complex variation in right ventricular outflow tract arrhythmias after ... Characteristics of diurnal ventricular premature complex variation in right ventricular outflow tract arrhythmias after ... Characteristics of diurnal ventricular premature complex variation in right ventricular outflow tract arrhythmias after ...
Premature ventricular contractions (PVCs) are extra heartbeats that begin in one of the hearts two lower pumping chambers ( ... Premature ventricular complexes. *Ventricular premature beats. *Ventricular extrasystoles. Occasional premature ventricular ... Premature ventricular contractions often cause few or no symptoms. But the extra beats can cause unusual sensations in the ... Most people with premature ventricular contractions (PVCs) who dont have heart disease wont need treatment. If you have heart ...
We searched on Pubmed for ventricular premature complexes (VPC) in pregnant women and found no case reporting increased ... Premature extra beats and sustained tachyarrhythmias are the most common arrhythmias in pregnancy. Symptomatic episodes occur ... Atrial premature complexes (APC), ventricular premature complexes (VPC), and supraventricular tachyarrhythmias are more ... We searched on Pubmed for ventricular premature complexes (VPC) in pregnant women and found no case reporting increased ...
... frequent premature ventricular complexes, and scooped ST segments consistent with cardenolide toxicity. At the direction of ...
... stimulation in this specific region reduced the formation of conduction block and the probability of premature ventricular ... which typically precedes ventricular tachycardia and fibrillation. MI is also associated with remodeling of the sympathetic ... which typically precedes ventricular tachycardia and fibrillation. MI is also associated with remodelling of the sympathetic ... Relationship of frequent postinfarction premature ventricular complexes to the reentry circuit of scar-related ventricular ...
This is known as a premature ventricular complex (PVC) or ventricular premature beat. When this occurs in a three-beat pattern ... 2017). Ventricular premature beats.. https://www.uptodate.com/contents/ventricular-premature-beats. ... its called a premature ventricular complex, or PVC. Treatments can correct an abnormal heartbeat. ... 2018). Premature ventricularcontractions (PVCs).. https://www.mayoclinic.org/diseases-conditions/premature-ventricular- ...
Ventricular premature complexes & heart failure. Take Quiz. Arterial stiffness & incident heart failure: Gender differences. ...
Atrial fibrillation, flutter and ventricular premature complexes can also be found.. b) Becker (BMD). is less common (3 per ... Right Ventricular Function Working Group on Aorta and Peripheral Vascular Diseases Working Group on Myocardial Function Working ... the sudden onset of cardiac symptoms accompanied by deterioration of left ventricular function in young DMD patients already on ...
Ventricular Premature Complexes. How long have you been taking it?. Choose one ...
Atrial Premature Complexes. *Tachycardia, Ventricular. *Ventricular Premature Complexes. How long have you been taking it?. ... Disopyramide is a prescription medications used to treat life-threatening ventricular (lower chambers of the heart) arrhythmias ... Disopyramide may widen the QRS complex. If this happens, your doctor will stop disopyramide. ... double-blind study in patients with asymptomatic non-life-threatening ventricular arrhythmias who had had a myocardial ...
Ventricular Premature Complexes. How long have you been taking it?. Choose one ...
The major disorder in the cardiac rhythm was the ventricular premature complex. Several dogs showed the R over T phenomenon. ... The females showed larger number of ventricular premature complex than the males. ...
Ethmozin, a new antiarrhythmic drug for suppressing ventricular premature complexes. Podrid, P.J., Lyakishev, A., Lown, B., ...
Sinus bradycardia, tachycardia, premature ventricular complexes & other arrhythmias. Permanent CNS or brain damage, and death ...
Premature ventricular complexes are the most common ventricular dysrhythmias during anesthesia. They are well tolerated in ... A good example is a healthy cat undergoing ovariohysterectomy which shows premature ventricular complexes on ECG. By simply ... Another ventricular dysrhythmia of concern will be ventricular tachycardia. This dysrhythmia can progress to ventricular ... Quinidine (0.2-0.5 mg/kg IV) can be used to treat atrial flutter/fibrillation). Premature atrial complexes can occur during ...
Catheter ablation of intramural outflow tract premature ventricular complexes: a multicentre study. Europace : European Pacing ... Small, G. R., & Beanlands, R. S. B. (2023). Right Ventricular Myocardial Blood Flow in Cardiac Amyloidosis: A Flag-Waving ... 2023). A 4D visualization tool for treatment planning of non-invasive radioablation in patients with ventricular tachycardia. ... Left Ventricular End-Diastolic Pressure for the Prediction of Contrast-Induced Nephropathy and Clinical Outcomes in Patients ...
Catheter ablation of intramural outflow tract premature ventricular complexes: a multicentre study. Europace : European Pacing ... Small, G. R., & Beanlands, R. S. B. (2023). Right Ventricular Myocardial Blood Flow in Cardiac Amyloidosis: A Flag-Waving ... 2023). A 4D visualization tool for treatment planning of non-invasive radioablation in patients with ventricular tachycardia. ... Left Ventricular End-Diastolic Pressure for the Prediction of Contrast-Induced Nephropathy and Clinical Outcomes in Patients ...
KEY WORDS: Ventricular fibrillation - Atrial fibrillation - Ventricular premature complexes - Tachycardia, ectopic atrial ... Panoramic mapping of ventricular fibrillation (VF) is helping to develop a better understanding of its physiology, with future ... but also as a guide for trans-catheter ablation of complex arrhythmias such as atrial fibrillation. It provides the clinician ...
Other less frequently observed arrhythmias included ventricular premature complexes (VPCs), supraventricular premature ... ventricular premature depolarizations, 2nd degree AV block, escape beats/rhythms, and supraventricular premature ... In one cat, incidental and reversible premature junctional complexes were seen at 1 and 2 hours after dosing which were ... followed by a few incidences of premature complexes and one incidence of atrioventricular block. Oxygen saturation, mucous ...
SH may lead to a predisposition to ventricular arrhythmias. Keywords : Hypothyroidism; Ventricular premature complexes; ... IMPACT OF SUBCLINICAL HYPOTHYROIDISM ON FREQUENCY OF PREMATURE VENTRICULAR CONTRACTIONS IN ELDERLY PATIENTS WITHOUT STRUCTURAL ... Premature ventricular contractions (PVCs) are common arrhythmias, most of them requiring no follow-up or treatment. Subclinical ...
Isoproterenol administration was associated with a few premature ventricular contractions and supraventricular complexes, but ... Comparison of the effects of four antiarrhythmic treatments for familial ventricular arrhythmias in Boxers. J Am Vet Med Assoc ... Comparison of the effects of four antiarrhythmic treatments for familial ventricular arrhythmias in Boxers. J Am Vet Med Assoc ... Comparison of the effects of four antiarrhythmic treatments for familial ventricular arrhythmias in Boxers. J Am Vet Med Assoc ...
1. Ventricular Premature Beats (Ventricular Extrasystoles) Ventricular premature beats are characterized by wide QRS complexes ... 2. Ventricular Tachycardia Introduction Ventricular tachycardia is defined as three or more consecutive ventricular premature ... Sudden death occurs more frequently (presumably as a result of ventricular fibrillation) when ventricular premature beats occur ... The class I and III agents are all effective in reducing ventricular premature beats but often cause side effects and may ...
... and premature ventricular complexes. If an arrhythmia develops, calcium administration should be discontinued until the heart ... Prolongation of the Q-T interval and ventricular premature contractions may be evident on an ECG. ...
... premature ventricular complexes and paroxysmal atrial fibrillation,compared to otherwise similar subgroup of 22 patients ... study showed in the subgroup of 26 patients with OSA a more prevalent presence of ACDs including premature atrial complexes, ...
... and/or nonsustained ventricular tachycardia (VT) on 24-hour Holter monitoring were randomly div … ... 90 ventricular premature complexes (VPCs) and/or nonsustained ventricular tachycardia (VT) on 24-hour Holter monitoring were ... Ventricular Premature Complexes / etiology* * Ventricular Premature Complexes / mortality * Ventricular Premature Complexes / ... Symptoms, a 6-minute walk test, left ventricular (LV) ejection fraction (EF), frequency and complexity of VPCs, and quality of ...
  • Observational studies and inferences from typical electrophysiology studies were initially focused on ventricular ectopy triggering ventricular tachycardia (VT), which, in turn, can degenerate into ventricular fibrillation, as a mechanisms for sudden cardiac death. (medscape.com)
  • We investigated the association between body mass index (BMI) and the risk for ectopic ventricular activity (defined as multiple ventricular premature beats (≥3), ventricular bigeminy, nonsustained ventricular tachycardia or sustained ventricular tachycardia) during exercise stress testing among 22,516 apparently healthy men and women who attended periodic health screening examinations between the years 2000 and 2014. (nih.gov)
  • Atrial premature complexes (APC), ventricular premature complexes (VPC), and supraventricular tachyarrhythmias are more prevalent than ventricular tachycardia, especially in women without structural heart disease. (hindawi.com)
  • Following myocardial infarction (MI), the myocardium is prone to calcium-driven alternans, which typically precedes ventricular tachycardia and fibrillation. (frontiersin.org)
  • Ventricular tachycardia is defined as three or more consecutive ventricular premature beats. (health.am)
  • Ventricular tachycardia is either nonsustained (lasting less than 30 seconds) or sustained. (health.am)
  • Ventricular tachycardia is a frequent complication of acute myocardial infarction and dilated cardiomyopathy but may occur in chronic coronary disease, hypertrophic cardiomyopathy, mitral valve prolapse, myocarditis, and in most other forms of myocardial disease. (health.am)
  • In nonacute settings, most patients with ventricular tachycardia have known or easily detectable cardiac disease, and the finding of ventricular tachycardia is an unfavorable prognostic sign. (health.am)
  • 90 ventricular premature complexes (VPCs) and/or nonsustained ventricular tachycardia (VT) on 24-hour Holter monitoring were randomly divided into 2 groups. (nih.gov)
  • Atrial fibrillation, flutter and ventricular premature complexes can also be found. (escardio.org)
  • ECM is emerging as an important tool not only for diagnostic mapping, but also as a guide for trans-catheter ablation of complex arrhythmias such as atrial fibrillation. (minervamedica.it)
  • Panoramic mapping of ventricular fibrillation (VF) is helping to develop a better understanding of its physiology, with future implications for the identification of therapeutic targets in patients with structural heart disease, as well as in idiopathic VF. (minervamedica.it)
  • Sudden death occurs more frequently (presumably as a result of ventricular fibrillation ) when ventricular premature beats occur in the presence of organic heart disease but not in individuals with no known cardiac disease. (health.am)
  • Of total 48 patients, the current study showed in the subgroup of 26 patients with OSA a more prevalent presence of ACDs including premature atrial complexes, sinus bradycardia, sinus pauses, premature ventricular complexes and paroxysmal atrial fibrillation,compared to otherwise similar subgroup of 22 patients without OSA. (easychair.org)
  • Alpha receptor blockade with either phentolamine or prazosin significantly reduced the number of premature ventricular complexes during coronary reperfusion (321 +/- 62-14 +/- 10 premature ventricular complexes, P less than 0.001), abolished early ventricular fibrillation (from 25% in controls to 0%), and prevented the increase in idioventricular rate seen with coronary reperfusion. (jci.org)
  • Electrical Substrate Ablation for Refractory Ventricular Fibrillation: Results of the AVATAR Study. (stanford.edu)
  • Background - Refractory ventricular fibrillation (VF) is a challenging clinical entity, for which ablation of triggering premature ventricular complexes (PVCs) is described. (stanford.edu)
  • CARL pioneered computational mapping of 'chaotic' heart rhythm disturbances (arrhythmias), targeting mechanistic drivers for atrial and ventricular fibrillation (AF, VF) using machine learning and computational models. (stanford.edu)
  • Multivariate binary logistic regression with repeated measures of 92,619 ESTs, showed that obese subjects were 33% more likely to have ectopic ventricular arrhythmias during exercise compared with subjects with normal weight (p = 0.005), and that each 1 kg/m(2) increase in BMI was associated with a significant 4% (p = 0.002) increased adjusted risk for exercise-induced ventricular arrhythmias. (nih.gov)
  • Premature extra beats and sustained tachyarrhythmias are the most common arrhythmias in pregnancy. (hindawi.com)
  • Disopyramide is a prescription medications used to treat life-threatening ventricular (lower chambers of the heart) arrhythmias. (rxwiki.com)
  • Premature ventricular contractions (PVCs) are common arrhythmias, most of them requiring no follow-up or treatment. (dergisi.org)
  • We concluded that serum TSH concentration has a role in the frequency of PVC, therefore, SH may lead to a predisposition to ventricular arrhythmias. (dergisi.org)
  • The class I and III agents are all effective in reducing ventricular premature beats but often cause side effects and may exacerbate serious arrhythmias in 5-20% of patients. (health.am)
  • Specific support has been via grants HL70529 and HL162260 for ventricular arrhythmias, and HL103800, HL83359, HL122384, HL149134, and several SBIR grants for atrial arrhythmias. (stanford.edu)
  • vided that no complex arrhythmias exist. (lu.se)
  • Introduction: Mechanical suppression of premature ventricular complexes (PVCs) is not a well-known observation. (psu.edu)
  • However, very frequent PVCs can be symptomatic of an underlying heart condition (such as arrhythmogenic right ventricular cardiomyopathy). (wikipedia.org)
  • Premature ventricular contractions (PVCs) are extra heartbeats that begin in one of the heart's two lower pumping chambers (ventricles). (nchmd.org)
  • To understand the cause of premature ventricular contractions (PVCs), it might help to learn more about how the heart typically beats. (nchmd.org)
  • Certain lifestyle choices and health conditions may make a person more likely to develop premature ventricular contractions (PVCs). (nchmd.org)
  • Idiopathic premature ventricular complexes (PVCs) are considered a benign form of cardiac arrhythmia. (juniperpublishers.com)
  • However, several reports demonstrated that frequent PVCs were associated with left ventricular (LV) dysfunction, increased LV dimensions and cardiomyopathy. (juniperpublishers.com)
  • Electrophysiology study using the Carto® 3 System (Biosense Webster, Irvine, CA) showed PVCs arising from anteroseptal right ventricular outflow tract. (juniperpublishers.com)
  • 3 ] in 1998 had shown that premature ventricular complexes (PVCs) was associated with cardiomyopathy and pharmacological suppression of PVCs in patients with presumed idiopathic dilated cardiomyopathy subsequently improved left ventricular (LV) systolic dysfunction. (juniperpublishers.com)
  • She was diagnosed with dilated cardiomyopathy based on echocardiography that showed a reduced left ventricular ejection fraction (LVEF) of 25% and LV systolic diameter 5.2cm with mild functional mitral regurgitation (MR). Her baseline 12-lead ECG showed inferior axis PVCs with left bundle branch block morphology ( Figure 1 ). (juniperpublishers.com)
  • Our article is an excellent reference point to learn a little more about premature ventricular complexes (PVCs) so that you can recognise the differences between this condition and others which are similar. (drsanjayguptacardiologist.com)
  • We retrospectively reviewed this phenomenon in the Ventricular Arrhythmia (VA) ablation procedures performed at Richard L. Roudebush Veterans Health Administration (VHA) center. (psu.edu)
  • Treatment options range from medication to atrial and ventricular ablation to pacemaker and defibrillator implantation (ICD) and the WATCHMAN ™ device. (uchealth.org)
  • Activation mapping with a contact-force ablation catheter (7.5 French, ThermoCool® SmartTouch® Catheter, Biosense-Webster) was performed and the earliest activation was detected from the anteroseptal right ventricular outflow tract ( Figure 2 ). (juniperpublishers.com)
  • Heart sounds of a 16 year old girl with premature ventricular contractions and other types of ectopic beats. (wikipedia.org)
  • Premature ventricular contractions may be associated with underlying heart disease, and certain characteristics are therefore elicited routinely: the presence of signs of heart disease or a known history of heart disease (e.g. previous myocardial infarction), as well as heart disease or sudden cardiac death in close relatives. (wikipedia.org)
  • Premature ventricular contractions occur in healthy persons of any age, but are more prevalent in the elderly and in men. (wikipedia.org)
  • There are three main physiological explanations for premature ventricular contractions: enhanced ectopic nodal automaticity, re-entry signaling, and toxic/reperfusion triggered. (wikipedia.org)
  • The diurnal variation type (group 1, n = 36) was defined as those patients who had most ventricular premature contractions (VPCs) during the night hours by preprocedure Holter recordings. (tmu.edu.tw)
  • Premature ventricular contractions are a common type of irregular heartbeat (arrhythmia). (nchmd.org)
  • Occasional premature ventricular contractions in people without heart disease usually aren't a concern and likely don't need treatment. (nchmd.org)
  • You might need treatment if the premature ventricular contractions are very frequent or bothersome, or if you have an underlying heart condition. (nchmd.org)
  • Premature ventricular contractions often cause few or no symptoms. (nchmd.org)
  • The cause of premature ventricular contractions isn't always clear. (nchmd.org)
  • VPCs are the most common ventricular arrhythmia. (medscape.com)
  • In animal models, focal mechanisms without evidence of macro-reentry play a major role in the origin of ventricular arrhythmia associated with ischemic cardiomyopathy. (medscape.com)
  • Obesity is independently associated with increased likelihood of ectopic ventricular arrhythmia during exercise. (nih.gov)
  • We searched on Pubmed for ventricular premature complexes (VPC) in pregnant women and found no case reporting increased incidence of this arrhythmia while supine. (hindawi.com)
  • The electrophysiological characteristics of diurnal variations in idiopathic right ventricular outflow tract (RVOT) VA have not previously been elucidated. (tmu.edu.tw)
  • Electrocardiogram (ECG) results demonstrated sinus bradycardia, frequent premature ventricular complexes, and scooped ST segments consistent with cardenolide toxicity. (cdc.gov)
  • Ambulatory electrocardiographic monitoring or monitoring during graded exercise may reveal more frequent and complex ventricular premature beats than occur in a single routine ECG. (health.am)
  • Frolkis JP et al: Frequent ventricular ectopy after exercise as a predictor of death. (health.am)
  • It was previously thought that frequent ventricular complexes in patients without structural heart disease was considered to be a benign condition without prognostic significance [ 1 , 2 ]. (juniperpublishers.com)
  • Diurnal variations in ventricular tachyarrhythmias (VAs) have been demonstrated in idiopathic arrhythmogenic heart disease. (tmu.edu.tw)
  • VPCs also are common in patients with hypertension, ventricular hypertrophy, cardiomyopathy, and mitral valve prolapse. (medscape.com)
  • Mitral regurgitation is related to posterior papillary muscle dysfunction.Additionally, the sudden onset of cardiac symptoms accompanied by deterioration of left ventricular function in young DMD patients already on treatment with a prednisone derivate, that postpones the development of DMD cardiomyopathy, should raise the suspicion of possible myocarditis (3). (escardio.org)
  • If the underlying condition is mitral prolapse, hypertrophic cardiomyopathy, left ventricular hypertrophy, or coronary disease - or if the QT interval is prolonged - ß-blocker therapy is appropriate. (health.am)
  • Nandakumar R, Koh K W, Alan K C C, Yap L B, Razali O, Zulkeflee M. Successful Treatment of Cardiomyopathy Induced by Premature Ventricular Complexes. (juniperpublishers.com)
  • Some patients are aware of virtually every premature ventricular beat, but others are unaware of even complex atrial or ventricular tachyarrhythmias. (msdmanuals.com)
  • The purpose of this clinical study is to test the hypothesis that market released Cardiac Resynchronization Therapy (CRT) devices which contain the AdaptivCRT® (aCRT) algorithm have a superior outcome compared to standard CRT devices in CRT indicated patients with normal atrio-ventricular (AV) conduction and left bundle branch block (LBBB). (stanford.edu)
  • However, there are limited data on the association between excess weight and the risk of ectopic ventricular activity. (nih.gov)
  • nbsp;When the conduction impulses from SA nodes do not reach the ventricles for some reason or when the rate of these impulses falls below the base rate determined by the ventricular pacemaker cells, the impulses are generated by lower heart region, resulting in idioventricular rhythm or ventricle escape complexes commonly known as irregular heartbeats. (petyourdog.com)
  • A decrese in the frequency of sinus node pacemaker impulses or its blockage to the ventricles results in taking over of pacemaker role by lower heart region, which results in ventricular escape complexes or an idioventricular rhythm. (petyourdog.com)
  • Simulations also demonstrated that β-adrenergic receptor stimulation in this specific region reduced the formation of conduction block and the probability of premature ventricular activation propagation. (frontiersin.org)
  • Les troubles du rythme et de la conduction cardiaque peuvent mettre en jeu le pronostic vital à court terme au cours de la COVID -19. (bvsalud.org)
  • In cases of isolated extrasystoles, the patient may actually perceive the augmented postextrasystolic beat as the "skipped" beat rather than the premature beat itself, probably because the extrasystole blocks the next sinus beat and allows longer ventricular filling and thus a higher stroke volume. (msdmanuals.com)
  • Ventricular premature complexes (VPCs) are ectopic impulses originating from an area distal to the His Purkinje system. (medscape.com)
  • Assessment and treatment of VPCs is challenging and complex, and is highly dependent on the clinical context. (medscape.com)
  • Symptoms, a 6-minute walk test, left ventricular (LV) ejection fraction (EF), frequency and complexity of VPCs, and quality of life were assessed after 8 weeks of treatment and during a mean 24-month follow-up. (nih.gov)
  • Jouven X et al: Long-term outcome in asymptomatic men with exercise-induced premature ventricular depolarizations. (health.am)
  • Automaticity: This is the development of a new site of depolarization in nonnodal ventricular tissue, which can lead to a VPC. (medscape.com)
  • Notice the unconducted atrial premature complexes (APCs) superimposed on the ST segment of the previous depolarization (arrows) that appear in a bigeminal pattern. (avma.org)
  • The P wave represents atrial depolarization and the QRS represents ventricular depolarization. (petyourdog.com)
  • Ventricular dysrhythmias induced by coronary occlusion alone (without reperfusion) were attenuated markedly by alpha-receptor blockade under conditions in which perfusion (measured with radiolabeled microspheres) within ischemic zones was not affected. (jci.org)
  • Ventricular premature beats are characterized by wide QRS complexes that differ in morphology from the patient's normal beats. (health.am)
  • Exercise generally abolishes premature beats in normal hearts, and the rhythm becomes regular. (health.am)
  • An increased frequency of ventricular premature beats during exercise is associated with a higher risk of cardiovascular mortality, though there is no evidence that specific therapy has a role. (health.am)
  • Heart rate (a) and ventricular premature complexes (b) during 24-hour Holter monitoring. (hindawi.com)
  • We also consult with the Advanced Heart Failure team at UCHealth University of Colorado Hospital when we have patients who might need LVAD (left ventricular assist device) or a heart transplant. (uchealth.org)
  • A premature ventricular contraction (PVC) is a common event where the heartbeat is initiated by Purkinje fibers in the ventricles rather than by the sinoatrial node. (wikipedia.org)
  • When your heartbeat does't follow the typical heartbeat pattern and instead is slightly offbeat, it's called a premature ventricular complex, or PVC. (healthline.com)